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PERIODONTAL POCKET
GINGIVAL RECESSION &
TOOTH MOBILITY
PERIODONTAL POCKET
• A periodontal pocket is defined as
the pathologically deeped gingival sulcus.
• Deepening of gingival sulcus may occur by
coronal movement of gingival margin, apical
displacement of gingival attachment.
CLASSIFICATION
Gingival pocket Periodontial pocket
Suprabony pocket Infrabony Pocket
Gingival Pocket
• It is formed by gingival enlargement
without destruction of underlying
peridontal tissue.
• The sulcus is deepend because of the
increased bulk of gingiva.
• Seen in gingivitis
Periodontal Pocket
• It produces destruction of supporting
periodontal tissue
• It leads to the loosening and
exfoliation of the teeth.
• Seen in peridontitis
Suprabony Pocket
• Base of pocket coronal to the crest of
alveolar bone.
• Horizontal bone loss.
• PDL fibres follow their normal horizontal -
oblique course between the tooth and the
bone
Infrabony Pocket
• Base of pocket is apical to the crest of
alveolar bone .
• Vertical bone loss
• PDL fibres follow angular pattern of the
adjacent bone .
PATHOGENISIS
Bacterial challenge : Diseased gingiva is associated with increased number of spirochetes .
Pocket formation starts as inflamatory in connective tissue wall of gingival sulcus
Formation of inflammatory exudate
Degeneration of surrounding connective tissue including gingival fibres
Just apical to the junctional epithelium , collagen fibres are destroyed and the area is occupied by
inflammatory cells
HISTOPATHOLOGY
1. Epithelium :
Densely infiltrated by leukocytes and edema .
Vascular degeneration
Ulceration of suppuration
2. Connective Tissue :
Edemmatous and densely infiltrated with plasma
cells lymphocytes and scattering of PMNs.
Blood vessels
Degenerative changes
CLINICAL FEATURES
• Bluish red discoloration
• Flaccidity
• Smooth and shinny surface
• Pink and firm gingival wall
• Bleeding
• Inner aspect of the pocket is painful
• Puss may be expressed out
PERIODONTAL POCKET CONTAINS
• Microorganism
• Gingival Fluid
• Food remment
• Salivary Musin
• Leukocytes
ROOT SURFACE WALL
• As the pockets deepens the cementum bocomes exposed to oral
environment .
• Collageneous remnents of sherpey fibres in the cementum
degenerates
• Penetration of bacteria results in fragmentation and breakdown of
cementum surface results in area of necrotic cementum.
TREATMENT
• Root planing
• Scanling
• Educating the pateint about oral hygine practices and habbit
changes
GINGIVAL RECESSION
• It is defined as the apical shift of the marginal gingiva for its normal position to
beyond the CEJ.
• Approx. 88% of people of from the age group >= 65yr have gingival recession
in one or more sites.
• Approx. 50% of people from the age group 18-64 yrs had gingival recession in
one or more sites.
CLASSIFICATION (Acc. to
Sullivan and Atkins )
• Deep Wide
• Shallow Wide
• Deep Narrow
• Shallow Narrow
Drawback : Doesn't enable clinician to
predict the outcome of therapy
CLASSIFICATION (Acc. to P.D.
Miller)
• Class 1:
1. Tissue recession with the attacted gingiva.
2. no bone or soft tissue loss
• Class 2 :
1. Recession extends to or beyond
mucogingival junction.
2. no bone or soft tissue loss
• Class 3 :
1. Extends beyond MGJ
2. partial bone and soft tissue loss
3. partial root coverage expected
• Class 4 :
1. Extends beyond MGJ
ETIOLOGY
• Anatomical Factor : Abnormal tooth position
Thin gingival biotype
High frenum attachment
• Pathological Factor : Periodontal Disease
Bacterial Plaque
Dental Calculus
• Traumatic Factor : Tooth brushing
Flossing
• Iatrogenic Factor : Orthodontic Treatment
Restorative and prosthetic Treatment
TECHNIQUE FOR ROOT COVERAGE
• Free gingival autograft
• Connective tissue autograft
• Subepithelial connective tirssue graft
• GTR produces
• Pouch &Tunnel's procedure
• Pedicle autograft
• laterally positioned
• Coronally positioned flap
TOOTH MOBILITY
Introduction
• Kenry AAP 1986 : It is defined as the degree of looseness of tooth .
• It is the movement of tooth in the socket resulting from an applied force .
• Mobility occures in two stages :
- Initial Stage
- Secondary stage
Types of Tooth Mobility :
• Physiologic : Moderate force exerted on the tooth surrounded by a healthy
peridontium and tooth shows tipping movement .
• Pathological : Any degree of movement perceived either faciolingually , mesiodistally
or axially when force is applied .
Factors for Pathological Tooth Mobility :
• Extent of inflammation
• Loose of tooth support
• Trauma from occlusion
• Periodontial Injury
• Pathologic process of the jaw
Classification (By Miller) :
• Class 0 : No movement on application of force
• Class 1 : Tooth movement is barely distinguishable.
• Class 2 : Tooth movement in any direction .
• Class 3 : Tooth movement in any direction tooth is depressible in the socket .
Classification (By Carranza) :
• Grade 1 : Slightly more than normal
• Grade 2 : Moderately more than normal
• Grade 3 : Severly more than normal in faciolingual or mesiodistal direction
combination with vertical dispalacement
Method To Assess Tooth Mobility :
Use of Handle of Mouth Mirror Use of Handle of Mouth mirror
and Handle of Probe and Finger
Use of Fingers Use of Peridontiometer
Signs and Symptoms
• Patient awareness of mobility
• Functional discomfort
• Esthetic
Clinical Impact
• Various degree of gingival inflamation
• Loss of attachment
• Pocket Formation
• Gingival recession
Treatment :
• Splinting of Tooth
• By using Night Guard
• Correction of occlusal surface
• Replacement of missing tooth
THANK YOU

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Periodontal pocket , Gingival Recession , Tooth mobility

  • 2. PERIODONTAL POCKET • A periodontal pocket is defined as the pathologically deeped gingival sulcus. • Deepening of gingival sulcus may occur by coronal movement of gingival margin, apical displacement of gingival attachment.
  • 3. CLASSIFICATION Gingival pocket Periodontial pocket Suprabony pocket Infrabony Pocket
  • 4. Gingival Pocket • It is formed by gingival enlargement without destruction of underlying peridontal tissue. • The sulcus is deepend because of the increased bulk of gingiva. • Seen in gingivitis Periodontal Pocket • It produces destruction of supporting periodontal tissue • It leads to the loosening and exfoliation of the teeth. • Seen in peridontitis
  • 5. Suprabony Pocket • Base of pocket coronal to the crest of alveolar bone. • Horizontal bone loss. • PDL fibres follow their normal horizontal - oblique course between the tooth and the bone Infrabony Pocket • Base of pocket is apical to the crest of alveolar bone . • Vertical bone loss • PDL fibres follow angular pattern of the adjacent bone .
  • 6. PATHOGENISIS Bacterial challenge : Diseased gingiva is associated with increased number of spirochetes . Pocket formation starts as inflamatory in connective tissue wall of gingival sulcus Formation of inflammatory exudate Degeneration of surrounding connective tissue including gingival fibres Just apical to the junctional epithelium , collagen fibres are destroyed and the area is occupied by inflammatory cells
  • 7. HISTOPATHOLOGY 1. Epithelium : Densely infiltrated by leukocytes and edema . Vascular degeneration Ulceration of suppuration 2. Connective Tissue : Edemmatous and densely infiltrated with plasma cells lymphocytes and scattering of PMNs. Blood vessels Degenerative changes
  • 8. CLINICAL FEATURES • Bluish red discoloration • Flaccidity • Smooth and shinny surface • Pink and firm gingival wall • Bleeding • Inner aspect of the pocket is painful • Puss may be expressed out
  • 9. PERIODONTAL POCKET CONTAINS • Microorganism • Gingival Fluid • Food remment • Salivary Musin • Leukocytes ROOT SURFACE WALL • As the pockets deepens the cementum bocomes exposed to oral environment . • Collageneous remnents of sherpey fibres in the cementum degenerates • Penetration of bacteria results in fragmentation and breakdown of cementum surface results in area of necrotic cementum.
  • 10. TREATMENT • Root planing • Scanling • Educating the pateint about oral hygine practices and habbit changes
  • 11. GINGIVAL RECESSION • It is defined as the apical shift of the marginal gingiva for its normal position to beyond the CEJ. • Approx. 88% of people of from the age group >= 65yr have gingival recession in one or more sites. • Approx. 50% of people from the age group 18-64 yrs had gingival recession in one or more sites.
  • 12. CLASSIFICATION (Acc. to Sullivan and Atkins ) • Deep Wide • Shallow Wide • Deep Narrow • Shallow Narrow Drawback : Doesn't enable clinician to predict the outcome of therapy
  • 13. CLASSIFICATION (Acc. to P.D. Miller) • Class 1: 1. Tissue recession with the attacted gingiva. 2. no bone or soft tissue loss • Class 2 : 1. Recession extends to or beyond mucogingival junction. 2. no bone or soft tissue loss • Class 3 : 1. Extends beyond MGJ 2. partial bone and soft tissue loss 3. partial root coverage expected • Class 4 : 1. Extends beyond MGJ
  • 14. ETIOLOGY • Anatomical Factor : Abnormal tooth position Thin gingival biotype High frenum attachment • Pathological Factor : Periodontal Disease Bacterial Plaque Dental Calculus • Traumatic Factor : Tooth brushing Flossing • Iatrogenic Factor : Orthodontic Treatment Restorative and prosthetic Treatment
  • 15. TECHNIQUE FOR ROOT COVERAGE • Free gingival autograft • Connective tissue autograft • Subepithelial connective tirssue graft • GTR produces • Pouch &Tunnel's procedure • Pedicle autograft • laterally positioned • Coronally positioned flap
  • 16. TOOTH MOBILITY Introduction • Kenry AAP 1986 : It is defined as the degree of looseness of tooth . • It is the movement of tooth in the socket resulting from an applied force . • Mobility occures in two stages : - Initial Stage - Secondary stage
  • 17. Types of Tooth Mobility : • Physiologic : Moderate force exerted on the tooth surrounded by a healthy peridontium and tooth shows tipping movement . • Pathological : Any degree of movement perceived either faciolingually , mesiodistally or axially when force is applied . Factors for Pathological Tooth Mobility : • Extent of inflammation • Loose of tooth support • Trauma from occlusion • Periodontial Injury • Pathologic process of the jaw
  • 18. Classification (By Miller) : • Class 0 : No movement on application of force • Class 1 : Tooth movement is barely distinguishable. • Class 2 : Tooth movement in any direction . • Class 3 : Tooth movement in any direction tooth is depressible in the socket . Classification (By Carranza) : • Grade 1 : Slightly more than normal • Grade 2 : Moderately more than normal • Grade 3 : Severly more than normal in faciolingual or mesiodistal direction combination with vertical dispalacement
  • 19. Method To Assess Tooth Mobility : Use of Handle of Mouth Mirror Use of Handle of Mouth mirror and Handle of Probe and Finger Use of Fingers Use of Peridontiometer
  • 20. Signs and Symptoms • Patient awareness of mobility • Functional discomfort • Esthetic Clinical Impact • Various degree of gingival inflamation • Loss of attachment • Pocket Formation • Gingival recession Treatment : • Splinting of Tooth • By using Night Guard • Correction of occlusal surface • Replacement of missing tooth