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URINARY OBSTRUCTION AT UPPER AND
        LOWER URINARY TRACT




Bayu F. Wibowo

Bag. Ilmu Bedah FK UNAND
 Classified According:
    Cause (Congenital Or Acquired),

    Duration (Acute Or Chronic),

    Degree (Partial Or Complete), And

    Level (Upper Or Lower Urinary Tract).
 Meatal Stenosis               Damage To Sacral Roots 2–4
                                 In Spina Bifida And
 Stenosis Distal Urethra,       Myelomeningocele.
 Posterior Urethral Valves,

 Ectopic Ureters,

 Ureteroceles,

 The Ureterovesical And
  Ureteropelvic Junctions
 Urethral stricture secondary to infection or injury;

 Benign prostatic hyperplasia or cancer of the prostate;

 Vesical tumor involving the bladder neck or one or both ureteral orifices;

 Local extension of cancer of the prostate or cervix into the base of the
  bladder, occluding the ureters;

 Compression of the ureters at the pelvic brim by metastatic nodes from
  cancer of the prostate or cervix;

 Ureteral stone;

 Retroperitoneal fibrosis or malignant tumor;

 Pregnancy
 Lower Tract
   Obstruction  Hydrostatic pressure proximal  dilation
    of the urethra  The wall of the urethra become thin 
    form of diverticulum  Infected urine + urinary
    extravasation  periurethral abscess.
 Mid Tract

    Stage of compensation

       The bladder musculature hypertrophies  the thickness may
        double or triple.

       Hypertrophied muscle may be seen endoscopically 
        superimposed with secondary infection (edema of the submucosa,
        infiltrated with plasma cells, lymphocytes, and polymorphonuclear
        cells)

    Stage of decompensation

       Large obstructing gland can be palpated rectally and observed
        cystoscopically;

       May appears as a mild obstruction cystoscopically.
 Upper Tract

 Ureter

    In the early stages of obstruction  intravesical pressure is
     normal

    Later  added stretch effect at the lower end of the ureter 
     induces further hydroureteronephrosis.

    Finally  increasing pressure  the ureteral wall becomes
     attenuated and therefore loses its contractile power (stage of
     decompensation).
 SYMPTOMS
   Lower And Midtract (Urethra And Bladder)

   Typified by the symptoms of urethral stricture, benign prostatic
    hyperplasia, neurogenic bladder, and tumor of the bladder
    involving the vesical neck.

   The principal symptoms are hesitancy in starting urination,
    lessened force and size of the stream, and terminal dribbling;
    hematuria, which may be partial, initially, with stricture or total
    with prostatic obstruction or vesical tumor, cloudy urine (due to
    complicating infection), acute urinary retention.
 SYMPTOMS
   Upper Tract (Ureter And Kidney)

   Symptoms of obstruction of the upper tract are typified by the
    symptoms of ureteral stricture or ureteral or renal stone.

   The principal complaints are pain in the flank radiating along the
    course of the ureter, gross total hematuria (from stone),
    gastrointestinal symptoms, chills, fever, burning on urination, and
    cloudy urine with onset of infection, which is the common sequel to
    obstruction or vesicoureteral reflux.

   Nausea, vomiting, loss of weight and strength, and pallor are due to
    uremia secondary to bilateral hydronephrosis.
 SIGNS
   Lower and midtract

      Palpation of the urethra  induration about a stricture.

      Rectal examination  atony of the anal sphincter (damage to the
       sacral nerve roots) or benign or malignant enlargement of the
       prostate.

      Vesical distention may be found
A flow rate under 10 mL/s is indicative of obstruction
 or weak detrusor function

Atonic neurogenic (neuropathic), bladder
 (diminished detrusor power), or with urethral
 stricture or prostatic obstruction (increased urethral
 resistance)  3–5 mL/s
 Upper tract

    An enlarged kidney may be discovered by palpation or percussion.

    Renal tenderness (+) if infection is present.

    A large pelvic mass (tumor, pregnancy) can displace and compress
     the ureters.

    Children with advanced urinary tract obstruction (usually Due to
     posterior urethral valves) may develop ascites
 Anemia  secondary to chronic infection or in advanced
  bilateral hydronephrosis (stage of uremia).

 Leukocytosisis  acute stage of infection.

 Microscopic hematuria may indicate renal or vesical
  infection, tumor, or stone.

 Bilateral hydronephrosis  urine flow through is slowed.

 An urea-creatinine ratio  above the normal 10:1.
X-RAY FINDINGS
 RELIEF OF OBSTRUCTION

 ERADICATION OF INFECTION
Urinary Tract Obstruction

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Urinary Tract Obstruction

  • 1. URINARY OBSTRUCTION AT UPPER AND LOWER URINARY TRACT Bayu F. Wibowo Bag. Ilmu Bedah FK UNAND
  • 2.  Classified According:  Cause (Congenital Or Acquired),  Duration (Acute Or Chronic),  Degree (Partial Or Complete), And  Level (Upper Or Lower Urinary Tract).
  • 3.  Meatal Stenosis  Damage To Sacral Roots 2–4 In Spina Bifida And  Stenosis Distal Urethra, Myelomeningocele.  Posterior Urethral Valves,  Ectopic Ureters,  Ureteroceles,  The Ureterovesical And Ureteropelvic Junctions
  • 4.  Urethral stricture secondary to infection or injury;  Benign prostatic hyperplasia or cancer of the prostate;  Vesical tumor involving the bladder neck or one or both ureteral orifices;  Local extension of cancer of the prostate or cervix into the base of the bladder, occluding the ureters;  Compression of the ureters at the pelvic brim by metastatic nodes from cancer of the prostate or cervix;  Ureteral stone;  Retroperitoneal fibrosis or malignant tumor;  Pregnancy
  • 5.  Lower Tract  Obstruction  Hydrostatic pressure proximal  dilation of the urethra  The wall of the urethra become thin  form of diverticulum  Infected urine + urinary extravasation  periurethral abscess.
  • 6.  Mid Tract  Stage of compensation  The bladder musculature hypertrophies  the thickness may double or triple.  Hypertrophied muscle may be seen endoscopically  superimposed with secondary infection (edema of the submucosa, infiltrated with plasma cells, lymphocytes, and polymorphonuclear cells)  Stage of decompensation  Large obstructing gland can be palpated rectally and observed cystoscopically;  May appears as a mild obstruction cystoscopically.
  • 7.
  • 8.  Upper Tract  Ureter  In the early stages of obstruction  intravesical pressure is normal  Later  added stretch effect at the lower end of the ureter  induces further hydroureteronephrosis.  Finally  increasing pressure  the ureteral wall becomes attenuated and therefore loses its contractile power (stage of decompensation).
  • 9.
  • 10.
  • 11.
  • 12.  SYMPTOMS  Lower And Midtract (Urethra And Bladder)  Typified by the symptoms of urethral stricture, benign prostatic hyperplasia, neurogenic bladder, and tumor of the bladder involving the vesical neck.  The principal symptoms are hesitancy in starting urination, lessened force and size of the stream, and terminal dribbling; hematuria, which may be partial, initially, with stricture or total with prostatic obstruction or vesical tumor, cloudy urine (due to complicating infection), acute urinary retention.
  • 13.  SYMPTOMS  Upper Tract (Ureter And Kidney)  Symptoms of obstruction of the upper tract are typified by the symptoms of ureteral stricture or ureteral or renal stone.  The principal complaints are pain in the flank radiating along the course of the ureter, gross total hematuria (from stone), gastrointestinal symptoms, chills, fever, burning on urination, and cloudy urine with onset of infection, which is the common sequel to obstruction or vesicoureteral reflux.  Nausea, vomiting, loss of weight and strength, and pallor are due to uremia secondary to bilateral hydronephrosis.
  • 14.  SIGNS  Lower and midtract  Palpation of the urethra  induration about a stricture.  Rectal examination  atony of the anal sphincter (damage to the sacral nerve roots) or benign or malignant enlargement of the prostate.  Vesical distention may be found
  • 15. A flow rate under 10 mL/s is indicative of obstruction or weak detrusor function Atonic neurogenic (neuropathic), bladder (diminished detrusor power), or with urethral stricture or prostatic obstruction (increased urethral resistance)  3–5 mL/s
  • 16.  Upper tract  An enlarged kidney may be discovered by palpation or percussion.  Renal tenderness (+) if infection is present.  A large pelvic mass (tumor, pregnancy) can displace and compress the ureters.  Children with advanced urinary tract obstruction (usually Due to posterior urethral valves) may develop ascites
  • 17.  Anemia  secondary to chronic infection or in advanced bilateral hydronephrosis (stage of uremia).  Leukocytosisis  acute stage of infection.  Microscopic hematuria may indicate renal or vesical infection, tumor, or stone.  Bilateral hydronephrosis  urine flow through is slowed.  An urea-creatinine ratio  above the normal 10:1.
  • 19.  RELIEF OF OBSTRUCTION  ERADICATION OF INFECTION