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Complication of traumatology theme lecture
1. COMPLICATIONS IN TRAUMATOLOGY
DR. UTKARSH SHAHI
ASSISTANT PROFESSOR IN ORTHOPEDICS
DEPARTMENT OF SURGERY
KING FAISAL UNIVERSITY
AL AHSA, KSA
2. LEARNING QUESTIONS
• What are the immediate complications of a fracture? Describe the diagnosis &
treatment.
• What are the early complications of a fracture? Describe the diagnosis & treatment.
• What are the late complications of a fracture? Describe the diagnosis & treatment.
• What are the advantages, disadvantages and complications of a fracture treatment
using traction, plaster cast, external fixator, plate & screw osteosynthesis, cerclage &
intramedullary pin fixation?
7. HEMORRHAGE: INTERVENTIONS
Direct pressure
Surgical intervention as indicated
Use of autologous or synthetic clotting material
Vitamin K or clotting replacement factors
Volume replacement and blood transfusion as necessary
Iron supplementation
9. BLOOD VESSELS : VASCULAR INJURY
Relatively uncommon event when associated with fractures.
When it occurs, it is always an emergent situation.
Injury to the artery is classically associated with several
specific fractures involving such sites as :
• Clavicle
• Supracondylar region of the elbow (especially in children)
• Femoral shaft
• Around the knee
10. NERVE INJURY
Typically, a nerve is compressed, contused, or
stretched.
Classic examples include:
• Radial nerve injury secondary to fractures of the distal humerus
• Sciatic nerve injury following posterior fracture dislocations of
hip
11. Neuropraxia.
• Death of the axon does not occur.
• The condition is generally caused by pressure or contusion and usually
improves by itself in a few weeks.
• The nerve is anatomically intact and physiologically nonfunctional.
Axonotmesis.
• Is an anatomic disruption of the axon in its sheath.
• Improvement follows regeneration, the axon growing at a slow rate of 1
mm a day along the existing axonal sheath.
Neurotmesis.
• Is an anatomic disruption of the nerve itself.
• Surgical repair is required if recovery is to be anticipated.
12.
13. MUSCLE AND TENDON INJURY
With any fracture or dislocation there is always some associated
muscle damage.
The extent of this damage and the results will vary depending on
the site in consideration.
Myositis ossificans is a specific complication of muscle damage in
which heterotopic bone forms within the damaged muscle.
The quadriceps and brachialis are specifically predisposed to
develop this complication.
14. ADULT RESPIRATORY DISTRESS SYNDROME
Also known as Shock lung or wet lung.
Can follow slight fluid overload and is made worse if:
• There is any damage to the lungs
• Aspiration into the lung or over-transfusion.
Oedema and electrolyte retention secondary to the trauma also contribute to it.
Treatment is by oxygen and ventilation.
Do not over-transfuse with crystalloids!
15. FAT EMBOLI SYNDROME (FES)
Mechanical blockage of blood
vessels by circulating fat particles
Occurs following
• Long bone fracture
• Pelvic fracture
• Total hip arthroplasty
16. FES: CLINICAL MANIFESTATIONS
Signs and symptoms can appear 12-
72 hours post injury
• Change in mental status
• Increased respiratory distress
• Petechial of skin & mucosa (appear
above nipple line and blanch)
17. FES: DIAGNOSTICS
No specific labs
Fat globules may be detected in blood, urine or sputum
PO2 drops to < 50 mm HG
Chest X Ray with diffuse “snowstorm” effect
VQ scan to r/o PE
18. FES: INTERVENTIONS
Early recognition to prevent morbidity and mortality
Minimize movement of long bone fractures
Respiratory support
• Intubation
• Ventilator management
• ICU monitoring
19. DEEP VEIN THROMBOSIS (DVT)
AND PULMONARY EMBOLISM (PE)
Formation of fibrin leads to development of a thrombus (fibrin clot)
Clinical symptoms appear when thrombus is large enough to
impede blood flow in a large vessel
When venous thrombosis or part of a thrombus dislodges from its
primary site, it becomes an embolus
Embolus can enter pulmonary circulation and perfusion distal to the
embolus can be partially or completely occluded
20. DVT: CLINICAL MANIFESTATIONS
Unilateral swelling of thigh/lower leg
Discomfort in leg
Erythema
Warmth
Tenderness
Palpable, tender venous cord in popliteal space
23. DVT & PE: PREVENTION
External pneumatic compression
Graduated compression stockings
Early ambulation and range of motion
Elevation of lower extremities
25. DVT & PE: PREVENTION
Full dose anticoagulation with heparin/warfarin
Oxygen therapy for PE
Thrombolytic therapy
• Urokinase
• streptokinase
Surgical embolectomy
Inferior vena cava filter
26. CRUSH SYNDROME
Also known as traumatic rhabdomyolysis or Bywaters' syndrome
It is characterized by major shock and renal failure after a crushing injury to skeletal
muscle.
It is compression of extremities or other parts of the body that causes muscle swelling
and/or neurological disturbances in the affected areas of the body.
Cases occur commonly in catastrophes such as earthquakes, to victims that have been
trapped under fallen masonry.
Treatment is complex & it should be started at the earliest to avoid irreversible damage
27. DISSEMINATED INTRAVASCULAR COAGULATION
Also known as Diffuse intravascular coagulation (DIC)
Can follow any injury and is due to a disturbance of the
clotting mechanism.
Treatment is done with:
• Fresh frozen plasma
• Platelets
• Heparin.
28. COMPARTMENT SYNDROME
“A condition in which the circulation and function
of tissues within a closed space compromised by
an increased pressure within that space”
The muscles and nerves of the extremity are
enclosed in fascial spaces or compartments and are
therefore susceptible to this condition
33. COMPARTMENT SYNDROME: DIAGNOSTICS
Muscle damage indicated by:
• Myoglobin in urine
• Elevated CPK, LDH and SGOT
Intracompartmental pressure monitor
• Pressures of 30-45 mm HG a concern
34. INTRACOMPARTMENTAL PRESSURES (ICP)
Pain can be unreliable especially in the trauma patient.
It can range from being mild to severe, and in the unconscious patient important
clinical symptoms and signs can be difficult to elicit.
Techniques have been developed to measure ICPs.
The normal tissue pressure within closed compartments is 0–10mmHg.
This pressure markedly increases in compartment syndrome
35. COMPARTMENT SYNDROME
PREVENTION
Early recognition is key to preventing or minimizing negative
outcome
Astute nursing intervention to identify pathologic pain in the
presence of good pain control (epidural, PCA)
The key to successful treatment of acute compartment syndrome is
early diagnosis and decompression of the affected compartments
38. WOUND/SSI
CLINICAL MANIFESTATIONS
Increased pain
Fever or chills
Foul smell from wound
Edema
Increased temperature around incision or wound
Erythema around wound or incision
Purulent exudate, poor wound healing
Elevated WBC, ESR, C-reactive protein
41. NON-UNION
• Delayed union non-union.
• No progression of healing over serial radiographs
• On x-ray :
• Hypertrophic …due to excessive mobility
• Atrophic…due to poor blood supply
• Needs intervention.
42.
43. NON-UNION TREATMENT
• Conservative;
• No symptoms no treatment
or removable splint.
• Symptomatic
• Hypertrophic (functional
Bracing)
• EMG Stimulation
• Pulsed US
• Operative
• Hypertrophic fixation
• Atrophic excision , bone
graft and fixation
44. MALUNION
• Union in an unsatisfactory position:
• Angulation
• Rotation
• Shortening
• Causes:
• Failure to reduce fracture adequately
• Failure to hold reduction while healing
• Gradual collapse of comminuted or osteoporotic bone
45. MALUNION
• Clinical Features:
• Deformity
• Usually visible in X - ray
• May be not apparent in some cases
• On x-ray:
• Check the position in the 1st 3 weeks
46. MALUNION TREATMENT
• Can be started before the fracture is fully united.
• Adults reduce to the anatomical position
• More than 10 to15 degrees may need;
• Re-manipulation
• Osteotomy
• Internal fixation
47. MALUNION TREATMENT
• Children
• Angular deformity resolve without treatment
• Rotational deformity needs intervention
• In lower limb shortening ( 2cm or more ) : lengthening
48. AVASCULAR NECROSIS
• Bone death due to interruption of blood supply
• Certain regions more than others include:
• Head of femur
• Proximal part of scaphoid
• Lunate
• Body of talus
• It is an early complication because of ischemia but
radiological findings are seen later