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BILIARY TRACT DISORDER
Ms. Anshu
M.Sc Nursing
KGMU Institute of
Nursing
INTRODUCTION-
Disorders of gallbladder and ducts are
extremely common.
Its more commonly affect people of
sedentary life style and obesity.
EPIDEMIOLOGY
In the united states alone, it is estimated
that 20 million people have gall stones
with approximately 1 million new cases
developing each year.
The most common conditions are gall
stones and associated cholecystitis.
About 98% of clients who present with
symptomatic gall bladder disease have
gall stones.
Malignancies and congenital anomalies
are very rare.
ANATOMY OF GALL BLADDER
Pear shape sac
7-10 cm long
Average capacity 30-
50 ml
When obstructed 300
ml
PHYSIOLOGY OF GALL BLADDER
The smooth muscles in the gallbladder
wall contract, leading to the bile
being secreted into the duodenum to
rid the body of waste stored in the bile as
well as aid in the absorption of
dietary fat by solubilising them using bile
acids.
BILE COMPOSITION
 Bile consists of water, electrolytes, bile
acids, cholesterol, phospholipids and
conjugated bilirubin
Bile is secreted by the liver into small
ducts that join to form the common hepatic
duct and get stored in gall bladder.
BILIARY TRACT DISORDERS INCLUDES-
Cholelithiasis
Acute cholecystitis
Chronic cholecystitis
CHOLELITHIASIS/ GALLSTONES
Gallstones are collections of cholesterol,
bile pigment or a combination of the two,
which can form in the gallbladder or within
the bile ducts of the liver.
Gallstones . .
The presence of
gallstones in the
gallbladder is called
cholelithiasis.
INCIDENCE
Common duct stones are found in about
10% tom15% of client with cholelithiasis.
The incidence increases with age, and the
frequency of gallstones it the common
duct in the older population may be as
high as 25%.
ETIOLOGY
Change in bile composition-
Gallbladder stasis
supersaturation of bile with cholesterol
Infection and tissue injury
Genetics
Those who are most at risk.
These are all adjectives to describe the person most at
risk of developing symptomatic gallstones.
FAIR FAT FORTY FEMALE
PATHOPHYSIOLOGY
TYPES OF GALLSTONES
Cholesterol
Pigmented
mixed
Cholesterol stones
Most common type
Incidence increases
with age
Female>male
Smooth & whitish
yellow to tan colour
Pigment stones
Excess of
unconjugated bilirubin
May be black
colour(associated
with hemolysis and
cirrhosis) or earthy
calcium bilirubinate
(associated with
infection)
Mixed stones
Combination of
cholesterol and
pigment stones or
other substance
Calcium
carbonate,phosphate,
bile salts, and
palmitate
SIGNS AND SYMPTOMS.
Complaints of
indigestion after
eating high fat foods.
Localized pain in the
right-upper quadrant
epigastric region.
Anorexia, nausea,
vomiting and
flatulence.
Increased heart
and respiratory
rate – causing
patient to become
diaphoretic which
in turn makes them
think they are
having a heart
attack.
SIGNS AND SYMPTOMS.
 Low grade fever.
 Elevated leukocyte count.
 Mild jaundice.
 Stools that contain fat – steatorrhea.
 Clay colored stools caused by a lack of
bile in the intestinal tract.
 Urine may be dark amber- to tea-colored.
DIAGNOSTICS TEST
History of patient
Physical examination
Laboratory test for-
Elevated conjugated bilirubin.
Elevated alkaline phosphate
Serum amylase and lipase
Elevated WBC count
Fecal studies.
Ultrasound of
the gallbladder
CT Scan
MRI
HIDA (hepato- iminodiacetic acid)
Cholangiography
ERCP (endoscopic retrograde
cholangiopancreatography)
Diagnostics.
 HIDA
(hydroxyiminod
iacetic acid
scan) - imaging
test used to examine
the gallbladder and
the ducts leading into
and out of the
gallbladder - also
referred to as
cholescintigraphy.
MEDICAL MANAGEMENT
GOALS-
To resolve symptoms
To remove stones
To prevent complications
1. PAIN MANAGEMENT
Give analgesics
Antacids, H2 blockers or proton pump
inhibitors- to neutralize gastric acid
2.For nausea and vomiting
Antiemetics given
3. antibiotics
Endoscopy
Gall stone dissolution
Oral administration of agents-
chenodeoxycholic acid (CDCA) or
chenodal
ursodeoxycholic acid (UDCA) or ursodiol
Action- reduces the amount of cholesterol
in bile
Medical Management.
Lithotripsy Extracorporeal shock
wave lithotripsy
(ESWL)-
1500 shock waves
directed at stones
Used for fewer than 4
stones, each smaller
than 3cm.
Medical Management.
 If stones are present in the
common bile duct, an
endoscopic sphincterotomy
must be performed to remove
them BEFORE a
cholecystectomy is done.
A number of various
instruments are inserted
through the endoscope in
order to "cut" or stretch the
sphincter.
Once this is done, additional
instruments are passed that
enable the removal of stones
and the stretching of
narrowed regions of the
ducts.
Drains (stents) can also be
used to prevent a narrowed
area from rapidly returning to
its previously narrowed state.
Surgical Management.
Cholecystectomy
or
Laparoscopic Cholecystectomy
– removal of the gallbladder.
This is the treatment of choice.
The gallbladder along with the cystic
duct, vein and artery are ligated.
Laproscopic cholecystectomy
NURSING MANAGEMENT
ASSESSMENT
Assess the general condition of patient
Assess pain of patient.
Observe for bleeding.
NURSING DIAGNOSIS
Acute pain related to surgical procedure.
Impaired skin integrity Invasion of body
structure
Ineffective breathing pattern Pain
Risk for deficient fluid volume related to
surgical procedure
NURSING INTERVENTION
1. Acute pain related to surgery
Monitor and record vital signs.
 administer medication as ordered.
assess the severity,frequency, and
characteristic of pain.
Provide divertional activities such as
reading newspapers.
2. Impaired skin integrity
Observe the color and character of the
drainage.
Change dressings as often as necessary.
Place patient in low- or semi-Fowler’s
position.
Monitor puncture sites (3–5) if endoscopic
procedure is done.
3. Ineffective breathing pattern
Observe respiratory rate, depth.
Auscultate breath sounds.
Assist patient to turn, cough, and deep
breathe periodically.
Show patient how to splint incision.
Instruct in effective breathing techniques.
Elevate head of bed, maintain low-
Fowler’s position.
4. Risk for deficient fluid volume
Monitor vital signs. Assess mucous
membranes, skin turgor, peripheral
pulses, and capillary refill.
Monitor I&O, including drainage from NG
tube ,T-tube, and wound. Weigh patient
periodically.
Observe for signs of bleeding:
hematemesis, melena, petechiae,
ecchymosis.
Administer IV fluids, blood products, as
indicated
What is it cholecystitis?
 By definition,
cholecystitis is an
inflammation of the
gallbladder wall and
nearby abdominal
lining.
Abdominal wall
Gallbladder
TYPES OF CHOLECYSTITIS
Acute cholecystitis
Chronic cholecystitis
ACUTE CHOLECYSTITIS
DEFINITION-
Acute cholecystitis refers to acute
inflammation of the gallbladder wall.
ETIOLOGY
Gall stone in cystic duct
Obstruction in cystic duct
Bacterial infection (gram positive and
gram negative aerobes and anaerobes:-
E. Coli, klebsiella, Clostredium and
streptococcus)
RISK FACTORS
Sedentary lifestyle
obesity
PATHOPHYSIOLOGY
PROGRESSION OF ACUTE
CHOLECYSTITIS.
- Gallbladder has a
grayish appearance & is
edematous.
-There is an obstruction
of the cystic duct and
the gallbladder begins
to swell.
- It no longer has the
"robin egg blue"
appearance of a normal
gallbladder.
- As acute
cholecystitis
progresses, the
gallbladder begins
to become necrotic
and gets a speckled
appearance as the
wall begins to die.
- Gallbladder
undergoes
gangrenous change
and the wall
becomes very dark
green or black.
- This is the stage
when perforation
occurs.
SIGNS AND SYMPTOMS
Complain of pain
In right upper quadrant
In epigastric region
In right subscapular
Onset sudden
Peak in 30min
Nausea and vomiting
Low grade fever
Mild jaundice
CHRONIC CHOLECYSTITIS
DEFINITION-
Repeated inflammation and infection of
gallbladder
SIGNS AND SYMPTOMS
Epigastric pain
Indigestion
Fat intolerance
Heart burn
Fibrosis of gall tissues
Inability to concentrate bile
MEDICAL MANAGEMENT
GOAL-
 to treat symptomatic causes
 to prevent complication
Antibiotic therapy-
Ampicillin
Ureidopenicillins – piperacillin or
mezlocillin
Third generation cephalosporins-
Ceftriaxone, cefixime, Cefotaxime
Aminoglycosides –
Gentamicin, Amikacin, Neomycin
SURGICAL MANAGEMENT
cholecystectomy
Nursing Interventions
Post Op - Cholesystectomy
1. Administer oral analgesics to facilitate movement
and deep breathing – and to stay ahead of pts pain.
2. Observe dressings frequently for exudate and hemorrhage.
3. Vitals are routinely checked.
4. Patient teaching:
-Must understand how to splint the abd. before
coughing.
-Report any abnormalities such as,
severe pain, tenderness in RUQ, increase in
pulse, etc . .
-Instructed that they usually can return to work in 3
days & can resume full activity in 1 week.
5. Fluid balance is maintained IV –
potassium added to compensate
for loss from surgery.
Nursing Interventions
1. Urine and stool should be observed for alterations
in the presence of bilirubin.
2. NG tube must be monitored for amount, color & consistency
of output.
Also, tube must be on LOW suction and nasal area should
be monitored for irritation and necrosis.
3. Anti-emetics may be administered if nausea persists.
4. I & O are measured and described carefully.
5. Pt. must understand how to splint the abdomen
for post op coughing, turning and deep breathing.
Interventions center on keeping patient comfortable by
carefully administering meds and watching for reactions.
CONCLUSION
Biliary disorders are extremely common
but diverse in nature.
Incidence rate of the disease is increasing
day by day.
Teaching and awareness is vital in
prevention and management of the
disease.
EVALUATION
A 45 yr old obese lady, complaining of
epigastric pain, right sided subscapular
pain which last for 3-4 hrs associated with
nausea and vomiting. She has mild icterus
and bilirubin is 3.3mg/dl.
Guess what could be the diagnosis of
patient?
Cholecystitis cholelithiasis-presentation

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Cholecystitis cholelithiasis-presentation

  • 1. BILIARY TRACT DISORDER Ms. Anshu M.Sc Nursing KGMU Institute of Nursing
  • 2. INTRODUCTION- Disorders of gallbladder and ducts are extremely common. Its more commonly affect people of sedentary life style and obesity.
  • 3. EPIDEMIOLOGY In the united states alone, it is estimated that 20 million people have gall stones with approximately 1 million new cases developing each year. The most common conditions are gall stones and associated cholecystitis.
  • 4. About 98% of clients who present with symptomatic gall bladder disease have gall stones. Malignancies and congenital anomalies are very rare.
  • 5. ANATOMY OF GALL BLADDER Pear shape sac 7-10 cm long Average capacity 30- 50 ml When obstructed 300 ml
  • 6. PHYSIOLOGY OF GALL BLADDER The smooth muscles in the gallbladder wall contract, leading to the bile being secreted into the duodenum to rid the body of waste stored in the bile as well as aid in the absorption of dietary fat by solubilising them using bile acids.
  • 7. BILE COMPOSITION  Bile consists of water, electrolytes, bile acids, cholesterol, phospholipids and conjugated bilirubin Bile is secreted by the liver into small ducts that join to form the common hepatic duct and get stored in gall bladder.
  • 8. BILIARY TRACT DISORDERS INCLUDES- Cholelithiasis Acute cholecystitis Chronic cholecystitis
  • 9. CHOLELITHIASIS/ GALLSTONES Gallstones are collections of cholesterol, bile pigment or a combination of the two, which can form in the gallbladder or within the bile ducts of the liver.
  • 10. Gallstones . . The presence of gallstones in the gallbladder is called cholelithiasis.
  • 11. INCIDENCE Common duct stones are found in about 10% tom15% of client with cholelithiasis. The incidence increases with age, and the frequency of gallstones it the common duct in the older population may be as high as 25%.
  • 12. ETIOLOGY Change in bile composition- Gallbladder stasis supersaturation of bile with cholesterol Infection and tissue injury Genetics
  • 13. Those who are most at risk. These are all adjectives to describe the person most at risk of developing symptomatic gallstones. FAIR FAT FORTY FEMALE
  • 15.
  • 17. Cholesterol stones Most common type Incidence increases with age Female>male Smooth & whitish yellow to tan colour
  • 18. Pigment stones Excess of unconjugated bilirubin May be black colour(associated with hemolysis and cirrhosis) or earthy calcium bilirubinate (associated with infection)
  • 19. Mixed stones Combination of cholesterol and pigment stones or other substance Calcium carbonate,phosphate, bile salts, and palmitate
  • 20. SIGNS AND SYMPTOMS. Complaints of indigestion after eating high fat foods. Localized pain in the right-upper quadrant epigastric region. Anorexia, nausea, vomiting and flatulence.
  • 21. Increased heart and respiratory rate – causing patient to become diaphoretic which in turn makes them think they are having a heart attack.
  • 22. SIGNS AND SYMPTOMS.  Low grade fever.  Elevated leukocyte count.  Mild jaundice.  Stools that contain fat – steatorrhea.  Clay colored stools caused by a lack of bile in the intestinal tract.  Urine may be dark amber- to tea-colored.
  • 23. DIAGNOSTICS TEST History of patient Physical examination Laboratory test for- Elevated conjugated bilirubin. Elevated alkaline phosphate Serum amylase and lipase Elevated WBC count Fecal studies.
  • 25. CT Scan MRI HIDA (hepato- iminodiacetic acid) Cholangiography ERCP (endoscopic retrograde cholangiopancreatography)
  • 26. Diagnostics.  HIDA (hydroxyiminod iacetic acid scan) - imaging test used to examine the gallbladder and the ducts leading into and out of the gallbladder - also referred to as cholescintigraphy.
  • 27. MEDICAL MANAGEMENT GOALS- To resolve symptoms To remove stones To prevent complications
  • 28. 1. PAIN MANAGEMENT Give analgesics Antacids, H2 blockers or proton pump inhibitors- to neutralize gastric acid 2.For nausea and vomiting Antiemetics given 3. antibiotics
  • 30. Gall stone dissolution Oral administration of agents- chenodeoxycholic acid (CDCA) or chenodal ursodeoxycholic acid (UDCA) or ursodiol Action- reduces the amount of cholesterol in bile
  • 31. Medical Management. Lithotripsy Extracorporeal shock wave lithotripsy (ESWL)- 1500 shock waves directed at stones Used for fewer than 4 stones, each smaller than 3cm.
  • 32. Medical Management.  If stones are present in the common bile duct, an endoscopic sphincterotomy must be performed to remove them BEFORE a cholecystectomy is done. A number of various instruments are inserted through the endoscope in order to "cut" or stretch the sphincter. Once this is done, additional instruments are passed that enable the removal of stones and the stretching of narrowed regions of the ducts. Drains (stents) can also be used to prevent a narrowed area from rapidly returning to its previously narrowed state.
  • 33. Surgical Management. Cholecystectomy or Laparoscopic Cholecystectomy – removal of the gallbladder. This is the treatment of choice. The gallbladder along with the cystic duct, vein and artery are ligated.
  • 35. NURSING MANAGEMENT ASSESSMENT Assess the general condition of patient Assess pain of patient. Observe for bleeding.
  • 36. NURSING DIAGNOSIS Acute pain related to surgical procedure. Impaired skin integrity Invasion of body structure Ineffective breathing pattern Pain Risk for deficient fluid volume related to surgical procedure
  • 37. NURSING INTERVENTION 1. Acute pain related to surgery Monitor and record vital signs.  administer medication as ordered. assess the severity,frequency, and characteristic of pain. Provide divertional activities such as reading newspapers.
  • 38. 2. Impaired skin integrity Observe the color and character of the drainage. Change dressings as often as necessary. Place patient in low- or semi-Fowler’s position. Monitor puncture sites (3–5) if endoscopic procedure is done.
  • 39. 3. Ineffective breathing pattern Observe respiratory rate, depth. Auscultate breath sounds. Assist patient to turn, cough, and deep breathe periodically. Show patient how to splint incision. Instruct in effective breathing techniques. Elevate head of bed, maintain low- Fowler’s position.
  • 40. 4. Risk for deficient fluid volume Monitor vital signs. Assess mucous membranes, skin turgor, peripheral pulses, and capillary refill. Monitor I&O, including drainage from NG tube ,T-tube, and wound. Weigh patient periodically. Observe for signs of bleeding: hematemesis, melena, petechiae, ecchymosis. Administer IV fluids, blood products, as indicated
  • 41. What is it cholecystitis?  By definition, cholecystitis is an inflammation of the gallbladder wall and nearby abdominal lining. Abdominal wall Gallbladder
  • 42. TYPES OF CHOLECYSTITIS Acute cholecystitis Chronic cholecystitis
  • 43. ACUTE CHOLECYSTITIS DEFINITION- Acute cholecystitis refers to acute inflammation of the gallbladder wall.
  • 44. ETIOLOGY Gall stone in cystic duct Obstruction in cystic duct Bacterial infection (gram positive and gram negative aerobes and anaerobes:- E. Coli, klebsiella, Clostredium and streptococcus)
  • 47. PROGRESSION OF ACUTE CHOLECYSTITIS. - Gallbladder has a grayish appearance & is edematous. -There is an obstruction of the cystic duct and the gallbladder begins to swell. - It no longer has the "robin egg blue" appearance of a normal gallbladder.
  • 48. - As acute cholecystitis progresses, the gallbladder begins to become necrotic and gets a speckled appearance as the wall begins to die.
  • 49. - Gallbladder undergoes gangrenous change and the wall becomes very dark green or black. - This is the stage when perforation occurs.
  • 50. SIGNS AND SYMPTOMS Complain of pain In right upper quadrant In epigastric region In right subscapular Onset sudden Peak in 30min
  • 51. Nausea and vomiting Low grade fever Mild jaundice
  • 52.
  • 54. SIGNS AND SYMPTOMS Epigastric pain Indigestion Fat intolerance Heart burn Fibrosis of gall tissues Inability to concentrate bile
  • 55. MEDICAL MANAGEMENT GOAL-  to treat symptomatic causes  to prevent complication
  • 56. Antibiotic therapy- Ampicillin Ureidopenicillins – piperacillin or mezlocillin Third generation cephalosporins- Ceftriaxone, cefixime, Cefotaxime Aminoglycosides – Gentamicin, Amikacin, Neomycin
  • 58. Nursing Interventions Post Op - Cholesystectomy 1. Administer oral analgesics to facilitate movement and deep breathing – and to stay ahead of pts pain. 2. Observe dressings frequently for exudate and hemorrhage. 3. Vitals are routinely checked. 4. Patient teaching: -Must understand how to splint the abd. before coughing. -Report any abnormalities such as, severe pain, tenderness in RUQ, increase in pulse, etc . . -Instructed that they usually can return to work in 3 days & can resume full activity in 1 week. 5. Fluid balance is maintained IV – potassium added to compensate for loss from surgery.
  • 59. Nursing Interventions 1. Urine and stool should be observed for alterations in the presence of bilirubin. 2. NG tube must be monitored for amount, color & consistency of output. Also, tube must be on LOW suction and nasal area should be monitored for irritation and necrosis. 3. Anti-emetics may be administered if nausea persists. 4. I & O are measured and described carefully. 5. Pt. must understand how to splint the abdomen for post op coughing, turning and deep breathing. Interventions center on keeping patient comfortable by carefully administering meds and watching for reactions.
  • 60. CONCLUSION Biliary disorders are extremely common but diverse in nature. Incidence rate of the disease is increasing day by day. Teaching and awareness is vital in prevention and management of the disease.
  • 61. EVALUATION A 45 yr old obese lady, complaining of epigastric pain, right sided subscapular pain which last for 3-4 hrs associated with nausea and vomiting. She has mild icterus and bilirubin is 3.3mg/dl. Guess what could be the diagnosis of patient?