Perplexities of Prameha and Parallel.pdf

PERPLEXITIES OF PRAMEHA AND
PARALLEL PERSPECTIVE
DR PRASANNA. N MOGASALE
MD(AYU)
SDM COLLEGE OF AYURVEDA
UDUPI

सामा यं ल णतया तेषां भूतािवलमू ता |
pnmogasale73@gmail.com

त आिवल भूतमू ल णाः सव एव मेहा भवि त ||
सु.िन.६/६
आिवलं समलम् | त ािवल वं मू य मेहदू याणां मेदोर मांसम जाशु ोदकवसालसीकौजसां
विचदेव मेहे क यिचदेव दू य य अवयव िम ीभावात् |
तेषामेव च दू याणां वैरेक भूत वात् मू भूत वम् |
अ. .िन.१०/७
मेह

( भूत आिवल)
( भूतमू ता)
( भूतािवलमू ता)

... मेदःसं यां तु च महे | िनि दतािन मेहाणां पूव पािण यािन च ||
च,िच.२८/१५
ि थवृि गलग डाबुदमेदोजौ कोप मधुमेहाित थौ याित वेद भृतयो मेदोदोषजाः;
सु.सू.२४/९
त मा मधुमेहश दः सव मेहे मधुमेहिवशेषे च वतते; यथा तृणश दः सवतृणे तृणिवशेषे च वतते|
च पािण च.िच.६/५७
Prameha is Meda pradoshaja vikara
मेह

मेदोवहानां ोतसां वृ कौ मूलं वपावहनं च |
च.िव.५/८
Vrikka - Adrenal gland
Adrenal glands produce hormones that help regulate metabolism,
immune system, blood pressure, response to stress and other
essential functions.
मेह

मेदोवहानां ोतसां वृ कौ मूलं वपावहनं च |
च.िव.५/८
वपावहनं वपा उदर था ि न धवितका
यामाहजना तैलवितक
े ित|
Vapavahana - Pancreas
The pancreas is a heterocrine gland. Functioning as an exocrine
gland, the pancreas excretes enzymes to break down the proteins,
lipids, carbohydrates, and nucleic acids in food. Functioning as
an endocrine gland, the pancreas secretes the hormones insulin
and glucagon to control blood sugar levels throughout the day.
मेह

अधोमुखोऽिप बि तिह मू वािहिसरामुखैः | पा यः पूयते सू मैः य दमानैरनारतम् ||
यै तैरेव िव यैनं दोषाः क
ु वि त िवंशितम् | मू ाघातान् मेहां क
ृ ा ममसमा यान् ||
अ. .िन.९/२-३
मेहा - मू रोग
Mootraroga are of two types:
§ Prabhoota mootrata: 20 Prameha.
§ Alpa mootrata: 20 mootraghata.

थूलः मेही बलवािनहैकः क
ृ श तथैकः प रदुबल |
च.िच.६/१५
मेह भेद
Prameha bheda:
§ Sthula prameha:
§ Krusha prameha:

ौ मेहौ भवतः- सहजोऽप यिनिम |
त सहजो मातृिपतृबीजदोषक
ृ तः, अिहताहारजोऽप यिनिम ः |
तयोः पूवणोप ुतः क
ृ शो ोऽ पाशी िपपासुभृशं प रसरणशील भवित;
उ रेण थूलो ब ाशी ि न धः श यासन व नशीलः ायेणेित ||
सु.िच.११/३
कृ शं का यािदल णयु ं सहजिम यथः|
थूलं थौ यािदधमयु मिहताहारिवहारजिम यथः|
मेह भेद
Prameha bheda:
§ Sahaja prameha:
§ Apathya nimittaja prameha:

क
ृ शं का यािदल णयु ं सहजिम यथः|
मेह भेद
Prameha bheda:
§ Sahaja prameha = Krusha pramehi
§ Apathya nimittaja prameha = Sthoola pramehi
थूलः मेही बलवािनहैकः क
ृ श तथैकः प रदुबल |
च.िच.६/१५

सपूव पाः कफिप मेहाः मेण ये वातकृ ता मेहाः |
सा या न ते, िप कृ ता तु या याः, सा या तु मेदो यिद न दु म् ||
च.िच.६/५६
त कफमेहाः सा या उ ा ते पूव पेण सह वतमाना असा या भवि त,
िप मेहा या या उ ा ते या येया भवि त|
तथा मेण च ये वातक
ृ ता ते पूव परिहता अ यसा याः|
मेह भेद
Prameha bheda:
§ Sa poorvaroopa prameha:
§ A poorvaroopa prameha:

उ ं च यथा- “अ य यािप च रोग य पूव पािण यं नरम् |
िवश यनेन क पेन त यािप मरणं ुवम्” (इ.अ.५) इित;
त सवपूव पानुब धे सवरोगाणामसा य वमु म्,
इह तु असम तपूव पानुब धेऽ यसा य वं भवतीित िवशेषवचना ल यते |
च पािण - च.िच.६/५६
मेह भेद
Prameha bheda:

पूव परिह येन िप कृ ते मेहे कदािचत् सा य वमिप भवतीित
अपवादिविधं दशियतुम्
उ सग ा ां पैि कया यताम् अनुवदित- िप कृ ता तु या या इित |
अव थासा यान् पैि कानाह- सा याि व यािद |
च पािण - च.िच.६/५६
मेह भेद
Prameha bheda:

च.िच.६/५६
अदु मेदसः पैि काः सा या भव ती यथः| िक
ं वा दु ाः
कफिप मेहा तावद् असा याः, तथा सपूव पाः स तोऽसा या भवि त |-
च पािण
मेह भेद
Prameha bheda:
§ Adusta meda prameha:
§ Pradusta meda prameha:

च.िच.६/५६
अदु मेदसः पैि काः सा या भव ती यथः| िक
ं वा दु ाः
कफिप मेहा तावद् असा याः, तथा सपूव पाः स तोऽसा या भवि त |-
च पािण
Prameha bheda:
§ Adusta meda prameha = A poorvaroopa prameha.
§ Pradusta meda prameha = Sa poorvaroopa prameha
मेह भेद

य तु खलु दोषाः क
ु िपताः मेहानिभिनवतिय य त इमािन पूव पािण दशयि त;
त था-
जिटलीभावं क
े शेषु, माधुयमा य य, करपादयोः सु तादाहौ, मुखतालुक ठशोषं,
िपपासाम्, आल यं, मलं काये, कायि छ ेषूपदेहं, प रदाहं सु तां चाङ
् गेषु,
षट्पदिपपीिलकािभ शरीरमू ािभसरणं, मू े च मू दोषान्, िव ं शरीरग धं, िन ां,
त ां च सवकालिमित ||
च.िन.४/४७
मेह भेद
Adusta meda prameha = Pradusta meda prameha

ADUSTA MEDA PRAMEHA = A POORVAROOPA PRAMEHA
In the early stages of the illness, when the glomerular filtration rate (GFR) is reduced but not
to levels below 35-50 per cent of normal, the patient is either asymptomatic or complains
only of generalized weakness. The clinical examination or investigations do not reveal any
significant abnormality. The patient is prescribed some tonics and his symptoms are often
dubbed as functional. On direct interrogation at this time many patients will come out with
the history of nocturia, which is one of the earliest manifestations of CRF and often ignored
by the patient as well as the physician.
Nocturia and polyuria are more likely to be early features of renal disease that
predominantly affect the medulla and in such conditions may be accompanied by
hyposthenuria rather than isosthenuria. Hyposthenuria when carefully looked for is a
common feature of CRF irrespective of the underlying disease.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5531136/
मेह भेद

ADUSTA MEDA PRAMEHA = A POORVAROOPA PRAMEHA
अधोमुखोऽिप बि तिह मू वािहिसरामुखैः | पा यः पूयते सू मैः य दमानैरनारतम् ||
यै तैरेव िव यैनं दोषाः क
ु वि त िवंशितम् | मू ाघातान् मेहां कृ ा ममसमा यान् ||
अ. .िन.९/२-३
अ. .िन.१०/७
मेह भेद

PRADUSTA MEDA PRAMEHA = SA POORVAROOPA PRAMEHA
मेह भेद
क
§ Sahaja prameha = Krusha pramehi
§ Apathya nimittaja prameha = Sthoola pramehi

SAHAJA PRAMEHA = KRUSHA PRAMEHI
मेह
तयोः पूवणोप ुतः क
ृ शो ोऽ पाशी िपपासुभृशं प रसरणशील भवित;
क
§ Beeja dosha is responsible for Sahaja prameha.
§ Generally emaciated/ Dhatu kshaya /not food craving/active.

स िह बीजदोषािदित; मेहार भकदोषदु बीजजात मेिह वात् |
च.िच.६/ ५७-च पािण
त सहजाः शु ातवदोषा वयाःI क
ु ाश मेहादयः |
िपतृजा मातृजा - अ.सं.सु.२२/१,२
क
ु लजा इित िपतृिपतामहािदकारणो ूताः|
च पािण – च.िच६/५७
§ Beeja dusti by Prameharambhaka dosha in the parents/grand parents.
मेह

सा यज ायं गभः| ...........
यािन ख व य गभ य सा यजािन, यािन चा य सा यतः स भवतः स भवि त, ता यनु या या यामः;
त था- आरो यमनाल यमलोलुप विमि य सादः वरवणबीजस पत् हषभूय वं चेित (सा यजािन)||
च.शा.३/११
असा यसेवां च गभ पघाितन दशयन् सा यसेवाया गभ ित कारण वं ढयित|
मेह
Saatmyaja bhava transforms information to the progeny.

त ो यते- य य य य ङ
् गावयव य बीजे बीज भाग उपत ो भवित, त य त याङ
् गावयव य
िवकृ ित पजायते, नोपजायते चानुपतापात्; त मादुभयोपपि र य |
च.शा.३/१७
बीज इित शु शोिणते | बीज याङ
् ग यङ
् गिनवतको भागो बीजभागः |
मनु यबीजं िह यङ
् गबीजभागसमुदाया मक
ं वस श यङ
् गसमुदाय पपु षजनकम्,
च पािण
§ Shukra shonita is Beeja which holds beeja bhaga.
§ Beeja bhaga - The factor which is responsible for different anga pratyanga.
§ Hence, Beeja is responsible for it’s replicate/ offspring.
§ Normalcy and abnormalcy of an Avayava depends on Beeja bhaga i.,e Beeja.
मेह

त ि िवधो बीजोपत ौ हेतुः- मातािप ोरपचारः, पूवक
ृ तं च कम;
तथाऽ येषामिप सहजानां िवकाराणाम् |
च.िच.१४/५
अपचार इित अनुिचताहारिवहार पः शु शोिणतभागिवशेषदूषकः|
पूवकृ तं च कमित जायमान य सहजाश ऽिभिनवतक
ं ज मा तरीयं कम |
त च कम दुबलं स मातािप पचारसिहतं बीजदूषक
ं भवित,
बलव ु िवनाऽपचारजनकमेव भवतीित ेयम् |
मेह
§ Karma decides Bheejopaghata
§ Karma results in
v Genetic disorders
v Mutation

त सहजाः शु ातवदोषा वयाःI क
ु ाश मेहादयः |
िपतृजा मातृजा
अ.सं.सु.२२/१,२
स िह बीजदोषािदित; मेहार भकदोषदु बीजजात मेिह वात् |
च.िच.६/ ५७-च पािण
क
ु लजा इित िपतृिपतामहािदकारणो ूताः|
च पािण – च.िच६/५७
मेह
§ Family history - Beeja dusti by Prameharambhaka dosha from the parents/grand parents.

मेह
APATHYA NIMITTAJA PRAMEHA = STHOOLA PRAMEHI
ि दोष कोपिनिम ा इित सवमेहे वेव ि दोषाः कारणम्,
अिधक वा च ैि मकािद यपदेश इित दशयित |
सु ुतेऽ यु ं - “सव एव मेहाः सवदोषजाः” (िन.अ.६) इित |
ि दोषकोपिनिम ा िवंशितः मेहा भवि त िवकारा ापरेऽप रसङ
् येयाः |
त यथा ि दोष कोपः मेहानिभिनवतयित तथाऽनु या या यामः ||
च.िन.४/३
§ Tridosha are responsible for all types of Prameha.
§ Vataja, Pittaja, Kaphaja prameha are based on Doshadhikyata.

य मपीनसमेहादीन् कफजान् कफसङ
् गतम् |
च.िच.१५/४८
आ यासुखं व नसुखं दधीिन ा यौदकानूपरसाः पयांिस |
नवा नपानं गुडवैक
ृ तं च मेहहेतुः कफक
ृ च सवम् ||
च.िच.६/४
§ Kapha dosha leads Prameha.
मेह

बह वः े मा दोषिवशेषः ||
च.िन.४/६
बह वः े मा दोषिवशेष इित बह व एव कफो मेहजनको ना प व इित |
त था- ै यशै यक डू थैयगौरव नेहसुि लेदोपदेहब धमाधुयिचरका र वािन े मणः कमािण;
च.सू.२०/१८
उदकादयो िह लेदनािदषु अ याः े ा इ यथः||
च.सू.२७/३५१-३५२-च पािणद
§ Kledadikyata of Prameha is because of Kapha dosha.
मेह

पुरीषमुप त भं वा वि नधारणं च, बि तपूरणिव लेदक
ृ मू ं, वेदः लेद व सौक
ु मायक
ृ त्; |
सु.सू.१५/५-२
बि तमू ाशयः, त य पूरणं, िव लेदमा वं च, मू ं करोती यथः|
ड हण क
ृ त िनब धसङ
् ह या या
अव भः पुरीष य, मू य लेदवाहनम् | वेद य लेदिवधृितः
अ. .सू.११/५
मू य सा यं ल यित-मू येित| लेदवाहनं- लेद य बिहिनगमनम्|
ीमद णद कृ तसवाङ
् गसु दरी या या
Basti - Avayava where
§ Mootram karoti – Formation of Urine
§ Kledasya bahir nirgamana – Elimination of Kleda/Mootra
BASTHI
मेह

... मेदःसं यां तु च महे |
िनि दतािन मेहाणां पूव पािण यािन च ||
च.सू.२८/१५
मेह
§ Medho dusti results in Sthoulya and Prameha.

कफः सिप ः पवन दोषा मेदोऽ शु ा बुवसालसीकाः |
म जा रसौजः िपिशतं च दू याः मेिहणां, िवंशितरेव मेहाः ||
च.िच.६/८
§ Dasha dooshya observed in Prameha.
मेह

य िप मेदः भृतयो दू या इह उ य ते, तथाऽिप
मेदोमांसशरीर लेदानामव य भािवतया प र हः |
म जादय सवमेहे न पुनरव यं दू य ते,
तोक
ं वा दु ा भवि त;
च पािण- च.िच.६/८
§ Among the dooshya - Meda, Mamsa and Shareeraja kleda are invariably involved
in all types of Prameha.
§ Involvement of other dooshya are not mandatory for all varieties of Prameha.
मेह

§ Kapha Prameha = Kapha pradhana dosha + Vata, Pitta, Meda (Mamsa, Kleda).
§ Pitta Prameha = Pitta pradhana dosha + Vata, Kapha, Shonita, Meda (Mamsa, Kleda).
§ Vata Prameha = Vata pradhana dosha + Kapha, Pitta,Vasa, Majja, Meda (Mamsa, Kleda).
त वातिप मेदोिभरि वतः े मा े म मेहा जनयित,
वातकफशोिणतमेदोिभरि वतं िप ं िप मेहान्,
कफिप वसाम जमेदोिभरि वतो वायुवात मेहान् ||
सु.िन.६/९
मेह

ब ब ं मेदो मांसं शरीरज लेदः शु ं शोिणतं वसा म जा लसीका रस ौजःसङ
् यात इित दू यिवशेषाः ||
च.िन.४/७
अब िमित असंहतम् | अ तु बह वमघन वं च यथायो यतया बो यम् |
बि त तु थूलगुदमु कसेवनीशु मू वािहनीनां नाडी(ली)नां म ये मू धारोऽ बुवहानां सव ोतसामुदिध रवापगानां ित ा,
च.सी.९/४
अ बुवहानां ोतसां ित ेित थानिम यथः, उदिधयथाऽऽपगानां पूरणीय थानं, तथा बि तर य बुवह ोतसां िव ाम थानम् |
च पािणद
मेह
§ An excess of Abaddha Medha, Mamsa, Shukra, Shonita, Vasaa, Majja, Laseeka,
Rasa, Ojas mixes with Shareeraja Kleda resulting excess of Shareeraja kleda.
§ Hence, Prabhoota Avila Mootrata.

त य च एवं वृ य अप रप वा एव वातिप े माणो यदा मेदसा सहैक वमुपे य
मू वािह ोतां यनुसृ याधो ग वा ब तेमुखमाि य िनिभ ते तदा मेहा जनयि त ||
सु.िन.६/४
अप रप वा आमाः | मेदसेित अ ािपश दो लु ो यः, तेन वसादयो ा ाः; अ ये ित ‘मेद ाप रप वं’ इित
पठि त, चकारेणानु ानिप वसादीन् समुि च वि त | उपे य ग वा| िनिभ ते िनःसर ती यथः |
मेह
§ Ahara and Achara Vitiates Kapha pradhana dosha acts as external milieu for the
disease Prameha.
§ Derangement in the Metabolism of Meda dhatu ( Medasca Aparipakwa) acts as
internal milieu for the disease Prameha.

रसिनिम मेव थौ यं का य च |
त े मलाहारसेिवनोऽ यशनशील य
अ यायािमनो िदवा व नरत य
चाम एव
अ नरसो मधुरतर
शरीरमनु ाम नित नेहान्
मेदो जनयित,
तदित थौ यमापादयित; ......
सु.सू.१५/३२
मेह
िदवा व ना यायामाल य स ं
शीति न धमधुरमे वा नपानसेिवनं पु षं
जािनयात् मेही भिव यतीित ||
त य च एवं वृ य अप रप वा एव (अप रप वा आमाः/ मेद ाप रप वं)
वातिप े माणो यदा मेदसा सहैक वमुपे य
मू वािह ोतां यनुसृ याधो ग वा ब तेमुखमाि य
िनिभ ते तदा मेहा जनयि त ||
सु.िन.६/3,४
आ यासुखं व नसुखं दधीिन ा यौदकानूपरसाः पयांिस | नवा नपानं गुडवैक
ृ तं च मेहहेतुः कफक
ृ च सवम् ||
च.िच.६/४
... मेदःसं यां तु च महे | िनि दतािन मेहाणां पूव पािण यािन च ||
च.सू.२८/१५

‘आम एवेित इवाथ ऽयमेवश दः, र ािद पेणाप रणततया अप व इवे यथः | न तु ‘आमाशय थः काया नेद ब यादिवपािचतः’
इ यािदनो ः, त य रोगहेतुतयाऽऽमाशय थ वेन च मेदोजनक वायोगात्’ इित |
च पािण
स यं, जाठरेणाि नना रसः क ावेन क
ृ त एव, िक तु धा वि निभरपाकादाम इ यु यते |
ड हण
§ चाम एव/ अप रप वा एव / ‘मेद ाप रप वं = Metabolic error – Not digestive error
मेह

िवंशित मेह

ि दोषकोपिनिम ा िवंशितः मेहा भवि त िवकारा ापरेऽप रसङ
् येयाः |
त यथा ि दोष कोपः मेहानिभिनवतयित तथाऽनु या या यामः ||
च.िन.४/३
दोषदू यािवशेषेऽिप त संयोगिवशेषतः || मू वणािदभेदेन भेदो मेहेषु क यते |
अ. .िन.१०/७,८
दोषदू यािवशेषेऽिप-समान वेऽिप सित,
तयोः-दोषदू ययोः, संयोगः-सं ेषः, त संयोगः,
त य िवशेषः- ा क
ृ तकमवशा ेदो, दोषदू ययोः संयोगानेक विम यथः|
त मा मू य यो वणािदभेदो-वणग धरस पशािदिवशेषः,
तेन भेदेन भेदो मेहेषु क यते-िन यते|
मेह

उदकमेह - DIABETES INSIPIDUS
अ छं बह िसतं शीतं िनग धमुदकोपमम् | े मकोपा नरो मू मुदमेही मेहित ||
च.िन.४/१३
उदकोपमिमित उदकवणािदतु यम्|
त शीतं तेमग धम छमुदकिमवोदकमेही मेहित |
अ.सं.िन१०/९
Urine excreted in large quantity which is transparent, white, cold and odourless

Diabetes insipidus (DI) is defined as the passage of large volumes (>3 L/24 hr) of dilute
urine. Urinary specific gravity is between 1 to 1.005. It has 2 major forms:
Central (neurogenic, pituitary, or neurohypophyseal) DI, characterized by decreased
secretion of antidiuretic hormone (ADH; also referred to as arginine vasopressin [AVP])
Nephrogenic DI, characterized by decreased ability to concentrate urine because of
resistance to ADH action in the kidney.
Etiology
DI is usually an acquired disorder, with central DI having different causes than does
nephrogenic DI. In rare cases, central or nephrogenic DI may be an inherited disorder.
https://emedicine.medscape.com/article/117648-overview
उदकमेह - DIABETES INSIPIDUS

अ यथमधुरं शीतमीषि पि छलमािवलम् | का डे ुरसमङ
् काशं े मकोपात् मेहित ||
च.िन.४/१४
इ ुवािलकारसमेह - KETOACIDOSIS
Moderately turbid urine which is cold, little slimy, extremely sweet like sugarcane juice.

इ ुवािलकारसमेह - KETOACIDOSIS
Diabetic ketoacidosis (DKA) is an acute, major, life-threatening complication of
diabetes characterized by hyperglycemia, ketoacidosis, and ketonuria.
It occurs when absolute or relative insulin deficiency inhibits the ability of glucose to
enter cells for utilization as metabolic fuel, the result being that the liver rapidly breaks
down fat into ketones to employ as a fuel source. The overproduction of ketones ensues,
causing them to accumulate in the blood and urine and turn the blood acidic.
DKA occurs mainly in patients with type 1 diabetes, but it is not uncommon in some
patients with type 2 diabetes.

शीतमेह- KETOACIDOSIS
अ यथमधुरं शीतं मू ं मेहित यो भृशम् | शीतमेिहनमाह तं पु षं े मकोपतः ||
च. िन.४/१९
शीतमेही सुबहशो मधुरं भृशशीतलम् ||
अ. .िन.१०/१२
शीतमेही सु बहशो मधुरमितशीतलं च मेहित|
Turbid urine passed frequently which is cold and extremely sweet.

शीतमेह- KETOACIDOSIS
Hypothermia is reduction in core temperature below 95°F (35°C), with the most
accurate reading taken by rectally. (Med Sci Law 1969;9[4]:231.)
Hypothermia can be caused by starvation, malnutrition, immobility, myocardial
infarction, pulmonary embolism, and endocrine disorders such as hypopituitarism
and hypothyroidism.
A British study determined that DKA-induced hypothermia (11.8% of all cases of
hypothermia admissions) is more common than hypothyroidism-induced
hypothermia (8% of all cases of hypothermia admissions). (Br Med J
1978;2[6149]:1387.)

य य पयुिषतं मू ं सा ीभवित भाजने | पु षं कफकोपेन तमाहः सा मेिहनम् ||
च.िन.४/१३,१४, १५
आिवलं सा ं सा मेही;
सु.िन.६/१०
आिवलम यथ कलुषं,
सा ीभवे पयुिषतं सा मेहेन मेहित|
अ. .िन.१०/१०
सा मेहेन य मेहित त पयुिषतं-राि ि थतं, घनीभवेत्|
सा मेह - PROTEINURIA
Turbid urine which gets condensed over time/over night.

Proteinuria identifies patients with renal damage and those at risk for worsening
renal disease and increased cardiovascular morbidity. An individual with
proteinuria in the setting of a normal glomerular filtration rate (GFR) is at high risk
of progressive loss of renal function.
The normal mean albumin excretion rate (AER) is 5-10 mg/day, with an AER of
>30 mg/day considered abnormal. AER between 30 to 300 mg/day is called
moderately increased albuminuria. Levels greater than 300 mg/day are called
severely increased albuminuria.
Albuminuria that persists for 3 months is considered CKD. Nephrotic-range
proteinuria is defined as greater than 3.5 g of protein excreted in the urine over 24
hours.

Proteinuria can be differentiated on the basis of any of the following:
Amount of protein (nephrotic or non-nephrotic)
Type of protein (albuminuria or low molecular weight proteinuria)
Underlying pathological damage (glomerular vs non-glomerular).
Pathophysiologically, most cases of proteinuria are classified into one or more of
the following categories:
Tubular
Overflow
Glomerular

सा सादमेह - PROTEINURIA
य य संह यते मू ं िकि चत् िकि चत् सीदित | सा सादमेहीित तमाहः े मकोपतः ||
च. िन.४/१६
संह यते यानीभवित |
घनं चोप र िव स निम यनेन सा सादमेहमाह |
Urine becomes partly viscous and partly clear when kept overtime.

सुरामेह - PROTEINURIA
सुरामेही सुरातु यमुपय छमधो घनम् ||
अ. .िन.१०/१०
सुरामेही सुरातु यं मेहित| त च मू ं पयुिषतमेवोप रभागेऽ छमधः-अध तात्, घनं भवेत्|
सुरामेही सुरातु यमुपय छमधो घनम् || मा.िन.३३/९
चरक
े सुरामेह थाने सा सादमेहः पिठतः
Looks like a spirituous liquor that is transparent above and dense below.

फ
े नमेह - PROTEINURIA
तोक
ं तोक
ं सफ
े नम छं फ
े नमेही मेहित ||
Eliminates urine little by little, being frothy and clear.

Polyuria/ Nocturia is observed in CKD even at grade three and four.
In early stage it is the osmotic diuresis. But, it is suggested that at later stage of
chronic renal failure the mechanism of a diuresis increase is not due to osmotic
diuresis but rather to secretion of prostaglandin E2 which inhibits cation
reabsorption and stimulates diuresis.
https://pubmed.ncbi.nlm.nih.gov/10900642/
सा मेह/ सा सादमेह / सुरामेह/ फ
े नमेह - PROTEINURIA

शु मेह - SPERMATURIA
शु ाभं शु िम ं वा मुहमहित यो नरः | शु मेिहनमाह तं पु षं े मकोपतः ||
च. िन.४/१८
Turbid urine as a consequence of Spermaturia.

शु मेह - SPERMATURIA
Etiology:
Retrograde ejaculation
Enlarged prostate
Alcohol
Diabetes mellitus
https://www.lybrate.com/topic/spermaturia-causes-prevention-of-it/8659428dcf044c2483ff33f263959916

त तुब िमवालालं िपि छलं यः मेहित | आलालमेिहनं िव ा ं नरं े मकोपतः ||
च. िन.४/२२
त तुब ं त तुव ीघिम यथः| लालािमवालालं, सम ता लाला पिम यथः|
आलालमेह - CHYLURIA
Turbid urine appears thready and contains slimy object.

म दं म दमवेगं तु क
ृ ं यो मू ये छनैः | शनैमिहनमाह तं पु षं े मकोपतः |
च. िन.४/२१
शनैः सकफ
ं मृ नं शनैमही;
मृ नं िपि छलम्|
शनैमह– CHYLURIA
Turbid urine passed frequently with difficulty.
Mostly urine contains Kapha/slimy content.

आलालमेह/शनैमह– CHYLURIA
Chyluria denotes the urinary excretion of chyle, which is a lymphatic fluid rich in
chylomicrons.
Chyle flows from the intestinal lacteals to the left subclavian vein through the
thoracic duct. When an abnormal connection between these structures and the urinary
tract develops, chyluria appears.
Chyluria is classified as parasitic or nonparasitic, the former being induced by
lymphatic filariasis, whereas the latter is caused by medical, traumatic or inherited
diseases.
The patient usually reports excretion of milky urines, monolateral flank pain,
malnutrition, weight loss and weakness. Urinalysis demonstrates nephrotic-range
proteinuria, lymphocyturia associated with chylomicrons and triglycerides in the
supernatant.
https://www.karger.com/article/fulltext/329154

िसकतामेह- CYSTINURIA
मूता मू गतान् दोषानणू मेहित यो नरः | िसकतामेिहनं िव ा ं नरं े मकोपतः ||
च. िन.४/२०
मूतािनित किठनान् | दोषािनित जातौ बहवचनं, येन दोषोऽ ैक एव भूतः कफः|
स जं िसकतानुिव ं िसकतामेही;
िसकतानुिव ं वालुकािम म् |
Turbid urine is passed along with small gravels.

Cystinuria is an autosomal-recessive defect in re-absorptive transport of cystine
and the dibasic amino acids ornithine, arginine, and lysine from the luminal fluid
of the renal proximal tubule and small intestine. The only phenotypic
manifestation of cystinuria is cystine urolithiasis, which often recurs throughout an
affected individual’s lifetime.
Worldwide, the overall prevalence is 1 person per 7000 population but varies
significantly by population.

The presentation in patients with cystinuria is similar to that of patients with other
types of renal calculi and includes the following:
Renal colic, Chronic urinary tract infections in a young person with a family
history of kidney stones, Passage of stones or gravel, Hematuria & Dysuria.
Cystine is one of the sulfur-containing amino acids; therefore, the urine may have
the characteristic odor of rotten eggs.
Urinalysis may show typical hexagonal or benzene crystals, which are essentially
pathognomonic of cystinuria. Microscopic crystalluria is present in 26%-83% of
patients. Disappearance of cystine crystals in the first morning urine is a good
index of treatment efficacy.

िप मेह - PHOSPHATURIA
रोमा िप रसतु यं िप मेही;
रोमा ा रोम चः| िप रसतु यं िप िम ोदकतु यम् |
सं रोमा िप ेन िप व हलं िसतम् |
अ. .िन.१०/११
िप ेन-िप मेहेन, सं रोमा सन् िप वत्-िप स शं, बहलं शु लं च मेहित |
Turbid urine resembling flour solution and accompanied with horripilation.

शु लमेह- PHOSPHATURIA
शु लं िप िनभं मू मभी णं यः मेहित | पु षं कफकोपेन तमाहः शु लमेिहनम् ||
च. िन.४/१७
Turbid urine having the opaque, white color like that of pasted flour.

िप मेह/शु लमेह - PHOSPHATURIA
Phosphaturia is caused due to excess of excretion of phosphate in urine. Presence
of too much of phosphate makes the urine appear milky or cloudy in appearance.
Any cause of primary hyperparathyroidism will cause phosphaturia and tend to
cause hypophosphatemia. Hyperparathyroidism caused by hypocalcemia or
vitamin D deficiency or resistance is similarly associated with hypophosphatemia.
Extracellular volume expansion increases the filtered phosphorus load and dilutes
the luminal concentration of phosphorus, resulting in phosphaturia. Ethanol and
glycosuria both decrease proximal tubule phosphate reabsorption.

िप मेह/शु लमेह - PHOSPHATURIA
Fanconi syndrome is a syndrome of inadequate reabsorption in the proximal renal
tubules of the kidney. The syndrome can be caused by various underlying congenital or
acquired diseases, by toxicity (for example, from toxic heavy metals), or by adverse
drug reactions.
It results in various small molecules of metabolism being passed into the urine instead
of being reabsorbed from the tubular fluid such as glucose, amino acids, uric acid,
phosphate, and bicarbonate.
Different forms of Fanconi syndrome can affect different functions of the proximal
tubule, and result in different complications. The loss of phosphate results in the bone
diseases rickets and osteomalacia (even with adequate vitamin D and calcium levels),
because phosphate is necessary for bone development in children and even for ongoing
bone metabolism in adults.

ग धवणरस पशयथा ार तथािवधम् | िप कोपा नरो मू ं ारमेही मेहित ||
च.िन.४/२९
ारमेह - ALKALINURIA
Urine has the smell, color, taste and touch similar to those of alkali.

ारमेह - ALKALINURIA
Alkaline urine has traditionally been associated with Proteus urinary tract infections.
In the absence of an infection, clinical findings might suggest other diagnoses.
The following are some of the traditional causes of alkaline urine:
(1) Urinary tract infection (Proteus and others)
(2) Metabolic alkalosis (pyloric stenosis and others)
(3) Failure of acidification (renal tubular acidosis, chronic renal failure, or aldosterone
abnormalities)
(4) Ingestion (salicylate, sodium bicarbonate, acetazolamide, etc); and
(5) Respiratory alkalosis (hyperventilation).
https://jamanetwork.com/journals/jamapediatrics/article-abstract/515482

कालमेह -ALKAPTONURIA (BLACK URINE DISEASE)
मसीवणमज ं यो मू मु णं मेहित | िप य प रकोपेण तं िव ात् कालमेिहनम् ||
च.िन.४/३०
Turbid urine passed in large quantity of black or dark urine

कालमेह -ALKAPTONURIA (BLACK URINE DISEASE)
Alkaptonuria is a rare inherited disorder. It occurs when body can’t produce
enough of an enzyme called homogentisic dioxygenase (HGD). This enzyme is
used to break down a toxic substance called homogentisic acid. The buildup of
homogentisic acid causes bones and cartilage to become discolored and brittle.
This typically leads to osteoarthritis, especially in spine and large joints. People
with alkaptonuria also have urine that turns dark brown or black when it’s
exposed to air.

चाषप िनभं मू म लं मेहित यो नरः| िप य प रकोपेण तं िव ा नीलमेिहनम् ||
च.िन.४/३१
सफ
े नम छं नीलं नीलमेही मेहित |
सु.िन.६/११
नीलमेह - INDICANURIA
Turbid urine with sour taste and color like that of the feather of the blue jay.

नीलमेह - INDICANURIA
Blue diaper syndrome is a rare, autosomal or X-linked recessive trait metabolic
disorder characterized in infants by bluish urine-stained diapers. It is also
known as Drummond's syndrome, and Hypercalcemia, Familial, with
Nephrocalcinosis and Indicanuria.
Indican (potassium indoxyl sulphate) is a normal constituent of urine in
amounts up to 30 mg per day. It is formed from indole derived from the action
of intestinal bacteria on tryptophan. It is usually invisible, but urines containing
a large excess of indican may darken on exposure to air and very occasionally
oxidation to indigo in alkaline urine may produce a bluish tinge to the
phosphate deposit. In such cases, typical blue rectangular or needlelike crystals
may be seen in the deposit.

िव ं लवणमु णं च र ं मेहित यो नरः| िप य प रकोपेण तं िव ा मेिहनम् ||
च.िन.४/३२
र मेह - HAEMATURIA
Turbid urine having red color, saline taste and smell like that of raw meat.

POLYCYSTIC KIDNEY DISEASE
It is a genetic disorder in which the renal tubules become structurally abnormal,
resulting in the development and growth of multiple cysts within the kidney. These cysts
may begin to develop in utero, in infancy, in childhood, or in adulthood. Cysts are non-
functioning tubules filled with fluid pumped into them, which range in size from
microscopic to enormous, crushing adjacent normal tubules and eventually rendering
them non-functional as well.
PKD is a general term for two types, each having their own pathology and genetic cause:
autosomal dominant polycystic kidney disease (ADPKD) and autosomal recessive
polycystic kidney disease (ARPKD).
Signs and symptoms include high blood pressure, headaches, abdominal pain, blood in
the urine, and excessive urination. Other symptoms include pain in the back, and cyst
formation (renal and other organs).

ALPORT SYNDROME
The term Alport syndrome encompasses a group of inherited, heterogeneous
disorders involving the basement membranes of the kidney and frequently
affecting the cochlea and eye as well.
There are various forms of the disease which includes Autosomal recessive Alport
syndrome (ARAS), X-linked Alport syndrome (XLAS), Autosomal dominant
Alport syndrome (ADAS).

ALPORT SYNDROME
Kidney-related manifestations
Hematuria - Most common and earliest manifestation of Alport syndrome
Proteinuria - Develops in males with XLAS and in males and females with ARAS
and ADAS
Hypertension - Usually present in males with XLAS and in males and females with
ARAS and ADAS.
Edema and nephrotic syndrome are present in 30-40% of young adults with Alport
syndrome; they are not common in early childhood, but their incidence
progressively increases with age. With onset of kidney insufficiency, symptoms of
chronic anemia and osteodystrophy may become evident.

FAMILIAL GLOMERULAR HEMATURIA – TBM
Thin basement membrane nephropathy (TBM) disease also known as benign
familial hematuria along with IgA nephropathy, the most common cause of blood
in the urine without any other symptoms.
The only abnormal finding in this disease is a thinning of the basement membrane
of the glomeruli (filters) in the kidneys.
Most patients with TBM disease maintain normal kidney function throughout
their lives.

माि ज मेह - HEMOGLOBINURIA
मि ज ोदकसङ
् काशं भृशं िव ं मेहित | िप य प रकोपा ं िव ा माि ज मेिहनम् ||
च.िन.४/३३
Turbid urine passed frequently, which smells like raw flesh and looks like the
decoction of Manjistha.

Hemoglobinuria is a condition in which the oxygen transport protein hemoglobin
is found in abnormally high concentrations in the urine.
The condition is caused by excessive intravascular hemolysis, in which large
numbers of red blood cells (RBCs) are destroyed, thereby releasing free
hemoglobin into the plasma.
Excess hemoglobin is filtered by the kidneys, which excrete it into the urine,
giving urine a purple color.

§ Acute glomerulonephritis
§ Burns
§ Renal cancer
§ Malaria
§ Paroxysmal nocturnal hemoglobinuria
§ Microangiopathies, e.g. hemolytic-uremic syndrome (HUS), thrombotic thrombocytopenic
purpura (TTP) leading to microangiopathic hemolytic anemia
§ Transfusion reactions
§ IgM autoimmune hemolytic anemia
§ Glucose-6-phosphate dehydrogenase deficiency
§ Pyelonephritis
§ Sickle cell anemia
§ Tuberculosis of the urinary tract
§ March hemoglobinuria secondary to repetitive impacts on the body, usually the feet
§ Athletic nephritis secondary to strenuous exercise
§ Acute lead poisoning

हा र मेह - UROBILINOGENURIA
ह र ोदकसङ
् काशं कटुक
ं यः मेहित | िप य प रकोपा ं िव ा ा र मेिहनम् ||
च.िन.४/३४
कटुक
ं सदाहं ह र ायु िमव हा र मेही |
अ.सं.िन.१०/१३
Turbid urine having pungent taste and color like that of the juice of Haridra

हा र मेह - UROBILINOGENURIA
Excess of urobilinogen may be due to excessive formation of bilirubin by cells
of the reticulo-endothelial system on account of
(1)Excessive red cell destruction, as in acute malaria.
(2)Excessive hemolysis, as in pernicious anemia or poisoning by drugs, etc.
(3)Excessive absorption of the products of blood disintegration, as in internal
hemorrhage anywhere, or in lobar pneumonia.

वसामेह - LIPIDURIA
वसािम ं वसाभं वा मुहमहित यो नरः| वसामेिहनमाह तमसा यं वातकोपतः||
च.िन.४/४१
Turbid urine mixed with Vasaa (serum of flesh) or having the appearance of Vasaa.

वसामेह - LIPIDURIA
Lipiduria or lipuria is the presence of lipids in the urine.
Lipiduria is most frequently observed in nephrotic syndrome where it is passed
as lipoproteins along with other proteins.
Congenital nephrotic syndrome is a rare kidney disease which manifests in
infants during the first 3 months of life, and is characterized by high levels of
protein in the urine (proteinuria), low levels of protein in the blood, and
swelling. This disease is primarily caused by genetic mutations which result in
damage to components of the glomerular filtration barrier and allow for leakage
of plasma proteins into the urinary space

म जानं सह मू ेण मुहमहित यो नरः| म जमेिहनमाह तमसा यं वातकोपतः||
च.िन.४/४२
सिपः काशं सिपमही मेहित |
सु.िन.६/१२
वातजे सिपमह रक
े म जमेहः
म जमेह / सिपमह – LIPIDURIA/CHYLURIA
Turbid urine mixed with Majja (marrow of bones)

म जमेह / सिपमह - LIPIDURIA/CHYLURIA
Nephrotic syndrome is the combination of nephrotic-range proteinuria with a low
serum albumin level and edema. Nephrotic-range proteinuria is the loss of 3 grams or
more per day of protein into the urine or, on a single spot urine collection, the
presence of 2 g of protein per gram of urine creatinine.
Nephrotic syndrome has many causes, including primary kidney diseases such as
minimal-change disease, focal segmental glomerulosclerosis, and membranous
glomerulonephritis. Nephrotic syndrome can also result from systemic diseases that
affect other organs in addition to the kidneys, such as diabetes, amyloidosis, and
lupus erythematosus.
Nephrotic syndrome may affect adults and children of both sexes and of any race.

हि तमेह - AMINOACIDURIA
ह ती म इवाज ं मू ं रित यो भृशम् | हि तमेिहनमाह तमसा यं वातकोपतः||
च.िन.४/४३
ह ती म इवाज ं मू ं वेगिवविजतम् | सलसीक
ं िवब ं च हि तमेही मेहित ||
अ. .िन.१०/ १७-१८; मा.िन.३३/१७
हि तमेहमाह– ह ती यािद| अज मनवरतं, वेगिवविजतं वेगरिहतं, सलसीक
ं लसीकया सह, िवब ं िप डीभूतिमव |
लसीका िवलीनमेदः ायम्|
Large quantities of turbid urine frequently like an elephant gone amuck.

The term aminoaciduria is applied when more than 5% of the filtered load is detected
in the urine.
It may result from an inherited metabolic abnormality, in which case the
aminoaciduria is a permanent finding, or it may be an acquired abnormality which
may either be transient or become permanent.
Only negligible amounts of amino acids and glucose are normally present in the final
urine, reflecting very efficient reabsorption mechanisms for these organic solutes in
the proximal tubule. Renal tubular transport defects or specific metabolic
abnormalities result in excretion of significant quantities of amino acids & glucose in
the urine.

Fanconi's syndrome is the most frequently studied inherited aminoaciduria. The
syndrome is characterized by a generalized aminoaciduria and by other renal
tubular defects affecting reabsorption of phosphate and glucose.
Hartnup disease is a rare autosomal recessive metabolic disorder where renal
tubular transport is defective and causes gross aminoaciduria.

मधुमेह / ौ मेह – DIABETES MELLITUS
कषायमधुरं पा डु ं मेहित यो नरः | वातकोपादसा यं तं तीया मधुमेिहनम् ||
च.िन.४/४४
मधुमेही मधुसमम्--------- | अ. .िन.१०/१८
ौ रसवण ौ मेही; …..| सु.िन.६/१२
ौ मेहो मधुमेहः|
Diabetes comes from a Greek
word that means to siphon. The
most obvious sign of diabetes is
excessive urination. Water passes
through the body of a person with
diabetes as if it were being
siphoned from the mouth through
the urinary system and out of the
body.

मधुमेह / ौ मेह – DIABETES MELLITUS
कषायमधुरं पा डु ं मेहित यो नरः | वातकोपादसा यं तं तीया मधुमेिहनम् ||
च.िन.४/४४
मधुमेही मधुसमम्--------- | अ. .िन.१०/१८
ौ रसवण ौ मेही; …..| सु.िन.६/१२
ौ मेहो मधुमेहः|
Mellitus comes from a Latin word
that means sweet like honey. The
urine of a person with diabetes
contains extra sugar (glucose). In
1679,the physician Thomas Willis,
tasted the urine of a person with
diabetes and described as
‘wonderfully sweet’ like honey.

मधुमेही मधुसमम् जायते स िकल ि धा ||
ु े धातु या ायौ दोषावृतपथेऽथवा |
अ. .िन.१०/१८,१९
आवृतो दोषिलङ
् गािन सोऽिनिम ं दशयेत् ||
ीणः णा णात् पूण भजते क
ृ सा यताम् |
अ. .िन.१०/१९,२०
अिनिम ं-अक मात्,
मेह
Modes of manifestation of Madhumeha:
1. Dhatu kshaya: Progressive manifestation - ीणः णा णात् पूण भजते
2. Margaavarana: Sudden onset - सोऽिनिम ं (अक मात्) दशयेत्

गु ि न धा ललवणा यितमा ं सम ताम् | नवम नं च पानं च िन ामा यासुखािन च ||
य यायामिच तानां संशोधनमक
ु वताम् |
च.सू.१७/७८, ७९
े मा िप ं च मेद मांसं चाित वधते || तैरावृतगितवायुरोज आदाय ग छित |
यदा बि तं तदा क
ृ ो मधुमेहः वतते || स मा त य िप य कफ य च मुहमुहः |
दशय याकृ ितं ग वा यमा यायते पुनः ||
च.सू.१७/७९ -८१
मेह
Modes of manifestation of Madhumeha:
Kapha Pitta Margavarana to Vata dosha.
Results in Madhumeha.

य तु मागावरणेन वायुः क
ु यित, स आवरकधिमतया ायेण सामो भवित, त च लङ
् घनं मा या कत यमेव |
च.िच.३/१३९ – च पािण
आमम नरसं क
े िचत्, क
े िच ु मलस चयम् | थमां दोषदुि ं च क
े िचदामं च ते ||
मधुकोश या या मा.िन.२५/१-५
मेह
§ Margaavarana: Sudden onset - सोऽिनिम ं (अक मात्) दशयेत् .
§ Saama dosha itself is considered as Margavarana.
§ Dosha dusti janya Ama is responsible for Prameha. But, onset must be sudden.

मेह
िव ैरिप न वेते गुणै नि त पर परम् | दोषाः सहजसा य वाि षं घोरमहीिनव ||
च.िच.२६/२९३
पर परगुणोपघात तु य िप दोषाणां ायो ना येव, तथाऽ य वशात् विच वतीित ेयं;
च.िव.१/१०-च पािण
§ Dosha are having opposite guna.
§ Dosha guna does not fight each other in an individual as they are Satmya to self.
§ Sometime, dosha guna may fight each other and hence disease.

मेह
अ ये दोषे य एवाितदु े योऽ यो यमू छनात् | को वे यो िवष येव वद याम य स भवम् ||
अ. .सू.१३/२६
य धातु देशे वि म दो भवित त ैवामस भवाि पिडका ु पि ः यात् |
मा.िन.६/२२ - आतङ
् कदपण या या
§ Anyonya moorchana of dusta dosha or fight between dosha guna results in
disease.
§ This causes dhatu dusti / dhatwagni dusti.
§ Hence, Prameha.

य तु मागावरणेन वायुः क
ु यित, स आवरकधिमतया ायेण सामो भवित, |
च.िच.३/१३९ – च पािण
अ ये दोषे य एवाितदु े योऽ यो यमू छनात् | को वे यो िवष येव वद याम य स भवम् ||
अ. .सू.१३/२६
Type 1 diabetes is a chronic illness characterized by the body’s inability to produce insulin due to the
autoimmune destruction of the beta cells in the pancreas. Although onset frequently occurs in childhood,
the disease can also develop in adults.
Currently, autoimmunity is considered the major factor in the pathophysiology of type 1 DM. In a
genetically susceptible individual, viral infection may stimulate the production of antibodies against a
viral protein that trigger an autoimmune response against antigenically similar beta cell molecules.
मेह

उप व & पूव प

उप वा तु खलु मेिहणां तृ णातीसार वरदाहदौब यारोचकािवपाकाः
पूितमांसिपडकालजीिव यादय त सङ
् गा वि त ||
च.िन.४/४८
मेह उप व
• Thirst, diarrhea, fever, burning sensation, weakness, anorexia, and indigestion.
• Carbuncles that putrefy muscle tissues, alaji and vidradhi, appear during the
chronic stage of the disease.

मि कोपसपणमाल यं मांसोपचयः ित यायः शैिथ यारोचकािवपाकाः कफ सेक छिदिन ाकास ासा ेित े मजानामुप वाः;
वृषणयोरवदरणं बि तभेदो मे तोदो िद शूलम लीका वरातीसारारोचका वमथुः प रधूपनं दाहो मू छा िपपासा िन ानाशः पा डु रोगः
पीतिव मू ने वं चेित पैि कानां;
द् हो लौ यमिन ा त भः क पः शूलं ब पुरीष वं चेित वातजानाम् |
एवमेते िवंशित मेहाः सोप वा या याताः ||
सु.िन.६/१३
‘शोषः कासः ास ेित वातजानामुप वा भवि त’ इित ता.|
मेह उप व
Sushruta briefed the complication under doshic classification.
Kaphaja – Mostly GIT & Cardio-respiratory symptoms.
Pittaja - Mostly GIT, Circulatory, Urinary, CNS, Haemopoietic system
Vataja – Cardio-respiratory, CNS, Locomotor and Anorectal symptoms.

वेदोऽङ
् गग धः िशिथलाङ
् गता च श यासन व नसुखे रित |
ने िज ा वणोपदेहो घनाङ
् गता क
े शनखाितवृि ः||
शीति य वं गलतालुशोषो माधुयमा ये करपाददाहः|
भिव यतो मेहगद य पं मू ेऽिभधावि त िपपीिलका ||
च.िच.६/१३,१४
य तु खलु दोषाः क
ु िपताः मेहानिभिनवतिय य त इमािन
पूव पािण दशयि त; त था- जिटलीभावं क
े शेषु, माधुयमा य य,
करपादयोः सु तादाहौ, मुखतालुक ठशोषं, िपपासाम्, आल यं,
मलं काये, कायि छ ेषूपदेहं, प रदाहं सु तां चाङ
् गेषु,
षट्पदिपपीिलकािभ शरीरमू ािभसरणं, मू े च मू दोषान्, िव ं
शरीरग धं, िन ां, त ां च सवकालिमित ||
च.िन.४/४७
मेह पूव प
LOOKS LIKE COMPLICATIONS:
§ करपादयोः सु तादाहौ/प रदाहं सु तां चाङ
् गेषु,
§ मलं काये, कायि छ ेषूपदेह
§ मू े च मू दोषान्,

मेह पूव प

तेषां यि कि चदाहारिवहारजातं े ममेदोमू जननं स हेतुः||
अ.सं.िन.१०/३
तेषां मेदोमू कफावहम् ||
अ नपानि याजातं य ाय त वतकम् |
वा ललवणि न धगु िपि छलशीतलम् ||
नवधा यसुरानूपमांसे ुगुडगोरसम् | एक थानासनरितः शयनं िविधविजतम् ||
अ. .िन.१०/१-३
मेह िनदान – मू जनक आहार
End product of fat digestion is not water soluble.
End product of Protein is mostly water soluble.
End product of carbohydrates are definitely water soluble.

Perplexities of Prameha and Parallel.pdf

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