PE AND TAMPONADE.pptx
•Transferir como PPTX, PDF•
0 gostou•4 visualizações
The document discusses different types of embolism, focusing on pulmonary embolism. Pulmonary embolism is defined as an embolism in the pulmonary arterial tree, where emboli most commonly originate from deep vein thromboses in the legs. Risk factors for pulmonary embolism include advanced age, obesity, prolonged surgery, and cancer. Consequences of pulmonary embolism depend on the size of the emboli and include pulmonary infarction, hemorrhage, and hypertension. Cardiac tamponade is also discussed, which is a medical emergency caused by fluid accumulation in the pericardial sac compressing the heart.
Denunciar
Compartilhar
Denunciar
Compartilhar
Recomendados
Pulmonary diseases of vascular origin(pulmonary embolism)
The most probable diagnosis is Pulmonary Saddle embolus. The key points are:
- Prolonged bed rest following pelvic fracture which is a risk factor for deep vein thrombosis
- Sudden onset of chest pain and collapse
- Rapid demise of the patient
This fits with the clinical presentation of a large pulmonary embolism causing sudden death as described in the document.
5 embolism
An embolus is a solid, liquid, or gaseous mass that breaks off and travels through the bloodstream, lodging in and blocking smaller blood vessels. Pulmonary embolisms originate from deep leg vein thrombi in 95% of cases and can cause infarction or blockage of lung tissue. Systemic embolisms originate from heart mural thrombi in 80% of cases and commonly impact the brain or lower extremities. Fat embolisms occur after bone fractures and burns, causing pulmonary insufficiency, neurological issues, and thrombocytopenia. Air embolisms enter the circulation through chest or obstetric injuries and can block major blood vessels. Amniotic fluid embolisms are a rare
Embolism - Causes, Symptoms, Treatment, Diagnosis
General Pathology Notes
Embolism Definition And Types
Paradoxical embolus
Retrograde embolus
Arterial (systemic) thromboembolism
Venous thromboembolism
Pulmonary Thromboembolism
Contrasting features of pulmonary thrombosis and pulmonary thromboembolism
Consequences Of Pulmonary Embolism
Natural history of pulmonary embolism
Systemic Embolism
Non-traumatic causes
Pulmonary fat embolism
Consequences Of Fat Embolism
Systemic fat embolism
Gas Embolism
Air Embolism
Venous Air Embolism
Arterial Air Embolism
Effects The effects of decompression sickness
Clinical effects of decompression sickness
Amniotic Fluid Embolism
Morphologic Features
Embolism
Embolism is the obstruction of blood vessels by foreign material carried by the bloodstream called an embolus. The most common type is thromboembolism caused by blood clots. Embolisms can be classified by the material causing the obstruction (solid, liquid, gas), whether infected or not, and the source of the embolus (cardiac, arterial, venous, lymphatic). Pulmonary embolism is a serious and potentially fatal condition where blood clots block arteries in the lungs. Risk factors include immobilization, and clots most often originate from veins in the legs. Consequences can include sudden death, lung infarction, hemorrhage and chronic lung disease.
02 respiratory vascular
Chronic passive pulmonary congestion (CVC lung)
Pulmonary edema
Pulmonary thrombo-embolism and infarction
Pulmonary hypertension
Pulmonary hemorrhage*
Thromboembolism,pulmonary embolism,general pathology
This document discusses different types of thromboembolism including pulmonary embolism, fat embolism, air embolism, and amniotic fluid embolism. It provides details on the definition, causes, risk factors, symptoms, diagnosis, treatment and prognosis of pulmonary embolism, which is the most common type of thromboembolism. The document also discusses the pathophysiology and mechanisms of the different embolism types.
416456384-6-Thrombosis-ppt.ppt
Thrombosis is caused by Virchow's triad of endothelial injury, stasis or turbulence of blood, and hypercoagulability. Endothelial injury is the most dominant influence and can lead to thrombosis in the heart or arteries. Alterations in normal blood flow such as stasis and turbulence bring platelets into contact with the endothelium and predispose to thrombosis. Hypercoagulability, either inherited or acquired, also increases the risk of thrombosis. Thrombi may embolize, causing partial or complete occlusion of vessels and ischemic necrosis of distal tissues. Pulmonary embolism is a common complication of deep vein thrombosis in the legs. Systemic embolism usually arises from intracard
EMBOLISM -a blockage in a blood vessel caused by a piece of material.pptx
This document summarizes thrombosis, embolism, and related topics. It discusses normal haemostasis and how thrombosis occurs due to endothelial injury, abnormal blood flow, hypercoagulability, and other factors. Thrombi can propagate, embolize, or dissolve. Embolism occurs when a detached mass is carried by the bloodstream, commonly causing pulmonary or systemic embolism. Specific types of embolism discussed are thromboembolism, fat embolism, air embolism, and amniotic fluid embolism.
Recomendados
Pulmonary diseases of vascular origin(pulmonary embolism)
The most probable diagnosis is Pulmonary Saddle embolus. The key points are:
- Prolonged bed rest following pelvic fracture which is a risk factor for deep vein thrombosis
- Sudden onset of chest pain and collapse
- Rapid demise of the patient
This fits with the clinical presentation of a large pulmonary embolism causing sudden death as described in the document.
5 embolism
An embolus is a solid, liquid, or gaseous mass that breaks off and travels through the bloodstream, lodging in and blocking smaller blood vessels. Pulmonary embolisms originate from deep leg vein thrombi in 95% of cases and can cause infarction or blockage of lung tissue. Systemic embolisms originate from heart mural thrombi in 80% of cases and commonly impact the brain or lower extremities. Fat embolisms occur after bone fractures and burns, causing pulmonary insufficiency, neurological issues, and thrombocytopenia. Air embolisms enter the circulation through chest or obstetric injuries and can block major blood vessels. Amniotic fluid embolisms are a rare
Embolism - Causes, Symptoms, Treatment, Diagnosis
General Pathology Notes
Embolism Definition And Types
Paradoxical embolus
Retrograde embolus
Arterial (systemic) thromboembolism
Venous thromboembolism
Pulmonary Thromboembolism
Contrasting features of pulmonary thrombosis and pulmonary thromboembolism
Consequences Of Pulmonary Embolism
Natural history of pulmonary embolism
Systemic Embolism
Non-traumatic causes
Pulmonary fat embolism
Consequences Of Fat Embolism
Systemic fat embolism
Gas Embolism
Air Embolism
Venous Air Embolism
Arterial Air Embolism
Effects The effects of decompression sickness
Clinical effects of decompression sickness
Amniotic Fluid Embolism
Morphologic Features
Embolism
Embolism is the obstruction of blood vessels by foreign material carried by the bloodstream called an embolus. The most common type is thromboembolism caused by blood clots. Embolisms can be classified by the material causing the obstruction (solid, liquid, gas), whether infected or not, and the source of the embolus (cardiac, arterial, venous, lymphatic). Pulmonary embolism is a serious and potentially fatal condition where blood clots block arteries in the lungs. Risk factors include immobilization, and clots most often originate from veins in the legs. Consequences can include sudden death, lung infarction, hemorrhage and chronic lung disease.
02 respiratory vascular
Chronic passive pulmonary congestion (CVC lung)
Pulmonary edema
Pulmonary thrombo-embolism and infarction
Pulmonary hypertension
Pulmonary hemorrhage*
Thromboembolism,pulmonary embolism,general pathology
This document discusses different types of thromboembolism including pulmonary embolism, fat embolism, air embolism, and amniotic fluid embolism. It provides details on the definition, causes, risk factors, symptoms, diagnosis, treatment and prognosis of pulmonary embolism, which is the most common type of thromboembolism. The document also discusses the pathophysiology and mechanisms of the different embolism types.
416456384-6-Thrombosis-ppt.ppt
Thrombosis is caused by Virchow's triad of endothelial injury, stasis or turbulence of blood, and hypercoagulability. Endothelial injury is the most dominant influence and can lead to thrombosis in the heart or arteries. Alterations in normal blood flow such as stasis and turbulence bring platelets into contact with the endothelium and predispose to thrombosis. Hypercoagulability, either inherited or acquired, also increases the risk of thrombosis. Thrombi may embolize, causing partial or complete occlusion of vessels and ischemic necrosis of distal tissues. Pulmonary embolism is a common complication of deep vein thrombosis in the legs. Systemic embolism usually arises from intracard
EMBOLISM -a blockage in a blood vessel caused by a piece of material.pptx
This document summarizes thrombosis, embolism, and related topics. It discusses normal haemostasis and how thrombosis occurs due to endothelial injury, abnormal blood flow, hypercoagulability, and other factors. Thrombi can propagate, embolize, or dissolve. Embolism occurs when a detached mass is carried by the bloodstream, commonly causing pulmonary or systemic embolism. Specific types of embolism discussed are thromboembolism, fat embolism, air embolism, and amniotic fluid embolism.
Embolism
An embolism occurs when a solid, liquid, or gas mass travels through the bloodstream and lodges in a blood vessel blocking circulation. Most embolisms originate from blood clots (thrombi) that dislodge and travel to other parts of the body. Pulmonary embolisms occur when clots travel to the lungs, potentially blocking vessels and causing respiratory issues. Systemic embolisms can travel anywhere in the body via arterial circulation and cause tissue damage depending on the size and location of the blockage. Other types of embolisms include fat, air, amniotic fluid, and debris that can similarly obstruct vessels.
EMBOLISM....Dr San Kauser (Pathology).pdf
Embolism is the obstruction of blood vessels by foreign material that travels through the bloodstream. There are several types of embolism classified by the material causing obstruction (solid, liquid, gas), source of obstruction (cardiac, arterial, venous), and whether infection is present. Common types include thromboembolism caused by blood clots, pulmonary embolism from clots blocking pulmonary arteries, fat/bone marrow embolism from tissue trauma, and amniotic fluid embolism during childbirth from amniotic material entering the mother's bloodstream. Embolisms can cause infarction of tissues and organs downstream from the obstruction depending on the location and size of the obstructing material.
Pulmonary embolism Nursing mx..(ESTHER)
Pulmonary embolism (PE) refers to obstruction of the pulmonary artery or its branches, usually by blood clots originating from the deep veins of the lower extremities. PE is a common and potentially lethal condition, with many patients dying within hours of onset from blockage of blood flow to the lungs. The diagnosis of PE involves evaluating risk factors, performing physical exams, blood tests like D-dimer, and imaging tests like CT pulmonary angiography or ventilation-perfusion scans. Effective treatment requires identifying patients at high risk of complications or death.
Chfppt
Congestive heart failure can cause two types of pulmonary edema seen on chest x-rays: pulmonary interstitial edema and pulmonary alveolar edema. Pulmonary interstitial edema appears as thickening of interlobular septa, Kerley B lines, peribronchial cuffing, thickening of fissures, and subpleural fluid. Pulmonary alveolar edema creates a fluffy, bat-wing pattern in the lungs. Left atrial pressure correlates with the severity and type of pulmonary edema seen on chest x-rays.
Cardiac tamponade
Cardiac tamponade is caused by fluid accumulation in the pericardial space, reducing heart filling and function. It requires urgent treatment to remove fluid via pericardiocentesis or surgery. Symptoms include low blood pressure, increased heart rate, and difficulty breathing due to restricted heart movement. A diagnosis is made through echocardiography identifying fluid and heart compression.
Lecture 10 valvular heart disease - Pathology
Disorders of the heart valves commonly result from infective endocarditis, rheumatic fever, ischemia, trauma, or congenital defects. The two major types are stenosis, where the valve opening narrows, and regurgitation, where the valve does not close properly. Aortic stenosis causes the left ventricle to work harder and hypertrophy. Mitral stenosis obstructs blood flow from the left atrium to the left ventricle. Aortic and mitral regurgitation cause the heart to pump blood twice per cycle, leading to dilation and failure over time. Imaging like echocardiography is used to diagnose valve disorders while treatment involves managing symptoms medically or replacing defective valves surgically.
Circulitary disturbance
Edema can be caused by increased hydrostatic pressure, increased vascular permeability, decreased colloid osmotic pressure, decreased protein synthesis or increased protein loss, or lymphatic obstruction. The major mechanisms are increased hydrostatic pressure, as seen in congestive heart failure, or increased vascular permeability during inflammation. Edema fluid is usually a protein-poor transudate when caused by hydrostatic or oncotic pressure changes, but is a protein-rich exudate with inflammatory causes due to higher vascular permeability.
Pulmonary embolism.pptx by mcac rms ct findings
Pulmonary embolism.pptx by mcac rms ct findings
What Is Pulmonary Embolism (Pulmonary Embolus)?
A pulmonary embolism is a sudden blockage in your pulmonary arteries, the blood vessels that send blood to your lungs. It usually happens when a blood clot in the deep veins in your leg breaks off and travels to your lungs.
A blood clot that travels to another part of your body is called an embolus. When an embolus blocks a blood vessel it's called an embolism. When a pulmonary embolus blocks blood flow to your lungs, it's called a pulmonary embolism
The most common cause of a pulmonary embolism is a the breaking off of a blood clot in your leg's deep veins, known as deep vein thrombosis (DVT). Other, rare causes of a pulmonary embolism include:
Air bubbles
DVT in the upper body
Fat embolus, usually from a severely broken bone
Tumors
You are more likely to develop blood clots if you have risk factors such as:
Cancer
Family history of blood clots or clotting disorders
Heart disease
Health history of blood clots or clotting disorders
Long-term use of birth control pills or estrogen therapy
A pulmonary embolism is also more likely to develop after:
Childbirth
Heart attack or stroke
Long plane or car rides
Long-term bedrest
Severe injuries, burns or fractures
Surgery
Pulmonary Embolism Symptoms
You may not have any symptoms of a pulmonary embolism, depending on the size of the clot and your overall health. As blood flow becomes more and more blocked, you may experience symptoms such as:
Coughing, including a cough that produces bloody mucus
Dizziness
Heart palpitations, sensations of your heart racing or pounding
Leg pain or swelling
Sharp and sudden chest pain
Shortness of breath that worsens with exertion
Pulmonary Embolism Complications
A pulmonary embolism can be life-threatening without treatment. It can cause several complications, including:
Cardiac arrest, when your heart suddenly stops beating
Cardiac arrhythmia, an irregular heart rhythm
Pleural effusion, fluid buildup in the membrane around your lungs (pleura
Pulmonary hypertension, high blood pressure in your lungs
Pulmonary infarction, when lung tissue dies
Diagnosing a Pulmonary Embolism
To diagnose a pulmonary embolism, your doctor does a physical exam, asks about your symptoms and orders one or more tests such as:
Blood tests to check if you have an increased risk of blood clotting
Chest X-rays to take pictures of your heart and lungs
CT pulmonary angiography, using X-rays and specialized computers to create cross-sectional, 3D images of your lungs and pulmonary arteries
Doppler ultrasound, using sound waves to look for deep vein thrombosis in your legs
Echocardiogram to view your heart's structures and blood vessels
Lab tests, such as pulse oximetry to measure oxygen levels in your blood or arterial blood gas tests to measure the balance of oxygen and carbon dioxide
Pulmonary angiogram, using a small, hollow tube (catheter) and a contrast dye to see how blood flows through the pulmonary A
Pulmonary edema & cardiogenic apo
Acute pulmonary edema refers to excess fluid in the lungs that can have cardiogenic or non-cardiogenic causes. Cardiogenic pulmonary edema is often due to left ventricular dysfunction that leads to increased pulmonary capillary pressure. Common causes of left ventricular dysfunction include congestive heart failure, myocardial infarction, cardiomyopathy, and valvular diseases. Pulmonary edema progresses through three stages as excess fluid builds up first in small blood vessels then the lung interstitium and alveoli, impairing gas exchange.
Embolism
This document defines and describes different types of embolism. The most common type is thromboembolism, which occurs when a thrombus or part of a thrombus breaks off and is carried by the bloodstream. Pulmonary thromboembolism is a significant type that occurs when thrombi travel to the lungs and obstruct the pulmonary arteries. Other types include fat, air, gas and paradoxical embolisms. Air embolism can be venous, entering systemic veins during surgery or trauma, or arterial, entering the lungs during procedures like angiography. Decompression sickness is a form of gas embolism that affects divers or those exposed to changes in atmospheric pressure.
Thrombosis, embolism and infarction
Thrombosis is the formation of a blood clot within a blood vessel or cavity of the heart. Virchow identified three main factors that contribute to thrombosis: endothelial injury, changes in blood flow, and hypercoagulability. Thrombi can propagate or embolize, becoming lodged in another vessel and resulting in infarction of downstream tissue. Infarctions appear pale/white in solid organs and red/hemorrhagic in lungs/other tissues. Over time, infarcted tissue progresses from coagulative necrosis to phagocytosis and scar formation.
thromboembolism
Thromboembolism refers to the blocking of blood vessels by particles that have broken away from clots in the body. It is common in patients with cardiac disease or impaired circulation. Intracardiac thrombi, which can be detected by echocardiogram, are especially common in patients with atrial fibrillation. Pulmonary embolism occurs when a thrombus breaks off and travels to the lung, often from a deep vein thrombosis. It can be treated with anticoagulants or thrombolytics. Pericardial effusion involves a buildup of fluid in the sac around the heart, which can lead to cardiac tamponade if pressure increases and compresses the heart.
Ed tech boholano
The document outlines a cardiovascular system course, covering anatomy and physiology of the heart and blood vessels, as well as history and assessment findings for cardiac diseases. Key topics include heart structure and function, blood supply, blood pressure control mechanisms, risk factors for heart disease, physical exam techniques, diagnostic tests, and common cardiac conditions such as coronary artery disease, heart failure, and infectious or inflammatory disorders of the heart.
INFARCTION
This document discusses infarction, which is localized ischemic necrosis of tissue due to decreased blood supply. Infarction can be caused by thrombi, emboli, vasospasm, expansion of atheroma, extrinsic compression of vessels, vessel twisting, or traumatic vessel rupture. There are three main types of infarction: red (hemorrhagic), white (anemic), and septic. Factors that influence infarction development include vulnerability to hypoxia, blood oxygen content, rate of occlusion, and blood supply. Myocardial, pulmonary, and cerebral infarctions are provided as examples and their characteristics and outcomes described.
Pulmonary embolism
Pulmonary embolism (PE) occurs when a blood clot gets lodged in an artery in the lung, blocking blood flow to part of the lung. Blood clots most often start in the legs and travel up through the right side of the heart and into the lungs.
Pulmonary Oedema - Pathophysiology - Approach & Management
Pulmonary edema is often caused by congestive heart failure. When the heart is not able to pump efficiently, blood can back up into the veins that take blood through the lungs. As the pressure in these blood vessels increases, fluid is pushed into the air spaces (alveoli) in the lungs.
thrombosisembolismandinfarction-180117180555.pptx
Thrombosis, embolism, and infarction are related pathological processes involving blood clots. Thrombosis is the formation of a blood clot within a blood vessel, while embolism occurs when a piece of a clot breaks off and travels to another location. Infarction results from obstruction of blood flow by a clot, causing tissue death. The document discusses the mechanisms, types, features, and progression of thrombosis, embolism, and infarction. It also covers related topics like Virchow's triad, hypercoagulable states, fat embolism, and amniotic fluid embolism.
thrombosisembolismandinfarction-180117180555.pptx
Thrombosis, embolism, and infarction are related pathological processes involving blood clots. Thrombosis is the formation of a blood clot within a blood vessel, while embolism occurs when a piece of a clot breaks off and travels to another location. Infarction results from obstruction of blood flow by a clot, causing tissue death. The document discusses the mechanisms, classifications, and morphological features of thrombosis, embolism, and infarction. It also covers related topics like Virchow's triad, hypercoagulable states, and the development and types of infarcts over time.
ECHOCARDIOGRAPHY IN CARDIAC TAMPONADE
This document discusses cardiac tamponade, which occurs when fluid rapidly accumulates in the pericardial sac, putting pressure on the heart and reducing cardiac function. Key points include:
- Pericardial effusion puts pressure on the heart, causing symptoms like chest pain and shortness of breath.
- Cardiac tamponade occurs when a rapid accumulation of fluid in the pericardial sac severely compresses the heart.
- Echocardiography is useful for diagnosing tamponade by showing findings like pericardial effusion, right ventricular collapse, and reduced respiratory variation in blood flow velocities.
- Tamponade is a medical emergency treated initially with medications and peric
Hemodynamic disorders
A short intoductory lecture for medical students on general pathology (basic terminology on hemodynamic disorders)
DATING OF ENDOMETRIUM - female genital tract.pptx
This document summarizes the histological changes that occur in the endometrium throughout the menstrual cycle and reproductive lifespan of females. It describes the development of the endometrium from embryonic origin through menarche, reproductive years with the proliferative and secretory phases, pregnancy, and post-menopause. Key histological features are highlighted for each phase, including changes to the endometrial glands and stroma, presence of mitotic figures, vacuolization, predecidualization, breakdown during menstruation, and atrophic changes after menopause.
Histology of uterine endometrium.pptxXXX
The uterus is a pear-shaped organ located in the pelvis. It has three layers - endometrium, myometrium, and perimetrium. The endometrium undergoes changes over the course of the menstrual cycle in response to hormones from the ovaries. During the proliferative phase, the endometrium thickens in response to rising estrogen levels. After ovulation, progesterone levels rise and the endometrium develops secretory changes in preparation for potential implantation. If implantation does not occur, the functional layer of the endometrium is shed during menstruation. Histological examination can be used to determine the stage of the menstrual cycle.
Mais conteúdo relacionado
Semelhante a PE AND TAMPONADE.pptx
Embolism
An embolism occurs when a solid, liquid, or gas mass travels through the bloodstream and lodges in a blood vessel blocking circulation. Most embolisms originate from blood clots (thrombi) that dislodge and travel to other parts of the body. Pulmonary embolisms occur when clots travel to the lungs, potentially blocking vessels and causing respiratory issues. Systemic embolisms can travel anywhere in the body via arterial circulation and cause tissue damage depending on the size and location of the blockage. Other types of embolisms include fat, air, amniotic fluid, and debris that can similarly obstruct vessels.
EMBOLISM....Dr San Kauser (Pathology).pdf
Embolism is the obstruction of blood vessels by foreign material that travels through the bloodstream. There are several types of embolism classified by the material causing obstruction (solid, liquid, gas), source of obstruction (cardiac, arterial, venous), and whether infection is present. Common types include thromboembolism caused by blood clots, pulmonary embolism from clots blocking pulmonary arteries, fat/bone marrow embolism from tissue trauma, and amniotic fluid embolism during childbirth from amniotic material entering the mother's bloodstream. Embolisms can cause infarction of tissues and organs downstream from the obstruction depending on the location and size of the obstructing material.
Pulmonary embolism Nursing mx..(ESTHER)
Pulmonary embolism (PE) refers to obstruction of the pulmonary artery or its branches, usually by blood clots originating from the deep veins of the lower extremities. PE is a common and potentially lethal condition, with many patients dying within hours of onset from blockage of blood flow to the lungs. The diagnosis of PE involves evaluating risk factors, performing physical exams, blood tests like D-dimer, and imaging tests like CT pulmonary angiography or ventilation-perfusion scans. Effective treatment requires identifying patients at high risk of complications or death.
Chfppt
Congestive heart failure can cause two types of pulmonary edema seen on chest x-rays: pulmonary interstitial edema and pulmonary alveolar edema. Pulmonary interstitial edema appears as thickening of interlobular septa, Kerley B lines, peribronchial cuffing, thickening of fissures, and subpleural fluid. Pulmonary alveolar edema creates a fluffy, bat-wing pattern in the lungs. Left atrial pressure correlates with the severity and type of pulmonary edema seen on chest x-rays.
Cardiac tamponade
Cardiac tamponade is caused by fluid accumulation in the pericardial space, reducing heart filling and function. It requires urgent treatment to remove fluid via pericardiocentesis or surgery. Symptoms include low blood pressure, increased heart rate, and difficulty breathing due to restricted heart movement. A diagnosis is made through echocardiography identifying fluid and heart compression.
Lecture 10 valvular heart disease - Pathology
Disorders of the heart valves commonly result from infective endocarditis, rheumatic fever, ischemia, trauma, or congenital defects. The two major types are stenosis, where the valve opening narrows, and regurgitation, where the valve does not close properly. Aortic stenosis causes the left ventricle to work harder and hypertrophy. Mitral stenosis obstructs blood flow from the left atrium to the left ventricle. Aortic and mitral regurgitation cause the heart to pump blood twice per cycle, leading to dilation and failure over time. Imaging like echocardiography is used to diagnose valve disorders while treatment involves managing symptoms medically or replacing defective valves surgically.
Circulitary disturbance
Edema can be caused by increased hydrostatic pressure, increased vascular permeability, decreased colloid osmotic pressure, decreased protein synthesis or increased protein loss, or lymphatic obstruction. The major mechanisms are increased hydrostatic pressure, as seen in congestive heart failure, or increased vascular permeability during inflammation. Edema fluid is usually a protein-poor transudate when caused by hydrostatic or oncotic pressure changes, but is a protein-rich exudate with inflammatory causes due to higher vascular permeability.
Pulmonary embolism.pptx by mcac rms ct findings
Pulmonary embolism.pptx by mcac rms ct findings
What Is Pulmonary Embolism (Pulmonary Embolus)?
A pulmonary embolism is a sudden blockage in your pulmonary arteries, the blood vessels that send blood to your lungs. It usually happens when a blood clot in the deep veins in your leg breaks off and travels to your lungs.
A blood clot that travels to another part of your body is called an embolus. When an embolus blocks a blood vessel it's called an embolism. When a pulmonary embolus blocks blood flow to your lungs, it's called a pulmonary embolism
The most common cause of a pulmonary embolism is a the breaking off of a blood clot in your leg's deep veins, known as deep vein thrombosis (DVT). Other, rare causes of a pulmonary embolism include:
Air bubbles
DVT in the upper body
Fat embolus, usually from a severely broken bone
Tumors
You are more likely to develop blood clots if you have risk factors such as:
Cancer
Family history of blood clots or clotting disorders
Heart disease
Health history of blood clots or clotting disorders
Long-term use of birth control pills or estrogen therapy
A pulmonary embolism is also more likely to develop after:
Childbirth
Heart attack or stroke
Long plane or car rides
Long-term bedrest
Severe injuries, burns or fractures
Surgery
Pulmonary Embolism Symptoms
You may not have any symptoms of a pulmonary embolism, depending on the size of the clot and your overall health. As blood flow becomes more and more blocked, you may experience symptoms such as:
Coughing, including a cough that produces bloody mucus
Dizziness
Heart palpitations, sensations of your heart racing or pounding
Leg pain or swelling
Sharp and sudden chest pain
Shortness of breath that worsens with exertion
Pulmonary Embolism Complications
A pulmonary embolism can be life-threatening without treatment. It can cause several complications, including:
Cardiac arrest, when your heart suddenly stops beating
Cardiac arrhythmia, an irregular heart rhythm
Pleural effusion, fluid buildup in the membrane around your lungs (pleura
Pulmonary hypertension, high blood pressure in your lungs
Pulmonary infarction, when lung tissue dies
Diagnosing a Pulmonary Embolism
To diagnose a pulmonary embolism, your doctor does a physical exam, asks about your symptoms and orders one or more tests such as:
Blood tests to check if you have an increased risk of blood clotting
Chest X-rays to take pictures of your heart and lungs
CT pulmonary angiography, using X-rays and specialized computers to create cross-sectional, 3D images of your lungs and pulmonary arteries
Doppler ultrasound, using sound waves to look for deep vein thrombosis in your legs
Echocardiogram to view your heart's structures and blood vessels
Lab tests, such as pulse oximetry to measure oxygen levels in your blood or arterial blood gas tests to measure the balance of oxygen and carbon dioxide
Pulmonary angiogram, using a small, hollow tube (catheter) and a contrast dye to see how blood flows through the pulmonary A
Pulmonary edema & cardiogenic apo
Acute pulmonary edema refers to excess fluid in the lungs that can have cardiogenic or non-cardiogenic causes. Cardiogenic pulmonary edema is often due to left ventricular dysfunction that leads to increased pulmonary capillary pressure. Common causes of left ventricular dysfunction include congestive heart failure, myocardial infarction, cardiomyopathy, and valvular diseases. Pulmonary edema progresses through three stages as excess fluid builds up first in small blood vessels then the lung interstitium and alveoli, impairing gas exchange.
Embolism
This document defines and describes different types of embolism. The most common type is thromboembolism, which occurs when a thrombus or part of a thrombus breaks off and is carried by the bloodstream. Pulmonary thromboembolism is a significant type that occurs when thrombi travel to the lungs and obstruct the pulmonary arteries. Other types include fat, air, gas and paradoxical embolisms. Air embolism can be venous, entering systemic veins during surgery or trauma, or arterial, entering the lungs during procedures like angiography. Decompression sickness is a form of gas embolism that affects divers or those exposed to changes in atmospheric pressure.
Thrombosis, embolism and infarction
Thrombosis is the formation of a blood clot within a blood vessel or cavity of the heart. Virchow identified three main factors that contribute to thrombosis: endothelial injury, changes in blood flow, and hypercoagulability. Thrombi can propagate or embolize, becoming lodged in another vessel and resulting in infarction of downstream tissue. Infarctions appear pale/white in solid organs and red/hemorrhagic in lungs/other tissues. Over time, infarcted tissue progresses from coagulative necrosis to phagocytosis and scar formation.
thromboembolism
Thromboembolism refers to the blocking of blood vessels by particles that have broken away from clots in the body. It is common in patients with cardiac disease or impaired circulation. Intracardiac thrombi, which can be detected by echocardiogram, are especially common in patients with atrial fibrillation. Pulmonary embolism occurs when a thrombus breaks off and travels to the lung, often from a deep vein thrombosis. It can be treated with anticoagulants or thrombolytics. Pericardial effusion involves a buildup of fluid in the sac around the heart, which can lead to cardiac tamponade if pressure increases and compresses the heart.
Ed tech boholano
The document outlines a cardiovascular system course, covering anatomy and physiology of the heart and blood vessels, as well as history and assessment findings for cardiac diseases. Key topics include heart structure and function, blood supply, blood pressure control mechanisms, risk factors for heart disease, physical exam techniques, diagnostic tests, and common cardiac conditions such as coronary artery disease, heart failure, and infectious or inflammatory disorders of the heart.
INFARCTION
This document discusses infarction, which is localized ischemic necrosis of tissue due to decreased blood supply. Infarction can be caused by thrombi, emboli, vasospasm, expansion of atheroma, extrinsic compression of vessels, vessel twisting, or traumatic vessel rupture. There are three main types of infarction: red (hemorrhagic), white (anemic), and septic. Factors that influence infarction development include vulnerability to hypoxia, blood oxygen content, rate of occlusion, and blood supply. Myocardial, pulmonary, and cerebral infarctions are provided as examples and their characteristics and outcomes described.
Pulmonary embolism
Pulmonary embolism (PE) occurs when a blood clot gets lodged in an artery in the lung, blocking blood flow to part of the lung. Blood clots most often start in the legs and travel up through the right side of the heart and into the lungs.
Pulmonary Oedema - Pathophysiology - Approach & Management
Pulmonary edema is often caused by congestive heart failure. When the heart is not able to pump efficiently, blood can back up into the veins that take blood through the lungs. As the pressure in these blood vessels increases, fluid is pushed into the air spaces (alveoli) in the lungs.
thrombosisembolismandinfarction-180117180555.pptx
Thrombosis, embolism, and infarction are related pathological processes involving blood clots. Thrombosis is the formation of a blood clot within a blood vessel, while embolism occurs when a piece of a clot breaks off and travels to another location. Infarction results from obstruction of blood flow by a clot, causing tissue death. The document discusses the mechanisms, types, features, and progression of thrombosis, embolism, and infarction. It also covers related topics like Virchow's triad, hypercoagulable states, fat embolism, and amniotic fluid embolism.
thrombosisembolismandinfarction-180117180555.pptx
Thrombosis, embolism, and infarction are related pathological processes involving blood clots. Thrombosis is the formation of a blood clot within a blood vessel, while embolism occurs when a piece of a clot breaks off and travels to another location. Infarction results from obstruction of blood flow by a clot, causing tissue death. The document discusses the mechanisms, classifications, and morphological features of thrombosis, embolism, and infarction. It also covers related topics like Virchow's triad, hypercoagulable states, and the development and types of infarcts over time.
ECHOCARDIOGRAPHY IN CARDIAC TAMPONADE
This document discusses cardiac tamponade, which occurs when fluid rapidly accumulates in the pericardial sac, putting pressure on the heart and reducing cardiac function. Key points include:
- Pericardial effusion puts pressure on the heart, causing symptoms like chest pain and shortness of breath.
- Cardiac tamponade occurs when a rapid accumulation of fluid in the pericardial sac severely compresses the heart.
- Echocardiography is useful for diagnosing tamponade by showing findings like pericardial effusion, right ventricular collapse, and reduced respiratory variation in blood flow velocities.
- Tamponade is a medical emergency treated initially with medications and peric
Hemodynamic disorders
A short intoductory lecture for medical students on general pathology (basic terminology on hemodynamic disorders)
Semelhante a PE AND TAMPONADE.pptx (20)
Pulmonary Oedema - Pathophysiology - Approach & Management
Pulmonary Oedema - Pathophysiology - Approach & Management
Mais de Manjula N
DATING OF ENDOMETRIUM - female genital tract.pptx
This document summarizes the histological changes that occur in the endometrium throughout the menstrual cycle and reproductive lifespan of females. It describes the development of the endometrium from embryonic origin through menarche, reproductive years with the proliferative and secretory phases, pregnancy, and post-menopause. Key histological features are highlighted for each phase, including changes to the endometrial glands and stroma, presence of mitotic figures, vacuolization, predecidualization, breakdown during menstruation, and atrophic changes after menopause.
Histology of uterine endometrium.pptxXXX
The uterus is a pear-shaped organ located in the pelvis. It has three layers - endometrium, myometrium, and perimetrium. The endometrium undergoes changes over the course of the menstrual cycle in response to hormones from the ovaries. During the proliferative phase, the endometrium thickens in response to rising estrogen levels. After ovulation, progesterone levels rise and the endometrium develops secretory changes in preparation for potential implantation. If implantation does not occur, the functional layer of the endometrium is shed during menstruation. Histological examination can be used to determine the stage of the menstrual cycle.
Anticoagulants.pptx
This document discusses various anticoagulants used for blood collection and their properties. It describes ethylene diamine tetra acetic acid (EDTA) as the most commonly used anticoagulant. EDTA works by chelating or removing calcium from the blood and prevents clotting. Other anticoagulants discussed include citrate, which complexes with calcium, and heparin, which interferes with coagulation factor activity. The appropriate anticoagulant to use depends on the subsequent tests to be performed on the blood sample.
platelet.pptx
Platelets are produced in bone marrow by large megakaryocytes cells and play a key role in hemostasis, coagulation, and wound healing. Normal platelet counts range from 150,000 to 450,000 per microliter of blood, with low platelet counts causing excessive bleeding due to thrombocytopenia and high platelet counts potentially leading to abnormal blood clot formation from thrombocytosis. Abnormalities in platelet count or function can result in bleeding or clotting disorders.
mitochondrial diseases.pptx
This document lists several mitochondrial diseases including MELAS (Mitochondrial Encephalomyopathy, Lactic Acidosis and Stroke-like episodes), MERRF (Myoclonic Epilepsy with Ragged Red Fibers), MIDD (Maternally Inherited Diabetes and Deafness), and LEIGH syndrome. All of these mitochondrial diseases listed are maternally inherited.
HL TYPES.pptx
This document discusses the different subtypes of Hodgkin's lymphoma, including nodular sclerosis, mixed cellularity, lymphocyte rich, lymphocyte depletion, and lymphocyte predominant. Each subtype is characterized by the morphology of the Reed Sternberg cells, immunophenotype, clinical features, and background cells. The subtypes differ in factors such as gender prevalence, Epstein-Barr virus association, common stage at presentation, and affected age groups.
cystic lesion of pancreas.pptx
This document summarizes different cystic and neoplastic lesions of the pancreas. It describes non-neoplastic lesions such as pseudocysts, lymphoepithelial cysts, duct retention cysts, and mucinous cysts. It also describes several neoplastic lesions - serous cystic neoplasms, mucinous cystic neoplasms, intraductal papillary mucinous neoplasms, intraductal tubulopapillary neoplasms, and solid pseudopapillary neoplasms. For each type of lesion, it provides brief details on pathogenesis, histopathological features, immunochemical markers, and genetic mutations when relevant.
cytoskeleton.pptx
This document describes the three main components of the cytoskeleton: microfilaments, microtubules, and intermediate filaments. Microfilaments are made of actin, microtubules are hollow tubes made of tubulin, and intermediate filaments are made of keratin or other proteins. The cytoskeleton helps maintain cell shape and provides support. Disorders of the cytoskeleton can cause diseases affecting muscles, skin, blood, and neurodegenerative conditions. Wiskott-Aldrich syndrome is an X-linked recessive immunodeficiency disorder caused by a defect on the WASP gene on the X chromosome.
MALARIA.pptx
The document outlines the life cycle of Plasmodium, the parasite that causes malaria. It starts with sporozoites infecting the liver where they multiply and release merozoites. The merozoites then invade red blood cells and undergo asexual reproduction, multiplying into merozoites within the red blood cells. Some parasites develop into gametocytes, the sexual form that can infect mosquitoes to continue the life cycle.
IMMUNOHEMATOLOGY BASICS.pptx
The document discusses blood grouping and cross matching in immunology. It covers the key points of ABO blood grouping systems discovered by Landsteiner in 1900. The four main blood groups are A, B, AB and O. Group O persons are considered universal donors as their red blood cells lack A and B antigens. Group AB persons are universal recipients as they possess both A and B antigens. Cross matching involves mixing recipient and donor blood samples to detect ABO incompatibility and clinically significant antibodies to ensure blood transfusion safety.
SEMEN ANALYSIS.pptx
Semen is produced in the male reproductive tract and contains sperm. A semen analysis evaluates several parameters of semen including volume, pH, sperm count, motility, viability, and morphology. It is used to assess male fertility and can help with infertility diagnosis. A semen sample is collected through masturbation after 2-7 days of abstinence and sent for analysis within an hour. The analysis involves examining the sample under a microscope to evaluate the number and health of sperm present. Abnormal results may indicate issues that could impact a man's fertility.
IMMUNODEFICIENCY.pptx
This document discusses various immunodeficiency disorders categorized as either primary or secondary immunodeficiencies. It focuses on defects in leukocyte function, including three types of leukocyte adhesion deficiency (LAD I, II, III) characterized by reduced integrins and other cell surface markers. One disorder discussed is chronic granulomatous disease, an X-linked disease caused by mutations in the CYBB gene that result in defects in the respiratory burst pathway. Another is Chediak-Higashi syndrome, an autosomal recessive condition caused by mutations in the CHS1 gene and LYST defects, leading to abnormalities in lysosomal enzyme structure/function and granule formation.
chemical mediators AHS cls.pptx
Chemical mediators initiate and regulate inflammatory reactions. They can be cell-derived or plasma-derived and include vasoactive amines, lysosomal components, platelet activating factor, cytokines, arachidonic acid metabolites, kinins, components of the clotting, fibrinolytic, and complement systems. Mediators are short-lived and stimulate the production of other mediators. They cause effects like increased vascular permeability, vasodilation, chemotaxis, and pain.
CELL DEATH - 2.pptx
There is no inflammation in apoptosis because:
- Apoptosis is an orderly and controlled process of programmed cell death that does not release intracellular contents into the surrounding tissue.
- In apoptosis, the cell shrinks and its contents are packaged into apoptotic bodies which are then phagocytosed by neighboring cells or macrophages in a non-inflammatory manner.
- There is no disruption of the cell membrane or release of cytoplasmic enzymes and other inflammatory mediators that would trigger an inflammatory response, as occurs in necrosis. The orderly packaging and removal of dead cells prevents collateral tissue damage and inflammation.
CELL COUNTING.pptx
This document discusses techniques for counting white blood cells (WBCs) and red blood cells (RBCs) using an improved Neubauer chamber. The chamber has a ruled area of 3x3 mm divided into large and small squares. WBCs are counted in the four corner large squares, each containing 16 small squares. RBCs are counted in the central large square containing 25 small squares. The techniques calculate WBC and RBC concentrations per mm3 of blood based on the cell counts within the squares and known volumes.
RETICULIN STAIN SEMINAR 24.5.pptx
1. Reticulin stain demonstrates reticular fibers and basement membrane material by staining them black using a silver impregnation process.
2. It is useful for studying lesions affecting organs rich in reticulin fibers like the liver and bone marrow, and can help diagnose conditions like cirrhosis, fibrosis, and tumors.
3. The staining process involves oxidizing, sensitizing, and impregnating slides with silver salts before developing and counterstaining to visualize the black reticulin fibers.
STAINING TECHNIQUES manju.pdf
This document discusses staining techniques used in histopathology. It describes the purpose of staining as outlining tissues and cellular components to identify tissues and establish the presence or absence of disease. The most common staining technique is hematoxylin and eosin (H&E) staining. Hematoxylin stains nuclei blue and eosin stains cytoplasm pink, allowing clear visualization of tissue structures. The document provides details on the preparation and use of hematoxylin and eosin stains as well as other specialized staining techniques.
cystitis.pptx
Cystitis is an inflammation of the bladder. The document discusses cystitis and its treatment with antibiotics, which are prescribed to fight the bacterial infection causing the symptoms. Antibiotics are the primary treatment recommended to address cystitis.
MICROANGIOPATHIC HEMOLYTIC ANEMIA - MAHA.pptx
This document discusses microangiopathic hemolytic anemia (MAHA), which results from the fragmentation of red blood cells as they pass through the microcirculation. Some key causes of MAHA include disseminated intravascular coagulation, thrombotic thrombocytopenic purpura, and hemolytic uremic syndrome. Laboratory findings of MAHA include decreased hemoglobin, peripheral blood smears showing fragmented red blood cells called schistocytes, and sometimes thrombocytopenia.
MOLECULAR PROFILING OF OSTEOARTICULAR NEOPLASM.pptx
Primary bone tumors can be benign or malignant and affect people of a wide age range. Molecular profiling using techniques like next generation sequencing has helped distinguish tumors with overlapping features and identified recurrent genetic alterations specific to different tumor types. For example, chondrosarcomas commonly harbor mutations in IDH1 and IDH2, while giant cell tumor of bone has H3F3A alterations in over 95% of cases. This molecular information can provide diagnostic, prognostic and predictive insights and help guide targeted therapy development.
Mais de Manjula N (20)
MOLECULAR PROFILING OF OSTEOARTICULAR NEOPLASM.pptx
MOLECULAR PROFILING OF OSTEOARTICULAR NEOPLASM.pptx
Último
Hypotension and role of physiotherapy in it
This particular slides consist of- what is hypotension,what are it's causes and it's effect on body, risk factors, symptoms,complications, diagnosis and role of physiotherapy in it.
This slide is very helpful for physiotherapy students and also for other medical and healthcare students.
Here is the summary of hypotension:
Hypotension, or low blood pressure, is when the pressure of blood circulating in the body is lower than normal or expected. It's only a problem if it negatively impacts the body and causes symptoms. Normal blood pressure is usually between 90/60 mmHg and 120/80 mmHg, but pressures below 90/60 are generally considered hypotensive.
The Importance of Black Women Understanding the Chemicals in Their Personal C...
Certain chemicals, such as phthalates and parabens, can disrupt the body's hormones and have significant effects on health. According to data, hormone-related health issues such as uterine fibroids, infertility, early puberty and more aggressive forms of breast and endometrial cancers disproportionately affect Black women. Our guest speaker, Jasmine A. McDonald, PhD, an Assistant Professor in the Department of Epidemiology at Columbia University in New York City, discusses the scientific reasons why Black women should pay attention to specific chemicals in their personal care products, like hair care, and ways to minimize their exposure.
nhs fpx 4000 assessment 4 analyzing a current health care problem or issue.pdf
nhs fpx 4000 assessment 4 analyzing a current health care problem or issue.pdf
CAPNOGRAPHY and CAPNOMETRY/ ETCO2 .pptx
End-tidal carbon dioxide (ETCO2) is the level of carbon dioxide that is released at the end of an exhaled breath. ETCO2 levels reflect the adequacy with which carbon dioxide (CO2) is carried in the blood back to the lungs and exhaled.
Non-invasive methods for ETCO2 measurement include capnometry and capnography. Capnometry provides a numerical value for ETCO2. In contrast, capnography delivers a more comprehensive measurement that is displayed in both graphical (waveform) and numerical form.
Sidestream devices can monitor both intubated and non-intubated patients, while mainstream devices are most often limited to intubated patients.
Friendly Massage in Ajman - Malayali Kerala Spa Ajman
At Malayali Kerala Spa Ajman, Full Service includes individualized care for every client. We specifically design each massage session for the individual needs of the client. Our therapists are always willing to adjust the treatments based on the client's instruction and feedback. This guarantees that every client receives the treatment they expect.
By offering a variety of massage services, our Ajman Spa Massage Center can tackle physical, mental, and emotional illnesses. In addition, efficient identification of specific health conditions and designing treatment plans accordingly can significantly enhance the quality of massaging.
At Malayali Kerala Spa Ajman, we firmly believe that everyone should have the option to experience top-quality massage services regularly. To achieve that goal we offer cheap massage services in Ajman.
If you are interested in experiencing transformative massage treatment at Malayali Kerala Spa Ajman, you can use our Ajman Massage Center WhatsApp Number to schedule your next massage session.
Contact @ +971 529818279
Visit @ https://malayalikeralaspaajman.com/
VEDANTA AIR AMBULANCE SERVICES IN REWA AT A COST-EFFECTIVE PRICE.pdf
Air Ambulance Services In Rewa works in close coordination with ground-based emergency services, including local Emergency Medical Services, fire departments, and law enforcement agencies.
More@: https://tinyurl.com/2shrryhx
More@: https://tinyurl.com/5n8h3wp8
U Part Wigs_ A Natural Look with Minimal Effort Jokerwigs.in.pdf
Joker Wigs has been a one-stop-shop for hair products for over 26 years. We provide high-quality hair wigs, hair extensions, hair toppers, hair patch, and more for both men and women.
Sectional dentures for microstomia patients.pptx
Microstomia, characterized by an abnormally small oral aperture, presents significant challenges in prosthodontic treatment, including limited access for examination, difficulties in impression making, and challenges with prosthesis insertion and removal. To manage these issues, customized impression techniques using sectional trays and elastomeric materials are employed. Prostheses may be designed in segments or with flexible materials to facilitate handling. Minimally invasive procedures and the use of digital technologies can enhance patient comfort. Education and training for patients on prosthesis care and maintenance are crucial for compliance. Regular follow-up and a multidisciplinary approach, involving collaboration with other specialists, ensure comprehensive care and improved quality of life for microstomia patients.
R3 Stem Cell Therapy: A New Hope for Women with Ovarian Failure
Discover the groundbreaking advancements in stem cell therapy by R3 Stem Cell, offering new hope for women with ovarian failure. This innovative treatment aims to restore ovarian function, improve fertility, and enhance overall well-being, revolutionizing reproductive health for women worldwide.
一比一原版(USF毕业证)旧金山大学毕业证如何办理
USF毕业证offer【微信95270640】《旧金山大学毕业证购买》《如何办理USF毕业证旧金山大学文凭学历》Q微信95270640实体公司，专业可靠，办理毕业证办美国成绩单,做加拿大文凭学历认证,办新西兰学位证，学位证书是什么?《制作旧金山大学毕业证多少钱》《USF学历证书丢了怎么办理》办澳洲文凭认证，办留信网认证（网上可查，实体公司，专业可靠）
专业为留学生办理旧金山大学旧金山大学硕士学位证成绩单【100%存档可查】留学全套申请材料办理。本公司承诺所有毕业证成绩单成品全部按照学校原版工艺对照一比一制作和学校一样的羊皮纸张保证您证书的质量！
如果你回国在学历认证方面有以下难题请联系我们我们将竭诚为你解决认证瓶颈
1所有材料真实但资料不全无法提供完全齐整的原件。【如：成绩单丶毕业证丶回国证明等材料中有遗失的。】
2获得真实的国外最终学历学位但国外本科学历就读经历存在问题或缺陷。【如：国外本科是教育部不承认的或者是联合办学项目教育部没有备案的或者外本科没有正常毕业的。】
3学分转移联合办学等情况复杂不知道怎么整理材料的。时间紧迫自己不清楚递交流程的。
如果你是以上情况之一请联系我们我们将在第一时间内给你免费咨询相关信息。我们将帮助你整理认证所需的各种材料.帮你解决国外学历认证难题。
国外旧金山大学旧金山大学硕士学位证成绩单办理方法：
1客户提供办理信息：姓名生日专业学位毕业时间等（如信息不确定可以咨询顾问：我们有专业老师帮你查询旧金山大学旧金山大学硕士学位证成绩单）；
2开始安排制作旧金山大学毕业证成绩单电子图；
3旧金山大学毕业证成绩单电子版做好以后发送给您确认；
4旧金山大学毕业证成绩单电子版您确认信息无误之后安排制作成品；
5旧金山大学成品做好拍照或者视频给您确认；
6快递给客户（国内顺丰国外DHLUPS等快读邮寄）。头贼脑的倒也逗人喜爱日上三竿时山娃总爱窜进自家瓜棚里跟小伙伴们坐着聊天聊着聊着便忍不住往瓜田里逡巡一番抱起一只硕大的西瓜用石刀劈开抑或用拳头砸开每人抱起一大块就啃啃得满嘴满脸猴屁股般的红艳大家一个劲地指着对方吃吃地笑瓜裂得古怪奇形怪状却丝毫不影响瓜味甜丝丝的满嘴生津遍地都是瓜横七竖八的活像掷满了一地的大石块摘走二三只爷爷是断然发现不了的即便发现爷爷也不恼反而教山娃辨认孰熟孰嫩孰甜孰淡名义上是护瓜吃
CHAPTER 1 SEMESTER V COMMUNICATION TECHNIQUES FOR CHILDREN.pdf
Here are some key objectives of communication with children:
Build Trust and Security:
Establish a safe and supportive environment where children feel comfortable expressing themselves.
Encourage Expression:
Enable children to articulate their thoughts, feelings, and experiences.
Promote Emotional Understanding:
Help children identify and understand their own emotions and the emotions of others.
Enhance Listening Skills:
Develop children’s ability to listen attentively and respond appropriately.
Foster Positive Relationships:
Strengthen the bond between children and caregivers, peers, and other adults.
Support Learning and Development:
Aid cognitive and language development through engaging and meaningful conversations.
Teach Social Skills:
Encourage polite, respectful, and empathetic interactions with others.
Resolve Conflicts:
Provide tools and guidance for children to handle disagreements constructively.
Encourage Independence:
Support children in making decisions and solving problems on their own.
Provide Reassurance and Comfort:
Offer comfort and understanding during times of distress or uncertainty.
Reinforce Positive Behavior:
Acknowledge and encourage positive actions and behaviors.
Guide and Educate:
Offer clear instructions and explanations to help children understand expectations and learn new concepts.
By focusing on these objectives, communication with children can be both effective and nurturing, supporting their overall growth and well-being.
一比一原版(UoA毕业证)昆士兰科技大学毕业证如何办理
UoA毕业证学历书【微信95270640】办文凭{昆士兰科技大学毕业证}Q微Q微信95270640UoA毕业证书成绩单/学历认证UoA Diploma未毕业、挂科怎么办？+QQ微信：Q微信95270640-大学Offer（申请大学）、成绩单（申请考研）、语言证书、在读证明、使馆公证、办真实留信网认证、真实大使馆认证、学历认证
办理国外昆士兰科技大学毕业证书 #成绩单改成绩 #教育部学历学位认证 #毕业证认证 #留服认证 #使馆认证（留学回国人员证明） #（证）等
真实教育部认证教育部存档中国教育部留学服务中心认证（即教育部留服认证）网站100%可查.
真实使馆认证（即留学人员回国证明）使馆存档可通过大使馆查询确认.
留信网认证国家专业人才认证中心颁发入库证书留信网永久存档可查.
昆士兰科技大学昆士兰科技大学毕业证文凭证书毕业证 #成绩单等全套材料从防伪到印刷从水印到钢印烫金跟学校原版100%相同.
国际留学归国服务中心：实体公司注册经营行业标杆精益求精！
国外毕业证学位证成绩单办理流程：
1客户提供办理昆士兰科技大学昆士兰科技大学毕业证文凭证书信息：姓名生日专业学位毕业时间等（如信息不确定可以咨询顾问：我们有专业老师帮你查询）；
2开始安排制作昆士兰科技大学毕业证成绩单电子图；
3昆士兰科技大学毕业证成绩单电子版做好以后发送给您确认；
4昆士兰科技大学毕业证成绩单电子版您确认信息无误之后安排制作成品；
5昆士兰科技大学成品做好拍照或者视频给您确认；
6快递给客户（国内顺丰国外DHLUPS等快递邮寄昆士兰科技大学昆士兰科技大学毕业证文凭证书）。闷不乐的样子父亲特意带山娃去找三楼房东家的儿子小伍玩小伍比山娃小一岁虎头虎脑的很霸气父亲让山娃跟小伍去夏令营听课山娃很高兴夏令营就设在附近一所小学山娃发现那所小学比自己的学校更大更美操场上还铺有塑胶跑道呢里面很多小朋友一班一班的快快乐乐原来城里娃都藏这儿来了怪不得平时见不到他们山娃恍然大悟起来吹拉弹唱琴棋书画山娃都不懂却什么都想学山娃怨自己太笨什么都不会斟酌再三山娃终于选定了学美术当听说每月要交感
Management of Post Operative Pain: to make doctors conscious about the benefi...
Multimodal analgesia.
Psychedelic Retreat Portugal - Escape to Lighthouse Retreats for an unforgett...
Our aim is to organise conscious gatherings and retreats for open and inquisitive minds and souls, with and without the assistance of sacred plants.
Michigan HealthTech Market Map 2024 with Policy Makers, Academic Innovation C...
Michigan HealthTech Market Map 2024. Includes 7 categories: Policy Makers, Academic Innovation Centers, Digital Health Providers, Healthcare Providers, Payers / Insurance, Device Companies, Life Science Companies, Innovation Accelerators. Developed by the Michigan-Israel Business Accelerator
Último (20)
The Importance of Black Women Understanding the Chemicals in Their Personal C...
The Importance of Black Women Understanding the Chemicals in Their Personal C...
nhs fpx 4000 assessment 4 analyzing a current health care problem or issue.pdf
nhs fpx 4000 assessment 4 analyzing a current health care problem or issue.pdf
Friendly Massage in Ajman - Malayali Kerala Spa Ajman
Friendly Massage in Ajman - Malayali Kerala Spa Ajman
VEDANTA AIR AMBULANCE SERVICES IN REWA AT A COST-EFFECTIVE PRICE.pdf
VEDANTA AIR AMBULANCE SERVICES IN REWA AT A COST-EFFECTIVE PRICE.pdf
U Part Wigs_ A Natural Look with Minimal Effort Jokerwigs.in.pdf
U Part Wigs_ A Natural Look with Minimal Effort Jokerwigs.in.pdf
R3 Stem Cell Therapy: A New Hope for Women with Ovarian Failure
R3 Stem Cell Therapy: A New Hope for Women with Ovarian Failure
CHAPTER 1 SEMESTER V COMMUNICATION TECHNIQUES FOR CHILDREN.pdf
CHAPTER 1 SEMESTER V COMMUNICATION TECHNIQUES FOR CHILDREN.pdf
Management of Post Operative Pain: to make doctors conscious about the benefi...
Management of Post Operative Pain: to make doctors conscious about the benefi...
Psychedelic Retreat Portugal - Escape to Lighthouse Retreats for an unforgett...
Psychedelic Retreat Portugal - Escape to Lighthouse Retreats for an unforgett...
Michigan HealthTech Market Map 2024 with Policy Makers, Academic Innovation C...
Michigan HealthTech Market Map 2024 with Policy Makers, Academic Innovation C...
PE AND TAMPONADE.pptx
- 4. EMBOLISM Detached intravascular solid / liquid / gaseous mass that is carried by blood to a distant site from point of origin. 20XX presentation title 4
- 5. Definition - Pulmonary embolism (PE) It is an embolism in which emboli occlude pulmonary arterial tree. 20XX presentation title 5
- 6. Pulmonary Thromboembolism Most fatal form of venous embolism in which there is occlusion of the pulmonary arterial tree. More than 95% of the cases – Venous emboli originate – DVT of LowerLimb. 20XX presentation title 6
- 7. PE They are carried through larger channels & usually pass through the right side of heart into the pulmonary vasculature. 20XX presentation title 7
- 8. Site of Origin of Emboli • Deep leg veins: DVTs are the source in more than 95% of cases of pulmonary emboli. • Deep leg veins include popliteal, femoral or iliac veins. • Other sites: Pelvic veins, vena cava. 20XX presentation title 8
- 10. Risk of pulmonary embolism Major risk factor is after surgery. The risk increases with:- advancing age, obesity, prolonged operative procedure, postoperative infection, cancer, and pre-existing venous disease. 20XX presentation title 10
- 11. Mechanism 20XX presentation title 11 DVTs undergo fragmentati on Thromboem boli - carried through larger vascular channels right side of the heart right ventricle pulmonary arterial vasculature
- 12. EFFECTS OF PE The effects of PE depend on: 1.Size of the emboli 2.Presence of Presence or absence of pulmonary congestion at the time of impaction. 20XX presentation title 12
- 13. Fate of Pulmonary Embolism Fate depends on the size of the embolus. 1. Resolution or organization: Small pulmonary emboli may travel into the smaller branches of pulmonary arteries and may resolve completely. 2. Massive pulmonary embolism: When emboli obstruct 60% or more of the pulmonary circulation, it is known as massive pulmonary embolism. 20XX presentation title 13
- 14. SADDLE EMBOLUS It is a large pulmonary embolus which lodges at the bifurcation of the main pulmonary artery. It produces acute massive obstruction of the blood flow to both lungs. 20XX presentation title 14
- 15. 20XX presentation title 15
- 16. CONSEQUENCES 1. Pulmonary infarction 2. Pulmonary hemorrhage 3. Pulmonary hypertension 20XX presentation title 16
- 17. CARDIAC TAMPONADE Cardiac tamponade is a medical emergency that takes place when abnormal amounts of fluid accumulate in the pericardial sac compressing the heart and leading to a decrease in cardiac output and shock. 20XX presentation title 17 Compression of the heart due to pericardial effusion
- 18. 20XX presentation title 18
- 19. 20XX presentation title 19
- 20. Causes of cardiac tamponade MYOCARDIAL INFARCTION CHEST TRAUMA PERICARDITIS KIDNEY FAILURE CANCER
- 22. areas of focus B2B MARKET SCENARIOS o Develop winning strategies to keep ahead of the competition o Capitalize on low-hanging fruit to identify a ballpark value o Visualize customer directed convergence CLOUD-BASED OPPORTUNITIES o Iterative approaches to corporate strategy o Establish a management framework from the inside 20XX presentation title 22
- 23. 20XX presentation title 23
- 25. 20XX presentation title 25
- 26. THANK YOU