2. HISTORY
• 1842- Italian scientist Carlo Matteucci realizes that
electricity is associated with the heart beat
• 1876- Irish scientist Marey analyzes the electric
pattern of frog’s heart
• 1895 - William Einthoven , credited for the invention
of EKG
• 1906 - using the string electrometer EKG,
William Einthoven diagnoses some heart problems
6. What is an EKG?
•The electrocardiogram (EKG) is a representation
of the electrical events of the cardiac cycle.
•Each event has a distinctive waveform
•the study of waveform can lead to greater insight
into a patient’s cardiac pathophysiology.
7. With EKGs we can identify
Arrhythmias
Myocardial ischemia and infarction
Pericarditis
Electrolyte disturbances (i.e. hyperkalemia,
hypokalemia)
Drug toxicity (i.e. digoxin and drugs which
prolong the QT interval)
8. Depolarization
• Contraction of any muscle is associated with
electrical changes called depolarization
• These changes can be detected by electrodes
attached to the surface of the body
9. Pacemakers of the Heart
• SA Node - Dominant pacemaker with an
intrinsic rate of 60 - 100 beats/minute.
• AV Node - Back-up pacemaker with an
intrinsic rate of 40 - 60 beats/minute.
• Ventricular cells - Back-up pacemaker with an
intrinsic rate of 20 - 45 bpm.
10. Impulse Conduction & the ECG
Sinoatrial node
AV node
Bundle of His
Bundle Branches
Purkinje fibers
11. The “PQRST”
• P wave - Atrial
depolarization
• T wave - Ventricular
repolarization
• QRS - Ventricular
depolarization
12. The PR Interval
Atrial depolarization
+
delay in AV junction
(AV node/Bundle of His)
(delay allows time for
the atria to contract
before the ventricles
contract)
16. Parts of ECG
Waves
Duration in ms
Amplitude
mV
P Wave 80 ms 0.25 mV
QRS complex 80 to 120ms 1.60 mV
ST segment 80 to 120ms
T wave 160ms 0.1 to 0.5 mV
ECG SIGNAL FREQUENCY RANGE:
0.05 – 150 Hz (diagnostic)
0.5 – 40 Hz (monitoring)
17. Einthoven's triangle
• Einthoven's triangle is an imaginary
formation of three limb leads in a triangle
used in electrocardiography, formed by the
two shoulders and the pubis. The shape
forms an inverted equilateral triangle with
the heart at the center that produces zero
potential when the voltages are summed. It
is named after Willem Einthoven, who
theorized its existence.
18. EKG Leads
which measure the difference in electrical potential
between two points
1. Bipolar Leads: Two different points on the body1. Bipolar Leads: Two different points on the body
2. Unipolar Leads: One point on the body and a virtual2. Unipolar Leads: One point on the body and a virtual
reference point with zero electrical potential, located inreference point with zero electrical potential, located in
the center of the heartthe center of the heart
19. EKG Leads
The standard EKG has 12 leads:
3 Standard Limb Leads
3 Augmented Limb Leads
6 Precordial Leads
47. RULE 6
The R wave must grow from V1 to at least V4
The S wave must grow from V1 to at least V3
and disappear in V6
48. RULE 7
The ST segment should start isoelectric
except in V1 and V2 where it may be elevated
49. RULE 8
The P waves should be upright in I, II, and V2 to V6
50. RULE 9
There should be no Q wave or only a small q less
than 0.04 seconds in width in I, II, V2 to V6
51. RULE 10
The T wave must be upright in I, II, V2 to V6
52. P wave
• Always positive in lead I and II
• Always negative in lead aVR
• < 3 small squares in duration
• < 2.5 small squares in
amplitude
• Commonly biphasic in lead V1
• Best seen in leads II
56. QRS Complexes
• Non-pathological Q waves may present in I, III, aVL,
V5, and V6
• R wave in lead V6 is smaller than V5
• Depth of the S wave, should not exceed 30 mm
• Pathological Q wave > 2mm deep and > 1mm wide or
> 25% amplitude of the subsequent R wave
58. ST Segment
• ST Segment is flat (isoelectric)
• Elevation or depression of ST segment by
1 mm or more
• “J” (Junction) point is the point between
QRS and ST segment
59. Variable Shapes Of ST Segment
Elevations in AMI
Goldberger AL. Goldberger: Clinical Electrocardiography: A Simplified Approach. 7th
ed: Mosby Elsevier; 2006.
60. T wave
• Normal T wave is asymmetrical, first half having a
gradual slope than the second
• Should be at least 1/8 but less than 2/3 of the
amplitude of the R
• T wave amplitude rarely exceeds 10 mm
• Abnormal T waves are symmetrical, tall, peaked,
biphasic or inverted.
• T wave follows the direction of the QRS deflection.
62. QT interval
1. Total duration of Depolarization and
Repolarization
2. QT interval decreases when heart rate
increases
3. For HR = 70 bpm, QT<0.40 sec.
4. QT interval should be 0.35 0.45 s,
5. Should not be more than half of the interval
between adjacent R waves (RR interval).
Atrial depolarisation
Electrically both atria act almost as one.
They have relatively little muscle and generate a single, small P wave.
P wave amplitude rarely exceeds two and a half small squares (0.25 mV).
The duration of the P wave should not exceed three small squares (0.12 s).
The wave of depolarisation is directed inferiorly and towards the left, and thus the P wave tends to be upright in leads I and II and inverted in lead aVR.
Sinus P waves are usually most prominently seen in leads II and V1.
A negative P wave in lead I may be due to incorrect recording of the electrocardiogram (that is, with transposition of the left and right arm electrodes), dextrocardia, or abnormal atrial rhythms.
Normal P waves may have a slight notch, particularly in the precordial (chest) leads. Bifid P waves result from slight asynchrony between right and left atrial depolarisation.
A pronounced notch with a peaktopeak interval of &gt; 1 mm (0.04 s) is usually pathological, and is seen in association with a left atrial abnormality—for example, in mitral stenosis.
The R wave in lead V6 is smaller than the R wave in V5, since the V6 electrode is further from the left ventricle.
The depth of the S wave, generally, should not exceed 30 mm in a normal individual (although &gt; 30 mm are occasionally recorded in normal young male adults)
In another website it is also shown that small q wave seen in leads III and aVF
Normal q-waves reflect normal septal activation (beginning on the LV septum); they are narrow (&lt;0.04s duration) and small (&lt;25% the amplitude of the R wave). They are often seen in leads I and aVL when the QRS axis is to the left of +60o, and in leads II, III, aVF when the QRS axis is to the right of +60o. Septal q waves should not be confused with the pathologic Q waves of myocardial infarction (http://medstat.med.utah.edu/kw/ecg/ecg_outline/Lesson3/index.html)
ST segment depression is always an abnormal finding, although often nonspecific (http://medstat.med.utah.edu/kw/ecg/ecg_outline/Lesson3/index.html)
As a general rule, T wave amplitude corresponds with the amplitude of the preceding R wave, though the tallest T waves are seen in leads V3 and V4. Tall T waves may be seen
in acute myocardial ischaemia and are a feature of hyperkalaemia.
Poor Man&apos;s Guide to upper limits of QT:
For HR = 70 bpm, QT&lt;0.40 sec; for every 10 bpm increase above 70 subtract 0.02 sec, and for every 10 bpm decrease below 70 add 0.02 sec. For example:
QT &lt; 0.38 @ 80 bpm
QT &lt; 0.42 @ 60 bpm