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Diseases of the Pulp
Presented by
Dr Rahul Katyayan
For the- Faculty of
Pediatric dentistry.
CONTENTS
⚫ INTRODUCTION
⚫ PULPITIS
⚫ SEQUELAE
⚫ CAUSES
⚫ CLASSIFICATION
⚫ REVERSIBLE PULPITIS
⚫ FOCAL REVERSIBLE PULPITIS
⚫ IRREVERSIBLE PULPITIS
⚫ ACUTE PULPITIS
⚫ CHRONIC PULPITIS
⚫ NECROSIS
⚫ REFERENCES
⮚ formative organ of tooth
⮚ builds primary dentin
during development of tooth
⮚ secondary dentin after
tooth eruption
⮚ reparative dentin in response
to stimulation as long as
odontoblast remain vital
Pulp
⮚ most common cause of
dental pain
⮚ loss of teeth in younger
persons
⮚ usual cause is caries
penetrating the dentin
Pulpitis
Pulpitis
UNTREATED
Death of pulp
Spread of Infection through
apical foramina into periapical tissues
Causes Periapical Periodontitis
⮚ (1)Physical Cause
a)Mechanical
b)Thermal
⮚ (2) Chemical Cause
⮚ (3) Bacterial Cause
Causes of Pulpal
Inflammation
⮚ (a) Mechanical Cause
A. trauma
a) accident (contact sports)
b) iatrogenic damage due to dental procedure
(during cavity or crown preparation)
B. Pathologic wear ( atrrition, abrasion)
C. Cracked tooth syndrome
D. Barometric changes
Causes of Pulpal
Inflammation
⮚ (b) Thermal Cause
✔ uninsulated metallic
restoration
✔ during cavity preparation
✔ polishing
Causes of Pulpal
Inflammation
⮚ (2) Chemical Cause
✔ arise from erosion
✔ or inappropriate use
of acidic dental material
Causes of Pulpal
Inflammation
⮚ (3) Bacterial Cause
✔ can damage pulp
through toxins secreted
by bacteria from caries
✔Microbial colonization in
the pulp by blood borne
microorganisms
(anachoresis).
Causes of Pulpal
Inflammation
⮚ (1) Based on Severity of
Inflammation
⮚ (2) According to Involvement
Classification
⮚ (1) Reversible Pulpitis
⮚ (2) Irreversible Pulpitis
⮚ (3) Pulp Degeneration
⮚ (4) Pulp Necrosis
(1) Based on Severity
of Inflammation
⮚ (1) Reversible Pulpitis
✔ Symptomatic (acute)
✔ Aysptomatic (chronic)
⮚ (2) Irreversible Pulpitis
✔ Acute
• Abnormally responsive to cold
• Abnormally responsive to heat
(1) Based on Severity
of Inflammation
⮚ (2) Irreversible Pulpitis
✔ Chronic
• Asymptomatic with
pulp exposure
• Hyperplastic
• Internal resorption
(1) Based on Severity
of Inflammation
⮚ (3) Pulp Degeneration
✔ Calcific
⮚ (4) Pulp Necrosis
(1) Based on Severity
of Inflammation
⮚ (1) According to extent of inflammation
⮚ (2) According to Severity
⮚ (3) According to presence or
absence of direct
communication
between dental pulp +
oral environment
(2) According to
Involvement
⮚ (1) According to extent of inflammation
✔ Focal or Subtotal or
Partial Pulpitis
✔ Total or Generalized
Pulpitis
(2) According to
Involvement
⮚ (2) According to Severity
✔ Acute
✔ Chronic
(2) According to
Involvement
⮚ (3) According to presence or
absence of direct
communication
between dental pulp +
oral environment
✔ Pulpitis Aperts (open pulpitis)
✔ Pulpitis Clausa (closed pulpitis)
(2) According to
Involvement
⮚ mild to moderate inflammatory
condition of pulp
✔ caused by noxious stimuli
✔ pulp is capable of returning
to un-inflammed state following removal of stimuli
Reversible Pulpitis
⮚ Causes
✔ agent capable of
injuring pulp like:
• trauma
• disturbed occlusal
relationship
• thermal shock
Reversible Pulpitis
⮚ Clinical Features
✔ sharp pain lasting for
a moment
✔ often brought on by cold
than hot food or beverages
and by cold air
Reversible Pulpitis
⮚ Clinical Features
✔ does not continue
when the cause has been
removed
✔ tooth responds to electric
pulp testing at lower
current
Reversible Pulpitis
⮚ Management
✔ prevention
✔ periodic care
✔ early insertion of filling
if a cavity has developed
✔ removal of noxious
stimuli
Reversible Pulpitis
⮚earliest form
⮚ also known as pulp hyperemia
⮚ excessive accumulation of
blood within pulp tissue
⮚ leads to vascular congestion
Focal Reversible
Pulpitis
⮚ Clinical Features
✔ sensitive to thermal
changes
✔ particularly to cold
✔ application of ice or cold
fluids to tooth result in pain
Focal Reversible
Pulpitis
⮚ Clinical Features
✔ disappears upon removal
of thermal irritant or
restoration .
✔ responds to electrical test
stimulant at lower level
of current
Focal Reversible
Pulpitis
⮚ Clinical Features
✔ indicates lower pain
threshold than that of
adjacent normal
teeth
Focal Reversible
Pulpitis
⮚ Clinical Features
✔ teeth show:
• deep carious lesion
• large metallic restoration
• restoration with defective
margins
Focal Reversible
Pulpitis
⮚ Management
✔ removal of irritants
before the pulp is
severely damaged
Focal Reversible
Pulpitis
⮚ persistent inflammatory
condition of pulp
⮚ may be symptomatic or
asymptomatic
⮚ caused by noxious stimulus
Irreversible Pulpitis
⮚ Causes
✔ bacteria involvement of
pulp through caries
✔ chemical
✔ thermal
✔ mechanical injury
Irreversible Pulpitis
⮚ Clinical Features
Early Stage
✔ paroxysm of pain
caused by:
• sudden temperature
changes like cold,
sweet, acid foodstuffs
Irreversible Pulpitis
⮚ Clinical Features
Early Stage
✔ pain often continues
when cause has been
removed
✔ may come and go
spontaneously
Irreversible Pulpitis
⮚ Clinical Features
Early Stage
✔ pain
• sharp
• piercing
• shooting
• generally severe
Irreversible Pulpitis
⮚ Clinical Features
Early Stage
✔ pain
• bending over exacerbates pain which
• lying down is due to change in
• change of position intrapulpal pressure
Irreversible Pulpitis
⮚ Clinical Features
Late Stage
✔ pain
• more severe as if tooth is under
• throbbing constant pressure
Irreversible Pulpitis
⮚ Clinical Features
Late Stage
✔ pain
• patient is often awake
at night due to pain
• increased by heat and
sometimes relieved by cold,
although continued application
of cold may intensify pain
Irreversible Pulpitis
⮚ Management
✔ complete removal of pulp
or pulpectomy
✔ if the time is a factor, pulpotomy
should be done as an emergency
procedure
✔ Surgical removal should be considered if the tooth
is not restorable.
Irreversible Pulpitis
Clinical Difference
Reversible Pulpitis Irreversible Pulpitis
⮚ pain generally lasts for few
seconds
⮚ the pain produced by thermal
stimulus disappears as soon as
the stimulus is removed
Pain may last several
minutes or later
⮚ pain lingers even after
the stimulus is removed
⮚ pain may come without
any apparent stimulus
⮚ extensive acute inflammation
of pulp
⮚ frequent sequel of focal
reversible pulpitis
Acute Pulpitis
⮚ Causes
✔ tooth with large carious
lesion
✔ defective restoration
where there has been
recurrent caries
✔ pulp exposure due to
faulty cavity preparation
Acute Pulpitis
⮚ Clinical Features
✔ severe pain is elicited by
thermal changes
✔ pain persists even after
thermal stimulus
disappears or been
removed
Acute Pulpitis
⮚ Clinical Features
✔ may be continuous
✔ intensity may be increased
when patient lies down
✔ application of heat
may cause acute
exacerbation of pain
Acute Pulpitis
⮚ Clinical Features
✔ tooth reacts to electric
pulp vitality tester at a
lower level of current
than adjacent normal
teeth
Acute Pulpitis
⮚ Clinical Features
✔ pressure increases
because of lack of
escape of inflammatory
exudate
✔ rapid spread of inflammation
through pulp with pain
+ necrosis
Acute Pulpitis
⮚ Management
✔ early stages of acute pulpitis pulpotomy
(removal of coronal pulp)
✔ placing material that
favors calcification such
as:
• calcium hydroxide
over entrance of
root canals
Acute Pulpitis
⮚ Management
✔ root canal filling with
inert material like
gutta percha should be
done
Acute Pulpitis
⮚ may develop with or
without episodes of
acute pulpitis
⮚ many pulps under large
carious cavities die painlessly
⮚ 1st indication is then
development of periapical
periodontitis, either with pain
or seen by chance in radiograph
Chronic Pulpitis
⮚ Clinical Features
✔ dull aching type
✔ more often intermittent
than continuous
Chronic Pulpitis
⮚ Management
✔ root canal therapy
✔ followed by crown
restoration
Chronic Pulpitis
⮚ also called as pulp polyp
or pulpitis aperta
⮚ essentially an excessive
exuberant proliferation
of chronically inflammed
dental pulp tissue
Chronic Hyperplastic
Pulpitis
⮚ pulpal inflammation due
to an extensive carious
exposure of a young pulp
⮚ development of granulation
tissue
⮚ covered at times by epithelium
⮚ resulting from long standing
low grade infection
Chronic Hyperplastic
Pulpitis
⮚ Causes
✔ slow progressive
exposure of pulp
✔ bacterial infection
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ most commonly involved
are deciduous molars +
1st permanent molar
• excellent blood supply
• large root opening
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ asymptomatic
✔ seen only in teeth of children
+ young adults
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ polypoid tissue appears
• fleshy
• reddish pulpal mass filling
most of pulp chamber
or cavity
• or even extend beyond
confines of tooth
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ polypoid tissue appears
• sometimes, if mass is
large enough
• interferes with closure
of mouth
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ polypoid tissue appears
• may cause discomfort
during mastication
• due to pressure of food
bolus
Chronic Hyperplastic
Pulpitis
⮚ Clinical Features
✔ polypoid tissue appears
• tissue easily bleeds
because of rich network
of blood vessels
• tooth may respond or
not at all to thermal test
Chronic Hyperplastic
Pulpitis
⮚ Management
✔ elimination of polypoid tissue
✔ followed by extirpation of pulp
✔ hyperplastic tissue bleeeding
can be controlled by pressure
✔ pulpectomy or extraction of tooth can also
be done if the tooth is not restorable.
Chronic Hyperplastic
Pulpitis
⮚ death of pulp
⮚ may be partial or total
depending on whether part
or the entire pulp is
involved
Necrosis
⮚ Causes
✔ sequeala of inflammation
✔ can also occur following
trauma
• pulp is destroyed before
an inflammatory reaction
Necrosis
⮚ Types
✔ (1) Coagulation Necrosis
✔ (2) Liquefaction Necrosis
Necrosis
⮚ Types
✔ (1) Coagulation Necrosis
• soluble portion of
tissue is precipitated
or converted into a solid
material
Necrosis
⮚ Types
✔ (1) Coagulation Necrosis
• tissue is converted into
tissue mass consisting
chiefly of coagulated
▪ proteins
▪ fats
▪ water
Necrosis
⮚ Types
✔ (2) Liquefaction Necrosis
• results when proteolytic
enzymes convert the
tissue into softened mass
liquid or amorphous debris
Necrosis
⮚ Clinical Features
✔ no painful symptoms
✔ discoloration of tooth
• 1st indication that the pulp
is dead
Necrosis
⮚ Clinical Features
✔ history of pain lasting from
a few minutes to a few
hours followed by
complete + sudden
cessation of pain
Necrosis
⮚ Management
✔ preparation + obturation of
root canals
Necrosis
References:
❖ Books
⮚ Grossman’s endodontic practice twelfth edition
⮚Textbook of oral pathology shafers
⮚Cawson, R.A: Cawson’s Essentials of Oral Pathology and
Oral Medicine, 8th Edition.(page 60)
⮚ Ghom, Ali & Mhaske, Shubhangi: Textbook of Oral
Pathology. (pages 420-425)
References:
⮚Adrian,J.C.,et al.:J. Am. Dent. Assoc.,83:113,
1971.
⮚Allard, U., et al.:Oral Surg.,48:454, 1979.
⮚Austin, L.T.:J. Am. Dent. Assoc.,17:1930, 1930.
⮚Baume, L.: SSO Schweiz. Monatsschr.
Zahnheilkd.,77:1082, 1965.
References:
⮚Baume, L.: Transactions of the Fourth
International Conference on Endodontics.
Philadelphia: University of Pennsylvania
press, 1968, p. 66 .
⚫Baume, L.J.: Monogr. Oral Sci.,8:1-
220, 1980.
THANK
YOU

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Diseases of pulp

  • 1. Diseases of the Pulp Presented by Dr Rahul Katyayan For the- Faculty of Pediatric dentistry.
  • 2. CONTENTS ⚫ INTRODUCTION ⚫ PULPITIS ⚫ SEQUELAE ⚫ CAUSES ⚫ CLASSIFICATION ⚫ REVERSIBLE PULPITIS ⚫ FOCAL REVERSIBLE PULPITIS ⚫ IRREVERSIBLE PULPITIS ⚫ ACUTE PULPITIS ⚫ CHRONIC PULPITIS ⚫ NECROSIS ⚫ REFERENCES
  • 3. ⮚ formative organ of tooth ⮚ builds primary dentin during development of tooth ⮚ secondary dentin after tooth eruption ⮚ reparative dentin in response to stimulation as long as odontoblast remain vital Pulp
  • 4. ⮚ most common cause of dental pain ⮚ loss of teeth in younger persons ⮚ usual cause is caries penetrating the dentin Pulpitis
  • 5. Pulpitis UNTREATED Death of pulp Spread of Infection through apical foramina into periapical tissues Causes Periapical Periodontitis
  • 6. ⮚ (1)Physical Cause a)Mechanical b)Thermal ⮚ (2) Chemical Cause ⮚ (3) Bacterial Cause Causes of Pulpal Inflammation
  • 7. ⮚ (a) Mechanical Cause A. trauma a) accident (contact sports) b) iatrogenic damage due to dental procedure (during cavity or crown preparation) B. Pathologic wear ( atrrition, abrasion) C. Cracked tooth syndrome D. Barometric changes Causes of Pulpal Inflammation
  • 8. ⮚ (b) Thermal Cause ✔ uninsulated metallic restoration ✔ during cavity preparation ✔ polishing Causes of Pulpal Inflammation
  • 9. ⮚ (2) Chemical Cause ✔ arise from erosion ✔ or inappropriate use of acidic dental material Causes of Pulpal Inflammation
  • 10. ⮚ (3) Bacterial Cause ✔ can damage pulp through toxins secreted by bacteria from caries ✔Microbial colonization in the pulp by blood borne microorganisms (anachoresis). Causes of Pulpal Inflammation
  • 11. ⮚ (1) Based on Severity of Inflammation ⮚ (2) According to Involvement Classification
  • 12. ⮚ (1) Reversible Pulpitis ⮚ (2) Irreversible Pulpitis ⮚ (3) Pulp Degeneration ⮚ (4) Pulp Necrosis (1) Based on Severity of Inflammation
  • 13. ⮚ (1) Reversible Pulpitis ✔ Symptomatic (acute) ✔ Aysptomatic (chronic) ⮚ (2) Irreversible Pulpitis ✔ Acute • Abnormally responsive to cold • Abnormally responsive to heat (1) Based on Severity of Inflammation
  • 14. ⮚ (2) Irreversible Pulpitis ✔ Chronic • Asymptomatic with pulp exposure • Hyperplastic • Internal resorption (1) Based on Severity of Inflammation
  • 15. ⮚ (3) Pulp Degeneration ✔ Calcific ⮚ (4) Pulp Necrosis (1) Based on Severity of Inflammation
  • 16. ⮚ (1) According to extent of inflammation ⮚ (2) According to Severity ⮚ (3) According to presence or absence of direct communication between dental pulp + oral environment (2) According to Involvement
  • 17. ⮚ (1) According to extent of inflammation ✔ Focal or Subtotal or Partial Pulpitis ✔ Total or Generalized Pulpitis (2) According to Involvement
  • 18. ⮚ (2) According to Severity ✔ Acute ✔ Chronic (2) According to Involvement
  • 19. ⮚ (3) According to presence or absence of direct communication between dental pulp + oral environment ✔ Pulpitis Aperts (open pulpitis) ✔ Pulpitis Clausa (closed pulpitis) (2) According to Involvement
  • 20. ⮚ mild to moderate inflammatory condition of pulp ✔ caused by noxious stimuli ✔ pulp is capable of returning to un-inflammed state following removal of stimuli Reversible Pulpitis
  • 21. ⮚ Causes ✔ agent capable of injuring pulp like: • trauma • disturbed occlusal relationship • thermal shock Reversible Pulpitis
  • 22. ⮚ Clinical Features ✔ sharp pain lasting for a moment ✔ often brought on by cold than hot food or beverages and by cold air Reversible Pulpitis
  • 23. ⮚ Clinical Features ✔ does not continue when the cause has been removed ✔ tooth responds to electric pulp testing at lower current Reversible Pulpitis
  • 24. ⮚ Management ✔ prevention ✔ periodic care ✔ early insertion of filling if a cavity has developed ✔ removal of noxious stimuli Reversible Pulpitis
  • 25. ⮚earliest form ⮚ also known as pulp hyperemia ⮚ excessive accumulation of blood within pulp tissue ⮚ leads to vascular congestion Focal Reversible Pulpitis
  • 26. ⮚ Clinical Features ✔ sensitive to thermal changes ✔ particularly to cold ✔ application of ice or cold fluids to tooth result in pain Focal Reversible Pulpitis
  • 27. ⮚ Clinical Features ✔ disappears upon removal of thermal irritant or restoration . ✔ responds to electrical test stimulant at lower level of current Focal Reversible Pulpitis
  • 28. ⮚ Clinical Features ✔ indicates lower pain threshold than that of adjacent normal teeth Focal Reversible Pulpitis
  • 29. ⮚ Clinical Features ✔ teeth show: • deep carious lesion • large metallic restoration • restoration with defective margins Focal Reversible Pulpitis
  • 30. ⮚ Management ✔ removal of irritants before the pulp is severely damaged Focal Reversible Pulpitis
  • 31. ⮚ persistent inflammatory condition of pulp ⮚ may be symptomatic or asymptomatic ⮚ caused by noxious stimulus Irreversible Pulpitis
  • 32. ⮚ Causes ✔ bacteria involvement of pulp through caries ✔ chemical ✔ thermal ✔ mechanical injury Irreversible Pulpitis
  • 33. ⮚ Clinical Features Early Stage ✔ paroxysm of pain caused by: • sudden temperature changes like cold, sweet, acid foodstuffs Irreversible Pulpitis
  • 34. ⮚ Clinical Features Early Stage ✔ pain often continues when cause has been removed ✔ may come and go spontaneously Irreversible Pulpitis
  • 35. ⮚ Clinical Features Early Stage ✔ pain • sharp • piercing • shooting • generally severe Irreversible Pulpitis
  • 36. ⮚ Clinical Features Early Stage ✔ pain • bending over exacerbates pain which • lying down is due to change in • change of position intrapulpal pressure Irreversible Pulpitis
  • 37. ⮚ Clinical Features Late Stage ✔ pain • more severe as if tooth is under • throbbing constant pressure Irreversible Pulpitis
  • 38. ⮚ Clinical Features Late Stage ✔ pain • patient is often awake at night due to pain • increased by heat and sometimes relieved by cold, although continued application of cold may intensify pain Irreversible Pulpitis
  • 39. ⮚ Management ✔ complete removal of pulp or pulpectomy ✔ if the time is a factor, pulpotomy should be done as an emergency procedure ✔ Surgical removal should be considered if the tooth is not restorable. Irreversible Pulpitis
  • 40. Clinical Difference Reversible Pulpitis Irreversible Pulpitis ⮚ pain generally lasts for few seconds ⮚ the pain produced by thermal stimulus disappears as soon as the stimulus is removed Pain may last several minutes or later ⮚ pain lingers even after the stimulus is removed ⮚ pain may come without any apparent stimulus
  • 41. ⮚ extensive acute inflammation of pulp ⮚ frequent sequel of focal reversible pulpitis Acute Pulpitis
  • 42. ⮚ Causes ✔ tooth with large carious lesion ✔ defective restoration where there has been recurrent caries ✔ pulp exposure due to faulty cavity preparation Acute Pulpitis
  • 43. ⮚ Clinical Features ✔ severe pain is elicited by thermal changes ✔ pain persists even after thermal stimulus disappears or been removed Acute Pulpitis
  • 44. ⮚ Clinical Features ✔ may be continuous ✔ intensity may be increased when patient lies down ✔ application of heat may cause acute exacerbation of pain Acute Pulpitis
  • 45. ⮚ Clinical Features ✔ tooth reacts to electric pulp vitality tester at a lower level of current than adjacent normal teeth Acute Pulpitis
  • 46. ⮚ Clinical Features ✔ pressure increases because of lack of escape of inflammatory exudate ✔ rapid spread of inflammation through pulp with pain + necrosis Acute Pulpitis
  • 47. ⮚ Management ✔ early stages of acute pulpitis pulpotomy (removal of coronal pulp) ✔ placing material that favors calcification such as: • calcium hydroxide over entrance of root canals Acute Pulpitis
  • 48. ⮚ Management ✔ root canal filling with inert material like gutta percha should be done Acute Pulpitis
  • 49. ⮚ may develop with or without episodes of acute pulpitis ⮚ many pulps under large carious cavities die painlessly ⮚ 1st indication is then development of periapical periodontitis, either with pain or seen by chance in radiograph Chronic Pulpitis
  • 50. ⮚ Clinical Features ✔ dull aching type ✔ more often intermittent than continuous Chronic Pulpitis
  • 51. ⮚ Management ✔ root canal therapy ✔ followed by crown restoration Chronic Pulpitis
  • 52. ⮚ also called as pulp polyp or pulpitis aperta ⮚ essentially an excessive exuberant proliferation of chronically inflammed dental pulp tissue Chronic Hyperplastic Pulpitis
  • 53. ⮚ pulpal inflammation due to an extensive carious exposure of a young pulp ⮚ development of granulation tissue ⮚ covered at times by epithelium ⮚ resulting from long standing low grade infection Chronic Hyperplastic Pulpitis
  • 54. ⮚ Causes ✔ slow progressive exposure of pulp ✔ bacterial infection Chronic Hyperplastic Pulpitis
  • 55. ⮚ Clinical Features ✔ most commonly involved are deciduous molars + 1st permanent molar • excellent blood supply • large root opening Chronic Hyperplastic Pulpitis
  • 56. ⮚ Clinical Features ✔ asymptomatic ✔ seen only in teeth of children + young adults Chronic Hyperplastic Pulpitis
  • 57. ⮚ Clinical Features ✔ polypoid tissue appears • fleshy • reddish pulpal mass filling most of pulp chamber or cavity • or even extend beyond confines of tooth Chronic Hyperplastic Pulpitis
  • 58. ⮚ Clinical Features ✔ polypoid tissue appears • sometimes, if mass is large enough • interferes with closure of mouth Chronic Hyperplastic Pulpitis
  • 59. ⮚ Clinical Features ✔ polypoid tissue appears • may cause discomfort during mastication • due to pressure of food bolus Chronic Hyperplastic Pulpitis
  • 60. ⮚ Clinical Features ✔ polypoid tissue appears • tissue easily bleeds because of rich network of blood vessels • tooth may respond or not at all to thermal test Chronic Hyperplastic Pulpitis
  • 61. ⮚ Management ✔ elimination of polypoid tissue ✔ followed by extirpation of pulp ✔ hyperplastic tissue bleeeding can be controlled by pressure ✔ pulpectomy or extraction of tooth can also be done if the tooth is not restorable. Chronic Hyperplastic Pulpitis
  • 62. ⮚ death of pulp ⮚ may be partial or total depending on whether part or the entire pulp is involved Necrosis
  • 63. ⮚ Causes ✔ sequeala of inflammation ✔ can also occur following trauma • pulp is destroyed before an inflammatory reaction Necrosis
  • 64. ⮚ Types ✔ (1) Coagulation Necrosis ✔ (2) Liquefaction Necrosis Necrosis
  • 65. ⮚ Types ✔ (1) Coagulation Necrosis • soluble portion of tissue is precipitated or converted into a solid material Necrosis
  • 66. ⮚ Types ✔ (1) Coagulation Necrosis • tissue is converted into tissue mass consisting chiefly of coagulated ▪ proteins ▪ fats ▪ water Necrosis
  • 67. ⮚ Types ✔ (2) Liquefaction Necrosis • results when proteolytic enzymes convert the tissue into softened mass liquid or amorphous debris Necrosis
  • 68. ⮚ Clinical Features ✔ no painful symptoms ✔ discoloration of tooth • 1st indication that the pulp is dead Necrosis
  • 69. ⮚ Clinical Features ✔ history of pain lasting from a few minutes to a few hours followed by complete + sudden cessation of pain Necrosis
  • 70. ⮚ Management ✔ preparation + obturation of root canals Necrosis
  • 71. References: ❖ Books ⮚ Grossman’s endodontic practice twelfth edition ⮚Textbook of oral pathology shafers ⮚Cawson, R.A: Cawson’s Essentials of Oral Pathology and Oral Medicine, 8th Edition.(page 60) ⮚ Ghom, Ali & Mhaske, Shubhangi: Textbook of Oral Pathology. (pages 420-425)
  • 72. References: ⮚Adrian,J.C.,et al.:J. Am. Dent. Assoc.,83:113, 1971. ⮚Allard, U., et al.:Oral Surg.,48:454, 1979. ⮚Austin, L.T.:J. Am. Dent. Assoc.,17:1930, 1930. ⮚Baume, L.: SSO Schweiz. Monatsschr. Zahnheilkd.,77:1082, 1965.
  • 73. References: ⮚Baume, L.: Transactions of the Fourth International Conference on Endodontics. Philadelphia: University of Pennsylvania press, 1968, p. 66 . ⚫Baume, L.J.: Monogr. Oral Sci.,8:1- 220, 1980.