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A seminar I made during my first year of medical college on the pathophysiology of RDS. feel free to tell me what you think.
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A seminar I made during my first year of medical college on the pathophysiology of RDS. feel free to tell me what you think.
Respiratory distress syndrome [RDS]
Respiratory distress syndrome [RDS]
Alan Mizwari
Nrds
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Deep Deep
DR. ROOPAM JAIN, Professor & Head, Department of Pathology & Blood Bank
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Rds ppt.
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RESPIRATORY DISTRESS SYNDROME, PREVIOUSLY HYALINE MEMBRANE DISEASE IS A COMMON COMPLICATION OF PREMATURITY WITH MORTALITY ALMOST 100% IN THE ABSENCE OF PULMONARY SURFACTANT ADMINISTRATION, ESPECIALLY IN LOW RESOURCE SETTINGS LIKE OURS.
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Acute respiratory distress syndrome is a type of respiratory failure characterized by rapid onset of widespread inflammation in the lungs.
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the common type of respiratory distress in newborn. To be in paeds, we have to understand these properly. I tried my best to make proper presentation.
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Hi guys, This ppt shows the pathophysiology of pulmonary surfactant in newborn and respiratory distress syndrome. Main focus is towards management of RDS esp. exogenous surfactant administration. Your comments are welcome. Thank you.
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Smart parking sensor networks can provide accurate data drawn directly from actual parking behaviors in every space – not just estimates. The benefit of this knowledge allows communities large and small to arrive at tangible applications to help them thrive and grow. For example, optimized parking in retail areas, i.e. less empty spots and fair enforcement of time limits, can improve turnover of spaces (number of cars that occupy a space per day). Increased turnover in retail areas typically leads to increased sales for that area's retailers. This then leads to greater revenue through taxes and property values for a community. That in turn attracts new businesses and residents, which then further helps enhance the overall economic value of the area. This is just one example of how better parking can enable a better, more vibrant and thriving community. --- These slides are from a presentation I gave at Converge Bangalore organized by @thoughtworks on November 8, 2014 in Bangalore. http://info.thoughtworks.com/converge-bangalore-november-8-registration.html
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From the Fall 2013 CASEX Talk at Indiana CASE Conference – IVY Tech. This presentation is a brief introduction to developing personas in education marketing and communications to better understand and engage with prospects.
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Respiratory Distress Sydrome is a condition that affects the lungs of newborn infants predominantly. Not much is known about the condition in the tropics. In this presentation Daniel Faithful Miebaka provides detailed review of the condition that has fatal potential.
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a ground-glass appearance and air bronchogram on the chest X-ray. Finally, because the baby Pao2 was less than 45 torr shortly after birth, the ductus arteriosus remained patent, producing the syndrome known as persistent pulmonary hypertension of the neonate (PPHN). As the infant's condition improved and his PaO2 increased, the ductus arteriosus closed and the signs and symptoms associated with PPHN disappeared. At the time of this writing, the baby was a perfectly normal 3 year-old boy who was attending half-day preschool sessions 5 days per week. 1. In this case, what are the major anatomic alterations of the lung associated with the respiratory disease that developed in the infant? 2. Describe the pathophysiology that develops as the conditions listed in question 1 worsen. 3. Describe how the following conditions are manifested in the clinical setting: Decreased pulmonary diffusion: Decreased lung compliance: 4. Why did PPHN develop in the infant in this case? How did this condition improve? Discussion This case illustrater the possible adverse effects of a premature birth on the infant's alveolar-apillary gas exchange units and pulmonary circulation. During fetal development, the alveolar-capillary system and the quantity of pulmonary surfactant usually are not sufficient to support life until the 28th week of gestation or beyond. In this case, the baby was born at the very beginning of this time period. Thus, because of the immaturity of the baby's alveolar- capilary system, the ability of the type II cells to produce pulmonary surfactant was inadequate. As a result of the insufficient amount of pulmonary surfactant, the pathologic processes of a common newborn respiratory disorder called respiratory distress syndrome (RDS) developed. The anatomic alterations of the lungs associated with RDS indude interstitial and intra-alveolar edema and hemorrhage, alveolar consoldation, intra-alveolar hyaline membrane formation, and atelectasis. All of these pathologic processes cause the alveolar-capillary membranes thickness to increase. As this condtion progressively worsened, the diffusion of oxygen between the alveoli and the pulmonary capillary blood decreased, and the infant's lung compliance decreased. Clinically, the decreased diffusion of oxygen was manifested by cyanosis, increased respiration rate and heart rate, and decreased Pa. The decreased lung compliance was manifested by nasal flaring, intercostal retractions, exhalation grunting, bilateral crackles, and.
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Table 2
(composition)
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Table 3
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Table 4
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64.
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67.
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71.
72.
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74.
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76.
Nonphysilogic jaundice Pathological
jaundice
77.
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81.
82.
83.
84.
85.
86.
87.
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90.
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107.
108.
109.
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115.
116.
117.
118.
119.
Cold injury
120.
Intake absence
121.
122.
123.
124.
125.
126.
127.
128.
129.
130.
131.
132.
133.
134.
Degree
135.
136.
137.
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142.
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