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Dr Hamid Rahman Sabawoon 1
Prepared and presented by: Dr Hamid
Rahman Sabawoon, MD, LMIH,MN,
Supervised by: Dr Mirwais Jalal, MD,
Trainer of G.Surgery
‫افغانســــــــتان‬ ‫اســـــــالمی‬ ‫جمــــــــــهوری‬
‫عـــــــــــــــامه‬ ‫صحـــــت‬ ‫وزارت‬
‫تخــــصـــــص‬ ‫اکمــــــــــال‬ ‫ریاســــت‬
‫بلــــــخی‬ ‫رابعـــــــه‬ ‫کمپــــلکس‬ ‫شفـــــاخانه‬
‫عمــــــومی‬ ‫جــــــراحی‬ ‫دیپــــــارتمنـــت‬
Surgical Infection
A Public Health Approach
Surveillance
Risk Factor
Identification
Intervention
Evaluation
Implementation
4
Illness, Sickness, and
Disease
 illness’ to refer to the subjective
sense of feeling unwell;
 illness does not define a specific
pathology, but refers to a person’s
subjective experience of it, such as
discomfort, tiredness, or general
malaise.
 The way a patient reports symptoms is
influenced by his or her cultural
background,
 Sickness’ to refer to socially and
culturally held conceptions of health
conditions (e.g., the dread of cancer or
the stigma of mental illness), which in
turn influence how the patient reacts
 Disease’ implies a focus on
pathological processes that may or
may not produce symptoms and that
result in a patient’s illness.
Superficial soft tissue
suppurative infection
Surgical site infection
Definition: infection in surgical site.
Classification l:
Classification ll:
Major SSI Minor SSI
Significant quantity of pus Small purulent or serous discharge
Delayed return home No delayed return home
SIRS i.e. tachycardia, pyrexia, leukocytosis No SIRS
Superficial Skin and subcutaneous tissue
Deep Musculofacial layers
Organ/space infection e.g. Intra-abdominal
SSI
 SIRS(Systemic inflammatory Response
syndrome)
 It’s a body response to SSI
features
Hyperthermia >38’c
Hypothermia <36’c
Tachycardia >90/min
Bradycardia <20/min
WBC count >12000 or <4000
SSI
 Sepsis= SIRS + infection
 Severe Sepsis= Sepsis + with one or more
organ failure.
 MODS(Multiple Organ Dysfunction Syndrome)
 It’s the effectthat the infection produces
systemically
 MSOF(Multiple System Organ Failure)
 It’s the end-stage of uncontrolled MODS
Step-ladder of SSI leading to death
Surgical procedure
SSI
SIRS
Sepsis
Severe Sepsis
MODS
MSOF
Graveyard
Furuncle (boil)
 Is a staphylococcal infection of a hair follicle or a
sebaceous gland.
 Pathology: acute suppurative inflammation
 Furunculosis: infection of several hair
follicles in a circumscribed area.
 The common sites:
face, neck and axilla.
Boil (Furuncles)
Clinical Picture
A small painful indurated swelling which is
- red
- hot
- and very tender
Common in diabetics.
Boil (Furuncles)
Complications
 boil may lead to cellulitis, particularly in those whose
power of immunity is less.
 boils may also lead to infection of the neighboring
hair follicles where numbers of hair follicles are too
many (e.g. axilla) leading to hydradenitis.
 boils usually secondarily infect the regional lymph
nodes.
 Cavernous sinus thrombosis if boil is on the face and
infection spreads through veins to intracranial
cavernous sinuses.
Treatment
 1.Antibiotics.
 2.Antiseptic.
Treatment
 The general health of the patient has to be improved, as boils
often occur in individuals with debility and ill-health.
 Incision is usually unnecessary as the pustule is very small.
Only a touch of iodine on the skin pustule will hasten necrosis
of the overlying skin and help the pus to drain out.
 If escape of pus does not occur spontaneously or with
application of iodine, removal of the affected hair allows
ready escape of pus.
 Antibiotic is usually not required.
 After escape of pus this placet should be cleaned twice with a
suitable disinfectant e.g. hexachlorophene.
 Analgesics (painkillers)
Carbuncle
 Is infective gangrene
of the subcutaneous
tissues usually secondary
to infection by
Staphylococcus aureus.
 It is common in immunocompromised
patients as in diabetics.
 The common sites:
face, nape of the neck, and the back
Pathology
 Infection usually starts in a hair follicle
 Extends to the subcutaneous fat where other
hair follicles get the infection.
 Multiple areas of necrosis and thrombosis of
blood vessels occur.
 Patches of skin undergo sloughing and
separate from the underlying granulation
tissue
Carbuncle
Clinical Picture
 There is usually sever toxemia.
 Starts as a painful induration of the skin and
subcutaneous tissues.
 The skin is red.
 Swelling its central part becomes soft.
 Multiple areas of skin thin out and separate
forming multiple sinuses.
Complications
 Local spread of infection.
 Pyaemia and septicemia.
 Cavernous sinus thrombosis
 Epidural abscess or meningitis
Antrax Carbuncle
Treatment
 improvement of the general health of the patient
should be brought about.
 proper antibiotic should be started immediately
from the culture and sensitivity test.
Operation may be required:
1. when toxaemia and pain persist even after a course
of antibiotics.
2. when the carbuncle is more than 2 inches in
diameter. It must be remembered that incision is
never made unless there is softening in the centre.
Treatment
1.Antibiotics.
2.culture and sensitivity of the discharge.
3.control of diabetes.
4.surgical excision of sloughs.
Hydradenitis Suppurativa
Mixed staph. And streptococcal infection
of the apocrine sweat glands, in the
perineum or the axilla,produces multiple
abscesses and pus discharging sinuses.
Hiradenitis
Treatment
 Surgical drainage of abscesses.
 Antiseptic and antifungal applications.
 Surgical excision of the apocrine sweat-
bearing skin following by skin grafting is
essential.
Cellulitis
 Is an invasive non suppurative infection of
the loose connective tissue
 Organism :
 streptococci [common]
 staphylococci [occasionally]
 Mix
 Anaerobic cellulites: crepitation
Clinical picture
 The affected area is red,indurated,hot and
painful
 It spreads rapidly with ill defined edge
 The skin may be the seat of blisters
 Fever
 Lymphangitis in the form of red streaks
 No suppuration
 In severe cases patches of skin necrosis with
sloughing of subcutaneous tissues
Differential Diagnosis
 Contact allergy
 Chemical inflammation
 DVT 4 “Nos”
No edge No pus
No Fluctuation No limit
Treatment
I. Bandaging to support the area
II. Rest and elevation of the affected part
III. Antibiotic (penicillin iv )
IV. Analgesics (painkillers)
Erysipelas
Skin wound local inflammation
lymphadenitis systemic inflammation
Redness of skin with clear boundary
Edema of proximal lymph node
Systemic sepsis
Clinical Picture
 Toxemia
 Locally : similar to cellulitis, but there
are the following differences:
1. The color of the skin is rose-pink
2. The edge is well defined
3. There may islets of inflammation beyond the
spreading margin
Complications
 1. Facial erysipelas may lead to cavernous
sinus thrombosis
 2. Septicemia
 3. Recurrent erysipelas may block the
lymphatics leading to elephantiasis.
Erysipelas
Treatment
 Isolation
 Similar to cellulitis
Erysipelas
Abscess
 Characterized by a necrotic center without a blood
supply and composed of debris from local tissues,
dead and dying leukocytes, components of blood
and plasma and bacteria.
 Avenues
 Direct spread :micro-organism get entry through
some breach in the surface epithelium.
 Lymphatic spread :from some distant focus
 Hematogenous spread:from some distant focus.
Abscess
 An abscess is a cavity
filled with pus and lined
by a pyogenic membrane.
 This pyogenic membrane
consists of dead tissue
cells and a wall of
granulation tissue
consisting for the most
part of phagocytic
histiocytes.
Abscess
 Types
1. Pyogenic abscess
2. Pyaemic abscess
3. Cold abscess
Abscess
Pyogenic abscess:
Pain: throbbing in character
Fever: swinging rise of body temperature with or
without shivering.
Area is red, hot, smooth, soft and tender
Brawny induration and oedema(pitting on pressure)
Flactuation is present
Regional lymph nodes maybe enlarged and tender
Abscess
Pyaemic abscess:
 Generally multiple
 Maybe synchronous(occurring at the same time) or
metachronous(in succation one after the other)
 Pyeamia is present(infective emboli in the blood)
 Non-reacting in nature.ie local features are absent
 Flactuation will be present
 Constitutional disturbances are great e.g pyrexi,
rigors,tachycardia and sweating.
Abscess
 Cold abscess:
 Its always a sequel of tuberculosis infection(caseation
necrosis) commonly seen in lymph node, joints and bone
 Non-reacting in nature
 Fluctuation is present
 Common site are:
 Neck and axilla-tuberculos lymphadenitis
 Right/left iliac fossa-tuberculosis of spine
 Bones- tuberculous osteomyelitis
 Joints- tuberculous arthritis
 Lion- tuberculous pyonephrosis
Abscess
Treatment
The basic principle of treatment of an abscess are:
 to drain the pus;
 to send a sample of pus for culture and
sensitivity test;
 Surgical decompression by incision/drainage,
curettage or finger breaking of loculi followed
by daily antiseptic dressing
 to give proper antibiotic.
 To give proper Analgesic for pain control
Acute Lymphangitis and
Lyphadenitis
 Acute lymphangitis:is due to infection of
lymph vessels by organisms usually
streptococci.
 Acute lymphadenitis: is due to spread of
infection along lymphatics from a septic
focus in the drainage area to the lymph-
nodes.
Lymphangitis
 A spread of infection along the lymphatic
system is manifested in a disease of the
lymphatic vessels and lymph nodes.
Inflammation of the lymphatic vessels
(lymphangitis) is one of the frequent
complications of infected wounds, especially
during the first weeks following injury, and
of local purulent diseases.
Lymphangitis
Treatment
 Antibiotics.
 Hot applications.
 Surgical drainage if suppuration
occurs.
Treatment
 The treatment of lymphangitis consists
primarily in elimination of its cause
(incision of the abscess, pockets of the
wound, etc.) and in giving the affected
organ complete rest.
Paraproctitis
 is the purulent inflammation of around-
rectal cellular tissue.
Paraproctitis
Perirectal abscess
Treatment
 In the stage of infiltration conservative
therapy usually used (antibiotics,
sparing thet). At the phlegmon or
abscess there is indicated urgent
operation.
Mastitis
 Mastitis
 is inflammation of lactic
gland tissue. There is
distinguished lactation
mastitis at nursing
mothers, mastitis of
newborns and in period
of pubescence.
Acute suppurative
infection
of the hand
paronychia
lateral nail fold trauma redness, pain
suppurative infection
Treatment:
 incision and drainage
 removal of the nail: infection extend
deep to the nail
 antibiotics
felon
paronychia spread or
penetration wound
causes: pain fever
WBC elevation
Treatment:
 incision and drainage
 antibiotics
Gas gangrene

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Surgical infections

  • 1. Dr Hamid Rahman Sabawoon 1
  • 2. Prepared and presented by: Dr Hamid Rahman Sabawoon, MD, LMIH,MN, Supervised by: Dr Mirwais Jalal, MD, Trainer of G.Surgery ‫افغانســــــــتان‬ ‫اســـــــالمی‬ ‫جمــــــــــهوری‬ ‫عـــــــــــــــامه‬ ‫صحـــــت‬ ‫وزارت‬ ‫تخــــصـــــص‬ ‫اکمــــــــــال‬ ‫ریاســــت‬ ‫بلــــــخی‬ ‫رابعـــــــه‬ ‫کمپــــلکس‬ ‫شفـــــاخانه‬ ‫عمــــــومی‬ ‫جــــــراحی‬ ‫دیپــــــارتمنـــت‬
  • 4. A Public Health Approach Surveillance Risk Factor Identification Intervention Evaluation Implementation 4
  • 5. Illness, Sickness, and Disease  illness’ to refer to the subjective sense of feeling unwell;  illness does not define a specific pathology, but refers to a person’s subjective experience of it, such as discomfort, tiredness, or general malaise.  The way a patient reports symptoms is influenced by his or her cultural background,
  • 6.  Sickness’ to refer to socially and culturally held conceptions of health conditions (e.g., the dread of cancer or the stigma of mental illness), which in turn influence how the patient reacts  Disease’ implies a focus on pathological processes that may or may not produce symptoms and that result in a patient’s illness.
  • 8. Surgical site infection Definition: infection in surgical site. Classification l: Classification ll: Major SSI Minor SSI Significant quantity of pus Small purulent or serous discharge Delayed return home No delayed return home SIRS i.e. tachycardia, pyrexia, leukocytosis No SIRS Superficial Skin and subcutaneous tissue Deep Musculofacial layers Organ/space infection e.g. Intra-abdominal
  • 9. SSI  SIRS(Systemic inflammatory Response syndrome)  It’s a body response to SSI features Hyperthermia >38’c Hypothermia <36’c Tachycardia >90/min Bradycardia <20/min WBC count >12000 or <4000
  • 10. SSI  Sepsis= SIRS + infection  Severe Sepsis= Sepsis + with one or more organ failure.  MODS(Multiple Organ Dysfunction Syndrome)  It’s the effectthat the infection produces systemically  MSOF(Multiple System Organ Failure)  It’s the end-stage of uncontrolled MODS
  • 11. Step-ladder of SSI leading to death Surgical procedure SSI SIRS Sepsis Severe Sepsis MODS MSOF Graveyard
  • 12. Furuncle (boil)  Is a staphylococcal infection of a hair follicle or a sebaceous gland.  Pathology: acute suppurative inflammation  Furunculosis: infection of several hair follicles in a circumscribed area.  The common sites: face, neck and axilla.
  • 14. Clinical Picture A small painful indurated swelling which is - red - hot - and very tender Common in diabetics.
  • 16. Complications  boil may lead to cellulitis, particularly in those whose power of immunity is less.  boils may also lead to infection of the neighboring hair follicles where numbers of hair follicles are too many (e.g. axilla) leading to hydradenitis.  boils usually secondarily infect the regional lymph nodes.  Cavernous sinus thrombosis if boil is on the face and infection spreads through veins to intracranial cavernous sinuses.
  • 18. Treatment  The general health of the patient has to be improved, as boils often occur in individuals with debility and ill-health.  Incision is usually unnecessary as the pustule is very small. Only a touch of iodine on the skin pustule will hasten necrosis of the overlying skin and help the pus to drain out.  If escape of pus does not occur spontaneously or with application of iodine, removal of the affected hair allows ready escape of pus.  Antibiotic is usually not required.  After escape of pus this placet should be cleaned twice with a suitable disinfectant e.g. hexachlorophene.  Analgesics (painkillers)
  • 19.
  • 20. Carbuncle  Is infective gangrene of the subcutaneous tissues usually secondary to infection by Staphylococcus aureus.  It is common in immunocompromised patients as in diabetics.  The common sites: face, nape of the neck, and the back
  • 21. Pathology  Infection usually starts in a hair follicle  Extends to the subcutaneous fat where other hair follicles get the infection.  Multiple areas of necrosis and thrombosis of blood vessels occur.  Patches of skin undergo sloughing and separate from the underlying granulation tissue
  • 23. Clinical Picture  There is usually sever toxemia.  Starts as a painful induration of the skin and subcutaneous tissues.  The skin is red.  Swelling its central part becomes soft.  Multiple areas of skin thin out and separate forming multiple sinuses.
  • 24. Complications  Local spread of infection.  Pyaemia and septicemia.  Cavernous sinus thrombosis  Epidural abscess or meningitis
  • 26. Treatment  improvement of the general health of the patient should be brought about.  proper antibiotic should be started immediately from the culture and sensitivity test. Operation may be required: 1. when toxaemia and pain persist even after a course of antibiotics. 2. when the carbuncle is more than 2 inches in diameter. It must be remembered that incision is never made unless there is softening in the centre.
  • 27. Treatment 1.Antibiotics. 2.culture and sensitivity of the discharge. 3.control of diabetes. 4.surgical excision of sloughs.
  • 28.
  • 29. Hydradenitis Suppurativa Mixed staph. And streptococcal infection of the apocrine sweat glands, in the perineum or the axilla,produces multiple abscesses and pus discharging sinuses.
  • 31. Treatment  Surgical drainage of abscesses.  Antiseptic and antifungal applications.  Surgical excision of the apocrine sweat- bearing skin following by skin grafting is essential.
  • 32. Cellulitis  Is an invasive non suppurative infection of the loose connective tissue  Organism :  streptococci [common]  staphylococci [occasionally]  Mix  Anaerobic cellulites: crepitation
  • 33. Clinical picture  The affected area is red,indurated,hot and painful  It spreads rapidly with ill defined edge  The skin may be the seat of blisters  Fever  Lymphangitis in the form of red streaks  No suppuration  In severe cases patches of skin necrosis with sloughing of subcutaneous tissues
  • 34.
  • 35. Differential Diagnosis  Contact allergy  Chemical inflammation  DVT 4 “Nos” No edge No pus No Fluctuation No limit
  • 36. Treatment I. Bandaging to support the area II. Rest and elevation of the affected part III. Antibiotic (penicillin iv ) IV. Analgesics (painkillers)
  • 37. Erysipelas Skin wound local inflammation lymphadenitis systemic inflammation Redness of skin with clear boundary Edema of proximal lymph node Systemic sepsis
  • 38.
  • 39. Clinical Picture  Toxemia  Locally : similar to cellulitis, but there are the following differences: 1. The color of the skin is rose-pink 2. The edge is well defined 3. There may islets of inflammation beyond the spreading margin
  • 40. Complications  1. Facial erysipelas may lead to cavernous sinus thrombosis  2. Septicemia  3. Recurrent erysipelas may block the lymphatics leading to elephantiasis.
  • 44. Abscess  Characterized by a necrotic center without a blood supply and composed of debris from local tissues, dead and dying leukocytes, components of blood and plasma and bacteria.  Avenues  Direct spread :micro-organism get entry through some breach in the surface epithelium.  Lymphatic spread :from some distant focus  Hematogenous spread:from some distant focus.
  • 45.
  • 46. Abscess  An abscess is a cavity filled with pus and lined by a pyogenic membrane.  This pyogenic membrane consists of dead tissue cells and a wall of granulation tissue consisting for the most part of phagocytic histiocytes.
  • 47. Abscess  Types 1. Pyogenic abscess 2. Pyaemic abscess 3. Cold abscess
  • 48. Abscess Pyogenic abscess: Pain: throbbing in character Fever: swinging rise of body temperature with or without shivering. Area is red, hot, smooth, soft and tender Brawny induration and oedema(pitting on pressure) Flactuation is present Regional lymph nodes maybe enlarged and tender
  • 49. Abscess Pyaemic abscess:  Generally multiple  Maybe synchronous(occurring at the same time) or metachronous(in succation one after the other)  Pyeamia is present(infective emboli in the blood)  Non-reacting in nature.ie local features are absent  Flactuation will be present  Constitutional disturbances are great e.g pyrexi, rigors,tachycardia and sweating.
  • 50. Abscess  Cold abscess:  Its always a sequel of tuberculosis infection(caseation necrosis) commonly seen in lymph node, joints and bone  Non-reacting in nature  Fluctuation is present  Common site are:  Neck and axilla-tuberculos lymphadenitis  Right/left iliac fossa-tuberculosis of spine  Bones- tuberculous osteomyelitis  Joints- tuberculous arthritis  Lion- tuberculous pyonephrosis
  • 52. Treatment The basic principle of treatment of an abscess are:  to drain the pus;  to send a sample of pus for culture and sensitivity test;  Surgical decompression by incision/drainage, curettage or finger breaking of loculi followed by daily antiseptic dressing  to give proper antibiotic.  To give proper Analgesic for pain control
  • 53. Acute Lymphangitis and Lyphadenitis  Acute lymphangitis:is due to infection of lymph vessels by organisms usually streptococci.  Acute lymphadenitis: is due to spread of infection along lymphatics from a septic focus in the drainage area to the lymph- nodes.
  • 54. Lymphangitis  A spread of infection along the lymphatic system is manifested in a disease of the lymphatic vessels and lymph nodes. Inflammation of the lymphatic vessels (lymphangitis) is one of the frequent complications of infected wounds, especially during the first weeks following injury, and of local purulent diseases.
  • 56. Treatment  Antibiotics.  Hot applications.  Surgical drainage if suppuration occurs.
  • 57. Treatment  The treatment of lymphangitis consists primarily in elimination of its cause (incision of the abscess, pockets of the wound, etc.) and in giving the affected organ complete rest.
  • 58. Paraproctitis  is the purulent inflammation of around- rectal cellular tissue.
  • 61. Treatment  In the stage of infiltration conservative therapy usually used (antibiotics, sparing thet). At the phlegmon or abscess there is indicated urgent operation.
  • 62. Mastitis  Mastitis  is inflammation of lactic gland tissue. There is distinguished lactation mastitis at nursing mothers, mastitis of newborns and in period of pubescence.
  • 64. paronychia lateral nail fold trauma redness, pain suppurative infection Treatment:  incision and drainage  removal of the nail: infection extend deep to the nail  antibiotics
  • 65.
  • 66. felon paronychia spread or penetration wound causes: pain fever WBC elevation Treatment:  incision and drainage  antibiotics
  • 67.