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Nutrition and the Diseases
of
Lifestyle
The practical modifications
that should be made in our day to day diet
to reduce risk of lifestyle diseases
Nutrition - the foundation of good
health and freedom from disease.
Significance of nutrition
 Adequate nutrition - for growth, development and maintenance
 Under nutrition contributes to 60% deaths amongst U5C (WHO,
2002)
 More than 44% children in India suffer from malnutrition
 About 55% men and 75% NPNLWomen are anaemic
 Chronic degenerative disorders like CVD, Hypertension, Type
2DM , certain cancers, etc are related to diet and nutritional status
 Obesity association with higher risk of developing CaBr, colon,
endometrium, gallbladder, esophagus, pancreas, etc.
 Consumption of foods rich in dietary fiber and antioxidants is
associated with reduced risk of certain cancers
 Under nutrition during fetal and early childhood is known to be
Role of Food
 a) Food builds body tissues
 b) Food regulates body processes
 c) Food supplies energy
 d) Food gives us enjoyment
Interplay between
Malnutrition and Infection !!
 RDA are the estimates of nutrient intakes which
individuals in a population group need to
consume to ensure that the physiological needs
of all subjects in that population are met.
 Corresponds to mean intake of the given
nutrient + 2 Standard Deviation (that is about
25% of the mean has been added). It covers the
requirement of 97.5% of the population. This is
the safe level of intake and the chances of this
level being inadequate is not more than 2.5%.
 Can the RDA be Applied to Individuals?
 For RDA of energy only the average
requirement is considered.
 Reference Indian adult man is between
20-39 years of age and weighs 60 Kg.
He is free from disease and physically fit
for work.
On each working day he is employed for 8
hours in occupation that usually involves
moderate activity.
While not at work he spends 8 hours in
bed, 4-6 hours sitting & moving about and
2 hours in walking and in active recreation
or household duties.
 Reference Indian Adult Woman :
is between 20-39 years of age and
healthy; she weighs 50 Kg.
She may be engaged in general
household work, in light industry or in any
other moderately active work for 8 hours.
While not at work she spends 8 hours in
bed, 4-6 hours sitting and moving about in
light activity and 2 hours in walking or
active household chores.
Energy Requirements of Reference Indian
Man and Woman
Sex Body
weight
(Kg)
Activity levels
Sedentary Moderate Heavy
Male 60 2425 2875 3800
Female 50 1875 2225 2925
As per the principles of Ayurveda, the diet
is supposed to change with the time of
the day and seasons. ‘Hot’ and ‘cold’
temperaments of the food are supposed
to be balanced with the weather,
seasons and climate.
These dictums were ingrained in the
lifestyle along with good and noble
thoughts (vichara) and the discipline of
life (Yoga) for a long lasting good health
and the ultimate union with the supreme
power.
Price of unregulated urbanization,
industrialization and increasing
level of affluence the so called
“modernization” is tremendous
load of “Non-Communicable
diseases ”/ “Chronic - diseases” /
“Lifestyle Diseases”.
The issue is a global phenomena
and not simply restricted to the
developed, rich countries.
Major “Lifestyle Diseases”
 Obesity,
 Heart Disease
 Hypertension, Diabetes Mellitus
 Oral cancer, Lung cancer
 Breast cancer, Colonic cancer & Other
cancers,
 Sexually Transmitted Dis.
 HIV & AIDS, Mental Stress and its sequel
 Osteoarthritis, Osteoporosis
 Liver Disease, Asthma and Bronchitis
 Road accidents
What is “Lifestyle”
“Lifestyle”, indicates the behavioural
patterns which we routinely adopt and
the way we tend to (involuntarily) live
our daily life, unless coerced to
change by some external stimulus.
Lifestyle is thus mainly dependent on
psycho - social and environmental
factors and, to a smaller extent, on
genetic influences.
The Major Components of Unhealthy
Lifestyle
 Lack of physical activity
 Faulty dietary habits
 Tobacco use
 Excessive alcohol intake
 Mental Stress
 Disregard to personal safety regarding
- Accidents
- Personal hygiene
- Promiscuous Sex
- Insect Vectors of Diseases
Eat adequately
Cereals : 350 ~ 400 g
Pulses : 50 ~ 100 g
Eat liberally
Veg : 200 ~ 250 g
Fruits : 200 ~ 250 g
Eat Moderately
Non veg < 100 g
Sugar : 50 ~ 60 g
Eat
Less
Salt 6g
Fats 30g
The principles of a healthy diet for an average adult
(consuming 2500 to 2800 Kcal per day)
 Healthy Diet include Foods with
◦ low calories (and low refined
sugars),
◦ enough proteins,
◦ low fats and salts but lots of
antioxidants, vitamins and natural
fibre.
And Should Provide
 The Required Energy, No More
and No Less, and
 Psychological satiety
X
Epidemiology of Obesity
Obesity:
 Commonest expressions of unhealthy
diet,
 Often combined with lack of physical
activity.
Burden Of Disease
WHO –
 Globally, over 1 billion (16%) adults are
overweight and 300 million (5%) are obese.
 The highest rise is in fast growing economies
especially of South East Asia.
 250 million obese in the third world countries
 India - > 100 million,
(12.6% women/ 9.3% men)
We are truly in the midst of an obesity
epidemic, with serious health ramifications.
Epidemiological Determinants
Obesogenic environment :
 Affluent lifestyle
 Wrong food
 Sedentary home environment,
 Vanishing old family traditions, replaced
by the ‘couch - potato’ culture
 Cut throat competition
 Quick money
 LOSS OF VALUES
Epidemiological Determinants
Age :
 The incidence increases with age till about
60years.
 Vulnerability is maximum around 40 years
Gender :
 More in females - inherent hormonal
differences.
Ethnicity :
 Large unexplained variations in different
ethnic groups.
Epidemiological Determinants
Education & Income levels :
 In Indian setting, higher education & Income
level-obese,(likely to be more affluent).
 In the west, however, scene is reverse.
Marital status :
 Married are more likely to be obese
Parity :
 Women with higher parity are more likely
Epidemiological Determinants
Diet :
 A diet rich in fats, refined sugar and
carbohydrates
 Consumption of as little as 100 extra
calories per day would increase the weight
of an individual by 4 kg in one year.
Smoking :
 An anorexic agent, not to be
promoted!!!!!!!!
Alcohol :
 Alcohol provides 7kcal per gm (+
Snacking!)
Physical Inactivity :
Causes of Obesity
 Increased energy intake and
decreased expenditure :
 Passive overeating :
 Binge eating :
 Metabolic factors :
 Genetic factors :
 Foetal programming :
Critical Periods for Weight Gain
Weight gained during certain critical
periods !
◦ Age range of 12 to 18 months
◦ Age range of 12 to 16 years
◦ Gain of 60% (or more) of his ideal
weight by an adult
◦ Weight gain during pregnancy
Quantifying Obesity
Body Mass Index = Weight (Kg) / Height (m)2
Grades of obesity based on BMI
BMI Classification Risk of Co-morbidities
< 18.5 Underweight Low to Average
18.5-24.9 Normal weight Average
25 - 29.9 Pre-obese
(Overweight)
Mildly increased
30 - 34.9 Obesity Class I Moderate
35 - 39.9 Obesity Class II Severe
> 40 Obesity Class III Very severe
Grades of Obesity based on BMI for Asians
BMI Classification Risk of Co-
morbidities
< 18.5 Underweight Low
18.5-23 Normal weight Increasing but
acceptable
23 -
27.5
Pre-obese
(Overweight)
Increased
> 27.5 Obese High
Waist circumference :
Normal, ≤ 90 cm for men and 80 cm for
women
Waist - Hip Ratio :
A ratio of < 0.9 for men and < 0.8 for women
Types of obesity
Gynoid / ‘Pear shaped’ :
 The fat is evenly (globally) distributed.
Android/‘Apple shaped’ :
 The fat is centrally distributed
 Commonly seen in men of the South
East Asian region, including India.
 Visceral (peritoneal) deposition of fat -
Higher waist circumference or higher
WHR
Hazards of obesity
 Higher risk of mortality and morbidity.
 The LE of a morbidly obese individual is
about a 10 yrs lower
 Most overweight and obese individuals
have difficulty in day to day activities
and are
 Prone for Life Style Diseases
Some Specific clinical Consequences
: Metabolic & Degenerative :
 Diabetes type II (50 to 100 times more in obese)
 Hyperlipidaemia, IHD, Hypertension (5 to 6 times
commoner),
 Stroke (2.5 to 6 times commoner),
 Gall stones, breast and colon cancer, infertility,
gout and polycystic ovary syndrome
Physical :
 Osteoarthritis, chronic back pain, respiratory
problems, limited mobility, higher accidents, slee
apnoea and skin problems.
Psychological :
 Depression, low self - esteem, social isolation,
poor employment status, impaired relationships
and discrimination.
!
“Most obese people won’t
enter treatment, most
who do;
won’t lose weight and
most who lose weight
regain it”
Prevention of Obesity
Prevention is the only viable long term
strategy
Losing 10 kg is associated with :
 A reduction in total mortality by 20%
 A reduction in systolic BP by 10 mmHg
 A reduction in diastolic BP by 20 mmHg
 A reduction in fasting glucose by upto
50%
 A reduction in total cholesterol by 10%
 A beneficial rise of 8% in HDL cholesterol
 An improved self - esteem
Prevention
Universal Measures:
Meant for all irrespective of their weight status.
Selective Measures:
High risk individuals are targeted;
Indicated Measures:
Or the Secondary Preventive (Early Dx & Tt)
measures are to be taken for those with
existing problems of overweight and obesity.
How to Reduce Weight?
Nearly 2500 years ago, Socrates had
very aptly said
‘Eat only when hungry and
drink only when thirsty, and
never to leave the table with a
feeling of satiety’.
How to Reduce Weight?
Aim for BMI < 25 kg/m2 (preferably <23.5)
The origin of obesity is multi-
factorial.
Modalities for treatment/prevention:
 The dietary therapy most practical and
effective
 Other measures are :
◦ (a) Behaviour therapy
◦ (b) Drug therapy
◦ (c) Surgical intervention
◦ (d) Genetic approach.
Dietary therapy (dieting) :
 Educate on nutritional and health aspects:
◦ How to make safe, sensible and gradual
change in eating pattern
◦ Increase the intake of complex
carbohydrates (unrefined cereals and
sugars, fibre rich foods) and decrease fats
and simple carbohydrates (refined sugars,
excessively milled cereals e.g. white
bread, maida, besan etc).
◦ Fruits and vegetables- an integral part of
diet.
◦ Low calorie and low fat foods.
Four areas to be considered
 Ascertain the activity status :
◦ Assess the present BMI and the desired BMI to
find out the weight (in Kg) to be reduced.
 Set a practical time frame for weight loss
 Assess the daily calorie intake.
◦ Translate weight to be reduced, to calorie
restriction.
◦ These calories are distributed between
carbohydrates, protein and fat so as to cut down
calories preferably from fats and carbohydrates (in
that order).
 Suitable substitutions should be made
◦ The frequency with which the foods are to be
eaten
Reducing weight - An example
Let us take a 1.66 m tall, sedentary male,
weighing 80 kg.
Step 1 :
His present BMI is 29. Let us presume that
his desired target BMI is 25.
To achieve this BMI his weight must be about
69 kg i.e. he must reduce 11 kg.
Step 2 :
It is recommended that he reduces 1.5 kg
weight per month, i.e. he would be able to
reduce 11 kg in about 7 months.
Step 3 :
Assess his total daily calorie intake.
Reducing weight –example…..
As a rule, generally; a reduction of about 500 Kcal
per day brings about a weight loss of about 500g
per week.
Conservatively, let us assume that a reduction
of 1.5 kg per month can be achieved.
500 Kcal per day can be reduced by cutting down –
15 g oil (135 Kcal), and about 90g (360 Kcal)
carbohydrates/ day.
Step 4 : Make suitable substitutions as applicable
For example, replace saturated fats with
PUFA/MUFA,
Replace whole milk with skimmed milk, and refined
flour with whole-wheat flour. More fruits and
vegetables could be included.
Making Some Wise Substitutions -
From Fat to Fit
 Refined carbohydrates with complex
carbohydrates
 High starch foods with high fibre ones
 Fried nuts with plain nuts
 Whole milk with low fat skimmed milk
 Mutton and beef with lean meat (e.g. chicken)
 Oily meat preparations with non-fried stews,
soups
 Chips, wafers, burgers, samosa, cutlets with
plain toast, fruits, salad and fruit juices
 Sweet biscuits with plain ones and nuts
 Saturated fats (ghee, butter) with vegetable oils?
Some more tips on dieting
 Do not skip meals to reduce weight
 Do not eat to finish leftovers
 Eat many small but measured meals
(minimum 3)
 Do not snack while watching TV or using
computer.
 It is a myth that some foods can burn fat
 Do not shop when hungry
 Do not use food as relaxation
 Develop a positive attitude and be cheerful
 Exercise regularly, Strengthen muscles
 Slow and steady weight loss of about 0.5 -
1kg per week is safest
Remember:
 Food labels claiming ‘low-fat’ or ‘no-fat’
may still have lot of calories
 Food labels claiming ‘zero cholesterol’
may not mean ‘zero oil’
 Drink enough water each day @
minimum of 8-10 glasses
 Alcohol has high calorie content; Snacks
taken along with drinks add to calories
 Fruits and vegetables are low calorie
food and source of antioxidants
Fad diets and their role in weight reduction :
 More popular ones are the Atkins diet, Ornish
diet, Weight watchers diet and the Zone diet.
These are based on different
‘principles’ -
 Atkins diet, the most popular of the lot,
restricts intake of carbohydrates to less
than 30 g a day and permits consumption
of fats (fatty meat, butter, and other high -
fat dairy products).
 The Ornish diet restricts fat,
 Weight watcher’s diet restricts portion size
and calories,
 Zone diet modulates macronutrient
balance and glycemic load
 The Atkins diet books have sold more than
45 million copies over 40 years all over the
world.
 It eliminates carbohydrates from food without
restricting protein and fat intake.
 Deprived of carbohydrates, the body uses fat for
fuel.
 A small part of metabolized fat is eliminated in
the urine as ketone bodies, and this is why such
diets are called “ketogenic”.
 In the short run, such diets produce rapid weight
loss due to polyuria.
 The apparent paradox that ad - libitum intake of
high - fat foods produces weight loss might be
(i) Severe restriction of carbohydrate
depleting glycogen stores
(ii) This leads to excretion of bound water
causing weight loss
(iii) The ketogenic nature of the diet being
appetite suppressing
(iv) The high protein - content being highly
satiating
(v) High fat / protein diet reduces
spontaneous food intake
(vi) In the absence of carbohydrates the food
choices are limited, leading to decreased
energy intake
Consequences
Wrong dietary habit- due to less appetite
patients eat without feeling severe hunger
and without measuring their food intake!
Orthostatic hypotension, fatigue and nausea
are frequent.
The diet increases plasma cholesterol and
uric acid. May be dangerous in diabetes
(anorexia, acidosis) and in heart or kidney
disease.
On the long run, re - feeding
carbohydrates cause water retention and
weight gain.
Wrap up
We are amidst an epidemic of
obesity.
Right eating habits
Balance Diet combined with
Right kind of Physical Activity
for age/ sex are the only
saviors
Eat with Awareness-why, what,
when, where, how much

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Epidemiology of obesity

  • 1. Nutrition and the Diseases of Lifestyle The practical modifications that should be made in our day to day diet to reduce risk of lifestyle diseases
  • 2. Nutrition - the foundation of good health and freedom from disease. Significance of nutrition  Adequate nutrition - for growth, development and maintenance  Under nutrition contributes to 60% deaths amongst U5C (WHO, 2002)  More than 44% children in India suffer from malnutrition  About 55% men and 75% NPNLWomen are anaemic  Chronic degenerative disorders like CVD, Hypertension, Type 2DM , certain cancers, etc are related to diet and nutritional status  Obesity association with higher risk of developing CaBr, colon, endometrium, gallbladder, esophagus, pancreas, etc.  Consumption of foods rich in dietary fiber and antioxidants is associated with reduced risk of certain cancers  Under nutrition during fetal and early childhood is known to be
  • 3. Role of Food  a) Food builds body tissues  b) Food regulates body processes  c) Food supplies energy  d) Food gives us enjoyment Interplay between Malnutrition and Infection !!
  • 4.  RDA are the estimates of nutrient intakes which individuals in a population group need to consume to ensure that the physiological needs of all subjects in that population are met.  Corresponds to mean intake of the given nutrient + 2 Standard Deviation (that is about 25% of the mean has been added). It covers the requirement of 97.5% of the population. This is the safe level of intake and the chances of this level being inadequate is not more than 2.5%.  Can the RDA be Applied to Individuals?  For RDA of energy only the average requirement is considered.
  • 5.  Reference Indian adult man is between 20-39 years of age and weighs 60 Kg. He is free from disease and physically fit for work. On each working day he is employed for 8 hours in occupation that usually involves moderate activity. While not at work he spends 8 hours in bed, 4-6 hours sitting & moving about and 2 hours in walking and in active recreation or household duties.
  • 6.  Reference Indian Adult Woman : is between 20-39 years of age and healthy; she weighs 50 Kg. She may be engaged in general household work, in light industry or in any other moderately active work for 8 hours. While not at work she spends 8 hours in bed, 4-6 hours sitting and moving about in light activity and 2 hours in walking or active household chores.
  • 7. Energy Requirements of Reference Indian Man and Woman Sex Body weight (Kg) Activity levels Sedentary Moderate Heavy Male 60 2425 2875 3800 Female 50 1875 2225 2925
  • 8. As per the principles of Ayurveda, the diet is supposed to change with the time of the day and seasons. ‘Hot’ and ‘cold’ temperaments of the food are supposed to be balanced with the weather, seasons and climate. These dictums were ingrained in the lifestyle along with good and noble thoughts (vichara) and the discipline of life (Yoga) for a long lasting good health and the ultimate union with the supreme power.
  • 9. Price of unregulated urbanization, industrialization and increasing level of affluence the so called “modernization” is tremendous load of “Non-Communicable diseases ”/ “Chronic - diseases” / “Lifestyle Diseases”. The issue is a global phenomena and not simply restricted to the developed, rich countries.
  • 10. Major “Lifestyle Diseases”  Obesity,  Heart Disease  Hypertension, Diabetes Mellitus  Oral cancer, Lung cancer  Breast cancer, Colonic cancer & Other cancers,  Sexually Transmitted Dis.  HIV & AIDS, Mental Stress and its sequel  Osteoarthritis, Osteoporosis  Liver Disease, Asthma and Bronchitis  Road accidents
  • 11. What is “Lifestyle” “Lifestyle”, indicates the behavioural patterns which we routinely adopt and the way we tend to (involuntarily) live our daily life, unless coerced to change by some external stimulus. Lifestyle is thus mainly dependent on psycho - social and environmental factors and, to a smaller extent, on genetic influences.
  • 12. The Major Components of Unhealthy Lifestyle  Lack of physical activity  Faulty dietary habits  Tobacco use  Excessive alcohol intake  Mental Stress  Disregard to personal safety regarding - Accidents - Personal hygiene - Promiscuous Sex - Insect Vectors of Diseases
  • 13. Eat adequately Cereals : 350 ~ 400 g Pulses : 50 ~ 100 g Eat liberally Veg : 200 ~ 250 g Fruits : 200 ~ 250 g Eat Moderately Non veg < 100 g Sugar : 50 ~ 60 g Eat Less Salt 6g Fats 30g The principles of a healthy diet for an average adult (consuming 2500 to 2800 Kcal per day)
  • 14.  Healthy Diet include Foods with ◦ low calories (and low refined sugars), ◦ enough proteins, ◦ low fats and salts but lots of antioxidants, vitamins and natural fibre. And Should Provide  The Required Energy, No More and No Less, and  Psychological satiety
  • 15. X
  • 16. Epidemiology of Obesity Obesity:  Commonest expressions of unhealthy diet,  Often combined with lack of physical activity.
  • 17. Burden Of Disease WHO –  Globally, over 1 billion (16%) adults are overweight and 300 million (5%) are obese.  The highest rise is in fast growing economies especially of South East Asia.  250 million obese in the third world countries  India - > 100 million, (12.6% women/ 9.3% men) We are truly in the midst of an obesity epidemic, with serious health ramifications.
  • 18. Epidemiological Determinants Obesogenic environment :  Affluent lifestyle  Wrong food  Sedentary home environment,  Vanishing old family traditions, replaced by the ‘couch - potato’ culture  Cut throat competition  Quick money  LOSS OF VALUES
  • 19. Epidemiological Determinants Age :  The incidence increases with age till about 60years.  Vulnerability is maximum around 40 years Gender :  More in females - inherent hormonal differences. Ethnicity :  Large unexplained variations in different ethnic groups.
  • 20. Epidemiological Determinants Education & Income levels :  In Indian setting, higher education & Income level-obese,(likely to be more affluent).  In the west, however, scene is reverse. Marital status :  Married are more likely to be obese Parity :  Women with higher parity are more likely
  • 21. Epidemiological Determinants Diet :  A diet rich in fats, refined sugar and carbohydrates  Consumption of as little as 100 extra calories per day would increase the weight of an individual by 4 kg in one year. Smoking :  An anorexic agent, not to be promoted!!!!!!!! Alcohol :  Alcohol provides 7kcal per gm (+ Snacking!) Physical Inactivity :
  • 22. Causes of Obesity  Increased energy intake and decreased expenditure :  Passive overeating :  Binge eating :  Metabolic factors :  Genetic factors :  Foetal programming :
  • 23. Critical Periods for Weight Gain Weight gained during certain critical periods ! ◦ Age range of 12 to 18 months ◦ Age range of 12 to 16 years ◦ Gain of 60% (or more) of his ideal weight by an adult ◦ Weight gain during pregnancy
  • 24. Quantifying Obesity Body Mass Index = Weight (Kg) / Height (m)2 Grades of obesity based on BMI BMI Classification Risk of Co-morbidities < 18.5 Underweight Low to Average 18.5-24.9 Normal weight Average 25 - 29.9 Pre-obese (Overweight) Mildly increased 30 - 34.9 Obesity Class I Moderate 35 - 39.9 Obesity Class II Severe > 40 Obesity Class III Very severe
  • 25. Grades of Obesity based on BMI for Asians BMI Classification Risk of Co- morbidities < 18.5 Underweight Low 18.5-23 Normal weight Increasing but acceptable 23 - 27.5 Pre-obese (Overweight) Increased > 27.5 Obese High Waist circumference : Normal, ≤ 90 cm for men and 80 cm for women Waist - Hip Ratio : A ratio of < 0.9 for men and < 0.8 for women
  • 26. Types of obesity Gynoid / ‘Pear shaped’ :  The fat is evenly (globally) distributed. Android/‘Apple shaped’ :  The fat is centrally distributed  Commonly seen in men of the South East Asian region, including India.  Visceral (peritoneal) deposition of fat - Higher waist circumference or higher WHR
  • 27. Hazards of obesity  Higher risk of mortality and morbidity.  The LE of a morbidly obese individual is about a 10 yrs lower  Most overweight and obese individuals have difficulty in day to day activities and are  Prone for Life Style Diseases
  • 28. Some Specific clinical Consequences : Metabolic & Degenerative :  Diabetes type II (50 to 100 times more in obese)  Hyperlipidaemia, IHD, Hypertension (5 to 6 times commoner),  Stroke (2.5 to 6 times commoner),  Gall stones, breast and colon cancer, infertility, gout and polycystic ovary syndrome Physical :  Osteoarthritis, chronic back pain, respiratory problems, limited mobility, higher accidents, slee apnoea and skin problems. Psychological :  Depression, low self - esteem, social isolation, poor employment status, impaired relationships and discrimination.
  • 29. ! “Most obese people won’t enter treatment, most who do; won’t lose weight and most who lose weight regain it”
  • 30. Prevention of Obesity Prevention is the only viable long term strategy Losing 10 kg is associated with :  A reduction in total mortality by 20%  A reduction in systolic BP by 10 mmHg  A reduction in diastolic BP by 20 mmHg  A reduction in fasting glucose by upto 50%  A reduction in total cholesterol by 10%  A beneficial rise of 8% in HDL cholesterol  An improved self - esteem
  • 31. Prevention Universal Measures: Meant for all irrespective of their weight status. Selective Measures: High risk individuals are targeted; Indicated Measures: Or the Secondary Preventive (Early Dx & Tt) measures are to be taken for those with existing problems of overweight and obesity.
  • 32. How to Reduce Weight? Nearly 2500 years ago, Socrates had very aptly said ‘Eat only when hungry and drink only when thirsty, and never to leave the table with a feeling of satiety’.
  • 33. How to Reduce Weight? Aim for BMI < 25 kg/m2 (preferably <23.5) The origin of obesity is multi- factorial. Modalities for treatment/prevention:  The dietary therapy most practical and effective  Other measures are : ◦ (a) Behaviour therapy ◦ (b) Drug therapy ◦ (c) Surgical intervention ◦ (d) Genetic approach.
  • 34. Dietary therapy (dieting) :  Educate on nutritional and health aspects: ◦ How to make safe, sensible and gradual change in eating pattern ◦ Increase the intake of complex carbohydrates (unrefined cereals and sugars, fibre rich foods) and decrease fats and simple carbohydrates (refined sugars, excessively milled cereals e.g. white bread, maida, besan etc). ◦ Fruits and vegetables- an integral part of diet. ◦ Low calorie and low fat foods.
  • 35. Four areas to be considered  Ascertain the activity status : ◦ Assess the present BMI and the desired BMI to find out the weight (in Kg) to be reduced.  Set a practical time frame for weight loss  Assess the daily calorie intake. ◦ Translate weight to be reduced, to calorie restriction. ◦ These calories are distributed between carbohydrates, protein and fat so as to cut down calories preferably from fats and carbohydrates (in that order).  Suitable substitutions should be made ◦ The frequency with which the foods are to be eaten
  • 36. Reducing weight - An example Let us take a 1.66 m tall, sedentary male, weighing 80 kg. Step 1 : His present BMI is 29. Let us presume that his desired target BMI is 25. To achieve this BMI his weight must be about 69 kg i.e. he must reduce 11 kg. Step 2 : It is recommended that he reduces 1.5 kg weight per month, i.e. he would be able to reduce 11 kg in about 7 months. Step 3 : Assess his total daily calorie intake.
  • 37. Reducing weight –example….. As a rule, generally; a reduction of about 500 Kcal per day brings about a weight loss of about 500g per week. Conservatively, let us assume that a reduction of 1.5 kg per month can be achieved. 500 Kcal per day can be reduced by cutting down – 15 g oil (135 Kcal), and about 90g (360 Kcal) carbohydrates/ day. Step 4 : Make suitable substitutions as applicable For example, replace saturated fats with PUFA/MUFA, Replace whole milk with skimmed milk, and refined flour with whole-wheat flour. More fruits and vegetables could be included.
  • 38. Making Some Wise Substitutions - From Fat to Fit  Refined carbohydrates with complex carbohydrates  High starch foods with high fibre ones  Fried nuts with plain nuts  Whole milk with low fat skimmed milk  Mutton and beef with lean meat (e.g. chicken)  Oily meat preparations with non-fried stews, soups  Chips, wafers, burgers, samosa, cutlets with plain toast, fruits, salad and fruit juices  Sweet biscuits with plain ones and nuts  Saturated fats (ghee, butter) with vegetable oils?
  • 39. Some more tips on dieting  Do not skip meals to reduce weight  Do not eat to finish leftovers  Eat many small but measured meals (minimum 3)  Do not snack while watching TV or using computer.  It is a myth that some foods can burn fat  Do not shop when hungry  Do not use food as relaxation  Develop a positive attitude and be cheerful  Exercise regularly, Strengthen muscles  Slow and steady weight loss of about 0.5 - 1kg per week is safest
  • 40. Remember:  Food labels claiming ‘low-fat’ or ‘no-fat’ may still have lot of calories  Food labels claiming ‘zero cholesterol’ may not mean ‘zero oil’  Drink enough water each day @ minimum of 8-10 glasses  Alcohol has high calorie content; Snacks taken along with drinks add to calories  Fruits and vegetables are low calorie food and source of antioxidants
  • 41. Fad diets and their role in weight reduction :  More popular ones are the Atkins diet, Ornish diet, Weight watchers diet and the Zone diet. These are based on different ‘principles’ -  Atkins diet, the most popular of the lot, restricts intake of carbohydrates to less than 30 g a day and permits consumption of fats (fatty meat, butter, and other high - fat dairy products).  The Ornish diet restricts fat,  Weight watcher’s diet restricts portion size and calories,  Zone diet modulates macronutrient balance and glycemic load
  • 42.  The Atkins diet books have sold more than 45 million copies over 40 years all over the world.  It eliminates carbohydrates from food without restricting protein and fat intake.  Deprived of carbohydrates, the body uses fat for fuel.  A small part of metabolized fat is eliminated in the urine as ketone bodies, and this is why such diets are called “ketogenic”.  In the short run, such diets produce rapid weight loss due to polyuria.  The apparent paradox that ad - libitum intake of high - fat foods produces weight loss might be
  • 43. (i) Severe restriction of carbohydrate depleting glycogen stores (ii) This leads to excretion of bound water causing weight loss (iii) The ketogenic nature of the diet being appetite suppressing (iv) The high protein - content being highly satiating (v) High fat / protein diet reduces spontaneous food intake (vi) In the absence of carbohydrates the food choices are limited, leading to decreased energy intake
  • 44. Consequences Wrong dietary habit- due to less appetite patients eat without feeling severe hunger and without measuring their food intake! Orthostatic hypotension, fatigue and nausea are frequent. The diet increases plasma cholesterol and uric acid. May be dangerous in diabetes (anorexia, acidosis) and in heart or kidney disease. On the long run, re - feeding carbohydrates cause water retention and weight gain.
  • 45. Wrap up We are amidst an epidemic of obesity. Right eating habits Balance Diet combined with Right kind of Physical Activity for age/ sex are the only saviors Eat with Awareness-why, what, when, where, how much

Notas do Editor

  1. Bullet 2 =(WHO, 2002) Gomez classification, NNMB 2007) non pregnant non lactating women = NPNLW
  2. This ‘safe level’ approach is not used for defining the energy requirement, as any excess of energy intake is as undesirable as its inadequate intake. Hence for defining the RDA of energy only the average requirement is considered.
  3. Lifestyle is developed in the form of a set pattern of behaviour, very gradually, over many years, in the way we eat, drink, exercise, use intoxicants, are predisposed to own health care and personal protection, sexual practices and so on. Since these behavioural patterns are acquired very gradually, changing them becomes a difficult proposition and needs a lot of persuasiveness as well as persistent approach on the part of the health care providers including health educators.
  4. Wrong food- Aggressive advertising, marketing and universal accessibility of chips, wafers and colas,
  5. Vulnerability is maximum around 40 years of age, owing to certain hormonal changes, affluence and a more sedentary lifestyle at this age.
  6. Alcohol provides 7kcal per gm, (carbohydrates or proteins (4kcal) Plus the snacks consumed along
  7. increased energy intake and decreased expenditure : will lead to excess calories being stored as fat and, ultimately to obesity. Passive overeating : without a biological need, Binge eating : overindulging in a party, on a weekend or with drinks Metabolic factors : Genetic factors : Obesogenic genes are under study, which alter the metabolism or alter the response to obesity limiting hormones like Leptins etc. Fetal programming : The Barker’s hypothesis proposes that under-nutrition during pregnancy may increase the susceptibility of that individual to obesity in adulthood.
  8. Weight gained during certain critical periods, usually lead to An increased number of fat cells and makes obesity difficult to treat. These periods include : Age range of 12 to 18 months Age range of 12 to 16 years Gain of 60% (or more) of his ideal weight by an adult Weight gain during pregnancy
  9. BMI does not measure the body fat but relates well with the degree of obesity.
  10. Gynoid / ‘Pear shaped’ : The fat is evenly distributed (globally distributed). Android/‘Apple shaped’ : In this type of obesity, the fat is centrally distributed or deposited preferentially in the abdominal region Commonly seen in men of the South East Asian region, including India. Higher waist circumference or higher WHR is a good indicator of visceral (peritoneal) deposition of fat.
  11. Higher risk of mortality and morbidity. The LE of a morbidly obese individual is about a 10 yrs lower than one with normal BMI. Most overweight and obese individuals have difficulty in walking, heavy breathing while walking, joint pains, snoring, morning headaches and shortness of breath.
  12. 1. Universal Prevention : As the name suggests, universal preventive measures are meant for all the individuals in the community, irrespective of their weight status. Theses measures include healthy lifestyle practices, like consuming a prudent and healthy diet. This includes low consumption of fat and refined carbohydrates. Active physical activity and shunning sedentary lifestyle also forms a part of this strategy. Health and nutritional education is also imparted to everyone in order to create awareness amongst masses for prevention of obesity. 2. Selective Prevention : High risk individuals are targeted under this preventive strategy. The high risk individuals are those who are more likely to gain weight. These include affluent people especially adolescents, pregnant women, middle aged people and those with a rich sedentary lifestyle consuming high energy food (fats) and those under psychological stress. Those with a hormonal disorder, family history of obesity or on certain drugs like Lithium, Sodium valproate, hormones etc. are also at a high risk of obesity. 3. Indicated Prevention : Indicated Prevention or the Secondary preventive measures are to be taken for those with existing problems of overweight and obesity.
  13. Ascertain the activity status : Assess the present BMI and the desired BMI. This would indicate the weight (in Kg) to be reduced. If Ht is 1.6m and wt is 80 kg (BMI = 80/2.56= 31.25), if desired BMI is 23 (a/2.56=23 then a=2.56*23 =58.8Kg) Set a practical time frame for weight reduction. Assess the daily calorie intake. The weight to be reduced is then translated to the calorie restriction. These calories are distributed between carbohydrates, protein and fat so as to cut down calories preferably from fats and carbohydrates (in that order). This also helps balance all nutrients. Suitable substitutions should be made The frequency with which the foods are to be eaten and The situation in which the food is ingested is also to be looked into.
  14. Refined carbohydrates (milled rice, white bread, biscuits) must be replaced with complex carbohydrates e.g. brown rice, wholewheat atta and whole-wheat bread etc. High starch foods (potatoes, rice) must be replaced with high fibre ones (whole grains, beans and some vegetables turnips, beet-root and carrots) Fried nuts with plain nuts Whole milk with low fat skimmed milk Substitute mutton and beef with lean meat (e.g. chicken) Substitute oily meat preparations with non-fried stews, soups Substitute chips, wafers, burgers, samosa, cutlets with plain toast, fruits, salad and fruit juices Sweet biscuits with plain ones and nuts Substitute saturated fats (ghee, butter, t-FA) with vegetable oils : sunflower, safflower, groundnut, linseed or cotton seed oils
  15. libitum
  16. Low - carbohydrate diets have been regarded as fad diets. A systematic review of low - carbohydrate diets found that the weight loss achieved is associated with the duration of the diet and restriction of energy intake, but not with restriction of carbohydrates, per se (18). Perhaps more long - term studies are needed to measure changes in nutritional status and body composition during the low - carbohydrate diet, and to assess fasting and postprandial cardiovascular risk factors and adverse effects of these diets (19). Without that information, low - carbohydrate diets cannot be recommended as a public health measure for weight reduction.