2. • Temporal bone fractures are extremely common with
head injuries.
• They present with hearing loss facial nerve
paralysis,vertigo leakage of CSF through the nose and
ear.
• The temporal bone fractures are
longitudinal,transverse or mixed.80% of temporal
bone fractures are longitudinal.
• Transverse fractures have more risk of permanent
facial nerve palsy than longitudinal fractures
3. Ketter et al states that inmmediate paralysis should be
explored as soon as the patient condition permits
because the palsy may be due to
1.Incomplete or complete transaction of nerve
2.Bony fragments compressing the nerve
3.Edema of the nerve due to generalized
inflammation due to trauma
4.Compression due to bands formed in the nerve
sheath which is caught between the fractured
fragments.
4. CASE DISCUSSION
• 2 patients had head trauma with temporal bone fractures
with presented with unilateral facial nerve palsy.
• Immediately ENoG was done which revealed 100%
degeneration.
• Both of them underwent endoscopic transcanal facial nerve
decompression under local anaesthesia.
• Fractured fragments removed from the horizontal segments
of facial nerve and decompression of horizontal segment was
done.
• Both patients are under followup and the results of nerve
exitability test and ENoG in preoperative and postoperative
period are recorded.
9. Traumatic Facial Nerve Paralysis
• Temporal bone fractures are classified as either
longitudinal(along the long axis of the petrous pyramid),
transverse(at right angles to the petrous pyramid),
mixed/complex/oblique
• Trauma to the temporal and parietal regions of the skull
tends to produce longitudinalfractures, which make up
the majority (90%) of temporalbone fractures. These
fractures cause conductive hearing loss by traversing the
tympanic membrane andthe tympanic cavity and by
causing a hemotympanum.
• Facial paralysis occurs in 20 to 25% of longitudinal
fractures, usually in the perigeniculate region.
10. • Transverse fractures, whichmake up less than
10% of temporal bone fractures.Transverse
fractures manifest with sensorineural hearing
loss and vestibular dysfunction owing to
involvement of the otic capsule or the IAC.
• They are more likely to cause facial nerve
paralysis in 50% patients.
11. Indication of Facial Nerve
Decompression
• If the patient either progresses to complete
paralysis or presents with complete paralysis, an
ENOG is obtained 4 days after occurrence of
complete paralysis. If degeneration is less than
90%, the corticosteroids are continued for the full
treatment course. Electroneurographic testing is
repeated every 1 to 3 days until > 90%
degeneration is detected and no voluntary motor
unit surgical decompression is indicated.
12. Approaches…
The facial nerve decompression can be carried
out by middle cranial fossa approach, trans
labrynthine approach,transmastoid
extralabrynthine subtemporal approach or
combination of above approaches.
13. Conclusion
In these patients we highlight the transcanal
approach to facial nerve decompression under
local anaesthesia and outcome of this
procedure.