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Head and Spinal Injury
Dr. Kayonde Nathan M.
Brain protection
• Bony skull
• Falx cerebri
• Tentorium cerebelli
• Cerebrospinal fluid
Injuries to brain arise due to differences in
motion of the skull and brain
Clinical details
• Loss of consciousness
• Glasgow coma scale
• Skull x-ray
• CT scan
Scalp injuries
• Injuries bleed profusely
• Blunt trauma may result in apparently “clean
cut” laceration, but bruising at edges and hair
driven into wound.
• Bruises easily palpable, shave hair to visualise
fully. “Hair hides”
“Black eye”
May be due to:
• Direct blow into the orbit
• An injury to the front of the scalp, draining
down over the supraorbital ridge
• A fracture of the base of the skull (direct or
contrecoup) allowing meningeal hemorrhage
to escape through the orbital roof
Skull Fracture
Skeleton of the head consists of three main
components:
• Mandible
• Facial skeleton
• Calvarium
Skull distorts when force is applied to it. Fracture
represents site of maximum stress on skull and
may not be at site of impact
Brain injury
• Skull fracture is a marker for the severity of force
applied to head.
• In general there is a correlation between them
• In the absence of fracture, rotary movements of
head cause shear strains within brain resulting in
diffuse axonal injury, contusion and laceration
• Cerebral oedema and haemorrhage are secondary
effects
• By direct intrusion, either by a foreign object
such as a penetrating weapon, bullet or other
missile – or fragments of skull in a compound
fracture where the skull is disrupted
• By deformation of the brain in closed head
injuries
Cerebral injuries (types)
• Cerebral contusion-When either linear or, more
often, laminar stresses are applied to the cortex,
this soft tissue may disrupt
• Cerebral laceration-Laceration of the cortex is an
extension in severity of contusion in which
mechanical separation of the tissue can be seen.
• Traumatic intracerebral hemorrhage-Substantial
areas of hemorrhage, either infiltrating the brain
tissue or forming actual hematomas
• Primary brainstem hemorrhage-Secondary
brainstem bleeding is dealt with under ‘cerebral
oedema’ so here we are concerned with
hemorrhage that occurs at the time of injury
• Coup and contrecoup damage
• Concussion-is a clinical, not a pathological entity,
but the pathologist must consider it, as it is
related to intracranial lesions and he is often
questioned about it in court proceeding
• Diffuse neuronal and axonal injury-diffuse
brain damage exists in four principal forms:
diffuse vascular injury, diffuse axonal injury,
hypoxic brain damage and diffuse brain
swelling
• Cerebral oedema-Swelling of the brain tissue
may be a local phenomenon around almost
any lesion, be it contusion, laceration, tumor
or infarct
Signs of diffuse axonal injury
• Diffuse petechial
• haemorrhages in corpus callosum
• Typically traumatic but may also occur in:
– Fat embolism
– Hypertensive encephalopathy
– Malaria
Signs of cerebral oedema
• Increased brain weight
• Flattening of gyri and obliteration of sulci
• Hippocampal herniation (unilateral)
• Cerebellar tonsillar herniation
• Mid brain haemorrhage as a consequence of
vascular occlusion and haemorrhagic infarction
Coup and Contre Coup
Contre coup
Linear fracture
• These are straight or curved fracture lines,
often of considerable length
• They either radiate out from a depressed zone,
or arise under or at a distance from the impact
area, from bulging deformation
• They may involve the inner or outer table, but
commonly traverse both
Hinge fracture
• Blow to the top of the head commonly results
in linear fractures that pass down the parietal
plates.
• May extend across base of skull in middle
fossa passing through pituitary fossa
• Indicates severe force as seen in road traffic
accidents (motor cyclist’s fracture) or fall from
a high building
Ring fracture
• These occur in the posterior fossa around the
foramen magnum and are most often caused
by a fall from a height onto the feet
• If the kinetic energy of the fall is not absorbed
by fractures of the legs, pelvis or spine, the
impact is transmitted up the cervical spine
• This may be rammed into the skull, carrying a
circle of occipital bone with it
Pond fracture
• This is merely a descriptive term for a shallow
depressed fracture forming a concave ‘pond’
• It is more common in the more pliable bones
of infants and, indeed, a depression can occur
in the absence of a fracture
Base of skull fracture
• Falls from a height on to the feet transmit
force through the legs and spine to the base of
skull forming a ring fracture around the
foramen magnum
• Similar fracture seen when the force from a
blow to the top of head is transmitted to base
of skull
Spider’s web fracture
• Localised blow to skull will cause “spider’s
web” fracture with radiating lines and
concentric circles
Spiders web
Depressed fracture
• Fragments of skull forced inwards
• Inner table fragmentation is greater than outer
• Fragments driven into underlying brain tissue
causing contusions and lacerations
Depressed fracture
“Blow-out” fracture
• Very thin orbital plates fracture easily and
may be only sign of trauma
• Direct injury to orbit
• Impact on occiput may cause contre coup
fracture of roof of orbit
Puppe’s rule
Intra-cranial haemorrhage
• Extra-dural haemorrhage
• Sub-dural haemorrhage
• Sub-arachnoid haemorrhage
• Traumatic sub-arachnoid haemorrhage
• Intra-cerebral haemorrhage
Pathological consequences of intra-cranial
haemorrhage
• Space occupying mass
• Displaces brain laterally and downward
• Raised intra-cranial pressure
• Decreased blood flow to brain
• Herniation of hippocampus below tentorium
• Coning of brain stem
Extra-dural haemorrhage
• Traumatic in origin, occurs in 2% of head injuries
• Fracture of temporal bone crossing branch of
middle meningeal artery
• Classical “lucid interval”
• Relatively rapid onset (arterial bleeding)
• Symptoms may be confused with alcohol
intoxication
• Drunk with head injury in police custody
Extra-dural haemorrhage
Sub-dural haemorrhage
• Traumatic in origin but degree of trauma may
be relatively trivial
• Rupture of cortical veins, relatively slow onset
(venous bleeding)
• Particularly seen in children (shaking) and in the
elderly (cerebral atrophy)
• May be chronic and histology can assist in
estimating age in relation to previous trauma
Sub-dural haemorrhage
Chronic sub-dural haemorrhage
Dating a Subdural hemorrhage
• 5 days-hematoma gradually changes from dark
red to a brownish color (obvious at 10-12/7)
• 12 days-presence of a membrane firm enough
to be picked off with forceps
Sub-arachnoid haemorrhage
• Spontaneous rupture of aneurysm on Circle of
Willis
• May occur as a complication of cerebral
contusion or as an extension of an intra-
cerebral haemorrhage
Traumatic Sub-arachnoid haemorrhage
• Kick or blow to the side of the neck
• Traumatic dissection of vertebral artery
within the cervical spine
• Most vulnerable at level of first cervical
vertebra
• Posterior fossa sub-arachnoid haemorrhage
Medical legal mimickers of injury
• Berry aneurysms
• AV malformations
• Heat hematomas
Spinal Injuries
• The spine and head should be thought of as
part of the same system in relation to trauma
• Compression damage-occurs when the victim
falls from a height either onto his feet or his
head, though in the latter case, head injuries
may overshadow damage to the spine, as well
as absorbing most of the impact
• Hyperflexion and hyperextension injury-
hyperextension is much more dangerous in
causing spinal damage, possibly because
flexion is protected by contraction of the
strong posterior neck muscles, whereas the
weak anterior longitudinal ligament is
incapable of preserving the integrity of the
cervical spine during hyperextension
• Spinal cord injury-Most damage to the spinal
cord arises from intrusion of some part of the
spinal column into the canal, be it bony
fragments or displacement, ligamentum
flavum, disc annulus or extruded nucleus
pulposus
• https://doi.org/10.1016/j.fsir.2021.100186
end

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Head Injury-final.pptx

  • 1. Head and Spinal Injury Dr. Kayonde Nathan M.
  • 2. Brain protection • Bony skull • Falx cerebri • Tentorium cerebelli • Cerebrospinal fluid Injuries to brain arise due to differences in motion of the skull and brain
  • 3. Clinical details • Loss of consciousness • Glasgow coma scale • Skull x-ray • CT scan
  • 4. Scalp injuries • Injuries bleed profusely • Blunt trauma may result in apparently “clean cut” laceration, but bruising at edges and hair driven into wound. • Bruises easily palpable, shave hair to visualise fully. “Hair hides”
  • 5. “Black eye” May be due to: • Direct blow into the orbit • An injury to the front of the scalp, draining down over the supraorbital ridge • A fracture of the base of the skull (direct or contrecoup) allowing meningeal hemorrhage to escape through the orbital roof
  • 6.
  • 7.
  • 8.
  • 9. Skull Fracture Skeleton of the head consists of three main components: • Mandible • Facial skeleton • Calvarium Skull distorts when force is applied to it. Fracture represents site of maximum stress on skull and may not be at site of impact
  • 10. Brain injury • Skull fracture is a marker for the severity of force applied to head. • In general there is a correlation between them • In the absence of fracture, rotary movements of head cause shear strains within brain resulting in diffuse axonal injury, contusion and laceration • Cerebral oedema and haemorrhage are secondary effects
  • 11. • By direct intrusion, either by a foreign object such as a penetrating weapon, bullet or other missile – or fragments of skull in a compound fracture where the skull is disrupted • By deformation of the brain in closed head injuries
  • 12.
  • 13. Cerebral injuries (types) • Cerebral contusion-When either linear or, more often, laminar stresses are applied to the cortex, this soft tissue may disrupt • Cerebral laceration-Laceration of the cortex is an extension in severity of contusion in which mechanical separation of the tissue can be seen. • Traumatic intracerebral hemorrhage-Substantial areas of hemorrhage, either infiltrating the brain tissue or forming actual hematomas
  • 14. • Primary brainstem hemorrhage-Secondary brainstem bleeding is dealt with under ‘cerebral oedema’ so here we are concerned with hemorrhage that occurs at the time of injury • Coup and contrecoup damage • Concussion-is a clinical, not a pathological entity, but the pathologist must consider it, as it is related to intracranial lesions and he is often questioned about it in court proceeding
  • 15. • Diffuse neuronal and axonal injury-diffuse brain damage exists in four principal forms: diffuse vascular injury, diffuse axonal injury, hypoxic brain damage and diffuse brain swelling • Cerebral oedema-Swelling of the brain tissue may be a local phenomenon around almost any lesion, be it contusion, laceration, tumor or infarct
  • 16. Signs of diffuse axonal injury • Diffuse petechial • haemorrhages in corpus callosum • Typically traumatic but may also occur in: – Fat embolism – Hypertensive encephalopathy – Malaria
  • 17. Signs of cerebral oedema • Increased brain weight • Flattening of gyri and obliteration of sulci • Hippocampal herniation (unilateral) • Cerebellar tonsillar herniation • Mid brain haemorrhage as a consequence of vascular occlusion and haemorrhagic infarction
  • 20. Linear fracture • These are straight or curved fracture lines, often of considerable length • They either radiate out from a depressed zone, or arise under or at a distance from the impact area, from bulging deformation • They may involve the inner or outer table, but commonly traverse both
  • 21.
  • 22. Hinge fracture • Blow to the top of the head commonly results in linear fractures that pass down the parietal plates. • May extend across base of skull in middle fossa passing through pituitary fossa • Indicates severe force as seen in road traffic accidents (motor cyclist’s fracture) or fall from a high building
  • 23.
  • 24. Ring fracture • These occur in the posterior fossa around the foramen magnum and are most often caused by a fall from a height onto the feet • If the kinetic energy of the fall is not absorbed by fractures of the legs, pelvis or spine, the impact is transmitted up the cervical spine • This may be rammed into the skull, carrying a circle of occipital bone with it
  • 25.
  • 26. Pond fracture • This is merely a descriptive term for a shallow depressed fracture forming a concave ‘pond’ • It is more common in the more pliable bones of infants and, indeed, a depression can occur in the absence of a fracture
  • 27.
  • 28. Base of skull fracture • Falls from a height on to the feet transmit force through the legs and spine to the base of skull forming a ring fracture around the foramen magnum • Similar fracture seen when the force from a blow to the top of head is transmitted to base of skull
  • 29.
  • 30. Spider’s web fracture • Localised blow to skull will cause “spider’s web” fracture with radiating lines and concentric circles
  • 32. Depressed fracture • Fragments of skull forced inwards • Inner table fragmentation is greater than outer • Fragments driven into underlying brain tissue causing contusions and lacerations
  • 34.
  • 35. “Blow-out” fracture • Very thin orbital plates fracture easily and may be only sign of trauma • Direct injury to orbit • Impact on occiput may cause contre coup fracture of roof of orbit
  • 36.
  • 38. Intra-cranial haemorrhage • Extra-dural haemorrhage • Sub-dural haemorrhage • Sub-arachnoid haemorrhage • Traumatic sub-arachnoid haemorrhage • Intra-cerebral haemorrhage
  • 39. Pathological consequences of intra-cranial haemorrhage • Space occupying mass • Displaces brain laterally and downward • Raised intra-cranial pressure • Decreased blood flow to brain • Herniation of hippocampus below tentorium • Coning of brain stem
  • 40.
  • 41. Extra-dural haemorrhage • Traumatic in origin, occurs in 2% of head injuries • Fracture of temporal bone crossing branch of middle meningeal artery • Classical “lucid interval” • Relatively rapid onset (arterial bleeding) • Symptoms may be confused with alcohol intoxication • Drunk with head injury in police custody
  • 43.
  • 44.
  • 45. Sub-dural haemorrhage • Traumatic in origin but degree of trauma may be relatively trivial • Rupture of cortical veins, relatively slow onset (venous bleeding) • Particularly seen in children (shaking) and in the elderly (cerebral atrophy) • May be chronic and histology can assist in estimating age in relation to previous trauma
  • 47.
  • 49. Dating a Subdural hemorrhage • 5 days-hematoma gradually changes from dark red to a brownish color (obvious at 10-12/7) • 12 days-presence of a membrane firm enough to be picked off with forceps
  • 50. Sub-arachnoid haemorrhage • Spontaneous rupture of aneurysm on Circle of Willis • May occur as a complication of cerebral contusion or as an extension of an intra- cerebral haemorrhage
  • 51.
  • 52. Traumatic Sub-arachnoid haemorrhage • Kick or blow to the side of the neck • Traumatic dissection of vertebral artery within the cervical spine • Most vulnerable at level of first cervical vertebra • Posterior fossa sub-arachnoid haemorrhage
  • 53. Medical legal mimickers of injury • Berry aneurysms • AV malformations • Heat hematomas
  • 54.
  • 55.
  • 56.
  • 57.
  • 58. Spinal Injuries • The spine and head should be thought of as part of the same system in relation to trauma • Compression damage-occurs when the victim falls from a height either onto his feet or his head, though in the latter case, head injuries may overshadow damage to the spine, as well as absorbing most of the impact
  • 59. • Hyperflexion and hyperextension injury- hyperextension is much more dangerous in causing spinal damage, possibly because flexion is protected by contraction of the strong posterior neck muscles, whereas the weak anterior longitudinal ligament is incapable of preserving the integrity of the cervical spine during hyperextension
  • 60. • Spinal cord injury-Most damage to the spinal cord arises from intrusion of some part of the spinal column into the canal, be it bony fragments or displacement, ligamentum flavum, disc annulus or extruded nucleus pulposus
  • 62. end