Red blue lesion diagnosis guide
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This document discusses different types of red or blue skin lesions, including congenital hemangiomas, vascular malformations, Kaposi's sarcoma, and lesions caused by metabolic deficiencies. Congenital hemangiomas are benign tumors present at birth that grow rapidly in infancy before regressing by puberty. Vascular malformations are structural anomalies of blood vessels present from birth. Kaposi's sarcoma is associated with human herpesvirus 8 and presents as reddish-brown nodules, affecting the skin and extremities. Metabolic deficiencies that can cause lesions include vitamin B deficiencies, vitamin B12 deficiency in pernicious anemia, and iron deficiency.
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Red blue lesion diagnosis guide
- 3. congenital: hemangioma Benign tumours of infancy :Characterized by Growth phase with endothelial cell proliferation of large (cavernous) or small (capillary) vascular channels Undergo gradual involvement by puberty Diagnosed at first 8 weeks of life female vs. 1 male 3 • • • • • •
- 4. .Cont Site : most common in head and neck Shape : pale macula with thread like telangieclasis Superficial skin lesion appear raised and bosselated Firm and rubbery to palpation • • • •
- 5. Congenital : vascular Structural anomaly of blood vessel without • endothelial proliferation Present at birth and persist throughout life • and grow with individual
- 6. .Cont :Capillary form • Part wine spot are common • Later in life lesion darken & becomes • nodular due to ectasia
- 7. .Cont :Venous form Blue in colour Compressible Swelling associated with increase venous pressure • • • •
- 8. .Cont :Anteriovenous form Persistent direct arterial & venous :communication Palpable thrill Overlying skin is hot to touch pain, bleeding and skin ulceration • • • • •
- 9. Neoplasms: kaposi’s sarcoma :Etiopathogenesis Human harpesvirus 8 Kaposi’s sarcoma herpesvirus Aids associated • • • •
- 10. .Cont : Clinical features Classic form ( indolent form & poor (prognosis Older male Multifocal reddish brown nodules Affect skin and lower extremities • • • • •
- 11. cont :Endomic form • Common in black population • Immunodeficiency form ( rapid aggressive & • (poor prognosis : Multifocal skin lesion not limited to extremities Oral and lymph node lesion are common Involve visceral organs Younger age group
- 12. .cont :Histopathology features Hypercellular foci Defined vascular channels Extarvasated RBCs Later may superficially resemble pyogenic granulomas • • • • •
- 13. Metabolic vitamin b complex deficiencies :Etiopathogenesis Decreased intake of vitamin b :Clinical feature Oral changes pain, burning sensation ,lip fissuring …est Keratitis of the eye Scaly dermatitis on nasolabial area • • • • • •
- 14. Vitamin b12 deficiency ((pernicious anaemia :Clinical feature Adult Weakness .pallor, shortness of breath and fatigue Headache, dizziness Nausea , diarrhoea& stomalitis :Oral manifestation Glossitis due to atrophy of the papillae • • • • • • •
- 15. Plummer vinson syndrome ((iron deficiency :Clinical feature • Weakness , pallor, shortness breath • ,brittle nails & hair atrophy of upper GIT and glossitis
- 16. Burning mouth syndrome :Etiologic Micro organisms fungi, bacteria Xerostomia Bit. B . Iron& zink definicicy • • • •