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Management of epistaxis
Susritha
Pg 2nd year
ASRAM;eluru.
Classification:
 Anterior epistaxis:
 Bleeding from a source anterior to the plane of the piriform
aperture.
 This includes bleeding from the anterior septum and rare
bleeds from the vestibular skin and mucocutaneous
junction.
 Posterior epistaxis:
 Bleeding from a vessel situated posterior to the piriform
aperture.
 This allows further subdivision into lateral wall, septal and
nasal floor bleeding.
Management of
epistaxis
direct
Bleeding point
specific
Endoscopic control
bipolar diathermy,
chemical cautery
(difficult in
posterior bleeds),
electrocautery
or direct pressure
from miniature
targeted packs
silver nitrate
cautery.
indirect
Donot require
identification of
bleeding points
Nasal packing:
Ribbon gauze
impregnated
with petroleum
jelly or
(BIPP) 24 and 72 hours.
sinusitis, septal
perforation, alar
necrosis,
hypoxia and
myocardial
infarction
Hot water irrigation:
vasodilatation
and reduction
in nasal lumen
dimensions
Hot water
irrigation 50C
Systemic medical therapy:
Tranexamic acid &
epsilon aminocaproic aci
reduce the severity and
risk of rebleeding
at a dose of 1.5 g three
times a day.
Preexisting
thromboembolicdisease
is a contraindication
Surgical
management
Posterior
packing
Ligation
techniques
Septal
surgery
embolisation
Posterior nasal packing
 General anaesthesia is preferrable.
 Nasopharyngeal tamponade is achieved using special
gauze packs inserted transorally and positioned by
means of tapes passed from the posterior choana to the
anterior nares bilaterally.
 Alternative is to insert a Foley urethral
catheter (size 12 or 14) along the floor
of the nasal cavity until the nasopharynx
is reached.
 The Foley catheter is inflated with up to
15mL of water, pulled forward to engage
in the posterior choana and anterior
packing is then inserted.
 The Foley catheter needs to be secured
anteriorly, taking care not to cause
pressure over the columella.
 Complications :
 Posterior packing causes considerable
pain and may cause hypoxia secondary to
soft palate oedema.
 Sinusitis and middle ear effusions are
common.
 More serious complications include
necrosis of the septum and columella.
Ligation:
 Ligation should be performed as close as possible to the
likely bleeding point.
 sphenopalatine artery;
 internal maxillary artery;
 external carotid artery;
 anterior/posterior ethmoidal artery.
sphenopalatine artery:
 An incision is made approximately 8mm anterior to and under
cover of the posterior end of the middle turbinate.
 The incision is carried down to the bone and a mucosal flap is
elevated.
 The foramen can be difficult to identify, but its location is
signalled by the crista ethmoidalis.
 Once the main vessel is identified, it can be ligated using
haemostatic clips and divided or coagulated using bipolar
diathermy
internal maxillary artery:
 The artery is exposed transantrally via
 anterior (sublabial) or
 combined anterior and medial(endoscopic) techniques.
 In the traditional sublabial approach, an antrostomy is
formed taking care to preserve the infraorbital nerve.
 The mucosa of the posterior wall of the antrum is then
elevated and a window is made through into the
pterygopalatine fossa.
 The branches of the internal maxillary artery are
identified pulsating within the fat of the fossa and are
carefully dissected out prior to clipping with haemostatic
clips.
 The proximal internal maxillary artery, descending
palatine and sphenopalatine branches are all clipped and
ideally divided.
 An endoscopic variation on this technique uses a middle
meatus antrostomy, as an instrument port with a 4-mm
endoscope is inserted through a small canine fossa
antrostomy.
 Transantral ligation controls haemorrhage in 89 percent
of cases.
 Complications include sinusitis, damage to the
infraorbital nerve, oroantral fistula, dental damage and
anaesthesia and rarely ophthalmoplegia and blindness.
ECAL:
 This procedure can be carried out
under local or general anaesthetic
using either a skin crease incision or a
longitudinal incision parallel with the
anterior border of the sternomastoid.
 The carotid bifurcation is identified
and the external carotid confirmed,
double-checked for arterial branches
and then ligated in continuity.
 Complications
 wound infection,
 haematoma and
 neurovasculardamage.
Anterior/posterior ethmoidal
artery ligation:
 The arteries are approached by a medial canthal incision
which is carried down to the bone of the anterior
lacrimal crest.
 Periosteal elevators are then used to elevate and
laterally retract the bulbar fascia.
 The anterior ethmoidal artery is seen as a
fibroneurovascular mesentry running from the bulbar
fascia into the anterior ethmoidal foramen.
 The vessel is clipped and divided and dissection is
continued to identify the posterior artery which is
located approximately 12mm behind.
Septal surgery:
 When epistaxis originates behind a prominent septal
deviation or vomeropalatine spur, septoplasty or
submucosal resection (SMR) may be required to access
the bleeding point.
 the blood supply to the septum is interrupted and
haemostasis secured.
embolization:
 It is essential to exclude arteriovenous malformations,
aneursyms and fistulae prior to embolization.
 Once the bleeding vessel is identified, a fine catheter is
passed into the internal maxillary circulation and
particles (polyvinyl alcohol, tungsten or steel microcoils)
are used to embolize the vessels.
 The ipsilateral facial artery is also embolized inorder to
prevent recirculation.
 Complications includeskin necrosis, paraesthesia,
cerebrovascular accident.
Secondary epistaxis:
 Epistaxis is commonly observed in patients with
coagulopathy secondary to liver disease, leukemia or
myelosuppression.
Hereditary haemorrhagic
telangiectasia
 (HHT)or Rendu–Osler–Weber disease, is an autosomal
dominant condition affecting blood vessels in the skin,
mucous membranes and viscera.
 The classical features are telangiectasia, arteriovenous
(AV) malformations and aneurysms. Recurrent epistaxis
occurs in 93 percent of cases.
 Management involves packing, cautery, antifibrinolytics,
systemic or topical oestrogens, coagulative lasers, septal
dermoplasty, ligation and embolization and, as a last
resort, permanent surgical closure of the nostrils
(Young’s operation)
Paediatric epistaxis:
 Enquire about the use of nasal sprays as intranasal
steroid sprays can cause localized nasal mucosal trauma
often in the region of the anterior end of the inferior
turbinate which can give rise to epistaxis.
 A concomitant vestibulitis should raise the suspicion of a
retained nasal foreign body.
 Idiopathic thrombocytopoenic purpura (ITPP) may
present as epistaxis.
 Nasal mycoses may need to be considered, particularly
in immunocompromised children, such as those
receiving chemotherapy.
 Commonly used methods for the management of
recurrent epistaxis in children are
 the prophylactic application of petroleum jelly to the nasal
vestibule and septum,
 the use of topical antiseptic creams, and
 cautery of Little’s area or the retrocollumellar veins.
 If the bleeding is persistently from one side, often we
find either a leash of vessels around Little’s area on the
affected side or a prominent retrocollumellar vein
 Cautery nowadays is most often undertaken using a silver
nitrate impregnated stick.
 Petroleum jelly is thought to be effective because it forms a
water-resistant film over the affected area of mucosa.
 In addition, antiseptic creams are thought to sterilize
localized infection in the region of the vestibule and the
nasal septum.
Summary:
 First line: direct therapy (bipolar/cautery, endoscopic if
required);
 Second line: indirect therapy (anterior packing);
 Third line: surgical therapy (ESPAL);
 Fourth line: angiography and embolization.
Thank you…
Journal club---04.04.2014
 By dr.bala teja (laryngeal amyloidosis)

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Management of epistaxis

  • 1. Management of epistaxis Susritha Pg 2nd year ASRAM;eluru.
  • 3.  Anterior epistaxis:  Bleeding from a source anterior to the plane of the piriform aperture.  This includes bleeding from the anterior septum and rare bleeds from the vestibular skin and mucocutaneous junction.  Posterior epistaxis:  Bleeding from a vessel situated posterior to the piriform aperture.  This allows further subdivision into lateral wall, septal and nasal floor bleeding.
  • 4. Management of epistaxis direct Bleeding point specific Endoscopic control bipolar diathermy, chemical cautery (difficult in posterior bleeds), electrocautery or direct pressure from miniature targeted packs silver nitrate cautery. indirect Donot require identification of bleeding points
  • 5.
  • 6. Nasal packing: Ribbon gauze impregnated with petroleum jelly or (BIPP) 24 and 72 hours. sinusitis, septal perforation, alar necrosis, hypoxia and myocardial infarction
  • 7.
  • 8. Hot water irrigation: vasodilatation and reduction in nasal lumen dimensions Hot water irrigation 50C
  • 9. Systemic medical therapy: Tranexamic acid & epsilon aminocaproic aci reduce the severity and risk of rebleeding at a dose of 1.5 g three times a day. Preexisting thromboembolicdisease is a contraindication
  • 11. Posterior nasal packing  General anaesthesia is preferrable.  Nasopharyngeal tamponade is achieved using special gauze packs inserted transorally and positioned by means of tapes passed from the posterior choana to the anterior nares bilaterally.
  • 12.
  • 13.  Alternative is to insert a Foley urethral catheter (size 12 or 14) along the floor of the nasal cavity until the nasopharynx is reached.  The Foley catheter is inflated with up to 15mL of water, pulled forward to engage in the posterior choana and anterior packing is then inserted.  The Foley catheter needs to be secured anteriorly, taking care not to cause pressure over the columella.  Complications :  Posterior packing causes considerable pain and may cause hypoxia secondary to soft palate oedema.  Sinusitis and middle ear effusions are common.  More serious complications include necrosis of the septum and columella.
  • 14. Ligation:  Ligation should be performed as close as possible to the likely bleeding point.  sphenopalatine artery;  internal maxillary artery;  external carotid artery;  anterior/posterior ethmoidal artery.
  • 15. sphenopalatine artery:  An incision is made approximately 8mm anterior to and under cover of the posterior end of the middle turbinate.  The incision is carried down to the bone and a mucosal flap is elevated.  The foramen can be difficult to identify, but its location is signalled by the crista ethmoidalis.  Once the main vessel is identified, it can be ligated using haemostatic clips and divided or coagulated using bipolar diathermy
  • 16.
  • 17. internal maxillary artery:  The artery is exposed transantrally via  anterior (sublabial) or  combined anterior and medial(endoscopic) techniques.  In the traditional sublabial approach, an antrostomy is formed taking care to preserve the infraorbital nerve.  The mucosa of the posterior wall of the antrum is then elevated and a window is made through into the pterygopalatine fossa.  The branches of the internal maxillary artery are identified pulsating within the fat of the fossa and are carefully dissected out prior to clipping with haemostatic clips.
  • 18.
  • 19.  The proximal internal maxillary artery, descending palatine and sphenopalatine branches are all clipped and ideally divided.  An endoscopic variation on this technique uses a middle meatus antrostomy, as an instrument port with a 4-mm endoscope is inserted through a small canine fossa antrostomy.  Transantral ligation controls haemorrhage in 89 percent of cases.  Complications include sinusitis, damage to the infraorbital nerve, oroantral fistula, dental damage and anaesthesia and rarely ophthalmoplegia and blindness.
  • 20. ECAL:  This procedure can be carried out under local or general anaesthetic using either a skin crease incision or a longitudinal incision parallel with the anterior border of the sternomastoid.  The carotid bifurcation is identified and the external carotid confirmed, double-checked for arterial branches and then ligated in continuity.  Complications  wound infection,  haematoma and  neurovasculardamage.
  • 21. Anterior/posterior ethmoidal artery ligation:  The arteries are approached by a medial canthal incision which is carried down to the bone of the anterior lacrimal crest.  Periosteal elevators are then used to elevate and laterally retract the bulbar fascia.  The anterior ethmoidal artery is seen as a fibroneurovascular mesentry running from the bulbar fascia into the anterior ethmoidal foramen.  The vessel is clipped and divided and dissection is continued to identify the posterior artery which is located approximately 12mm behind.
  • 22.
  • 23.
  • 24. Septal surgery:  When epistaxis originates behind a prominent septal deviation or vomeropalatine spur, septoplasty or submucosal resection (SMR) may be required to access the bleeding point.  the blood supply to the septum is interrupted and haemostasis secured.
  • 25. embolization:  It is essential to exclude arteriovenous malformations, aneursyms and fistulae prior to embolization.  Once the bleeding vessel is identified, a fine catheter is passed into the internal maxillary circulation and particles (polyvinyl alcohol, tungsten or steel microcoils) are used to embolize the vessels.  The ipsilateral facial artery is also embolized inorder to prevent recirculation.  Complications includeskin necrosis, paraesthesia, cerebrovascular accident.
  • 26. Secondary epistaxis:  Epistaxis is commonly observed in patients with coagulopathy secondary to liver disease, leukemia or myelosuppression.
  • 27. Hereditary haemorrhagic telangiectasia  (HHT)or Rendu–Osler–Weber disease, is an autosomal dominant condition affecting blood vessels in the skin, mucous membranes and viscera.  The classical features are telangiectasia, arteriovenous (AV) malformations and aneurysms. Recurrent epistaxis occurs in 93 percent of cases.  Management involves packing, cautery, antifibrinolytics, systemic or topical oestrogens, coagulative lasers, septal dermoplasty, ligation and embolization and, as a last resort, permanent surgical closure of the nostrils (Young’s operation)
  • 29.  Enquire about the use of nasal sprays as intranasal steroid sprays can cause localized nasal mucosal trauma often in the region of the anterior end of the inferior turbinate which can give rise to epistaxis.  A concomitant vestibulitis should raise the suspicion of a retained nasal foreign body.  Idiopathic thrombocytopoenic purpura (ITPP) may present as epistaxis.  Nasal mycoses may need to be considered, particularly in immunocompromised children, such as those receiving chemotherapy.
  • 30.  Commonly used methods for the management of recurrent epistaxis in children are  the prophylactic application of petroleum jelly to the nasal vestibule and septum,  the use of topical antiseptic creams, and  cautery of Little’s area or the retrocollumellar veins.  If the bleeding is persistently from one side, often we find either a leash of vessels around Little’s area on the affected side or a prominent retrocollumellar vein
  • 31.  Cautery nowadays is most often undertaken using a silver nitrate impregnated stick.  Petroleum jelly is thought to be effective because it forms a water-resistant film over the affected area of mucosa.  In addition, antiseptic creams are thought to sterilize localized infection in the region of the vestibule and the nasal septum.
  • 32. Summary:  First line: direct therapy (bipolar/cautery, endoscopic if required);  Second line: indirect therapy (anterior packing);  Third line: surgical therapy (ESPAL);  Fourth line: angiography and embolization.
  • 34. Journal club---04.04.2014  By dr.bala teja (laryngeal amyloidosis)