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WOUNDS
CONTENTS
• Introduction
• Classification of wounds
• Stages of wound healing
• Types of wound healing
• Factors affecting wound healing
• Examination of patient
• Treatment for wounds
• Complications of wounds
INTRODUCTION
Wound is the mechanical integrity of skin, mucosal membrane, more deep tissues
and organs. Wound is composed by wound cavity limited with walls and bottom of
wound and wound canal is deep and narrow.
Local signs of wound: pain, bleeding, open defect.
CLASSIFICATION
OF WOUNDS
A.Reason of wounding-
surgical is aseptic, accidental
wound is contaminated.
B.Type of injury and wound –
cut, punctured, contused,
bite, penetrated, lacerated.
5
6
STAGES OF WOUND HEALING
8
TYPES OF WOUND
HEALING PROCESS
• PRIMARY INTENTION - Clean
incised wounds in which the
edges of wounds are in close
apposition.
• SECONDARY INTENTION – When
the incision margins are not held
together and there is more than
10mm distance between them.
FACTORS AFFECTINGWOUND HEALING
SYSTEMIC
Age
Nutrition
Trauma
Metabolic Diseases
Immunosuppression
Connective tissue disorders
Smoking
Mechanical injury
Infection
Edema
Ischemia/necrotic tissue
Topical agents
Ionizing radiation
Low oxygen tension
Foreign bodies
LOCAL
9
EXAMINATION OF PATIENT
10
How was the wound created? If chronic, why is it still open?
What is the underlying etiology?
: Where on the body is it located? Is it in an area that is
difficult to offload or to keep clean? Is it in a region of high skin
tension? Is it near any vital structures such as a major artery?
: How long has this wound been present? (e.g., acute or
chronic)
• What: What anatomy does it involve (e.g., epidermis, dermis,
subcutaneous tissue, fascia, muscle, tendon, bone, arteries,
nerves)?
What comorbidities or social factors does the patient have
that might negatively impact their healing ability?Is it life-
threatening?
1
2
Local complications - delayed and
non-healing of the wound,
cellulitis, abscess formation,
osteomyelitis as well as further
wound breakdown.
Systemic complications -
bacteremia with the possibility of
distant hematogenous spread and
sepsis.
COMPLICATIONS OF WOUNDS
14

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WOUNDS.pptx

  • 2. CONTENTS • Introduction • Classification of wounds • Stages of wound healing • Types of wound healing • Factors affecting wound healing • Examination of patient • Treatment for wounds • Complications of wounds
  • 3. INTRODUCTION Wound is the mechanical integrity of skin, mucosal membrane, more deep tissues and organs. Wound is composed by wound cavity limited with walls and bottom of wound and wound canal is deep and narrow. Local signs of wound: pain, bleeding, open defect.
  • 4. CLASSIFICATION OF WOUNDS A.Reason of wounding- surgical is aseptic, accidental wound is contaminated. B.Type of injury and wound – cut, punctured, contused, bite, penetrated, lacerated.
  • 5. 5
  • 6. 6
  • 7. STAGES OF WOUND HEALING
  • 8. 8 TYPES OF WOUND HEALING PROCESS • PRIMARY INTENTION - Clean incised wounds in which the edges of wounds are in close apposition. • SECONDARY INTENTION – When the incision margins are not held together and there is more than 10mm distance between them.
  • 9. FACTORS AFFECTINGWOUND HEALING SYSTEMIC Age Nutrition Trauma Metabolic Diseases Immunosuppression Connective tissue disorders Smoking Mechanical injury Infection Edema Ischemia/necrotic tissue Topical agents Ionizing radiation Low oxygen tension Foreign bodies LOCAL 9
  • 10. EXAMINATION OF PATIENT 10 How was the wound created? If chronic, why is it still open? What is the underlying etiology? : Where on the body is it located? Is it in an area that is difficult to offload or to keep clean? Is it in a region of high skin tension? Is it near any vital structures such as a major artery? : How long has this wound been present? (e.g., acute or chronic) • What: What anatomy does it involve (e.g., epidermis, dermis, subcutaneous tissue, fascia, muscle, tendon, bone, arteries, nerves)? What comorbidities or social factors does the patient have that might negatively impact their healing ability?Is it life- threatening?
  • 11.
  • 12. 1 2 Local complications - delayed and non-healing of the wound, cellulitis, abscess formation, osteomyelitis as well as further wound breakdown. Systemic complications - bacteremia with the possibility of distant hematogenous spread and sepsis. COMPLICATIONS OF WOUNDS
  • 13.
  • 14. 14

Notas do Editor

  1. Acute wounds: an adequate angiogenesis promotes re-epithelialization, fibroblasts' proliferation, and neutrophils' anti-infection activities. Chronic wounds : persistent local bacterial infections hinder the formation of novel blood vessels. In turn, the restricted angiogenesis hampers fibroblasts' proliferation and the neutrophils' anti-infection activities.
  2. Haemostasis - A platelet plug prevents blood loss and a preliminary fibrin matrix is formed. Inflammation then ensues to remove debris and prevent infection, commencing with neutrophil influx, which is promoted by histamine release from mast cells. Monocytes arrive later and differentiate into tissue macrophages to clear remaining cell debris and neutrophils. During the proliferative phase, keratinocytes migrate to close the wound gap, blood vessels reform through angiogenesis, and fibroblasts replace the initial fibrin clot with granulation tissue. Macrophages and regulatory T cells are also vital for this stage of healing. The deposited matrix is remodelled further by fibroblasts, blood vessels regress and myofibroblasts cause overall wound contraction.