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This is the unedited version of the "Probiotics" presentation given at the University of Arizona's College of Medicine. Its intent was to demonstrate that even in the "hard sciences" base emotional biases can interfere with good scientific data and interpretation. To fully appreciate this presentation, you must be eating yogurt prior to beginning to view it, and proceed through the slides one at a time without skipping ahead. This is the actual slide show that was shown at the seminar and is not for the squeamish. I'll post a more benign version for those not wishing to test their boundaries and "good taste."
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Definition: “Living microorganisms when conferred in sufficient amount on the host, will render beneficial effects on health.”(FAO/WHO) Lactobacillus, Candida, Streptococcus, Enterococcus, Bifidobacterium, Aspergillus, and Saccharomyces spp. History Élie Metchnikoff (20th century) Werner Kolath(1953) Probiotics for poultry Need of Probiotics? Selection criteria Probiotics in poultry industry First decisive incident Commercial vs. wild chicken Selection criteria Probiotic requirement in poultry (concept) How probiotics act? Maintain normal intestinal microflora competitive exclusion and resistance Change metabolism speeding digestive enzyme activity Perk up feed intake and digestion Diminish bacterial enzyme activity and ammonia production Stimulate the immune system Evaluation of probiotics on poultry Growth performance Intestinal microbiota and morphology Immune response Meat quality/chicken caracass Side effects(toxicity of ingredients) Growth performance broilers fed with two probiotic species put on more weight(Lan et al.,2003 ) weight gain significantly higher in probiotic fed birds(Kabir et al.,2004) . inactivated probiotics have constructive actions on the production achievement (Huang et al.,2004). Cont’d values of giblets and dressing percentage elevated for probiotic fed broilers (Mahanjan et al.,1999) Intestinal microbiota and morphology Probiotics inhibited pathogens by dwelling on intestinal wall space(Kabir et al.,2005 ) Birds fed dietary B. subtilis for 28 days displayed better growth and prominent intestinal histologies. (Samanya and Yamauchi.,2002) Chicks given Lactobacillus strains had less amount of coliforms in cecal grindings(Watkins and Kratzer.,1983 ). Cont’d L. salivarius 3d strain decreased the number of Clostridium perfringens and Salmonella enteritidis (Kizerwetter-Swida and Binek., 2009). Probiotic species have an implicit action on regulation of intestinal microflora and pathogen occlusion (Higgins et al., 2007) Immune response Higher amount of antibody production(Kabir et al.,2005 ) Improved serum and intestinal antibodies to a foreign antigens in chickens (Haghighi et al.,2005) Probiotics protected broilers against Eimeria acervulina infection even with a moderate dose (Dalloul et al.,2003) Cont’d Better local immune defenses against coccidiosis. Splenocytes and cecal tonsil cells, STAT2 and STAT4 genes were greatly stimulated and the expression of STAT2, STAT4, IL-18, IFN-alpha, and IFN-gamma genes in cecal tonsil cells were up-regulated after treating with L. acidophilus DNA. Additive probiotic supplements were ineffective on systemic IgG (Midilli et al.,2008 ).
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At the start of the 20th century, Russian noble prize winner and father of modern immunology, Elie Metchnikoff, a scientist at the Pasteur institute, was the first conceptualize “probiotics”. In 1907 Metchnokoff proposed that the acid producing bacteria in fermented milk products could prevent “fouling” in the large intestine and if consumed regularly, lead to a longer, healthier life. In early 1930’s, in Japan, Minoru shirota developed a fermented milk product called Yakult (probiotic yogurt like product made by fermenting a mixture of skimmed milk with a special strain of Lactobacillus casei shirota). Probiotic term coined in 1965 by Lilly and StillwellThe human gastrointestinal (GI) tract is a highly specialised ecosystem that has evolved over time, both physiologically and microbiologically. At least in part, this is a consequence of the host and environmental pressures that it must counteract in order to maintain eubiosis. The GI tract is one of the most diverse and metabolically active organs in the human body. The human gut and its microbiota cannot be realistically considered as separate entities as they represent a dynamic biological system that has co-evolved from birth. The human GI tract is composed of highly adapted regions for mediation of its diverse functions, many of which impact markedly upon host health and welfare. Physiological considerations in each unique region infl uence the degree and type of colonisation and initial colonisers also modify the physiological conditions therein. This results in the development of distinct microhabitats along the length of the GI tract, which infl uence metabolism, protection and immune stimulation. Such effects are both local and systemic as the GI tract is connected to the vascular, lymphatic and nervous systems. The ability of the gut to sustain its benefi cial microbiota, against harmful or opportunistic microbiota, in a desirable community structure, is critical for host health and reduction of disease risk. The focus of this chapter is to discuss how the complex interplays between the human GI tract and its indigenous microbiota affect host health and how certain benefi cial microbial species, with their potential for manipulation, are crucial to this processThe human gastrointestinal tract is sterile up until birth, when microbial colonisation begins during the delivery process. The inoculum may be largely derived either from the mother’s vaginal or faecal fl ora (in a conventional birth) or from the environment (in a caesarean delivery).Hence, the microbiota that colonise the newborn tract are acquired post-natally. This is of extreme importance in the choice of delivery, as newborns delivered by caesarean section are exposed to a different microbiota than that of a vaginal delivery. Bacterial populations develop progressively during the fi rst few days of life; facultative anaerobes predominate initially and create a reduced environment that allows for the growth of strict
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An Investor Pitch Deck is a brief presentation, often created using PowerPoint, used to provide your audience with a quick overview of your cannabis business plan. You will also use your pitch deck during face-to-face or online meetings with your partners and co-founders. We offer a '70% ready to go' professional Cannabis Edibles Investor Pitch Deck Template. The template is completely open and editable, and all you have to do is add your company information. https://cannabusinessplans.com/product/cannabis-investor-pitch-deck-template-edibles/
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