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ORTHO
CONFERENCE
Ext pattraporn
HISTORY
Male 43 yr
cc: รถชน 3 hr PTA
PI : 3 hr PTA รถกระบะชนเสาไฟฟ้า มีอาการปวดต้นคอ มีอาการอ่อนแรงและชาที่แขนและขา ไม่มีแผลตามตัว สลบจา
เหตุการณ์ไม่ได้ ไม่มีอาเจียน ไม่หายใจหอบเหนื่อย ไม่ปวดท้อง
Past history : no underlying disease
PHYSICAL EXAMINATION
Primary survey
A : Can talk, tender at neck with limited ROM
B : Equal breath sound, CCT -ve, no subcutaneous
emphysema
C : BP 96/60 mmHg, PR 66 bpm, no active bleeding
D : E4V5M6, pupil 3 mm RTLBE
E : no external wound
PHYSICAL EXAMINATION
Vital sign : BP 96/60 mm Hg PR 90 bpm RR 20 /min Temp
37.2
GA : A Thai man , good consciousness
CVS : normal S1 , S2 , no murmur , cap refill < 2 secs
Lung : clear , equal both lung , no adventitious sound
Abd : soft , not tender , no guarding , no rebound
tenderness
PHYSICAL EXAMINATION
Can't flexion and extension
neck tender posterior
Decrease sensation below
C6
Bulbocarvernosus reflex -ve
Loose sphincter tone
RT LT
C5 II II
C6 II I
C7 II II
C8 0 0
T1 0 0
RT LT
L2 0 0
L3 0 0
L4 0 0
L5 0 0
S1 0 0
INVESTIGATION
Film C-spine AP, Lateral
Swimming view
SPINOUS PROCESS LINE
Spinolaminar line
posterior vertebral body line
anterior vertebral body line
facet joints appear as stacked
parallelograms
Prevertebral soft-tissue shadow
Disc C2-C3 < 7mm
Disc C6-C7 < 21 mm
AP TRANSLATION
3.5 mm of translational deformity is suggestive of mechanical
instability
COBB ANGLE
>11 degrees suggestive of posterior ligamentous injury
and potential instability
CT SCAN
• More sensitive for detecting fractures
• More consistently enables assessment of the
occipitocervical and cervicothoracic junctions
ALLEN & FERGUSON
CLASSIFICATION
Distraction flexion II
DISTRACTIVE FLEXION
DIAGNOSIS
C5-C6 unilateral facet dislocation with complete cord injury
INITIAL MANAGEMENT
High dose Methyl-
prednisolone Methyl
prednisolone 30mg/kg then
5.4 mg/kg over the next 24
hours
On skull traction
MRI c-spine
HIGH-DOSE METHYL
PREDNISOLONE
MRI
• Superiority in visualizing the spinal cord, intervertebral disc,
and spinal ligaments
• Detecting
• traumatic disc herniations
• epidural hematoma
• spinal cord edema or compression
• posterior ligamentous disruption
MRI
Indication
• patients with neurological
deficits
• patients with injuries in which
the integrity of the posterior
ligamentous complex is
unclear and would directly
influence the treatment plan
TREATMENT
SUBAXIAL CERVICAL SPINE INJURY
CLASSIFICATION (SLIC)
<= 3 : nonoperative
>= 5 : operative
TREATMENT
8 point
Operative treatment
FACET DISLOCATION
Non-operative treatment
• Indication : unilateral facet dislocations
without any signs of neurological injury
• Halo vest immobilization 3 month
• Flexion-extension views to confirm stability
FACET DISLOCATION
Operative treatment
• Closed reduction using cranial tong or halo
traction as early as possible in awake,
conscious, and able to be serially examined
patient
• Pre-reduction and post-reduction MRI
FACET DISLOCATION
Operative treatment
• If there the spinal cord is being indented by a
disc herniation, anterior surgery is preferred
• Anterior surgery followed by posterior
stabilization for patients with highly unstable
bilateral facet dislocations
TREATMENT
SPINAL CORD INJURY
ANATOMY
SPINAL CORD
SPINAL CORD INJURY
Complete cord injury syndrome
Incomplete cord injury syndrome
Conus medullaris syndrome
Clauda equine syndrome
COMPLETE CORD INJURY
SYNDROME
After presence of bulbocavernosus reflex : no sensation or
voluntary motor function is noted
INCOMPLETE CORD INJURY
SYNDROME
Some neurological function persist after return of
bulbocavernosus reflex
Sacral sparing : imply continuity between cerebral cortex
and lower sacral motor neuron.
Such as 1. Perianal sensation 2. Voluntary rectal motor
function 3. Big toe flexor activity
INCOMPLETE CORD INJURY
SYNDROME
INCOMPLETE CORD INJURY
SYNDROME
ANTERIOR CORD SYNDROME
Blood flow is reduced or
interrupted in the artery that runs
along the anterior portion of the
spinal cord.
May be the result of bone
fragments from traumatic injury
to the vertebra, spinal disc
herniations or
flexion/compression injury.
Most poor prognosis : recovery
rate 10%
CENTRAL CORD SYNDROME
Most common type
Characterized by impairment in the
arms and hands and, to a lesser extent,
in the legs.
Spare sacral spine thalamus and
corticospinal tracts
Recovery from distal to proximal [toe
flexion > toe extension > ankle > knee >
hip]
recovery rate 75%
BROWN SEQUARD
SYNDROME
Hemisection of the spinal cord
Motor paralysis , loss of vibration and
proprioception on the ipsilateral side as
the lesion and deficits in pain and
temperature sensation on the
contralateral side of the lesion.
The most common cause of Brown-
Séquard syndrome is penetrating trauma
such as a gunshot wound or stab wound
to the spinal cord.
Best prognosis : More than 90% of
people regain bladder & bowel control
and the ability to walk.
POSTERIOR CORD
SYNDROME
SPINAL SHOCK
Immediate temporary loss of total power , sensation and
reflexs below the level of injury
Loss of bulbocavernosus reflex
Usually recovery in 24-48 hrs

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C5 C6 dislocation

  • 2. HISTORY Male 43 yr cc: รถชน 3 hr PTA PI : 3 hr PTA รถกระบะชนเสาไฟฟ้า มีอาการปวดต้นคอ มีอาการอ่อนแรงและชาที่แขนและขา ไม่มีแผลตามตัว สลบจา เหตุการณ์ไม่ได้ ไม่มีอาเจียน ไม่หายใจหอบเหนื่อย ไม่ปวดท้อง Past history : no underlying disease
  • 3. PHYSICAL EXAMINATION Primary survey A : Can talk, tender at neck with limited ROM B : Equal breath sound, CCT -ve, no subcutaneous emphysema C : BP 96/60 mmHg, PR 66 bpm, no active bleeding D : E4V5M6, pupil 3 mm RTLBE E : no external wound
  • 4. PHYSICAL EXAMINATION Vital sign : BP 96/60 mm Hg PR 90 bpm RR 20 /min Temp 37.2 GA : A Thai man , good consciousness CVS : normal S1 , S2 , no murmur , cap refill < 2 secs Lung : clear , equal both lung , no adventitious sound Abd : soft , not tender , no guarding , no rebound tenderness
  • 5. PHYSICAL EXAMINATION Can't flexion and extension neck tender posterior Decrease sensation below C6 Bulbocarvernosus reflex -ve Loose sphincter tone RT LT C5 II II C6 II I C7 II II C8 0 0 T1 0 0 RT LT L2 0 0 L3 0 0 L4 0 0 L5 0 0 S1 0 0
  • 6. INVESTIGATION Film C-spine AP, Lateral Swimming view
  • 7.
  • 9.
  • 10. Spinolaminar line posterior vertebral body line anterior vertebral body line facet joints appear as stacked parallelograms Prevertebral soft-tissue shadow Disc C2-C3 < 7mm Disc C6-C7 < 21 mm
  • 11. AP TRANSLATION 3.5 mm of translational deformity is suggestive of mechanical instability
  • 12.
  • 13. COBB ANGLE >11 degrees suggestive of posterior ligamentous injury and potential instability
  • 14.
  • 15. CT SCAN • More sensitive for detecting fractures • More consistently enables assessment of the occipitocervical and cervicothoracic junctions
  • 16.
  • 17.
  • 20. DIAGNOSIS C5-C6 unilateral facet dislocation with complete cord injury
  • 21. INITIAL MANAGEMENT High dose Methyl- prednisolone Methyl prednisolone 30mg/kg then 5.4 mg/kg over the next 24 hours On skull traction MRI c-spine
  • 23. MRI • Superiority in visualizing the spinal cord, intervertebral disc, and spinal ligaments • Detecting • traumatic disc herniations • epidural hematoma • spinal cord edema or compression • posterior ligamentous disruption
  • 24. MRI Indication • patients with neurological deficits • patients with injuries in which the integrity of the posterior ligamentous complex is unclear and would directly influence the treatment plan
  • 25.
  • 27. SUBAXIAL CERVICAL SPINE INJURY CLASSIFICATION (SLIC) <= 3 : nonoperative >= 5 : operative
  • 29. FACET DISLOCATION Non-operative treatment • Indication : unilateral facet dislocations without any signs of neurological injury • Halo vest immobilization 3 month • Flexion-extension views to confirm stability
  • 30. FACET DISLOCATION Operative treatment • Closed reduction using cranial tong or halo traction as early as possible in awake, conscious, and able to be serially examined patient • Pre-reduction and post-reduction MRI
  • 31. FACET DISLOCATION Operative treatment • If there the spinal cord is being indented by a disc herniation, anterior surgery is preferred • Anterior surgery followed by posterior stabilization for patients with highly unstable bilateral facet dislocations
  • 33.
  • 37. SPINAL CORD INJURY Complete cord injury syndrome Incomplete cord injury syndrome Conus medullaris syndrome Clauda equine syndrome
  • 38. COMPLETE CORD INJURY SYNDROME After presence of bulbocavernosus reflex : no sensation or voluntary motor function is noted
  • 39. INCOMPLETE CORD INJURY SYNDROME Some neurological function persist after return of bulbocavernosus reflex Sacral sparing : imply continuity between cerebral cortex and lower sacral motor neuron. Such as 1. Perianal sensation 2. Voluntary rectal motor function 3. Big toe flexor activity
  • 42. ANTERIOR CORD SYNDROME Blood flow is reduced or interrupted in the artery that runs along the anterior portion of the spinal cord. May be the result of bone fragments from traumatic injury to the vertebra, spinal disc herniations or flexion/compression injury. Most poor prognosis : recovery rate 10%
  • 43. CENTRAL CORD SYNDROME Most common type Characterized by impairment in the arms and hands and, to a lesser extent, in the legs. Spare sacral spine thalamus and corticospinal tracts Recovery from distal to proximal [toe flexion > toe extension > ankle > knee > hip] recovery rate 75%
  • 44. BROWN SEQUARD SYNDROME Hemisection of the spinal cord Motor paralysis , loss of vibration and proprioception on the ipsilateral side as the lesion and deficits in pain and temperature sensation on the contralateral side of the lesion. The most common cause of Brown- Séquard syndrome is penetrating trauma such as a gunshot wound or stab wound to the spinal cord. Best prognosis : More than 90% of people regain bladder & bowel control and the ability to walk.
  • 46. SPINAL SHOCK Immediate temporary loss of total power , sensation and reflexs below the level of injury Loss of bulbocavernosus reflex Usually recovery in 24-48 hrs

Notas do Editor

  1. C, Retropharyngeal soft tissue more than 5 mm on midsagittal image (arrow). D, a : indicates hemorrhage causing widening of soft tissue density at C3 level. b : indicates anterior annulus disruption. c : indicates disruption of ligamentum flavum.
  2. Distractive flexion Stage 1: Facet subluxation, gapping of the spinous process ligaments, with or without some blunting of anterosuperior vertebral body (like CF stage 1) Stage 2: Unilateral facet dislocation, usually PLC is intact, rotational deformity Stage 3: Bilateral facet dislocations, 50% translation of upper vertebral body on lower one Stage 4: Close to 100% translation of upper vertebral body on lower one, so-called floating vertebra
  3. F, Arrow a indicates hemorrhage at C3 level. Arrow b indicates disruption through anterior anulus and through the disc space
  4. sagittal T2-weighted image of an uninjured cervical spine small arrow is pointing to the PLL large solid arrow is pointing to the ligamentum flavum