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GENERAL PHYSICAL EXAMINATION
1. B Y - D R N I T E S H K U M A R M D M E D I C I N E
GPE
2. GPE
Complete assessment of a patient’s physical and
mental status.
Systematic collection of objective information that is
directly observed or is elicited through examination
techniques.
3. OBJECTIVES
To understand the physical and mental well being of
the patients.
To detect diseases in its early stage.
To determine the cause and extent of the diseases.
To determine the nature of the treatment or nursing
care needed .
To contribute to the medical research.
4. GENERAL PRINCIPLES OF PHYSICAL
EXAMINATION
Introduce yourself
Explain the purpose and nature of the examination
Obtain the patient’s consent
Quite, warm and well light room.
Privacy
Reassure and relax the patient
Gentleness
Always plan the examination relevant to the patient
Further questioning if abnormal finding
Always examined from right side of the bed
5. General appearance
Consciousness,
cooperation &
orientation
Attitute and posture
Build and nutrition-
Height,Weight, BMI,
Anthropometric features,
Congenital anomalies.
Others--hair, face, oral
cavity, nail, extremites
6. FACIES
Specific diagnosis can be
made by just looking at a
patient’s face.
Some facial
characteristics are so
typical of certain diseases
that they immediately
suggest the
diagnosis….so called
diagnostic facies……
7.
8. Oral Cavity
The teeth and breath
Check the oral cavity looking for
MOUTH ULCERS
-Aphtous,drugs and trauma
-gastrointestinal disease;inflammatory bowel
disease,coeliac disease
-rheumatological;Behcets syndrome,reiter
-erythema multiforme
-infections;herpes zoster,simplex,syphilis,tuberculosis
9.
10. Gum hypertrophy
Phenytoin
Pregnancy
Scurvy(vitamin C
deficiency; gums become
swollen, spongy, red and
bleeds easily)
Gingivitis; smoking
leukemia
14. PULSE
The pulse is a wave form that is felt by fingers and
produced by cardiac systole which travels through
the peripheral arterial tree in peripheral direction at
a rate more faster than that of blood columm.
Time lag from cardiac systole:
Carotid: 30ms
Radial: 80ms
Femoral: 75ms
Brachial: 60ms
15. IMPORTANCE:
Also called the mirror of heart
Information about arterial wall condition
Rough estimation of SBP n DBP
State of heart n circulation
Detect and diagnosis of arrhythmia
Diagnosis in case of AR and acute LVF
16. The arterial pulse should be examined in all 4 limbs and
both sides of the neck
1. Radials
2. Brachials
3. Carotids
4. Femorals
5. Popliteals
6. Temporal
7. Facial
8. Peripheral arteries of the legs :Dorsalis Pedis Posterior
tibial
17. How to feel the Pulse
The Radial pulse:
The 3 middle fingers are used
The palmar surface of the fingers overlies the radial
A. and encircles the wrist
At first the artery is completely occluded, then
gradually release the pressure until maximum feeling
of the pulse wave is perceived.
18. Examination of pulses
Rate ---- radial pulse used generally
Rhythm --- radial pulse used generally
Character --The carotid artery
Volume -- The carotid artery
Symmetry -- Compare arteries both side
Radio femoral delay -- Compare radial and femoral
pulse
Condition of vessels
20. Pulse: Rhythm
Regular-- Count for at least half a minute
Irregular
Regularly irregular
Irregularly irregular
21. Carotid pulse
Be careful
Always one at a time
Stimulating its
baroreceptors with low
palpitation
Severe Bradycardia
Even stop the heart
Pulse;character/volume
22. Pulse Volume
• Volume is the movement imparted to your fingers
and reflects the pulse pressure - the difference
between systolic and diastolic blood pressure
best assessed by palpating the carotid artery
Pulse pressure ;
30 to 60 mm Hg--normal
< 30 mm Hg, ----small
>60 mm Hg, ---large
Pulse volume depends on stroke volume and arterial
compliance
23. Character
• Character is the impression of the pulse waveform
obtained
best assessed in the carotid arteries except.
bisferiens pulse , pulsus alternans, are more
evident in peripheral arteries
25. Hypokinetic Pulse --Small
weak pulse
(small volume and narrow
PP).
Hyperkinetic Pulse---A high
amplitude pulse with a rapid
rise
(large volume and wide PP)
26. Anacrotic Pulse-A low
amplitude pulse (parvus)
with a slow rising and late
peak (tardus).
Pulsus bisferiens --is a single
pulse wave with two peaks in
systolebest felt in brachial and
femoral artery
27. Collapsing Pulse (Water-Hammer
Pulse, Corrigans Pulse)
It is a large volume pulse
with a rapid upstroke
(systolic pressure is high)
and a rapid downstroke
(diastolic pressure is low).
The rapid upstroke is
because of an increased
stroke volume.
The rapid downstroke is
because of diastolic run-off
into the left ventricle, and
decreased peripheral
resistance and rapid run-
off to the periphery.
28. Condition of Arterial Wall
• Young adult : Not Palpable
Old Individuals , Hypertensive patients Palpable
First place the index and middle fingers of both
hands over the radial artery side by side and
exsanguinate the artery by moving the two fingers in
opposite direction. The radial artery is now rolled
over the radius by two index fingers
29. Brachial pulse
Flex the patient’s elbow
Feel the pulse just
medial to biceps tendon
Carotid Arteries
•At the level of thyroid
cartilage
Peripheral pulsations
30. The Carotids
The patient lies down with the head of the bed
elevated 30 degrees
Carotid pulsations may be visible just medial to
sternomastoid
Place the left thumb on the right carotid A. in the
lower third of the neck at the level of the cricoid
cartilage, just inside the medial border of the
sternomastoid and press posteriorly
Never press both carotids at same time
31. The dorsalis pedis artery
Lateral to the extensor hallucis
tendon
Absent in 10%
The posterior tibial pulse.
32. femoral artery- beneath the
inguinal ligament ,about mid
way between ASIS and SP
Popliteal artery- Patient knee
should be flexed –leg relaxed
Place the finger tips of both
hands so that they meet in the
middle line behind the knee
and press them deeply in the
popliteal fossa
33. RADIO-RADIAL & RADIO-FEMORAL
DELAY
Delay of the left radial
compared with the right
radial is found in
THORACIC INLET
SYNDROME :
CERVICAL RIB
,TAKAYASU’S DISEASE
Delay of the femoral
compared with the right
radial pulse is found in
coarctation of the aorta
34. 72 beats per min, regular rhythm, normal volume
and normal character, all peripheral pulses are well
felt, no radioradial or radiofemoral delay, no vessel
wall thickening
35. Blood pressure
The pressure exerted by circulating blood on the
walls of blood vessels
Affected by exertion, anxiety, excitement and
changes in body posture, cuff size
The first number is the systolic blood pressure
reading, and it represents the maximum pressure
exerted when the heart contracts
The second number is the diastolic blood pressure
reading, and it represents the pressure in the arteries
when the heart is at rest.
36. Cuff size guidelines a
device used for
measuring arterial
pressure. Mercury,
aneroid, electric
Cuff Arm cirum
Range at mid point
Adult 27-34 cm
Large Adult 35-44 cm
Adult thigh 45-52 cm
Cuff
Sphygmomanometer
39. TEMPERATURE
Whether or not a patient is febrile is often obvious by
palpation of the forehead with the dorsum of the
hand
But the exact temperature has to be recorded in
doubtful cases to document fever and to assess the
height of temp using a clinical thermometer
40. Temperature
.=Core
oral
rectal
=Surface
axillary
groin
Body temperature
Normal value
Oral 36.30C~37.20C
Rectal 36.50C~37.70C
Axillary 360C~370C
The normal body temperature varies from
person to person, by age, and through out day
Being lowest in the early hours of the
morning and highest in the afternoon
The variation may range within 10C
Rectal T >Oral T> Axillary T (each in 0.50C)
41. Respiratory rate
It is expansion and relaxation of of chest wall
Normal value is : 16 – 22 bpm (Adult)
Rate
Depth
Type
42. Respiratory rate– counted by placing the examiners
palm over the patient’s abdomen ,noting the rise and
fall of the abdomen
Note-simultaneously divert the patient’s attention
Types –abdominothoracic-(male)
thoracoabdominal(female)
Method- keep two hands flat , one on the chest and
other on the abdomen and watch for movements of
hands
44. Pallor
Paleness may be the result of decreased blood
supply to the skin (cold, fainting, shock,
hypoglycemia) or decreased number of red blood
cells (anemia)
Depends upon
Thickness of skin
Quality of skin
Amount and quality of blood in capillaries.
45. (must be compared with both side
and must be observed in day light)
1. Lower palpebral
conjuntiva
2. Dorsum of tongue
3. Mucous membrane
of mouth
4. Nail bed
5. Palm of the hand
6. Sole of the feet
Sites where pallor is seen
46. ICTERUS
Yellowish discolouration of skin,mucous
membrane and sclera.
Due to increased bilirubin.
Yellowish discolouration of sclera is due to the
high elastin content in the sclera
Normal level- o.3-1 mg/dl
Latent jaundice- below 3 mg/dl
Clinical jaundice- more than 3 mg/dl
47. Sites
Upper sclera
Ventral surface of
tongue (between lingual
vein and frenulum)
Nail bed
Palm of the hand
Sole of feet
48. Differential diagnosis of yellowish
discolouration of skin
Carotenoderma (Increased intake of carrots,
oranges and leafy vegetables): here there is no
yellowish discolouration of sclera
Quinacrine
Chronic exposure to phenols
Long standing anemia
49. CYANOSIS
Cyanosis is the bluish discolouration of skin and
mucous membrane.
It results from the increased amount of reduced
hemoglobin the the blood.
Manifests when the reduced hemoglobin is >5g/dL
Cyanosis may be masked in severe anemia
50. Types of cyanosis
Central
Imperfect
oxygenation of the
blood e.g Heart
failure , COAD
Admixture of
venous with arterial
blood e.g
Congenital heart
diseases
Peripheral
Excessive reduction
of oxyhaemoglobin in
the capillaries when
the blood flow is
slowed
Vasoconstrictionexpos
ure to cold
Arterial obstruction
Low cardiac output
51. Sites :
Tip of tongue (central cyanosis)
Lips
Tips fingers and toes
Tip and alae of nose Central+Peripheral
Earlobules Cyanosis
52. Central cyanosis Peripheral cyanosis
1)mechanism = Low SpO2
2)sites = Tip of tongues
3)Extremities= Warm
4)Application of
heat= No effect
5)Administration
of O2 = Cyanosis disappears
Poor peripheral circulation
Other sites
Cold
Cyanosis disappears
No effect
Differences
53. CLUBBING
It is bulous swelling of subungal
connective tissue at onychodermal
angle
It is loss of onychodermal angle
(Lovi’s Bond), Normally < 160
Increase in the soft tissue of the base of
the nail
Drumstick appearance of the tip of the
finger
HOW TO ELICIT?
i. Fluctuation test
. Schamroth’s sign (Diamond shape)
56. Oedema
Accumulation of fluid in interstitial space.
Types:
Pitting type: Apply firm pressure on the shin of
tibia or 2cm above the medial malleolus for 30 s and
see for pitting.
Causes: Congestive cardiac failure, nephrotic
syndrome, liver cirrhosis, hypoproteinemia
Non pitting type:Lymphatic obstruction , Deep
vein thrombosis, Myxoedema, Scleroderma
57. How edema can be recognized?
Inspection:
Pallid and glossy
appearance of the skin at
the swollen part
Palpation:
Doughy feeling
Pitting on finger pressure
(the pressure of the
finger should be
maintained for 30
seconds)
58. Lymph Node
Lymphadenopathy- Enlargement of lymphnode
Distributed all over the body
Important part in the body’s defense against
infection
59. Distribution of lymph nodes
neck
Submental
Submandibullar
Pre- Post-auricular
Ant- Post cervical
chain
Occipital
Supraclavicular
(Virchow’s gland)
61. Points to be noted
Site
Number of nodes
Size
Consistency- hard, firm, rubbery, soft
Tenderness
Discrete or confluent
Mobile or fixed
Condition of overlying skin (local temp,
discharging
sinuses)
62. JVP:
Jugular Venous Pulse ;
defined as the oscillating top of vertical column of
blood in right IJV that reflects the phasic pressure
changes in Right Atrium in cardiac cycle.
• Jugular Venous Pressure:
Vertical height of oscillating column of blood .
63. Right atrial pressure during
systole and right ventricular
filling pressure during diastole
are producing pulsation and
pressure waves in jugular veins.
Evaluation of JVP offers a
window into the right heart,
providing critical information
regarding its hemodynamics.
64. Causes of Elevated JVP
1. Unilateral non-pulsatile
Innominate vein thrombosis
2. Bilateral non-pulsatile
SVC obstruction
Massive right sided pleural
effusion
3. Bilateral pulsatile
a. Cardiac
Cardiac failure
Tricuspid stenosis
Tricuspid regurgitation
Constrictive pericarditis
Cardiac tamponade
b. Pulmonary : COPD/cor
pulmonale
c. Abdominal :
Ascites
Pregnancy
d. Iatrogenic Excess IV fluids
Low jugular venous
pressure
Hypovolaemia.
66. Lateral to carotid
artery & deep to
sternomastoid
muscle.
External jugular is
superficial to
sternomastoid
67.
68. Examination of JVP
Right IJV is usually assessed both for waveform and
estimation of venous pressure
Unlike EJV pulsation, it is not possible to see IJV
pulsation directly as it is deep.
We actually see the transmitted pulsations to
overlying skin between two heads of
sternocleidomastoid.
69. Why Internal Jugular Vein?
• IJV has a direct course to RA.
• IJV is anatomically closer to RA.
• IJV has no valves( Valves in EJV prevent
transmission of RA pressure)
• external jugular vein is more superficial & prone to
kinking and partial obstruction as it traverses
the deep fascia of the neck.
• Vasoconstriction Secondary to hypotension ( in
CCF) can make EJV small and barely visible.
70. Right IJV Preferred :Why?
Right IJV have straight line course through innominate
vein to the svc and right atrium
Less likely extrinsic compression from other structures in
neck.
Left innominate vein compressed by arch of Aorta and
Presence of Left SVC can falsely elevate the JVP on Left
side.
Left IJV drains into Lt innominate vein, which is not in
straight line from RA.
Why not EJV-
No or less numbers of valves in IJV than EJV
71. Differences between IJV and Carotid pulses
Superficial and lateral in
the neck
Better seen than felt
Has two peaks and two
troughs
Descents >obvious than
crests
Digital compression
abolishes venous pulse
Jugular venous pressure
falls during inspiration
Abdominal compression
elevates jugular pressure
Deeper and medial in the
neck
Better felt than seen
Has single upstroke only
Upstroke brisker and
visible
Digital compression has no
effect
Do not change with
respiration
Abdominal compression
has no effect on carotid
pulse
72. Method Of Examination
• The patient should be comfortable during the examination.
• Clothing should be removed from the neck and upper
thorax.
There should not be any tight bands around abdomen
• Patient reclining with head elevated 45 °
Ensure that the neck muscles are relaxed by resting the back
of the head on a pillow. Neck should not be sharply flexed &
slightly rotated towards the opposite side
• Examined effectively by shining a light tangentially across
the neck from the right side of the patient
• → Identify the internal jugular pulsation in between two
heads of SCM , (DIFFERENTIATE it from CAROTID
PULSATION)
73. WHY 45° ?? FOR BETTER
VISUALISATION
• (A) Supine: jugular vein
distended, pulsation not
visible.
(B) Reclining at 45°:
point of transition
between distended and
collapsed vein can
usually be seen to pulsate
just above the clavicle.
(C) Upright: upper part
of vein collapsed and
transition point obscured
74. Jugular venous pressure
Level of sternal angle is about 5 cm above the level of
mid right atrium IN ANY POSITION.
JVP is measured in ANY position in which top of the
column is seen easily.
Usually JVP is less than 8 cm water
< 3 cm column above level of sternal angle.
75. Measurement of JVP
Two scale method is commonly used ( one scale at
the upper level of the JVP ,parallel to the ground and
second scale at the level of sternal angle
,perpendicular to the first scale.
Normally JV pressure does not exceed 3- 4 cm above
the sternal angle
Since RA is approximately 5 cm below the sternal
angle ,
the jugular venous pressure corresponds to 9 cm
=7mmhg
Elevated JVP : JVP of >4 cm above sternal angle
76.
77.
78. Hepatojuglar reflux
Useful diagnostic maneuver when –
1. JVP is borderline elevated
2. Latent RVF
3. Silent TR is suspected
Maneuver:-
Gently apply firm pressure to the periumblical region for
10 – 30 sec with pt lying comfortably and breathing
quietly, while JVP is observed.
Pressure shouldn’t applied over the Liver in Rt
hypochondrium region ,as it may be painful in presence of
hepatic congestion.
79. What happened in Normal
Subjects:-
JV pressure rises transiently
(<15 sec.) to <3cm while
abdominal pressure is
continued, because Normal
RV is able to receive the
augmented venous return to
Rt heart without a rise in
mean venous Pressure.
80. Positive Response
A Sustained rise of >3cm in venous pressure for at
least 15 sec after resumption of spontaneous
respiration is a positive response.
A positive test result indicates the inability of the
right heart to handle an increased venous return.
81. Most common cause of a positive test is RHF/
(incipient Heart Failure )
• Positive test in: Borderline elevation of JVP
Silent TR
Latent RHF
• False Negative: SVC/IVC obstruction
Budd Chiari syndrome
• Positive Test imply SVC and IVC are patent