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BOWEL OBSTRUCTION
By,
DR.SRINIVASAN RAMAN
Tbilisi state medical university.
BOWEL OBSTRUCTION OVERVIEW
 CLASSIFICATION
 COMMON CAUSES OF OBSTRUCTION
 CLINICAL FEATURES
 INVESTIGATION
 TREATMENT
INTRODUCTION
 Accounts for 5% of all acute surgical admissions
 Patients are often extremely ill requiring prompt assessment, resuscitation
and intensive monitoring
 Types:
 Obstruction:
 A mechanical blockage arising from a structural abnormality that presents
a physical barrier to the progression of gut contents.
 Ileus:
 is a paralytic or functional variety of obstruction
 Obstruction is:
 Partial or complete
 Simple or strangulated
CLASSIFICATION
Result from atony of the
intestine with loss of
normal peristalsis, in the
absence of a mechanical
cause.
or it may be present
in a non-propulsive
form (e.g. mesenteric
vascular occlusion or
pseudo-obstruction)
ADYNAMIC
(FUNCTIONAL)
Result from atony of the
intestine with loss of
normal peristalsis, in the
absence of a mechanical
cause.
or it may be present
in a non-propulsive
form (e.g. mesenteric
vascular occlusion or
pseudo-obstruction)
ADYNAMIC
(FUNCTIONAL)
TYPES OF BOWEL OBSTRUCTION
TYPES AND CAUSES OF DYNAMIC
OBSTRUCTION
Intraluminal
•Impaction
•Foreign bodies
•Bezoars
•Gallstone
Intramural
•Congenital atresia
•Stricture
•Malignancy(15%)
Extramural
•Bands/
adhesion(40%)
•Hernia (12%)
•Volvulus
•Intussusception
•Tumor-
benign/malignant
 Peritoneal irritation local fibrin production produces adhesions between
apposed surfaces
 As early as 4 weeks post laparotomy. The majority of patients present
between 1-5 years
Colorectal Surgery 25%
Gynaecological 20%
Appendectomy 14%
 Prevention: good surgical technique, washing of the peritoneal cavity with
saline to remove clots, etc, minimizing contact w/ gauze, covering
anastomosis & raw peritoneal surfaces
TREATMENT OF ADHESIVE
OBSTRUCTION
Initially treat conservatively provided there is no signs of
strangulation; should rarely continue conservative
treatment for longer than 72 hours
At operation, divide only the causative adhesion and
limit dissection
Laparoscopic adhesiolysis in cases of chronic
subacute obstruction
Hernia
 Accounts for 20% of SBO
 Commonest 1. Femoral hernia
2. ID inguinal
3. Umbilical
4. Others: incisional
 The site of obstruction is the neck of hernia
 The compromised viscus is with in the sac.
 Ischaemia occurs initially by venous occlusion, followed by oedema and
arterial compromise.
 Attempt to distinguish the difference between:
 Incarceration
 Sliding
 Obstruction
 Strangulation is noted by:
 Persistent pain
 Discolouration
 Tenderness
 Constitutional symptoms
Volvulus
A twisting or axial rotation of
a portion of bowel about its
mesentery. When complete it
forms a closed loop
obstruction ischemia
Commonest spontaneous
type in adult is sigmoid, can
be relieved by
decompression per anum
Surgery is required to prevent
or relieve ischaemia
Features: palpable tympanic lump
(sausage shape) in the midline or
left side of abdomen.
Constipation, abdominal
distension (early & progressive)
ACUTE INTUSSUSEPTION
Occurs when one portion of the gut becomes invaginated within an
immediately adjacent segment.
Common in 1st year of life
Common after viral illness enlargement of Peyer’s patches
Ileocolic is the commonest variety in child.
Colocolic intussusception commonest in adult
 An intussusception is composed of three parts :
 the entering or inner tube;
 the returning or middle tube;
 the sheath or outer tube (intussuscipiens).
 Classically, a previously healthy infant presents
with colicky pain and vomiting (milk then bile).
 Between episodes the child initially appears well.
 Later, they may pass a ‘redcurrant jelly’ stool.
LARGE BOWEL OBSTRUCTION
 Distinguishing ileus from mechanical obstruction is challenging
 Caecum is at the greatest risk of perforation
 Perforation results in the release of formed feaces with heavy bacterial
contamination
Aetiology:
1. Carcinoma:
The commonest cause, 18% of colonic ca. present with obstruction
2. Benign stricture:
Due to Diverticular disease, Ischemia, Inflammatory bowel disease.
3. Volvulus:
-Sigmoid Volvulus/ Caecal Volvulus
4. Hernia.
5. Congenital : HirschPrung, anal stenosis and agenesis
CLINICAL FEATURES
Large bowel obstruction
distension is early and pronounced.
Pain is mild and vomiting and dehydration are late.
The proximal colon and caecum are distended on abdominal radiography
CARDINAL FEATURES:
Colicky pain
Vomiting
Abd distention
Constipation
OTHER FEATURES:
Dehydration
Hypokalaemia
Pyrexia
Abd tenderness
PHYSICAL EXAMINATION
INSPECTION
Abdominal distention, scars, visible peristalsis.
PALPATION
Mass, tenderness, guarding
PERCUSSION
Tymphanic, dullness
AUSCULTATION
Bowel sound are high pitch and increase in frequency
INVESTIGATIONS:
 Lab:
 FBC (leukocytosis, anaemia, hematocrit, platelets)
 Clotting profile
 Arterial blood gasses
 U& Crt, Na, K, Amylase, LFT and glucose, LDH
 Group and save (x-match if needed)
 Optional (ESR, CRP, Hepatitis profile)
 RadiOlogical:
 Plain ABDOMINAL xrays
 USS ( free fluid, masses, mucosal folds, pattern of paristalsis, Doppler of mesenteric
vasulature, solid organs)
 Other advanced studies (CT, MRI, Contrast studieS)
Fluid levels with gas above;
‘stepladder pattern’. Ileal
obstruction by adhesions;
patient erect.
Supine radiograph from a patient with
complete small bowel obstruction
shows distended small bowel loops in
the central abdomen with prominent
valvulae conniventes (small white
arrow)
Figure 3. Lateral decubitus
view of the abdomen, showing
air-fluid levels consistent with
intestinal obstruction (arrows).
 In small bowel
 Central 3cm thick
diameter
 Vulvulae coniventae
 Ileum may occur
tubeless
 In large bowel
 Peripheral diameter 6cm
 Presence of haustration
ROLE OF CT
 Used with iv contrast, oral and rectal
contrast (triple contrast).
 Able to demonstrate abnormality in the
bowel wall, mesentery, mesenteric vessels
and peritoneum.
 It can define:
 the level of obstruction
 The degree of obstruction
 The cause: volvulus, hernia, luminal and
mural causes
 The degree of ischaemia
 Free fluid and gas
 Ensure: patient vitally stable with no renal
failure and no previous alergy to iodine
• FIGURE: AXIAL COMPUTED TOMOGRAPHY SCAN SHOWING
DILATED, CONTRAST-FILLED LOOPS OF BOWEL ON THE
PATIENT’S LEFT (YELLOW ARROWS), WITH DECOMPRESSED
DISTAL SMALL BOWEL ON THE PATIENT’S RIGHT (RED
ARROWS). THE CAUSE OF OBSTRUCTION, AN
INCARCERATED UMBILICAL HERNIA, CAN ALSO BE SEEN
(GREEN ARROW), WITH PROXIMALLY DILATED BOWEL
ENTERING THE HERNIA AND DECOMPRESSED BOWEL
EXITING THE HERNIA.
CONTRAST STUDIES
Barium should not be used in
a patient with peritonitis
 As: follow through, enema
 Limited use in the acute setting
 Gastrografin is used in acute
abdomen but is diluted
 Useful in recurrent and chronic
obstruction
 May able to define the level and
mural causes.
 Can be used to distinguish
adynamic and mechanical
obstruction
TREATMENT OF INTESTINAL
OBSTRUCTION
 Supportive
1. Resuscitation
2. Ryle tube free flow with 4 hourly aspiration
-Decompression of proximal to the obstruction, reduce subsequent aspiration
during induction of anesthesia and post extubation.
3. IV drip normal saline / Hartmann (Sodium & water loss during IO)
4. Broad spectrum antibiotic (not mandatory but need in all patient
undergoing surgery.
MANAGEMENT FOR LARGE BOWEL
OBSTRUCTION
All patients require
•Adequate resuscitation
•Prophylactic antibiotics
•Consenting and marking for potential stoma formation
•At operation
•Full laparotomy should be performed
•Liver should be palpated for metastases
•Colon should be inspected for synchronous tumours
•Appropriate operations include:
•Right sided lesions – right hemicolectomy
•Transverse colonic lesion – extended right hemicolectomy
•Left sided lesions – various options
INDICATIONS FOR SURGERY
 Generalised peritonitis
 Localised peritonitis
 Visceral perforation
 Irreducible hernia
 Palpable mass lesion
 Failure to improve
 Advanced malignancy
 Incomplete obstruction
 Advanced malignancy
SURGERY
Three-staged procedure
 Defunctioning colostomy
 Resection and anastomosis
 Closure of colostomy
Two-staged procedure
 Hartmann’s procedure: the
surgical resection of
the rectosigmoid colon with
closure of the rectal stump and
formation of an end colostomy. It
was used to treat colon
cancer or diverticulitis
 Closure of colostomy
Complications associated with
intestinal obstruction repair
 include excessive bleeding
 infection
 formation of abscesses (pockets of pus)
 leakage of stool from an anastomosis
 adhesion formation
 paralytic ileus (temporary paralysis of the intestines)
 reoccurrence of the obstruction.
PARALYTIC ILEUS
A state in which there is a failure of transmission of peristaltic
waves 2° to neuromuscular failure ( in Auerbach’s and
Meissner’s plexuses)
Stasis  leads to accumulation of fluid and gas within bowel
a/w distension, vomiting, absence of bowel sound and
absolute constipation
Varieties factors: postoperative, infection, reflex ileus and
metabolic
Radiological: gas filled loops of intestines with multiple fluid
levels
Management:
Essence of treatment prevention with use of
nasogastric suction and restriction of oral intake until
bowel sound and passage of flatus return
Maintain electrolyte balance
Specific treatment:
Removed primary cause
Decompressed GI distension
If prolong paralytic ileus , consider laparotomy exclude
hidden cause and facilitate bowel decompression
REFERENCE
 http://emedicine.medscape.com/article/930576-overview#aw2aab6b2b4
 http://www.patient.co.uk/doctor/intestinal-obstruction-and-ileus
 https://www.youtube.com
 https://en.wikipedia.org/wiki/Bowel_obstruction
 http://www.webmd.com/digestive-disorders/tc/bowel-obstruction-topic-
overview
 http://www.mayoclinic.org/diseases-conditions/intestinal-
obstruction/basics/symptoms/con-20027567
Bowel obstruction

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Bowel obstruction

  • 2. BOWEL OBSTRUCTION OVERVIEW  CLASSIFICATION  COMMON CAUSES OF OBSTRUCTION  CLINICAL FEATURES  INVESTIGATION  TREATMENT
  • 3. INTRODUCTION  Accounts for 5% of all acute surgical admissions  Patients are often extremely ill requiring prompt assessment, resuscitation and intensive monitoring  Types:  Obstruction:  A mechanical blockage arising from a structural abnormality that presents a physical barrier to the progression of gut contents.  Ileus:  is a paralytic or functional variety of obstruction  Obstruction is:  Partial or complete  Simple or strangulated
  • 4. CLASSIFICATION Result from atony of the intestine with loss of normal peristalsis, in the absence of a mechanical cause. or it may be present in a non-propulsive form (e.g. mesenteric vascular occlusion or pseudo-obstruction) ADYNAMIC (FUNCTIONAL) Result from atony of the intestine with loss of normal peristalsis, in the absence of a mechanical cause. or it may be present in a non-propulsive form (e.g. mesenteric vascular occlusion or pseudo-obstruction) ADYNAMIC (FUNCTIONAL)
  • 5. TYPES OF BOWEL OBSTRUCTION
  • 6. TYPES AND CAUSES OF DYNAMIC OBSTRUCTION Intraluminal •Impaction •Foreign bodies •Bezoars •Gallstone Intramural •Congenital atresia •Stricture •Malignancy(15%) Extramural •Bands/ adhesion(40%) •Hernia (12%) •Volvulus •Intussusception •Tumor- benign/malignant
  • 7.
  • 8.  Peritoneal irritation local fibrin production produces adhesions between apposed surfaces  As early as 4 weeks post laparotomy. The majority of patients present between 1-5 years Colorectal Surgery 25% Gynaecological 20% Appendectomy 14%  Prevention: good surgical technique, washing of the peritoneal cavity with saline to remove clots, etc, minimizing contact w/ gauze, covering anastomosis & raw peritoneal surfaces
  • 9. TREATMENT OF ADHESIVE OBSTRUCTION Initially treat conservatively provided there is no signs of strangulation; should rarely continue conservative treatment for longer than 72 hours At operation, divide only the causative adhesion and limit dissection Laparoscopic adhesiolysis in cases of chronic subacute obstruction
  • 10. Hernia  Accounts for 20% of SBO  Commonest 1. Femoral hernia 2. ID inguinal 3. Umbilical 4. Others: incisional  The site of obstruction is the neck of hernia  The compromised viscus is with in the sac.  Ischaemia occurs initially by venous occlusion, followed by oedema and arterial compromise.  Attempt to distinguish the difference between:  Incarceration  Sliding  Obstruction  Strangulation is noted by:  Persistent pain  Discolouration  Tenderness  Constitutional symptoms
  • 11. Volvulus A twisting or axial rotation of a portion of bowel about its mesentery. When complete it forms a closed loop obstruction ischemia Commonest spontaneous type in adult is sigmoid, can be relieved by decompression per anum Surgery is required to prevent or relieve ischaemia Features: palpable tympanic lump (sausage shape) in the midline or left side of abdomen. Constipation, abdominal distension (early & progressive)
  • 12.
  • 13. ACUTE INTUSSUSEPTION Occurs when one portion of the gut becomes invaginated within an immediately adjacent segment. Common in 1st year of life Common after viral illness enlargement of Peyer’s patches Ileocolic is the commonest variety in child. Colocolic intussusception commonest in adult  An intussusception is composed of three parts :  the entering or inner tube;  the returning or middle tube;  the sheath or outer tube (intussuscipiens).
  • 14.  Classically, a previously healthy infant presents with colicky pain and vomiting (milk then bile).  Between episodes the child initially appears well.  Later, they may pass a ‘redcurrant jelly’ stool.
  • 15.
  • 16. LARGE BOWEL OBSTRUCTION  Distinguishing ileus from mechanical obstruction is challenging  Caecum is at the greatest risk of perforation  Perforation results in the release of formed feaces with heavy bacterial contamination Aetiology: 1. Carcinoma: The commonest cause, 18% of colonic ca. present with obstruction 2. Benign stricture: Due to Diverticular disease, Ischemia, Inflammatory bowel disease. 3. Volvulus: -Sigmoid Volvulus/ Caecal Volvulus 4. Hernia. 5. Congenital : HirschPrung, anal stenosis and agenesis
  • 17. CLINICAL FEATURES Large bowel obstruction distension is early and pronounced. Pain is mild and vomiting and dehydration are late. The proximal colon and caecum are distended on abdominal radiography CARDINAL FEATURES: Colicky pain Vomiting Abd distention Constipation OTHER FEATURES: Dehydration Hypokalaemia Pyrexia Abd tenderness
  • 18. PHYSICAL EXAMINATION INSPECTION Abdominal distention, scars, visible peristalsis. PALPATION Mass, tenderness, guarding PERCUSSION Tymphanic, dullness AUSCULTATION Bowel sound are high pitch and increase in frequency
  • 19. INVESTIGATIONS:  Lab:  FBC (leukocytosis, anaemia, hematocrit, platelets)  Clotting profile  Arterial blood gasses  U& Crt, Na, K, Amylase, LFT and glucose, LDH  Group and save (x-match if needed)  Optional (ESR, CRP, Hepatitis profile)  RadiOlogical:  Plain ABDOMINAL xrays  USS ( free fluid, masses, mucosal folds, pattern of paristalsis, Doppler of mesenteric vasulature, solid organs)  Other advanced studies (CT, MRI, Contrast studieS)
  • 20. Fluid levels with gas above; ‘stepladder pattern’. Ileal obstruction by adhesions; patient erect. Supine radiograph from a patient with complete small bowel obstruction shows distended small bowel loops in the central abdomen with prominent valvulae conniventes (small white arrow) Figure 3. Lateral decubitus view of the abdomen, showing air-fluid levels consistent with intestinal obstruction (arrows).
  • 21.  In small bowel  Central 3cm thick diameter  Vulvulae coniventae  Ileum may occur tubeless  In large bowel  Peripheral diameter 6cm  Presence of haustration
  • 22. ROLE OF CT  Used with iv contrast, oral and rectal contrast (triple contrast).  Able to demonstrate abnormality in the bowel wall, mesentery, mesenteric vessels and peritoneum.  It can define:  the level of obstruction  The degree of obstruction  The cause: volvulus, hernia, luminal and mural causes  The degree of ischaemia  Free fluid and gas  Ensure: patient vitally stable with no renal failure and no previous alergy to iodine • FIGURE: AXIAL COMPUTED TOMOGRAPHY SCAN SHOWING DILATED, CONTRAST-FILLED LOOPS OF BOWEL ON THE PATIENT’S LEFT (YELLOW ARROWS), WITH DECOMPRESSED DISTAL SMALL BOWEL ON THE PATIENT’S RIGHT (RED ARROWS). THE CAUSE OF OBSTRUCTION, AN INCARCERATED UMBILICAL HERNIA, CAN ALSO BE SEEN (GREEN ARROW), WITH PROXIMALLY DILATED BOWEL ENTERING THE HERNIA AND DECOMPRESSED BOWEL EXITING THE HERNIA.
  • 23. CONTRAST STUDIES Barium should not be used in a patient with peritonitis  As: follow through, enema  Limited use in the acute setting  Gastrografin is used in acute abdomen but is diluted  Useful in recurrent and chronic obstruction  May able to define the level and mural causes.  Can be used to distinguish adynamic and mechanical obstruction
  • 24.
  • 25.
  • 26. TREATMENT OF INTESTINAL OBSTRUCTION  Supportive 1. Resuscitation 2. Ryle tube free flow with 4 hourly aspiration -Decompression of proximal to the obstruction, reduce subsequent aspiration during induction of anesthesia and post extubation. 3. IV drip normal saline / Hartmann (Sodium & water loss during IO) 4. Broad spectrum antibiotic (not mandatory but need in all patient undergoing surgery.
  • 27. MANAGEMENT FOR LARGE BOWEL OBSTRUCTION All patients require •Adequate resuscitation •Prophylactic antibiotics •Consenting and marking for potential stoma formation •At operation •Full laparotomy should be performed •Liver should be palpated for metastases •Colon should be inspected for synchronous tumours •Appropriate operations include: •Right sided lesions – right hemicolectomy •Transverse colonic lesion – extended right hemicolectomy •Left sided lesions – various options
  • 28. INDICATIONS FOR SURGERY  Generalised peritonitis  Localised peritonitis  Visceral perforation  Irreducible hernia  Palpable mass lesion  Failure to improve  Advanced malignancy  Incomplete obstruction  Advanced malignancy
  • 29. SURGERY Three-staged procedure  Defunctioning colostomy  Resection and anastomosis  Closure of colostomy Two-staged procedure  Hartmann’s procedure: the surgical resection of the rectosigmoid colon with closure of the rectal stump and formation of an end colostomy. It was used to treat colon cancer or diverticulitis  Closure of colostomy
  • 30.
  • 31. Complications associated with intestinal obstruction repair  include excessive bleeding  infection  formation of abscesses (pockets of pus)  leakage of stool from an anastomosis  adhesion formation  paralytic ileus (temporary paralysis of the intestines)  reoccurrence of the obstruction.
  • 32. PARALYTIC ILEUS A state in which there is a failure of transmission of peristaltic waves 2° to neuromuscular failure ( in Auerbach’s and Meissner’s plexuses) Stasis  leads to accumulation of fluid and gas within bowel a/w distension, vomiting, absence of bowel sound and absolute constipation Varieties factors: postoperative, infection, reflex ileus and metabolic Radiological: gas filled loops of intestines with multiple fluid levels
  • 33. Management: Essence of treatment prevention with use of nasogastric suction and restriction of oral intake until bowel sound and passage of flatus return Maintain electrolyte balance Specific treatment: Removed primary cause Decompressed GI distension If prolong paralytic ileus , consider laparotomy exclude hidden cause and facilitate bowel decompression
  • 34. REFERENCE  http://emedicine.medscape.com/article/930576-overview#aw2aab6b2b4  http://www.patient.co.uk/doctor/intestinal-obstruction-and-ileus  https://www.youtube.com  https://en.wikipedia.org/wiki/Bowel_obstruction  http://www.webmd.com/digestive-disorders/tc/bowel-obstruction-topic- overview  http://www.mayoclinic.org/diseases-conditions/intestinal- obstruction/basics/symptoms/con-20027567