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Valvular
Disease, Conduction
Disorder &
Bradydysrhythmias
Arbi Ayvazian D.O PGY2
Emergency Medicine
ARMC 1/2014
Valvular Disorder
Valvular Disorder
 Things to know
 Endocarditiis presentation
 Murmurs, Rheumatic HD
 Specific high risk diseases
Infective Endocarditis
 Risk Factors: Abnormal or artificial valve
 Mitral valve most common, IVDA -> Tricuspid (staph)
 Most common bug -> Staph
 Tooth extraction -> Strep
 Acute -> high fever, murmur, flu like symp., younger
 Subacute -> Strep viridans, Anemia, older
 Prophylaxis? Depends on bug and procedure
Infective Endocarditis
 Vasculitis and Embolic manifestations
 Janeway lesions: Non-tender, hemorrhagic, flat, on
palms and soles.
 Osler nodes -> tender, tips of fingers and toes

 Roth spots and splinter hemorrhages
Infective Endocarditis
 Dx by echo, blood cultures, high ESR/CRP
 Rx: Vancomycin for Staph, PCN for Strep
 Prophylaxis if abnormal valve and procedure

 Procedure site determines bug and Abx
 Classic broad question -> dental and Amoxicilin,
GI/GU more gram negative coverage
 Controversial in mitral valve prolapse (no on boards)
End Point of Valve
Disease
 Heart Fails and dilates
 Valves become regurgitant
 ECG shows LVH as ventricles expand

 LBBB develops as heart and conduction system
stretches which is poor prognostic sgin
Murmurs: MR. ASS, MS.AID
Mitral
Regurgitation

Mitral
Stenosis

Aortic
Stenosis

Aortic
Insufficiency

SYSYTOLIC

DIASTOLIC
Aortic Stenosis
 Symptoms progress from : SOB, CHF, Syncope (bad!)
 Murmur: Systolic, up into the neck, slow carotid
upstroke

 ECG : LVH, LBBB
 Exercise-induced syncope
 Vasodilators can make it worse
 Rx: Surgical (moderate to severe)
Aortic Regurgitation
 THINK AORTIC DISECCTION
 Murmur: Diastolic, lower left, sternal border
 LOTS Signs: water hammer pulse, Austin Flint
Murmur, Duroziez’s Murmur, Quincke’s pulse, de
Musset’s sign, Lighthouse, Landolfi’s, Beck’s, etc, etc,
etc.
 Rx: Afterload reduction…..surgical
Mitral Stenosis
 Cardiovascular collapse in pregnant patient during
delivery
 Murmur: Diastolic, Opening SNAP

 Atrial fib common, blood backs up into left atrium ->
lungs = CHF, Chronic -> Hemoptysis
 AF can cause decompensation, crash quick due to
loss of KICK, CARDIOVERT if Acute.
Mitral Regurgitaion
 Ischemia + SHOCK + new MURMUR = ruptured
chordae tendineae/papillary muscle
 Murmur: Radiates widely, esp. into axilla

 Atrium stretches and produces A. Fib
 Mitral valve prolapse can get worse and overtime lead
to regurgitation
Conduction Disease
 Normal Conduction system
Bundle Branch and
Fascicular Blocks
 RBBB:
 ECD: Wide QRS, Abnormal QRS complexes in right
precordical leads (V1- V2) (rSR’). We know this.
 Incomplete RBBB

 RBBB block morphology with a normal QRS width
 Common finding in children and young adult
LBBB
 ECG: Wide QRS.
 Abnormal morphology: RR’ or large wide R (I, V5, V6)
Anormal repol., QS or RS pattern in right precordial leads
(V1,V2)
Hemi Blocks
 Left anterior vs posterior block
 Anterior more common (left coronary blood supply)
Ant: left axis deviation, QR (I, aVL), RS (II,III, aVF)

Post: Right axis, RS (I, aVL), QR (II,III, aVF)
 Bifascicular block
 Most common combination: LAF with RBBB
 Marker for advance cardiac disease
Heart Blocks
 SA node: Blood supply Rt corornary (65%), circumflex
(25%), both (10%)
 AV node: Post. Descending artery (rt coronary 90%)

 SA blocks (sick sinus, sinus pause, sinus arrest, etc.)
 Absence of P and ORS, and T cycles
 Ventricular activity -> dependent on escape rhythm
Rx: pacemaker + medication to suppress
tachydysrhythmias
AV node Blocks
 First –Degree AV Block – conduction delay in AV node, PR
prolong
 Second –Degree Block – intermittent loss of conduction
between artia and ventricle
 Mobitz I (Wenckebach) : PR increases until dropped beat,
generally goes not need emergency Tx
 Mobitz II: PR normal from beat to beat with an occ. Abrupt
dropped beat.
 Rx: Can progress to complete block, pacer.

 Third-degree AV Block – No conduction through AV
 No assos. of P and QRS
 Pace and pacemaker
Bradydysrhythmia
 Sinus Bradycardia
 <60bpm, high vagal tone, medications, hyothyroidism
 Signs and symptoms – generally asymptomatic, or
signs of hypoperfusion

 Rx: Direct towards degree of patient
symptoms, atropine, pacing, vasopressors.
Bradydsyrhythmia
Simplified!
Stable or Unstable?
Wide or Narrow?
Slow or VERY slow
Bradydysrhythmia
 WHY IS THIS PATIENT BRADYCARDIC

Ischemia
Drugs
Electrolytes
Stable or Unstable
 Same criteria as tachycardia
 BP, mentation, awake and talking? -> perfusion
Wide or Narrow
Wide (much worse than narrow)
= Block below AV node

= Slower
= More likely to Stop
= NOT atropine sensitive
Wide or Narrow
Narrow
= more stable

= Faster
=Atropine sensitive

=? Block at AV node
Treatment of Bradycardia
IVF, O2, Monitor
TCP (often fails) or TVP

Atropine (go slow, not good on wide QRS)
Epinephrine

Dopamine

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Arbi Ayvazian, DO- Valvular Disease, Conduction Disorders & Bradydysrhythmias- ARMC Emergency Medicine

  • 1. Valvular Disease, Conduction Disorder & Bradydysrhythmias Arbi Ayvazian D.O PGY2 Emergency Medicine ARMC 1/2014
  • 3. Valvular Disorder  Things to know  Endocarditiis presentation  Murmurs, Rheumatic HD  Specific high risk diseases
  • 4. Infective Endocarditis  Risk Factors: Abnormal or artificial valve  Mitral valve most common, IVDA -> Tricuspid (staph)  Most common bug -> Staph  Tooth extraction -> Strep  Acute -> high fever, murmur, flu like symp., younger  Subacute -> Strep viridans, Anemia, older  Prophylaxis? Depends on bug and procedure
  • 5. Infective Endocarditis  Vasculitis and Embolic manifestations  Janeway lesions: Non-tender, hemorrhagic, flat, on palms and soles.  Osler nodes -> tender, tips of fingers and toes  Roth spots and splinter hemorrhages
  • 6. Infective Endocarditis  Dx by echo, blood cultures, high ESR/CRP  Rx: Vancomycin for Staph, PCN for Strep  Prophylaxis if abnormal valve and procedure  Procedure site determines bug and Abx  Classic broad question -> dental and Amoxicilin, GI/GU more gram negative coverage  Controversial in mitral valve prolapse (no on boards)
  • 7. End Point of Valve Disease  Heart Fails and dilates  Valves become regurgitant  ECG shows LVH as ventricles expand  LBBB develops as heart and conduction system stretches which is poor prognostic sgin
  • 8. Murmurs: MR. ASS, MS.AID Mitral Regurgitation Mitral Stenosis Aortic Stenosis Aortic Insufficiency SYSYTOLIC DIASTOLIC
  • 9. Aortic Stenosis  Symptoms progress from : SOB, CHF, Syncope (bad!)  Murmur: Systolic, up into the neck, slow carotid upstroke  ECG : LVH, LBBB  Exercise-induced syncope  Vasodilators can make it worse  Rx: Surgical (moderate to severe)
  • 10. Aortic Regurgitation  THINK AORTIC DISECCTION  Murmur: Diastolic, lower left, sternal border  LOTS Signs: water hammer pulse, Austin Flint Murmur, Duroziez’s Murmur, Quincke’s pulse, de Musset’s sign, Lighthouse, Landolfi’s, Beck’s, etc, etc, etc.  Rx: Afterload reduction…..surgical
  • 11. Mitral Stenosis  Cardiovascular collapse in pregnant patient during delivery  Murmur: Diastolic, Opening SNAP  Atrial fib common, blood backs up into left atrium -> lungs = CHF, Chronic -> Hemoptysis  AF can cause decompensation, crash quick due to loss of KICK, CARDIOVERT if Acute.
  • 12. Mitral Regurgitaion  Ischemia + SHOCK + new MURMUR = ruptured chordae tendineae/papillary muscle  Murmur: Radiates widely, esp. into axilla  Atrium stretches and produces A. Fib  Mitral valve prolapse can get worse and overtime lead to regurgitation
  • 13. Conduction Disease  Normal Conduction system
  • 14. Bundle Branch and Fascicular Blocks  RBBB:  ECD: Wide QRS, Abnormal QRS complexes in right precordical leads (V1- V2) (rSR’). We know this.  Incomplete RBBB  RBBB block morphology with a normal QRS width  Common finding in children and young adult
  • 15. LBBB  ECG: Wide QRS.  Abnormal morphology: RR’ or large wide R (I, V5, V6) Anormal repol., QS or RS pattern in right precordial leads (V1,V2)
  • 16. Hemi Blocks  Left anterior vs posterior block  Anterior more common (left coronary blood supply) Ant: left axis deviation, QR (I, aVL), RS (II,III, aVF) Post: Right axis, RS (I, aVL), QR (II,III, aVF)  Bifascicular block  Most common combination: LAF with RBBB  Marker for advance cardiac disease
  • 17. Heart Blocks  SA node: Blood supply Rt corornary (65%), circumflex (25%), both (10%)  AV node: Post. Descending artery (rt coronary 90%)  SA blocks (sick sinus, sinus pause, sinus arrest, etc.)  Absence of P and ORS, and T cycles  Ventricular activity -> dependent on escape rhythm Rx: pacemaker + medication to suppress tachydysrhythmias
  • 18. AV node Blocks  First –Degree AV Block – conduction delay in AV node, PR prolong  Second –Degree Block – intermittent loss of conduction between artia and ventricle  Mobitz I (Wenckebach) : PR increases until dropped beat, generally goes not need emergency Tx  Mobitz II: PR normal from beat to beat with an occ. Abrupt dropped beat.  Rx: Can progress to complete block, pacer.  Third-degree AV Block – No conduction through AV  No assos. of P and QRS  Pace and pacemaker
  • 19. Bradydysrhythmia  Sinus Bradycardia  <60bpm, high vagal tone, medications, hyothyroidism  Signs and symptoms – generally asymptomatic, or signs of hypoperfusion  Rx: Direct towards degree of patient symptoms, atropine, pacing, vasopressors.
  • 21. Bradydysrhythmia  WHY IS THIS PATIENT BRADYCARDIC Ischemia Drugs Electrolytes
  • 22. Stable or Unstable  Same criteria as tachycardia  BP, mentation, awake and talking? -> perfusion
  • 23. Wide or Narrow Wide (much worse than narrow) = Block below AV node = Slower = More likely to Stop = NOT atropine sensitive
  • 24. Wide or Narrow Narrow = more stable = Faster =Atropine sensitive =? Block at AV node
  • 25.
  • 26. Treatment of Bradycardia IVF, O2, Monitor TCP (often fails) or TVP Atropine (go slow, not good on wide QRS) Epinephrine Dopamine

Notas do Editor

  1. SA node, vagal tone, AV node -&gt; normal slowing, if prolong something is wrong.