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Pressure
     ulcers

Dr. Doha Rasheedy
   Definition
   Risk factors
   Diagnosis, staging
   prevention
   Treatment
A 75yrs elderly male pt, with PH of DM,
 recurrent CVS, bed ridden of 2 yrs duration
 and has urinary incontince. On exam. There
 was an ulcer about 2×3×2 cm with necrotic
 non viable base , painless, on the sacrum.
What is the most likely diagnosis ?
Venous Ulcer
Diabetic ulcer
Pressure ulcer
 A localized  area of soft-tissue injury
  resulting from compression between
  a bony prominence and an external
  surface.
 It a type of a vascular necrosis
RISK
      FACTORS


Intrinsic   Extrinsic
Clinical picture
and Stages
The Norton Scale•

Patients at risk of





developing Pus can be
identified clinically by:
Norton Scale
It detect the physical
and mental condition,
the activity, mobility and
incontinent
A score 10-12------High
risk of PUS development
   Unstageable: Full thickness tissue loss
    in which slough (yellow, tan, gray,
    green or brown), eschar (tan, brown
    or black), or both in the wound bed
    cover the base of the ulcer.




         Pictures - Royal College of Surgeons of Edinburgh
Sites
   Any skin exposed to continuous
    pressure.

    Internal viscera exposed to
    unusual pressure, as trachea
    pressed by balloon of endotracheal
    tube
   Over Bony Prominences
    1. Occiput
    2. Ears
    3. Scapula              Internal
    4. Spinous Processes    organs
    5. Shoulder
    6. Elbow
    7. Iliac Crest
    8. Sacrum/Coccyx
    9. Ischial Tuberosity
    10.Trochanter
    11.Knee
    12.Malleolus
    13.Heel
    14.Toes
   Any skin surface
    subject to excess
    pressure
   Examples include
    skin surfaces under:
    ◦ Oxygen tubing
    ◦ Urinary catheter
      drainage tubing
    ◦ Casts
    ◦ Cervical collars
Differential diagnosis
Not all
      Pressure ulcer to heel
                                      ulcers are   Neuropathic diabetic foot ulcer
                                      pressure
                                        ulcers




Arterial ulcer on toes and forefoot                       Venous leg ulcer
                                                                                     19
Prevention
and
treatment
focuses on:

                  Skin care

              Mechanical loading

               Support surfaces
   Daily systematic skin inspection and cleansing

    factors that promote dryness
   Avoid massaging over bony prominences
    moisture (incontinence, perspiration, drainage)
It requires gentle washing and drying
   Minimize friction and shear
   Reposition at least every 2 hours (may use pillows,
    foam wedges)
   Keep head of bed at lowest elevation possible
   Use lifting devices to decrease friction and shear
   Remind patients in chairs to shift weight every 15
    min
      “Doughnut” seat cushions are contraindicated,
                may cause pressure ulcers
   Pay special attention to heels (heel ulcers account for
    20% of all pressure ulcers)
**Use for all older persons at risk for ulcers**
   Static
     Foam, static air, gel, water, combination (less expensive)

   Dynamic
     Alternating air, low-air-loss, or air-fluidized
Heal protector

                                  Air mattress; Alternate
                                  pressure / low air loss / air
                                  fluidized

                                  Other media; gel / water/
                                 foam
http://www.diamond-medical.com/images/database/medlinesupracxc.jpg
GENERAL
                 ASSESSMENT



             Risk factor elimination



             ULCER ASSESSMENT,
             MONITORING HEALING



MANAGEMENT       Cleaning
              Debridement


               Dressings
               SURGICAL
                REPAIR
   Health problems (e. g, urinary
    incontinence)
   Nutritional status
   Pain level
  ULCER            MONITORING
   ASSESSMENT:       HEALING
Location           Document all
 Stage
                     observations over
                     time
 Area
 Depth             Describe each ulcer
Drainage            to track progress of
 Necrosis           healing
 Granulation       Use validated tools
                     (eg, PUSH)
 Cellulitis
   Cleaning Avoid topical antiseptics because of their tissue toxicity

  Debridement
Is necessary to remove dead tissue it include
1.   Autolytic debridement using hydrocolloid or foam dressings
2.   Enzymatic debridement using exogenous collagenase
     (IRUXOL)
3.   Mechanical debridement
4.   Surgical, sharp Scalpel, scissor to remove dead tissue; laser
     debridement
5.   Bio surgery: Larvae to digest dead tissue
 Dressings
By wet to dry saline or hydrocolloid (duo-derm), or
  polyurethane, in exudative wounds fill the wound by
  aligniates or hydro gel.
 SURGICAL REPAIR
May be used for stage III and IV ulcers
    Direct closure, skin grafting, skin flaps,
    musculocutaneous flaps, free flaps
Complications
   Sepsis (aerobic or anaerobic bacteremia)
   Localized infection, cellulitis, osteomyelitis
   Pain
   Depression

    Mortality rate = 60% in older persons who
    develop a pressure ulcer within 1 year of
    hospital discharge
The mainstay in
  pressure ulcer
   treatment is
prevention of risk
      factors.
   Older adults are at high risk for development of
    pressure ulcers
   Pressure ulcers may result in serious morbidity
    and mortality
   Techniques that reduce pressure, moisture,
    friction, and shear can prevent pressure ulcers
   Pressure ulcers should be treated with proper
    cleansing, dressings, debridement, or surgery as
    indicated
a)Pressure ulcer = decubitus ulcer= bed sores.

b)Stage 1 PU is partial skin thickness loss.

c)One important risk factor for PU development
  is moisture.

d)One of the complications of PU is cellulitis.

e)A cornerstone in management of PU is
  debridement
  First line treatment for pressure ulcer
1. Surgical closure
2. Debridement
3. Pressure relief
4. Dressing
 Stage 3 pressure ulcer is:
1. Persistent non-blanchable erythema of intact
   skin
2. Full-thickness skin loss involving necrosis of
   subcutaneous tissue that may extend down
   to, but not through, underlying fascia.
3. Partial-thickness skin loss
4. Full-thickness skin loss with damage to
   muscle, bone, or supporting structures (e.g.
   tendon, joint capsule).
  First line treatment for pressure ulcer
1. Surgical closure
2. Debridement
3. Pressure relief
4. Dressing
 Stage 3 pressure ulcer is:
1. Persistent non-blanchable erythema of intact
   skin
2. Full-thickness skin loss involving necrosis of
   subcutaneous tissue that may extend down
   to, but not through, underlying fascia.
3. Partial-thickness skin loss
4. Full-thickness skin loss with damage to
   muscle, bone, or supporting structures (e.g.
   tendon, joint capsule).
Pressure ulcers presentation

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Pressure ulcers presentation

  • 1. Pressure ulcers Dr. Doha Rasheedy
  • 2.
  • 3. Definition  Risk factors  Diagnosis, staging  prevention  Treatment
  • 4. A 75yrs elderly male pt, with PH of DM, recurrent CVS, bed ridden of 2 yrs duration and has urinary incontince. On exam. There was an ulcer about 2×3×2 cm with necrotic non viable base , painless, on the sacrum. What is the most likely diagnosis ? Venous Ulcer Diabetic ulcer Pressure ulcer
  • 5.  A localized area of soft-tissue injury resulting from compression between a bony prominence and an external surface.  It a type of a vascular necrosis
  • 6.
  • 7. RISK FACTORS Intrinsic Extrinsic
  • 8.
  • 10. The Norton Scale• Patients at risk of  developing Pus can be identified clinically by: Norton Scale It detect the physical and mental condition, the activity, mobility and incontinent A score 10-12------High risk of PUS development
  • 11.
  • 12.
  • 13. Unstageable: Full thickness tissue loss in which slough (yellow, tan, gray, green or brown), eschar (tan, brown or black), or both in the wound bed cover the base of the ulcer. Pictures - Royal College of Surgeons of Edinburgh
  • 14. Sites
  • 15. Any skin exposed to continuous pressure.  Internal viscera exposed to unusual pressure, as trachea pressed by balloon of endotracheal tube
  • 16. Over Bony Prominences 1. Occiput 2. Ears 3. Scapula Internal 4. Spinous Processes organs 5. Shoulder 6. Elbow 7. Iliac Crest 8. Sacrum/Coccyx 9. Ischial Tuberosity 10.Trochanter 11.Knee 12.Malleolus 13.Heel 14.Toes
  • 17. Any skin surface subject to excess pressure  Examples include skin surfaces under: ◦ Oxygen tubing ◦ Urinary catheter drainage tubing ◦ Casts ◦ Cervical collars
  • 19. Not all Pressure ulcer to heel ulcers are Neuropathic diabetic foot ulcer pressure ulcers Arterial ulcer on toes and forefoot Venous leg ulcer 19
  • 21. focuses on: Skin care Mechanical loading Support surfaces
  • 22. Daily systematic skin inspection and cleansing  factors that promote dryness  Avoid massaging over bony prominences  moisture (incontinence, perspiration, drainage) It requires gentle washing and drying  Minimize friction and shear
  • 23. Reposition at least every 2 hours (may use pillows, foam wedges)  Keep head of bed at lowest elevation possible  Use lifting devices to decrease friction and shear  Remind patients in chairs to shift weight every 15 min “Doughnut” seat cushions are contraindicated, may cause pressure ulcers  Pay special attention to heels (heel ulcers account for 20% of all pressure ulcers)
  • 24. **Use for all older persons at risk for ulcers**  Static  Foam, static air, gel, water, combination (less expensive)  Dynamic  Alternating air, low-air-loss, or air-fluidized
  • 25. Heal protector Air mattress; Alternate pressure / low air loss / air fluidized Other media; gel / water/ foam http://www.diamond-medical.com/images/database/medlinesupracxc.jpg
  • 26. GENERAL ASSESSMENT Risk factor elimination ULCER ASSESSMENT, MONITORING HEALING MANAGEMENT Cleaning Debridement Dressings SURGICAL REPAIR
  • 27. Health problems (e. g, urinary incontinence)  Nutritional status  Pain level
  • 28.  ULCER  MONITORING ASSESSMENT: HEALING Location  Document all  Stage observations over time  Area  Depth  Describe each ulcer Drainage to track progress of  Necrosis healing  Granulation  Use validated tools (eg, PUSH)  Cellulitis
  • 29. Cleaning Avoid topical antiseptics because of their tissue toxicity  Debridement Is necessary to remove dead tissue it include 1. Autolytic debridement using hydrocolloid or foam dressings 2. Enzymatic debridement using exogenous collagenase (IRUXOL) 3. Mechanical debridement 4. Surgical, sharp Scalpel, scissor to remove dead tissue; laser debridement 5. Bio surgery: Larvae to digest dead tissue
  • 30.  Dressings By wet to dry saline or hydrocolloid (duo-derm), or polyurethane, in exudative wounds fill the wound by aligniates or hydro gel.  SURGICAL REPAIR May be used for stage III and IV ulcers Direct closure, skin grafting, skin flaps, musculocutaneous flaps, free flaps
  • 32. Sepsis (aerobic or anaerobic bacteremia)  Localized infection, cellulitis, osteomyelitis  Pain  Depression Mortality rate = 60% in older persons who develop a pressure ulcer within 1 year of hospital discharge
  • 33. The mainstay in pressure ulcer treatment is prevention of risk factors.
  • 34. Older adults are at high risk for development of pressure ulcers  Pressure ulcers may result in serious morbidity and mortality  Techniques that reduce pressure, moisture, friction, and shear can prevent pressure ulcers  Pressure ulcers should be treated with proper cleansing, dressings, debridement, or surgery as indicated
  • 35. a)Pressure ulcer = decubitus ulcer= bed sores. b)Stage 1 PU is partial skin thickness loss. c)One important risk factor for PU development is moisture. d)One of the complications of PU is cellulitis. e)A cornerstone in management of PU is debridement
  • 36.  First line treatment for pressure ulcer 1. Surgical closure 2. Debridement 3. Pressure relief 4. Dressing  Stage 3 pressure ulcer is: 1. Persistent non-blanchable erythema of intact skin 2. Full-thickness skin loss involving necrosis of subcutaneous tissue that may extend down to, but not through, underlying fascia. 3. Partial-thickness skin loss 4. Full-thickness skin loss with damage to muscle, bone, or supporting structures (e.g. tendon, joint capsule).
  • 37.  First line treatment for pressure ulcer 1. Surgical closure 2. Debridement 3. Pressure relief 4. Dressing  Stage 3 pressure ulcer is: 1. Persistent non-blanchable erythema of intact skin 2. Full-thickness skin loss involving necrosis of subcutaneous tissue that may extend down to, but not through, underlying fascia. 3. Partial-thickness skin loss 4. Full-thickness skin loss with damage to muscle, bone, or supporting structures (e.g. tendon, joint capsule).

Notas do Editor

  1. Intrinsic: physiologic factors or disease states that increase the risk for pressure ulcer developmentAgeNutritional statusDecreased arteriolar blood pressureLocal skin disorderSoft tissue edema, under nutrition, dehydration, atherosclerosis lead to impaired tissue repair and healing.DiabetesAnemia: decreases O2 to the woundNutritional State (Serum chemistries, Albumin, Prealbumin)Weight Loss (oxandrelone)Coagulopathic state Multiple comorbiditiesIncontinence;foleyImmobility:turning q2 hours Extrinsic: external factors that damage skinPressure, friction, shearMoisture, urinary, or fecal incontinence
  2. Epidermal turnover rates decrease by 30% to 50% by the age of 70, resulting in rougher skin with decreased barrier function, delayed wound healing.The dermal- epidermal junction fattens resulting in decreased contact between the two layers. As a result the two layers may easily separate, making older skin more likely to tear and blister.Basal and peak levels of cutaneous blood flow are reduced by about 60%, resulting in compromised vascular responsiveness during injury or infection.Collagen synthesis decreases and degradation increases, resulting in a loss of the connective tissue matrix and impaired wound healing.Elastic fibers decrease in number and size, resulting in decreased skin elasticity. Subcutaneous fat decreases with age, decreasing its ability to protect deeper structures from injury. Distribution of subcutaneous fat changes (decreasing in face and hands, increasing in thighs and abdomen), which decreases pressure diffusion over bony prominences.
  3. Stage I: Persistent non-blanchable erythema of intact skinStage II: Partial-thickness skin loss involving epidermis, dermis, or both. Ulcer is superficial and presents as an abrasion, blister, or shallow craterStage III: Full-thickness skin loss involving damage or necrosis of subcutaneous tissue that may extend down to, but not through, underlying fascia.Stage IV: Full-thickness skin loss with extensive destruction, tissue necrosis, or damage to muscle, bone, or supporting structures (e.g. tendon, joint capsule). Undermining and sinus tracts may also be present.
  4. by site, sensation, surrounding inflammation, underlying pressure points, viability of the floor