Austin Journal of Clinical Neurology is an open access, peer reviewed, scholarly journal dedicated to publish articles in all areas of neurology, neurophysiology and stroke. The aim of the journal is to provide a forum for neurologists, researchers, physicians, and other health professionals to find most recent advances in the areas of clinical and experimental neurology. Austin Journal of Clinical Neurology accepts original research articles, review articles, case reports, clinical images and rapid communication on all the aspects of clinical neurology and nervous system diseases. Austin Journal of Clinical Neurology strongly supports the scientific upgradation and fortification in related scientific research community by enhancing access to peer reviewed scientific literary works. Austin Publishing Group also brings universally peer reviewed journals under one roof thereby promoting knowledge sharing, mutual promotion of multidisciplinary science. Austin Journal of Clinical Neurology is an open access, peer reviewed, scholarly journal dedicated to publish articles in all areas of neurology, neurophysiology and stroke.

2. HISTORY
“CEREBRAL PALSY” was first identified
by a English Surgeon “WILLIAM LITTLE”
in 1860. It is also called as Little’s Disease

3. DEFINITION
Cerebral Palsy is considered as a
neurological disorder caused by a non-
progressive brain injury or malformation
that occurs by abnormal development or
damage to motor control centers of
the brain.

4. ETIOLOGY
Prenatal
Perinatal
Postnatal/childhood

5. 1. PRENATAL
Maternal diseases
Obstetrical complication
Abuse of drugs
Cerebral malformation
Maternal diseases
Prolonged rupture of
membranes
Gestational diseases
Premature delivery
Preeclampsia
Infections
Severe protienuria
Fetal heart rate
depression
Long labor
Asphyxia

6. 2. PERINATAL
Prematurity and associated problems
Low birth weight
Meconium aspiration
Cerebral trauma
Severe hyperglycemia
Blood incompatibilities
Hyperbilirubinemia
Meconium aspiration
Complicated delivery

7. 3. POSTNATAL
Brain injury
Stroke
Meningitis or encephalitis
Traumatic brain injury
Seizures
Vascular malformations
Cerebral anoxia
Sickle cell anemia
Near drowning
Toxins

8. CLASSIFICATION OF
CP
CEREBRAL PALSY is classified
into two types as below:
Depending upon Topographical
Distribution
Depending on tone or movement
patterns (physiologic)

9. DEPENDING UPON
TOPOGRAPHICAL DISTRIBUTION
Monoplegia
Diplegia
Paraplegia
Quadriplegia
Triplegic
Hemiplegic
Tetraplegic/Double hemiplegic

10. DEPENDING ON TONE OR
MOVEMENT PATTERNS
(PHYSIOLOGIC)
Spastic CP
Athetoid/Dyskinetic CP
Ataxic CP
Flaccid/Hypotonic CP
Choreiform CP
Rigid CP
Mixed CP

12. DIAGNOSIS
Physical Evaluation
Magnetic Resonance Imaging(MRI)
Computerized Tomography(CT) scan
EEG
X-ray skull-intracranial calcification
Neurological assessment
Ultrasound

13. TREATMENT
Physiotherapy
Occupational therapy
Speech therapy
Surgery
Adjunctive therapy
Recreation therapy

14.  Reduce spasticity
 Increase mobility and flexibility
 Prevent deformity
 Improve physical activity
 Improve functional skills
 Intake of proper diet and medications
MEASURES TAKEN BY A
CP PATIENT

15. MAINTAIN PROPER DIET