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Jessica Cuervo Bustamante
          Medicine Student
               III Semester
             ID 000171223
                      2011
 Time    takes to ce    u
                o p ro d
               ta mutation
 produce ation
 T
          kes
  ime ta ut
        a m
 depends of the
              d s of th
                        e
      depen er of
 number ofrred and
          numb
               ccu
     rrors o  Depending
 errors occurred and
    e
               d.
    co rrecte umber of
 corrected. Depending
                 n
       on the s, we will
               ion
       mutat f there is
 on the number of i
          know  fficiency
 mutations, we ms of
          a good syste will
                     e
                 air
 know ifetherellis
         of r p e ce .
                 th
  a good efficiency
 of repair systems of
 the cell.
 ScienceDaily   (Jan. 20,
 2011)

 The dual role
that Cdc7 plays
    in the cell
  lifecycle and
    DNA repair
      offers a
    promising
    target for
     potential
      cancer
    therapies.
 Studyof enzymes involved in DNA
 repair is essential to learn about
 new advances in treatments for cancer and
 other pathologies associated with the genetic
 mutation
ScienceDaily (Feb. 11,
2011) 
 Know the different methods of combination of
 Chk1 and Src inhibitors are important in
 explaining the behavior of pathologies such as
 multiple myeloma and to develop techniques to
 improve treatment and promote new
 discoveries about how to remove without
 damaging attack DNA healthy cells, in the
 mechanism of action of carcinogenic diseases.
 knowing molecules involve
 d in the development
 of skin cancer will allow us
 to approach the various
  prevention methods that
 can be implemented to
 reduce mortality rates
  DNA repair mechanisms are important
 markers of certain incurable diseases that
 cause great damage in cells, tissues and
 organs mutations are rapidly acquired resista
 nce to the repair of the genome which
 is usually lethal to human been.
The best condition is
associated with dama
ge to DNA repair is
cancer, and it takes s
pecial care and
operation of the
mechanisms that
control the damage
being as well
essential for the
diagnosis and
treatment.
   —MARTINEZ S.Lina María. Biología molecular. 6. ed.
    Medellín : UPB. Fac. de Medicina,  2009. 81 p.
 
   —University of North Carolina School of Medicine (2011,
    January 20). Chaperone enzyme provides new target for
    cancer treatments. ScienceDaily. Retrieved February 12,
    2011, from
    http://www.sciencedaily.com­/releases/2011/01/1101181
    43220.htm
 
   —Virginia Commonwealth University (2011, February 11).
    Scientists combine targeted agents to kill multiple
    myeloma cells.ScienceDaily. Retrieved February 12, 2011,
    from
    http://www.sciencedaily.com­/releases/2011/02/1102101
    53014.htm
Folding Jessica Cuervo Bustamante

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Folding Jessica Cuervo Bustamante

  • 1. Jessica Cuervo Bustamante Medicine Student III Semester ID 000171223 2011
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  • 4.  Time takes to ce u o p ro d  ta mutation produce ation T kes ime ta ut a m depends of the d s of th e depen er of number ofrred and numb ccu rrors o  Depending errors occurred and e d. co rrecte umber of corrected. Depending  n on the s, we will ion mutat f there is on the number of i know  fficiency mutations, we ms of  a good syste will  e air know ifetherellis of r p e ce . th  a good efficiency of repair systems of the cell.
  • 5.  ScienceDaily (Jan. 20, 2011)
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  • 8.  The dual role that Cdc7 plays in the cell lifecycle and DNA repair offers a promising target for potential cancer therapies.
  • 9.  Studyof enzymes involved in DNA repair is essential to learn about new advances in treatments for cancer and other pathologies associated with the genetic mutation
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  • 14.  Know the different methods of combination of Chk1 and Src inhibitors are important in explaining the behavior of pathologies such as multiple myeloma and to develop techniques to improve treatment and promote new discoveries about how to remove without damaging attack DNA healthy cells, in the mechanism of action of carcinogenic diseases.
  • 15.
  • 16.  knowing molecules involve d in the development of skin cancer will allow us to approach the various  prevention methods that can be implemented to reduce mortality rates
  • 17.   DNA repair mechanisms are important markers of certain incurable diseases that cause great damage in cells, tissues and organs mutations are rapidly acquired resista nce to the repair of the genome which is usually lethal to human been.
  • 18. The best condition is associated with dama ge to DNA repair is cancer, and it takes s pecial care and operation of the mechanisms that control the damage being as well essential for the diagnosis and treatment.
  • 19. —MARTINEZ S.Lina María. Biología molecular. 6. ed. Medellín : UPB. Fac. de Medicina,  2009. 81 p.    —University of North Carolina School of Medicine (2011, January 20). Chaperone enzyme provides new target for cancer treatments. ScienceDaily. Retrieved February 12, 2011, from http://www.sciencedaily.com­/releases/2011/01/1101181 43220.htm    —Virginia Commonwealth University (2011, February 11). Scientists combine targeted agents to kill multiple myeloma cells.ScienceDaily. Retrieved February 12, 2011, from http://www.sciencedaily.com­/releases/2011/02/1102101 53014.htm