Premium Bangalore Call Girls Jigani Dail 6378878445 Escort Service For Hot Ma...
Cardiology 2.1. ECG or EKG - by Dr. Farjad Ikram
1.
2.
3. OBJECTIVES
What is an ECG and why it is used?
What is the normal calibration of ECG?
What is the conductive system of the heart?
Where the electrodes are placed?
What are the polarities of the 12 leads?
What are parts of an ECG complex?
How to estimate heart rate from ECG?
What are features of normal sinus rhythm?
What is axis of heart and axis deviation?
What is normal P-QRS-T morphology in different leads?
What are changes seen in limb leads reversal?
4.
5. WHAT IS ECG?
It is the graphical recording of electrical conduction in
the heart over a period of time (usually 10 sec), using
electrodes placed over the skin.
In conventional 12 lead ECG, a total 10 electrodes are
placed: 6 over the chest and one on each limb.
The overall magnitude of electrical activity of heart is
measured from 12 different angles or “leads”.
The graph of voltage versus time produced is called
Electrocardiogram.
6. USES OF ECG?
It is used to detect:
Rate and rhythm disorders
Conduction problems
Myocardial ischemia
Myocardial infarcts
Chamber dilation
Chamber hypertrophy
Inflammation i.e. pericarditis
Electrolyte disturbances
Drug toxicity
Other: pulmonary embolism
7.
8. SPEED CALIBRATION
X-axis time
– 1 small square
• 40 ms (0.04 sec)
– 1 large square
• 200 ms (0.2 sec)
Speed: 25 mm/sec
9. VOLTAGE CALIBRATION
Y axis voltage
– 1 large square
0.5 mV
– 2 large squares
1.0 mV
– Therefore, 1 mV
produces deviation of 2
large squares (10 mm)
10.
11. CHAMBERS OF HEART
Heart has two electrically functional units: Atria and Ventricles.
Electrically divided by the properties of anulous fibrosus cordis.
15. AXIS OF THE HEART
The electrical axis of the heart is the mean direction
of the action potentials traveling through the
conductive system of the heart.
16. WAVEFORMS
Contraction of any muscle is associated with
electrical changes called depolarization.
This is followed by its relaxation, which is
associated with the reversal of these changes
called repolarization.
These electrical changes will produce
waveforms on the ECG. i.e. P-QRS-T waves.
No electrical changes will produce an flat
isoelectric baseline on the ECG.
17. ECG COMPLEX
Waveforms
– P wave
– QRS complex
– T wave
– U wave
Segments
– PR segment
– ST segment
Intervals
– PR interval
– QT interval
– RR interval
19. ELECTROMAGNETICS
1. depolarization toward the
positive electrode produces a
positive deflection
2. depolarization away from the
positive electrode produces a
negative deflection
3. repolarization toward the
positive electrode produces a
negative deflection
4. repolarization away from the
positive electrode produces a
positive deflection
20. ELECTROMAGNETICS
If direction of conduction is at right angle to the
positive electrode it will produce positive
deflection (depolarization) and then negative
deflection (repolarization).
24. CHEST ELECTRODES
Chest Electrode Placement
V1 4th ICS, right of sternum
V2 4th ICS, left of sternum
V3 Between V2 and V4
V4 5th ICS, in left mid
clavicular line
V5 Same height as V4, in
left anterior axillary line
V6 Same height as V4, in
left mid axillary line
25. LEADS
3 limb leads
– I - (+LA and -RA)
– II - (+LL and -RA) also called ‘sinus lead’
– III - (+LL and -LA)
3 augmented limb leads:
– aVR - (+RA and average of LA & LL)
– aVL - (+LA and average of RA & LL)
– aVF - (+LL and average of RA & LA)
6 Chest leads:
– V1, V2, V3, V4, V5, V6 (have +ve electrodes of same name)
– Negative electrode is WCT (Wilson’s central terminus), an
average of the three limb electrodes (RA, LA, LL)
Other Chest leads:
• Posterior chest leads: V7, V8, V9
• Right chest leads: V3R, V4R, V5R
34. REPORTING AN ECG
Estimated heart rate
Comment on the rhythm
Comment on the axis
Comment on:
– P wave morphology
– PR segment
– QRS morphology
– ST segment
– T wave morphology
– QT interval
Compare with a previous ECG
Conclusion
36. ESTIMATING HEART RATE
Alt. method
– count number of small boxes between two
consecutive R waves
– divide 1500 by that number to est. HR
3 second method
– count number of QRS complexes that fit into 3
seconds (15 large squares)
– multiply this number with 20 to est. HR
– preferred method in irregularly irregular rhythms
37. HEART RATE INTERPRETATION
HR of 60-100 beats/min Normal range
HR > 100 beats/min Tachycardia
Physiologic i.e. exercise
Inappropriate i.e. fever, anxiety, tachyarrhythmia
HR < 60 beats/min Bradycardia
Physiologic i.e. athletes at rest
Inappropriate i.e. heart blocks, vaso-vagal reflex
Paediatric values
New born i.e. 110 - 150 b/m
2 years i.e. 85 - 125 b/m
4 years i.e. 75 - 115 b/m
6 years + i.e. 60 - 100 b/m
38. NORMAL SINUS RHYTHM
Normal heart rate
Regular rhythm
P waves should be sinus
P wave is round and upward in lead I & II
Each QRS is preceded by a P wave
The PR interval should remain constant
QRS complexes should be narrow
39. AXIS DEVIATION (THUMB RULE)
QRS IN
LEAD I
QRS IN
LEAD AVF
AXIS
DEVIATION
SEE IN
POSITIVE POSITIVE NORMAL
POSITIVE NEGATIVE LEFT AXIS
DEVIATION
• Elevated diaphragm
(ascites, pregnancy)
• IWMI, hyperkalemia
• LVH alone, LVH with LBBB
• Occassionally LBBB alone
NEGATIVE POSITIVE RIGHT AXIS
DEVIATION
• Young, thin people
• LWMI
• RVH alone, RVH with RBBB
• Occassionally LBBB alone
NEGATIVE NEGATIVE NORTH WEST
AXIS
• Severe RVH
• Severe hyperkalemia
41. P WAVE MORPHOLOGY
sinus P wave is round and upward in lead I / II
always inverted in aVR
can be biphasic or inverted in lead V1
maximal height - 2.5 mm in lead II / III
duration is shorter than 0.12 sec (3 small sq)
42. PR SEGMENT & PR INTERVAL
PR segment
– an iso-electric line, due to conduction delay to AV node
– from end of P wave to start of QRS complex
– diffuse PR segment depression in acute pericarditis
PR interval
– includes P wave + PR segment
– from start of P wave to start of QRS
– normally 0.12 to 0.2 sec (3-5 small sq)
– short PR interval are seen in pre-excitation:
• MAT, WPW, junctional rhythms
– prolonged PR interval are seen in:
• 1st and 2nd degree AV block
• hypokalemia, digitalis toxicity, carditis
43. DELTA WAVE
In WPW syndrome, short PR interval manifests as a
“delta wave”, a slurred upstroke in the QRS complex
44. QRS COMPLEX
Normal duration is < 110 ms or < 3 small squares
– Q wave – 1st downward deflection after P wave
• seen in I, aVL, V5 and V6; usually absent in leads V1-V2
– R wave – 1st upward deflection
• short in V1-V2, long in V5-V6
– S wave – 2nd downward deflection
• long in V1-V2, short in V5-V6
R wave progression
from V1 to V6, R wave height but S wave depth
Q
S
R
TZ Normal Transition Zone is
at V3-V4, when S wave
equals R wave.
Represents apex of heart.
45. QRS ABNORMALITIES
Broad QRS complex (>120 ms)
– LBBB, RBBB, hyperkalemia, VT etc
Increased QRS height
– LVH, RVH
Poor R wave progression
– AWMI, LVH, LBBB, WPW
Dominant R wave in V1
– PWMI, RVH, RBBB, WPW, children
Pathologic Q waves
– Markers of previous MI
– Q wave width > 1 small sq. + Q wave depth > 2 small sq.
– Or, Q wave is ≥ 25% of the R wave
Pathologic Q
46. VENTRICULAR HYPERTROPHY
Sokolow Lyon index for LVH:
• S wave depth in V1/V2 + R wave height in V5/V6 ≥ 35 mm
• R wave height in aVL ≥ 11 mm
Criteria for RVH:
• dominant R in V1 + dominant S waves in V5/V6
• deep S waves in leads I, II, III, aVL, V5, V6
47. ST SEGMENT
Usually iso-electric (flat)
Measured from J point to beginning of T wave
J point is the junction between QRS and ST segment
ST elevation in STEMI (convex or obliquely straight upwards)
Diffuse ST elevation with PR depression in acute pericarditis
ST depression in unstable angina / NSTEMI
Sagging ST depression in digoxin effect and hypokalemia
ST segment
J point
P T “sagging” ST
depression
48. ST SEGMENT ELEVATION
“saddleback”
pattern of diffuse
ST elevation
& PR depression
in Pericarditis
obliquely straight
ST elevation in
STEMI
upward convex
ST elevation in
STEMI
notched J point
“fish-hook” pat. of
ST elevation seen
in BER
49. T WAVE AND U WAVE
Upwards in most leads
Can be inverted in V1, but always inverted in aVR
Should be less than 2/3rd the height of R wave
Abnormalities of T wave
Tall narrow T waves in hyperkalemia
Tall broad T waves in acute STEMI (hyperacute)
Generalized flat T waves in hypokalemia
Flattened or inverted T waves – early sign of ischemia
Deep T waves in chest leads – Wellen’s sign
Diffuse deep “cerebral” T waves – raised ICP
U wave
Late repolarization of ventricles
Usually seen in V6 after T wave
Prominent in hypokalemia
50. QT INTERVAL
From beginning of QRS to the ending of T wave
Duration 0.35 - 0.45 sec ( 9 -12 small sq. )
QT duration is inversely proportional to Heart Rate
QTc also with HR which can be corrected using Bazzet
formula:
Short QTc (< 340 ms)
• Hypercalcemia
• Digoxin effect
Long QTc (> 460 ms)
• Hypothermia
• Hypokalemia
• Hypomagnesiumia
• Hypocalcemia
• Drugs - amiodarone, quinidine, TCAs, erythromycin
51. Chamberline’s 10 RULES
1. PR interval should be 0.12-0.2 sec (3-5 small sq.)
2. With of QRS should not exceed 0.11 sec (3 small sq.)
3. QRS should be dominantly upwards in lead I and II
4. QRS and T waves have same direction in limb leads
5. All waves are negative in lead aVR
6. R wave must grow from V1 to V4 while S wave must
grow from V1 to at least V3 and disappear in V6
7. ST segment should start iso-electric except in V1 and
V2 where it may be elevated
8. P waves should be upright in I, II, and V2 to V6
9. No pathologic Q wave in I, II, and V2 to V6
10. T wave must be upright in I, II, and V2 to V6
52.
53. LA / RA REVERSAL
Lead I becomes inverted.
Leads II and III switch places.
Leads aVL and aVR switch places.
Lead aVF remains unchanged.
What will you see?
• Lead I is completely inverted.
• Lead aVR often becomes positive.
• There may be marked right axis
deviation.
54. LA / LL REVERSAL
Lead III becomes inverted.
Leads I and II switch places.
Leads aVL and aVF switch places.
Lead aVR remains unchanged.
What will you see?
• Lead III is completely inverted.
• P wave is unexpectedly larger in
lead I than in lead II.
55. RA / LL REVERSAL
Lead II becomes inverted.
Leads I and III become inverted
and switch places.
Leads aVR and aVF switch places.
Lead aVL remains unchanged.
What will you see?
• Lead I, II, III and aVF all are
completely inverted.
• Lead aVR is upright.
56. RA / RL (N) REVERSAL
Leads I and aVL become inverted.
Lead II will be flat.
Lead III is unchanged.
Lead aVR and aVL become
identical.
What will you see?
• Lead II is a flat line.
57. LA / RL (N) REVERSAL
Lead I becomes identical to lead II.
Lead II is unchanged.
Lead III is flat.
Lead aVR is an inverted lead II.
Lead aVL and aVF become identical.
What will you see?
• Lead III is a flat line.
58. LL / RL (N) REVERSAL
Einthoven’s triangle is preserved.
What will you see?
• ECG is unchanged.
59. LA with LL / RA with RL
Bilateral arm-leg electrode reversal.
Lead I is flat.
Lead II is an inverted lead III.
Lead III is inverted.
aVR and aVL become identical.
aVF looks like negative lead III.
What will you see?
• Lead I is a flat line.
60. QUICK SPOTTING
OF LEAD REVERSAL
Lead I is flat or completely inverted.
Lead II is flat or completely inverted.
Lead III is flat or completely inverted.
Lead aVR is positive.
P wave is larger in lead I than in lead II.
61. DEXTROCARDIA
What will you see?
Right axis deviation.
Complete inversion of lead I.
All waves in aVR are positive.
Absent R wave progression in chest leads
- S wave is dominant throughout