2. OUTLINE
Studies
definitions
Anatomy of the eye
Types of trauma
Corneal abrasion
Chemical burn
Blunt injury to the eye
Laceration of the eye,
Ocular foreign bodies( intraocular, conjunctiva, corneal)
Orbital wall fractures
Eyelid laceration
Case
6. Chambers of the eye:
Anterior chamber : the boundaries are the cornea and
the iris
posterior chamber :is demarcated by the iris and the
lens.
Vitreous chamber: filled gelatinous material which
serves principally to maintain the eye's shape
The eyeball is well protected by
the projecting margins of the bony orbit
the eyelids.
The blinking reflex
protective action of the eye lashes
8. DEFINTIONS
Eyewall: Sclera and cornea.
Closed globe injury: No full- thickness wound of eyewall.
Open globe injury: Full- thickness wound of the eyewall.
Contusion: There is no wound.
direct energy delivery by the object that caused damage inside
the wall, e. g., choroidal rupture
Lamellar laceration: Partial- thickness wound of the
eyewall.
Rupture: Full- thickness wound of the eyewall, caused by
a blunt object.
9. Laceration: Full- thickness wound of the eyewall,
caused by a sharp object.
The wound occurs at the impact site by an outside- in
mechanism
Penetrating injury: Entrance wound.
Perforating injury: Entrance and exit wounds.
11. CORNEAL ABRASION
is a medical condition involving
the loss of the surface epithelial
layer of the eye's cornea as a
result of physical forces
Causes
Fingernails
Pieces of paper or cardboard
Branches or leaves
Contact lenses that have been
left in too long
17. HISTORY
Ask about specific nature of the chemical (acid ,
alkali) ….why???
the mechanism of injury
Pain (often extreme)
Foreign body sensation
Blurred vision
Excessive tearing
Photophobia
Red eye(s)
SIGNS AND SYMPTOMS
18. IMMEDIATE INTERVENTION:
physical examination should be delayed until the
affected eye is irrigated and the pH of the ocular
surface is neutralized
Topical anesthesia
Irrigation with 1-2 liters of water or more (normal
saline) using special irrigating tubing Morgan lens
for 15 minuts.
Irrigate until pH of the ocular surface is
neutralized… litmus paper
20. PHYSICAL EXAMINATION
Decreased visual acuity: Initial visual acuity can be
decreased because of corneal epithelial defects
increased IOP: An immediate rise in IOP may result
from collagen deformation and shortening, thereby
shrinking the anterior chamber
Conjunctival inflammation
Corneal epithelial defect: Corneal epithelial defect:
Inspect carefully eyelids (foreign bodies)
Perilimbal ischemia: the limbal stem cells are
responsible for repopulating the corneal epithelium.
21. Injuries can be graded from 0-5, as follows:
Grade 0 - Minimal epithelial defect, clear corneal stroma, no
limbal ischemia
Grade 1 - Partial-complete epithelial defect, clear corneal
stroma, no limbal ischemia
Grade 2 - Partial-complete epithelial defect, mild stromal
haze, none or only mild limbal ischemia
Grade 3 - Complete epithelial defect, moderate stromal haze,
less than one third of the limbus is ischemic
Grade 4 - Complete epithelial defect, stromal haze blurring
iris details, one third to two thirds of the limbus is ischemic
Grade 5 - Complete epithelial defect, stromal opacification,
greater than two thirds of the limbus is ischemic
22. TREATMENTS
artificial tear : play an important role in healing.
Ascorbate: plays a fundamental role in collagen
remodeling, leading to an improvement in corneal
healing.
topical steroids : can help break this inflammatory
cycle.
aqueous suppressants: especially oral carbonic
anhydrase inhibitors and topical beta-adrenergic
blockers. To prevent increase IOP
Prophylactic topical antibiotics
24. HISTORY
exact time
mechanism of eye injury
How? Fight, sport, car accident, work accedents
Tool of assault if applicable. Sharp or blunt object
possible IO foreign body. Ex. Broken glass,
Drugs history
Any known comorbidities, blood disorders
25. BLUNT INJURY TO THE EYE
Causes: by fist, ball, stone, falling
Conditions secondary to blunt trauma
Hyphema ;Bleeding in the anterior chamber of the
eye
Retinal Detachment: Flashes, Floaters and visual
field defect
26. Eyelid Laceration
Globe Rupture
Lens Dislocation:
Normaly lens are clear
with edge of lens not
visible
Traumatic Glaucoma
29. LACERATING INJURY
Superficial minor or deep (involving the full
thickness of cornea or sclera)
Emergency !!!
Symptoms
Severe Eye Pain
Decreased Visual Acuity
Eye tearing
30. Clinical features
Inspection (with penlight or preferably a slit lamp):
• Obvious corneal or scleral laceration
• Volume loss to eye
• Uveal (iris or ciliary body) prolapse
• Other iris abnormalities (peaked pupil or eccentric pupil)
• 360 degree, bullous subconjunctival hemorrhage (posterior rupture)
• Intraocular or protruding foreign body
Decreased visual acuity by Snellen or handheld chart, assess
counting fingers, hand motion or light perception if unable to see chart
Relative afferent pupillary defect by swinging penlight technique
31. PHYSICAL EXAMINATION
If you suspect open globe, avoid any examination
procedure that might apply pressure to the eyeball.
ex, intraocular pressure measurement by
tonometry.
If you suspect globe rupture, avoid placing any
medication or diagnostic eye drops into the eye.
Any protruding foreign bodies should be left in
place. Removal should be referred to the
ophthalmologist.
32. EXAMINATION
In conscious and cooperative patients:
Visual acuity.
The anterior segment is ideally examined with a slit
lamp.
Pay particular attention to the corneoscleral laceration.
The location and the length of the laceration should be
noted.
The size and the shape of the pupil and its reaction.
Fundus examination.
33. PHYSICAL FINDINGS
Markedly decreased visual acuity
Eccentric or teardrop pupil
Increased anterior chamber depth
Gross deformity of the eye with obvious volume
loss is clear evidence of globe rupture
The presence of uvea (iris, ciliary body, or choroid)
prolapsing into or through the wound is diagnostic
of an open globe injury
If an open globe is apparent then the clinician
should not place dilating drops in the eye
34. Iris tissue prolapsing through a cornea or scleral wound is
pathognomonic for an open globe
35. INVESTIGATIONS
Orbital CT Scan, axial and coronal
Consider CT or XR of the orbits if an orbital wall
fracture is suspected.
36. Management
Superficial trauma : topical antibiotics and oral
analgesia
If you suspect open globe injury, then do the
following
Eye shield placement over the affected eye
Avoidance of any eye manipulation
Bed rest
Avoidance of any eye solutions (eg, fluorescein,
tetracaine, cycloplegics)
Antiemetic therapy
Pain medication …morphine
Sedation, as needed
Don’t remove any protruding object
37. Tetanus vaccine
Referral to ophthalmologist when:
Ex .. If globe rupture is suspected
surgical globe repair, ideally within 24 hours of
injury
39. FOREIGN BODY
Any material such as dust or sand that gets into the
eye
2 types:
Superficial foreign bodies
Penetrating foreign bodies
History: where(work, sport) and how and what
A history of working with power tools, blowers, or
weed-whackers may indicate a higher risk of an
intraocular foreign body
41. CORNEAL FOREIGN BODY
may have associated rust ring if metallic
patients may note tearing, photophobia, foreign
body sensation, red eye
signs include foreign body, epithelial defect that
stains with fluorescein,
Complications
abrasion, infection, scarring, rust ring, secondary iritis
42. CONJUNCTIVAL FOREIGN BODY
Symptoms
Scratchy sensation with each blink?
Foreign body sensation
Mild pain
Mild injection
43. EXAMINATION
Visual acuity
Inspect, upper and lower eyelid conjunctiva for foreign
bodies.
Fluorescein stain
Helps localize foreign body
(sand or other particle)
44. TREATMENT
Removal of foreign body
Irrigation
Cotton swab moistened with topical anesthetic
treat with an antibiotic ointment
Referral within 24h if:
Large corneal abrasion
Deeply embedded FB
45.
46. INTRAOCULAR FOREIGN BODY
History
What was the patient doing?
Metal on metal hammering, drilling
Was the patient exposed to high speed-missile?
Sudden impact on the eyelids or eye?
Pain or decreased vision?
47. EXAMINATION
Visual acuity
Inspection:
Corneal or scleral laceration, hyphema, irregular pupil or absent red
reflex.
Slit lamp
Referral: immediately if Hx suggests struck by a high
speed missile.
50. CAUSES
vehicle accidents, industrial accidents, sports-
related facial trauma, and assaults.
The hydraulic theory advocates that increased
intraorbital pressure causes a decompressing
fracture into an adjacent sinus
PATHOPHYSIOLOGY
51. Associated
structures
BonesOrbital structure
Frontal sinus,
supraorbital nerve
Frontal boneSuperior orbital rim,
roof of orbit
Lateral canthal
ligament
Sphenoid bone,
zygomotic bone
Lateral wall of orbit
inferior oblique and
inferior rectus
muscles, maxillary
sinus, infraorbital
nerve
Zygoma, maxillary
bone
Infraorbital rim and
floor of orbit
Medial rectus
muscle, ethmoid
sinus, medial
canthal ligament,
lacrimal duct
system
Maxillary and
ethmoid bones
Medial wall of orbit
52. FRACTURE TYPES
Orbital zygomatic fracture : The most common
fracture of the orbital rim is in the orbital zygomatic
region. This injury is typically the result of a high-impact
blow to the lateral orbit
Nasoethmoid fracture :Fracture in this portion of the
orbital rim can result in disruption of the medial canthal
ligament and the lacrimal duct system. In addition, the
medial rectus muscle may become trapped in fractures
of the medial wall of the orbit
Orbital floor fracture : sometimes known as "blowout.
The mechanism of fracture
Increased intraocular pressure (hydraulic theory)
A direct blow to the infraorbital rim
Orbital roof fracture:
53. ORBITAL ROOF FRACTURE
More common in young children:
- High cranium to midface ratio in children
- Pneumatization of the frontal sinus in adults
Orbital roof fractures have
high assciation with
intracranial injury.
54. CONSEQUENCES OF ORBITAL FLOOR
FRACTURE
Entrapment of the inferior rectus muscle and/or
orbital fat.
subsequent loss of inferior rectus muscle function is
due to
1. Entrapment of the muscle within the fracture.
2. Edema and hemorrhage of muscles and
extraocular fat
( prolapsed through the
fracture to the maxillary
sinus)
55. CONSEQUENCES OF ORBITAL FLOOR
FRACTURE
Orbital dystopia
- The affected eye is lower in the horizontal plane
-Due to entrapped muscle and orbital fat pull the eye downward.
56. CONSEQUENCES OF ORBITAL FLOOR
FRACTURE
Enophthalmos: (the eye is receded into the orbit)
may develop when the globe is displaced
posteriorly in association with an orbital floor
fracture and prolapse of tissue into the maxillary
sinus.
Injury to the infraorbital nerve (resulting in
numbness below the eye )
57. History — Specific information regarding when the
injury occurred, area of the face that was injured, and
the mechanism of injury should be obtained.
Where does it hurt?
Do you have blurry, double, or decreased vision?
Do you have difficulty with eye movement or double-
vision in a specific direction?
Do you have numbness of a particular region of your
face?
Hints
1. Diffuse pain occurs with an orbital hematoma
2. pain with eye movement suggests injury involving
extraocular muscles.
3. Any change in vision could indicate a serious intraocular
injury.
4. Diplopia, particularly with upward gaze, and numbness
below the eye may occur with fractures of the orbital floor.
5. Numbness of the forehead suggests damage to the
supraorbital nerve as the result of injury to the roof of the
orbit.
58. EXAMINATION
On inspection of the globe, the following features
are indications of significant injury:
Proptosis (orbital hematoma)
Extrusion of intraocular contents, severe
conjunctival hemorrhage, and/or a tear-shaped
pupil (ruptured globe)
Orbital dystopia and/or enophthalmos (orbital floor
fracture)
Pupillary reactivity, size, and shape
extraocular movements and visual acuity.
Funduscopic examination may identify vitreous
hemorrhage or retinal injury.
59. EYELID LACERATIONS
Simple eyelid lacerations:
simple lacerations that are horizontal and follow the
skin lines and that involve less than 25 percent of
the lid will usually heal well without suturing . The
clinician may dress these with a triple antibiotic
ointment
the clinician may apply an adhesive surgical tape
Uncomplicated lid lacerations of a greater extent:
repaired with sutures placed in similar fashion as for
other anatomic locations
60. MUST REFER TO OPHTHALMOLOGIST
Full-thickness lid lacerations — A high threshold
of suspicion for penetrating injury to the globe.
Lacerations with orbital fat prolapse —
Lacerations involving the tear drainage
system —
Orbital injury or foreign body
Laceration with poor alignment
61. CASE
A 12 years old male was referred to emergency
department for evaluation of possible glob injury
while hammering on a glass board; a glass shard
flew into his right eye. He complained of pain,
foreign body sensation and decrease of vision.
Boston 1988…. Emergency …..3000 patient over a period of 6 months….. Most less than 15 yrs… sport injury,, work environment
6000 students……… 12 have monoocular low vision caused by trauma…. Concluded : injury is the main reason for monocular loss of vision in childhood
At cornoe-scleral junction
often prevent the entrance of F.B. which might cause damage to the cornea.
http://isotonline.org/betts/
Acid cause protien denaturation and coagulationProtein coagulation prevents deeper penetration of acids Alkali can penetrate Deeper into and through the cornea
: The degree of limbal ischemia (blanching) is perhaps the most significant prognostic indicator for future corneal healing because
Once the inciting chemical has been completely removed, epithelial healing can begin. Chemically injured eyes have a tendency to poorly produce adequate tears; therefore,
you should make every attempt to avoid sudden intraocular hypertension with extrusion of ocular contents and further wound contamination.
Orbital roof fractures are more common in younger patients (less than 10 years) [5,6]. This phenomenon is probably related to the high cranium-to-midface ratio of young children as compared with adults (thus exposing a larger upper surface for injury).