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Flexor Tendon
Injuries




C. Noel Henley, MD
Flexor Tendon
Injuries


IU Orthopaedic Surgery


C. Noel Henley, MD
Flexor Tendon
Injuries


IU Orthopaedic Surgery
April 5, 2006

C. Noel Henley, MD
Talk Summary
Talk Summary
• Introduction
Talk Summary
• Introduction
• Basic science
   –   anatomy
   –   nutrition
   –   biomechanics
   –   healing
• Surgical considerations
   – lacerations
   – avulsions
• Rehabilitation
Introduction
Introduction
• History
Introduction
• History
  – no repair possible in the digit
Introduction
• History
  – no repair possible in the digit
  – “no man’s land”
Introduction
• History
  – no repair possible in the digit
  – “no man’s land”
• Current evidence
Introduction
• History
  – no repair possible in the digit
  – “no man’s land”
• Current evidence
  – 1970s: repair is possible without delayed grafting,
    reconstruction
Introduction
• History
  – no repair possible in the digit
  – “no man’s land”
• Current evidence
  – 1970s: repair is possible without delayed grafting,
    reconstruction
  – Now: adhesion-free, primarily healed tendon repair is
    possible
Introduction
Introduction
• General goals of repair
Introduction
• General goals of repair
  – primary tendon repair – avoid grafting
Introduction
• General goals of repair
  – primary tendon repair – avoid grafting
  – sufficient strength for passive motion rehab which will
    allow for
Introduction
• General goals of repair
  – primary tendon repair – avoid grafting
  – sufficient strength for passive motion rehab which will
    allow for
     • minimum adhesion formation
Introduction
• General goals of repair
  – primary tendon repair – avoid grafting
  – sufficient strength for passive motion rehab which will
    allow for
     • minimum adhesion formation
     • restoration of gliding surface
Introduction
• General goals of repair
  – primary tendon repair – avoid grafting
  – sufficient strength for passive motion rehab which will
    allow for
     • minimum adhesion formation
     • restoration of gliding surface
     • facilitation of repair site healing
Anatomy
Anatomy

 • FDS
Anatomy

 • FDS
   – O: volar humerus, radius, ulna
Anatomy

 • FDS
   – O: volar humerus, radius, ulna
   – divided muscle belly in mid forearm (FA)
Anatomy

 • FDS
   – O: volar humerus, radius, ulna
   – divided muscle belly in mid forearm (FA)
      • super cial – long and ring ngers (LF, RF)
Anatomy

 • FDS
   – O: volar humerus, radius, ulna
   – divided muscle belly in mid forearm (FA)
      • super cial – long and ring ngers (LF, RF)
      • deep – index and small ngers (IF, SF)
Anatomy

 • FDS
   – O: volar humerus, radius, ulna
   – divided muscle belly in mid forearm (FA)
      • super cial – long and ring ngers (LF, RF)
      • deep – index and small ngers (IF, SF)
 • FDP
Anatomy

 • FDS
   – O: volar humerus, radius, ulna
   – divided muscle belly in mid forearm (FA)
      • super cial – long and ring ngers (LF, RF)
      • deep – index and small ngers (IF, SF)
 • FDP
   – O: anteromedial ulna, IO membrane
Anatomy

 • FDS
   – O: volar humerus, radius, ulna
   – divided muscle belly in mid forearm (FA)
      • super cial – long and ring ngers (LF, RF)
      • deep – index and small ngers (IF, SF)
 • FDP
   – O: anteromedial ulna, IO membrane
   – dorsal to FDS in volar FA
Anatomy

 • FDS
   – O: volar humerus, radius, ulna
   – divided muscle belly in mid forearm (FA)
      • super cial – long and ring ngers (LF, RF)
      • deep – index and small ngers (IF, SF)
 • FDP
   – O: anteromedial ulna, IO membrane
   – dorsal to FDS in volar FA
   – four tendons usually arise from common belly
Anatomy
Anatomy

• In the hand
Anatomy

• In the hand
  – ve zones
Anatomy

• In the hand
  – ve zones
     • V: musculo-tendinous junction to
       proximal edge of carpal canal
Anatomy

• In the hand
  – ve zones
     • V: musculo-tendinous junction to
       proximal edge of carpal canal
     • IV: beneath (dorsal to) the
       transverse carpal ligament
Anatomy

• In the hand
  – ve zones
     • V: musculo-tendinous junction to
       proximal edge of carpal canal
     • IV: beneath (dorsal to) the
       transverse carpal ligament
        – IF, SF FDS are dorsal to LF, RF FDS
          here
Anatomy
Anatomy
• Zones (cont.)
Anatomy
• Zones (cont.)
  – III: distal edge of transverse
    carpal ligament to origin of
     bro-osseous sheath at the
    distal palmar crease
Anatomy
• Zones (cont.)
  – III: distal edge of transverse
    carpal ligament to origin of
     bro-osseous sheath at the
    distal palmar crease
  – II: origin of bro-osseous
    sheath to FDS insertion
Anatomy
• Zones (cont.)
  – III: distal edge of transverse
    carpal ligament to origin of
      bro-osseous sheath at the
    distal palmar crease
  – II: origin of bro-osseous
    sheath to FDS insertion
  – I: distal to FDS insertion
Anatomy
Anatomy




 • Super cialis
Anatomy




 • Super cialis
   – FDS divides into two slips, wrapping around
     FDP; reunite at Camper’s chiasma
Anatomy




 • Super cialis
   – FDS divides into two slips, wrapping around
     FDP; reunite at Camper’s chiasma
   – continues to insert along the proximal half of the
     middle phalanx (P2)
Anatomy
Anatomy




 • Profundus
Anatomy




 • Profundus
Anatomy




 • Profundus
   – passes through chiasma to insert at proximal base
     of distal phalanx (P3)
Anatomy




 • Profundus
   – passes through chiasma to insert at proximal base
     of distal phalanx (P3)
   – FDP = primary digital exor
Anatomy




 • Profundus
   – passes through chiasma to insert at proximal base
     of distal phalanx (P3)
   – FDP = primary digital exor
   – FDS + intrinsics combine for forceful exion
Anatomy
Anatomy
Anatomy
Anatomy

 • Sheath
Anatomy

 • Sheath
   – visceral and parietal synovial layers
     enclose the tendons
Anatomy

 • Sheath
   – visceral and parietal synovial layers
     enclose the tendons
   – Pulleys
Anatomy

 • Sheath
   – visceral and parietal synovial layers
     enclose the tendons
   – Pulleys
      • A2 and A4 arise from periosteum of
        proximal P1 and midportion of P2 (prox-
        prox and mid-middle)
Anatomy
Anatomy

 • Sheath
Anatomy

 • Sheath
   – Pulleys
Anatomy

 • Sheath
   – Pulleys
      • A1, A3, and A5 arise from volar plates of
        MCP, PIP, and DIP joints respectively
Anatomy
Anatomy

 • Sheath
Anatomy

 • Sheath
   – Pulleys
Anatomy

 • Sheath
   – Pulleys
      • cruciate pulleys collapse to allow annular
        pulley apposition during exion
Nutrition
Nutrition

 • Two sources of tendon nutrition
Nutrition

 • Two sources of tendon nutrition
   – vascular
Nutrition

 • Two sources of tendon nutrition
   – vascular
   – synovial
Nutrition

 • Two sources of tendon nutrition
   – vascular
   – synovial
 • Vascularity sources
Nutrition

 • Two sources of tendon nutrition
   – vascular
   – synovial
 • Vascularity sources
   – longitudinal vessels
Nutrition

 • Two sources of tendon nutrition
   – vascular
   – synovial
 • Vascularity sources
   – longitudinal vessels
   – proximal synovial fold vessels
Nutrition

 • Two sources of tendon nutrition
   – vascular
   – synovial
 • Vascularity sources
   – longitudinal vessels
   – proximal synovial fold vessels
   – segmental branches from paired digital vessels
     (vincular system)
Nutrition

 • Two sources of tendon nutrition
   – vascular
   – synovial
 • Vascularity sources
   – longitudinal vessels
   – proximal synovial fold vessels
   – segmental branches from paired digital vessels
     (vincular system)
   – insertional vessels at FDP, FDS insertions
Nutrition
Nutrition
Nutrition
Nutrition

 • Avascular segments
Nutrition

 • Avascular segments
   – FDP and FDS: over proximal phalanx
Nutrition

 • Avascular segments
   – FDP and FDS: over proximal phalanx
   – FDP: short zone over middle phalanx
Nutrition

 • Avascular segments
   – FDP and FDS: over proximal phalanx
   – FDP: short zone over middle phalanx
 • Synovial uid diffusion
Nutrition

 • Avascular segments
   – FDP and FDS: over proximal phalanx
   – FDP: short zone over middle phalanx
 • Synovial uid diffusion
   – imbibition
Nutrition
Nutrition
Nutrition
Nutrition

 • Vascularity
Nutrition

 • Vascularity
   – damage to vascular and uid nutritional systems
     may further hamper healing of repaired exor
     tendons
Nutrition

 • Vascularity
   – damage to vascular and uid nutritional systems
     may further hamper healing of repaired exor
     tendons
   – their integrity must be respected during all
     reparative efforts
Nutrition
Nutrition
Biomechanics
Biomechanics

 • Excursion
Biomechanics

 • Excursion
   – 9 cm of excursion for composite wrist, full digital
      exion
Biomechanics

 • Excursion
   – 9 cm of excursion for composite wrist, full digital
      exion
   – 2.5 cm required for full digital exion with wrist
     at neutral
Biomechanics
Biomechanics

 • Moment arm
Biomechanics

 • Moment arm
   – increased distance of tendon from joint center of
     rotation = higher moment arm = less motion per
     muscle contraction force
Biomechanics
Biomechanics

• Moment arm
Biomechanics

• Moment arm
  – pulley system constraint governs these parameters
Biomechanics

• Moment arm
  – pulley system constraint governs these parameters
  – loss of portions of pulley system may alter balance
    between exor, intrinsic, and extensor tendons
Biomechanics

• Moment arm
  – pulley system constraint governs these parameters
  – loss of portions of pulley system may alter balance
    between exor, intrinsic, and extensor tendons
  – loss of A2 or A4 pulleys may diminish motion/power
    or lead to contractures of IP joints
Tendon Healing
Tendon Healing

 • Phases
Tendon Healing

 • Phases
   – in ammatory – 48-72 hours
Tendon Healing

 • Phases
   – in ammatory – 48-72 hours
      • repair strength reliant upon suture itself
Tendon Healing

 • Phases
   – in ammatory – 48-72 hours
      • repair strength reliant upon suture itself
   – collagen-producing phase – 5 days to 4 weeks
Tendon Healing

 • Phases
   – in ammatory – 48-72 hours
      • repair strength reliant upon suture itself
   – collagen-producing phase – 5 days to 4 weeks
      • strength accelerates rapidly here
Tendon Healing

 • Phases
   – in ammatory – 48-72 hours
      • repair strength reliant upon suture itself
   – collagen-producing phase – 5 days to 4 weeks
      • strength accelerates rapidly here
   – remodeling phase – until 112 days
Tendon Healing

 • Phases
   – in ammatory – 48-72 hours
      • repair strength reliant upon suture itself
   – collagen-producing phase – 5 days to 4 weeks
      • strength accelerates rapidly here
   – remodeling phase – until 112 days
 • Predominance of extrinsic over intrinsic
   healing leads to more adhesions
Tendon Healing




        inflammatory
Tendon Healing




        inflammatory
Tendon Healing




      collagen-producing
Tendon Healing




      collagen-producing
Tendon Healing




        remodeling
Tendon Healing




        remodeling
Tendon Healing
Tendon Healing
• Adhesion formation and control
Tendon Healing
• Adhesion formation and control
  – Contributing factors
Tendon Healing
• Adhesion formation and control
  – Contributing factors
     • trauma (from injury and surgery)
Tendon Healing
• Adhesion formation and control
  – Contributing factors
     • trauma (from injury and surgery)
        – tendon surface crushing
Tendon Healing
• Adhesion formation and control
  – Contributing factors
     • trauma (from injury and surgery)
        – tendon surface crushing
        – vincula disruption
Tendon Healing
• Adhesion formation and control
  – Contributing factors
     • trauma (from injury and surgery)
        – tendon surface crushing
        – vincula disruption
     • tendon ischemia
Tendon Healing
• Adhesion formation and control
  – Contributing factors
     • trauma (from injury and surgery)
        – tendon surface crushing
        – vincula disruption
     • tendon ischemia
     • tendon immobilization
Tendon Healing
• Adhesion formation and control
  – Contributing factors
     • trauma (from injury and surgery)
        – tendon surface crushing
        – vincula disruption
     • tendon ischemia
     • tendon immobilization
     • repair gapping
Tendon Healing
• Adhesion formation and control
  – Contributing factors
     • trauma (from injury and surgery)
          – tendon surface crushing
          – vincula disruption
     •   tendon ischemia
     •   tendon immobilization
     •   repair gapping
     •   excision of tendon sheath components
Tendon Healing
Tendon Healing

 • How is healing related to motion?
Tendon Healing

             • How is healing related to motion?
                      – passive mobilization enhances healing by
                        stimulating tendon wound maturation and scar
                        remodeling*                      , 110




*Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA
(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:
American Academy of Orthopaedic Surgeons, 1988, pp 5-40.
Tendon Healing

             • How is healing related to motion?
                      – passive mobilization enhances healing by
                        stimulating tendon wound maturation and scar
                        remodeling*                      , 110



                      – use a strong, gap-resistant suture technique +
                        controlled-motion stress postoperatively




*Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA
(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:
American Academy of Orthopaedic Surgeons, 1988, pp 5-40.
Tendon Healing

             • How is healing related to motion?
                      – passive mobilization enhances healing by
                        stimulating tendon wound maturation and scar
                        remodeling*                      , 110



                      – use a strong, gap-resistant suture technique +
                        controlled-motion stress postoperatively
                      – 3-5 mm of excursion needed at repair site to
                        prevent adhesions                             144




*Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA
(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:
American Academy of Orthopaedic Surgeons, 1988, pp 5-40.
Tendon Repair
Tendon Repair
• Contraindications
Tendon Repair
• Contraindications
  – severe multiple tissue injuries to ngers, palm
Tendon Repair
• Contraindications
  – severe multiple tissue injuries to ngers, palm
  – gross wound contamination
Tendon Repair
• Contraindications
  – severe multiple tissue injuries to ngers, palm
  – gross wound contamination
  – signi cant skin loss over the exor system
Tendon Repair
• Contraindications
  – severe multiple tissue injuries to ngers, palm
  – gross wound contamination
  – signi cant skin loss over the exor system
• Reduce fractures, repair neurovascular injuries
  concomitantly
Tendon Repair
• Contraindications
  – severe multiple tissue injuries to ngers, palm
  – gross wound contamination
  – signi cant skin loss over the exor system
• Reduce fractures, repair neurovascular injuries
  concomitantly
• Repair tendons acutely/subacutely to avoid
  deterioration of tendon ends and extrinsic muscle
  shortening
Examination
Examination
• High index of suspicion for multiple structures
  injured through a small laceration or crush injury
Examination
• High index of suspicion for multiple structures
  injured through a small laceration or crush injury
• Complete neurovascular exam
Examination
• High index of suspicion for multiple structures
  injured through a small laceration or crush injury
• Complete neurovascular exam
   – division of two digital nerves indicates arterial division as
     well
Examination
• High index of suspicion for multiple structures
  injured through a small laceration or crush injury
• Complete neurovascular exam
   – division of two digital nerves indicates arterial division as
     well
   – dysvascularity could compromise repair or skin ap
     healing +/- cold intolerance
Examination
Examination
• Alterations in posture
Examination
Examination
• Alterations in posture
Examination
Examination
• Tenodesis effect
Examination
Examination
• Functional testing (FDS, FDP)
Examination
• Functional testing (FDS, FDP)
Examination
Examination
• Functional testing (FDS)
Examination
Examination
• Functional testing (FDP)
Surgery
Surgery
• Planning
Surgery
• Planning
  – inform the patient of the injury severity and potential
    need for more surgery
Surgery
• Planning
  – inform the patient of the injury severity and potential
    need for more surgery
  – incisions
Surgery
• Planning
  – inform the patient of the injury severity and potential
    need for more surgery
  – incisions
     • keep tendon retraction in mind
Surgery
• Planning
  – inform the patient of the injury severity and potential
    need for more surgery
  – incisions
     • keep tendon retraction in mind
     • no bonus points for small incisions
Surgery
• Planning
  – inform the patient of the injury severity and potential
    need for more surgery
  – incisions
     • keep tendon retraction in mind
     • no bonus points for small incisions
     • maintain ap viability
Surgery
• Planning
  – inform the patient of the injury severity and potential
    need for more surgery
  – incisions
     •   keep tendon retraction in mind
     •   no bonus points for small incisions
     •   maintain ap viability
     •   avoid skin/scar contracture
Surgery - planning
Surgery - planning
• Volar incisions
Surgery - planning
• Volar incisions   • Mid-axial incision
Surgery - principles
Surgery - principles
• Ideal repair
Surgery - principles
• Ideal repair
   – easy placement of sutures in the tendon
Surgery - principles
• Ideal repair
   – easy placement of sutures in the tendon
   – secure suture knots
Surgery - principles
• Ideal repair
   – easy placement of sutures in the tendon
   – secure suture knots
   – smooth junction of tendon ends
Surgery - principles
• Ideal repair
   – easy placement of sutures in the tendon
   – secure suture knots
   – smooth junction of tendon ends
   – minimal gapping at the repair site
Surgery - principles
• Ideal repair
   – easy placement of sutures in the tendon
   – secure suture knots
   – smooth junction of tendon ends
   – minimal gapping at the repair site
   – minimal interference with tendon vascularity
Surgery - principles
• Ideal repair
   – easy placement of sutures in the tendon
   – secure suture knots
   – smooth junction of tendon ends
   – minimal gapping at the repair site
   – minimal interference with tendon vascularity
   – sufficient strength for early motion stress to the tendon
Surgery
Surgery
• Core suture considerations
Surgery
• Core suture considerations
  – strength of a repair is proportional to the number of
    suture strands crossing the repair site
Surgery
• Core suture considerations
  – strength of a repair is proportional to the number of
    suture strands crossing the repair site
  – increasing crossing strands increases difficulty and risk of
    tendon damage, vascularity compromise
Surgery
• Core suture considerations
  – strength of a repair is proportional to the number of
    suture strands crossing the repair site
  – increasing crossing strands increases difficulty and risk of
    tendon damage, vascularity compromise
  – repairs usually rupture at the suture knots
Surgery
• Core suture considerations
  – strength of a repair is proportional to the number of
    suture strands crossing the repair site
  – increasing crossing strands increases difficulty and risk of
    tendon damage, vascularity compromise
  – repairs usually rupture at the suture knots
  – locking loop con gurations usually allow less gapping
    than grasping types
Surgery
• Core suture considerations
  – strength of a repair is proportional to the number of
    suture strands crossing the repair site
  – increasing crossing strands increases difficulty and risk of
    tendon damage, vascularity compromise
  – repairs usually rupture at the suture knots
  – locking loop con gurations usually allow less gapping
    than grasping types
  – larger caliber sutures increase repair strength
Surgery
Surgery
• Core suture methods
Surgery
Surgery
• Suture material
Surgery
• Suture material
  – absorbable materials seem attractive
Surgery
• Suture material
  – absorbable materials seem attractive
     • less foreign body reaction long-term
Surgery
• Suture material
  – absorbable materials seem attractive
     • less foreign body reaction long-term
     • less stress-shielding of the host tissue
Surgery
• Suture material
  – absorbable materials seem attractive
     • less foreign body reaction long-term
     • less stress-shielding of the host tissue
  – optimal rates of material absorption, strength reduction
    are unknown
Surgery
• Suture material
  – absorbable materials seem attractive
     • less foreign body reaction long-term
     • less stress-shielding of the host tissue
  – optimal rates of material absorption, strength reduction
    are unknown
  – 3-0, 4-0 braided polyester is most commonly used
Surgery
Surgery
• Circumferential (peripheral) sutures
Surgery
• Circumferential (peripheral) sutures
  – may provide a 10-15% increase in exor tendon repair
    strength
Surgery
• Circumferential (peripheral) sutures
  – may provide a 10-15% increase in exor tendon repair
    strength
  – reduces gapping between ends
Surgery
• Circumferential (peripheral) sutures
  – may provide a 10-15% increase in exor tendon repair
    strength
  – reduces gapping between ends
  – methods
Surgery
• Circumferential (peripheral) sutures
  – may provide a 10-15% increase in exor tendon repair
    strength
  – reduces gapping between ends
  – methods
     • running lock loop (Lin)
Surgery
• Circumferential (peripheral) sutures
  – may provide a 10-15% increase in exor tendon repair
    strength
  – reduces gapping between ends
  – methods
     • running lock loop (Lin)
     • horizontal mattress method (Mashadi)
Surgery
• Circumferential (peripheral) sutures
  – may provide a 10-15% increase in exor tendon repair
    strength
  – reduces gapping between ends
  – methods
     • running lock loop (Lin)
     • horizontal mattress method (Mashadi)
     • Halsted continuous horizontal mattress method (Wade)
Surgery
• Circumferential (peripheral) sutures
  – may provide a 10-15% increase in exor tendon repair
    strength
  – reduces gapping between ends
  – methods
     •   running lock loop (Lin)
     •   horizontal mattress method (Mashadi)
     •   Halsted continuous horizontal mattress method (Wade)
     •   cross-stich technique (Silfverskiold)
Surgery
Surgery
Surgery
Surgery
Surgery
Surgery
Surgery
• Strickland conclusion: Any 4-strand core suture +
  running lock or horizontal mattress circumferential
  suture should permit light composite grip during the
  entire healing period
Surgery
Surgery
• Sheath repair
Surgery
• Sheath repair
  – cumulative work of several investigators fails to give clear
    direction
Surgery
• Sheath repair
  – cumulative work of several investigators fails to give clear
    direction
  – advantages
Surgery
• Sheath repair
  – cumulative work of several investigators fails to give clear
    direction
  – advantages
     • provides a barrier to extrinsic adhesions
Surgery
• Sheath repair
  – cumulative work of several investigators fails to give clear
    direction
  – advantages
     • provides a barrier to extrinsic adhesions
     • quicker return of synovial nutrition
Surgery
• Sheath repair
  – cumulative work of several investigators fails to give clear
    direction
  – advantages
     • provides a barrier to extrinsic adhesions
     • quicker return of synovial nutrition
     • serves as a mold for the remodeling tendon
Surgery
• Sheath repair
  – cumulative work of several investigators fails to give clear
    direction
  – advantages
     •   provides a barrier to extrinsic adhesions
     •   quicker return of synovial nutrition
     •   serves as a mold for the remodeling tendon
     •   results in better sheath-tendon biomechanics
Surgery
• Sheath repair
  – cumulative work of several investigators fails to give clear
    direction
  – advantages
     •   provides a barrier to extrinsic adhesions
     •   quicker return of synovial nutrition
     •   serves as a mold for the remodeling tendon
     •   results in better sheath-tendon biomechanics
  – disadvantages
Surgery
• Sheath repair
  – cumulative work of several investigators fails to give clear
    direction
  – advantages
     •   provides a barrier to extrinsic adhesions
     •   quicker return of synovial nutrition
     •   serves as a mold for the remodeling tendon
     •   results in better sheath-tendon biomechanics
  – disadvantages
     • difficult
Surgery
• Sheath repair
  – cumulative work of several investigators fails to give clear
    direction
  – advantages
     •   provides a barrier to extrinsic adhesions
     •   quicker return of synovial nutrition
     •   serves as a mold for the remodeling tendon
     •   results in better sheath-tendon biomechanics
  – disadvantages
     • difficult
     • may restrict tendon gliding
Surgery
Surgery
• Partial lacerations
Surgery
• Partial lacerations
   – lacerations of 50% or less do not need to be sutured
Surgery
• Partial lacerations
   – lacerations of 50% or less do not need to be sutured
   – Grewal: nonrepaired partial lacerations had higher
     ultimate load and stiffness than repaired tendons
Surgery
• Partial lacerations
   – lacerations of 50% or less do not need to be sutured
   – Grewal: nonrepaired partial lacerations had higher
     ultimate load and stiffness than repaired tendons
   – entrapment, rupture, triggering of unrepaired partial
     severed tendons has been reported (Schlenker)
Surgery
Surgery
• Zone I injury – FDP avulsion
Surgery
• Zone I injury – FDP avulsion
  – occurs w/ forcible extension during strong DIP
     exion
Surgery
• Zone I injury – FDP avulsion
  – occurs w/ forcible extension during strong DIP
      exion
  – three types (Leddy and Packer) – relevant to
      nding the fragment/tendon end and to repair
    timing
Surgery
• Zone I injury – FDP avulsion
  – occurs w/ forcible extension during strong DIP
      exion
  – three types (Leddy and Packer) – relevant to
      nding the fragment/tendon end and to repair
    timing
  – tendon repair to cancellous bone provides the
    best result
Surgery
• Zone I injury – FDP avulsion
  – occurs w/ forcible extension during strong DIP
      exion
  – three types (Leddy and Packer) – relevant to
      nding the fragment/tendon end and to repair
    timing
  – tendon repair to cancellous bone provides the
    best result
     • done with sutures exiting dorsally tied over a button
Surgery
• Zone I injury – FDP avulsion
  – occurs w/ forcible extension during strong DIP
      exion
  – three types (Leddy and Packer) – relevant to
      nding the fragment/tendon end and to repair
    timing
  – tendon repair to cancellous bone provides the
    best result
     • done with sutures exiting dorsally tied over a button
     • OR with suture anchors
Surgery – FDP avulsion
Surgery – FDP avulsion
• Leddy and Packer classi cation
Surgery
Surgery
• FDP laceration – Zone I
Surgery
• FDP laceration – Zone I
  – laceration distal to FDS insertion = Zone I injury
Surgery
• FDP laceration – Zone I
  – laceration distal to FDS insertion = Zone I injury
  – distal stump < 1 cm long = perform FDP advancement
    and tendon to bone repair
Surgery
• FDP laceration – Zone I
  – laceration distal to FDS insertion = Zone I injury
  – distal stump < 1 cm long = perform FDP advancement
    and tendon to bone repair
  – > 1 cm distally = repair primarily
Surgery
• FDP laceration – Zone I
  – laceration distal to FDS insertion = Zone I injury
  – distal stump < 1 cm long = perform FDP advancement
    and tendon to bone repair
  – > 1 cm distally = repair primarily
     • laceration may be near A4  difficult repair
Surgery
• FDP laceration – Zone I
  – laceration distal to FDS insertion = Zone I injury
  – distal stump < 1 cm long = perform FDP advancement
    and tendon to bone repair
  – > 1 cm distally = repair primarily
     • laceration may be near A4  difficult repair
     • advancement of FDP > 1 cm may result in the quadrigia effect
Rehabilitation (zone ii)
Rehabilitation (zone ii)
Rehabilitation (zone ii)
Rehabilitation (zone ii)
Talk Summary
Talk Summary
• Basic science
  – anatomy
  – nutrition
  – biomechanics
  – healing
• Surgical considerations
  – lacerations
  – avulsions
• Rehabilitation
OITE
OITE
2004 #10

A partial laceration of the exor tendon should be repaired when the
   percentage of tendon lacerated is more than

1.   10%
2.   20%
3.   40%
4.   60%
5.   80%
OITE
2004 #10

A partial laceration of the exor tendon should be repaired when the
   percentage of tendon lacerated is more than

1.   10%
2.   20%
3.   40%
4.   60%
5.   80%
OITE
OITE
2005 #246
OITE
2005 #246
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
3.   Musculotendinous rupture in the forearm
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
3.   Musculotendinous rupture in the forearm
4.   Stretch injury to the ulnar nerve at the wrist
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
3.   Musculotendinous rupture in the forearm
4.   Stretch injury to the ulnar nerve at the wrist
5.   Ulnar artery thrombosis
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
3.   Musculotendinous rupture in the forearm
4.   Stretch injury to the ulnar nerve at the wrist
5.   Ulnar artery thrombosis
OITE
OITE
2005 #246
OITE
2005 #246
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
3.   Musculotendinous rupture in the forearm
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
3.   Musculotendinous rupture in the forearm
4.   Stretch injury to the ulnar nerve at the wrist
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
3.   Musculotendinous rupture in the forearm
4.   Stretch injury to the ulnar nerve at the wrist
5.   Ulnar artery thrombosis
OITE
2005 #246

A 21 year-old left-handed college student was playing rugby 3 days ago and
     felt the sudden onset of pain in his right ring nger when another
     played pulled away from him. A clinical photo is shown in Figure 86.
     What is the most likely diagnosis?

1.   Volar plate avulsion of the proximal interphalangeal joint
2.   Avulsion of the distal exor digitorum profundus tendon
3.   Musculotendinous rupture in the forearm
4.   Stretch injury to the ulnar nerve at the wrist
5.   Ulnar artery thrombosis
OITE
OITE
2003 #10

Active mobilization following exor tendon repair is best accomplished
   with the wrist in

1. exion and the metacarpophalangeal joints in exion.
2. exion and the metacarpophalangeal joints in extension.
3. neutral and the metacarpophalangeal joints in extension.
4. neutral and the metacarpophalangeal joints in exion.
5. extension and the metacarpophalangeal joints in exion.
OITE
OITE
2003 #10

Active mobilization following exor tendon repair is best accomplished
   with the wrist in

1. exion and the metacarpophalangeal joints in exion.
2. exion and the metacarpophalangeal joints in extension.
3. neutral and the metacarpophalangeal joints in extension.
4. neutral and the metacarpophalangeal joints in exion.
5. extension and the metacarpophalangeal joints in
    exion.
OITE
OITE
2003 #126

What is the major advantage of allowing early active motion of a repaired
 zone II exor tendon injury?

•   1- Increased tendon excursion
•   2- Greater repair strength
•   3- Less postoperative pain
•   4- Better patient compliance
•   5- Faster tendon healing
OITE
OITE
2003 #126

What is the major advantage of allowing early active motion of a repaired
 zone II exor tendon injury?

• 1- Increased tendon excursion
•   2- Greater repair strength
•   3- Less postoperative pain
•   4- Better patient compliance
•   5- Faster tendon healing
OITE
OITE
2005 #21

e ideal exor tendon rehab protocol that minimizes peritendinous
    adhesions includes

1.   Casting for 6 weeks
2.   A synergistic wrist and digit motion rehabilitation protocol
3.   Dorsal blocking splint with unrestricted active nger exion
4.   Dynamic extension outrigger splinting
5.   Early aggressive active motion and a strengthening program
OITE
OITE
2005 #21

e ideal exor tendon rehab protocol that minimizes peritendinous
    adhesions includes

1.   Casting for 6 weeks
2. A synergistic wrist and digit motion
   rehabilitation protocol
3.   Dorsal blocking splint with unrestricted active nger exion
4.   Dynamic extension outrigger splinting
5.   Early aggressive active motion and a strengthening program
OITE
OITE
2005 #103

A 3 year-old child undergoes repair of lacerated exor tendons of the
     ring and little ngers in zone II. Postoperative rehabilitation should
     consist of

1.   Passive exion and extension exercises
2.   Passive exion and active extension exercises
3.   Active exion and extension exercises
4.   Cast immobilization for 4 weeks
5.   Cast immobilization for 8 weeks
OITE
OITE
2005 #103

A 3 year-old child undergoes repair of lacerated exor tendons of the
     ring and little ngers in zone II. Postoperative rehabilitation should
     consist of

1.   Passive exion and extension exercises
2.   Passive exion and active extension exercises
3.   Active exion and extension exercises
4. Cast immobilization for 4 weeks
5.   Cast immobilization for 8 weeks
OITE
OITE
2002 #70

A 4 year old girl undergoes repair of both exor tendons in zone II.
  Initial postoperative physical therapy should consist of

1.   passive mobilization only.
2.   active extension and passive exion mobilization.
3.   active place and hold mobilization.
4.   immobilization.
5.   unrestricted active mobilization.
OITE
OITE
2002 #70

A 4 year old girl undergoes repair of both exor tendons in zone II.
  Initial postoperative physical therapy should consist of

1. passive mobilization only.
2. active extension and passive exion mobilization.
3. active place and hold mobilization.
4.immobilization.
5. unrestricted active mobilization.
Bibliography




 Trumble, ed. Hand Surgery Update 3., 2003.

 Green, ed. Green’s Operative Hand Surgery, 5th ed., 2003.

 Canale, ed. Campbell’s Operative Orthopaedic Surgery, 10th ed., 2003.
Bibliography




 Trumble, ed. Hand Surgery Update 3., 2003.

 Green, ed. Green’s Operative Hand Surgery, 5th ed., 2003.

 Canale, ed. Campbell’s Operative Orthopaedic Surgery, 10th ed., 2003.
Bibliography
Bibliography




        Schmidt. Surgical Anatomy of the Hand. 2004

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Flexor Tendons - Anatomy, Injury, Treatment

  • 2. Flexor Tendon Injuries IU Orthopaedic Surgery C. Noel Henley, MD
  • 3. Flexor Tendon Injuries IU Orthopaedic Surgery April 5, 2006 C. Noel Henley, MD
  • 6. Talk Summary • Introduction • Basic science – anatomy – nutrition – biomechanics – healing • Surgical considerations – lacerations – avulsions • Rehabilitation
  • 9. Introduction • History – no repair possible in the digit
  • 10. Introduction • History – no repair possible in the digit – “no man’s land”
  • 11. Introduction • History – no repair possible in the digit – “no man’s land” • Current evidence
  • 12. Introduction • History – no repair possible in the digit – “no man’s land” • Current evidence – 1970s: repair is possible without delayed grafting, reconstruction
  • 13. Introduction • History – no repair possible in the digit – “no man’s land” • Current evidence – 1970s: repair is possible without delayed grafting, reconstruction – Now: adhesion-free, primarily healed tendon repair is possible
  • 16. Introduction • General goals of repair – primary tendon repair – avoid grafting
  • 17. Introduction • General goals of repair – primary tendon repair – avoid grafting – sufficient strength for passive motion rehab which will allow for
  • 18. Introduction • General goals of repair – primary tendon repair – avoid grafting – sufficient strength for passive motion rehab which will allow for • minimum adhesion formation
  • 19. Introduction • General goals of repair – primary tendon repair – avoid grafting – sufficient strength for passive motion rehab which will allow for • minimum adhesion formation • restoration of gliding surface
  • 20. Introduction • General goals of repair – primary tendon repair – avoid grafting – sufficient strength for passive motion rehab which will allow for • minimum adhesion formation • restoration of gliding surface • facilitation of repair site healing
  • 23. Anatomy • FDS – O: volar humerus, radius, ulna
  • 24. Anatomy • FDS – O: volar humerus, radius, ulna – divided muscle belly in mid forearm (FA)
  • 25. Anatomy • FDS – O: volar humerus, radius, ulna – divided muscle belly in mid forearm (FA) • super cial – long and ring ngers (LF, RF)
  • 26. Anatomy • FDS – O: volar humerus, radius, ulna – divided muscle belly in mid forearm (FA) • super cial – long and ring ngers (LF, RF) • deep – index and small ngers (IF, SF)
  • 27. Anatomy • FDS – O: volar humerus, radius, ulna – divided muscle belly in mid forearm (FA) • super cial – long and ring ngers (LF, RF) • deep – index and small ngers (IF, SF) • FDP
  • 28. Anatomy • FDS – O: volar humerus, radius, ulna – divided muscle belly in mid forearm (FA) • super cial – long and ring ngers (LF, RF) • deep – index and small ngers (IF, SF) • FDP – O: anteromedial ulna, IO membrane
  • 29. Anatomy • FDS – O: volar humerus, radius, ulna – divided muscle belly in mid forearm (FA) • super cial – long and ring ngers (LF, RF) • deep – index and small ngers (IF, SF) • FDP – O: anteromedial ulna, IO membrane – dorsal to FDS in volar FA
  • 30. Anatomy • FDS – O: volar humerus, radius, ulna – divided muscle belly in mid forearm (FA) • super cial – long and ring ngers (LF, RF) • deep – index and small ngers (IF, SF) • FDP – O: anteromedial ulna, IO membrane – dorsal to FDS in volar FA – four tendons usually arise from common belly
  • 33. Anatomy • In the hand – ve zones
  • 34. Anatomy • In the hand – ve zones • V: musculo-tendinous junction to proximal edge of carpal canal
  • 35. Anatomy • In the hand – ve zones • V: musculo-tendinous junction to proximal edge of carpal canal • IV: beneath (dorsal to) the transverse carpal ligament
  • 36. Anatomy • In the hand – ve zones • V: musculo-tendinous junction to proximal edge of carpal canal • IV: beneath (dorsal to) the transverse carpal ligament – IF, SF FDS are dorsal to LF, RF FDS here
  • 39. Anatomy • Zones (cont.) – III: distal edge of transverse carpal ligament to origin of bro-osseous sheath at the distal palmar crease
  • 40. Anatomy • Zones (cont.) – III: distal edge of transverse carpal ligament to origin of bro-osseous sheath at the distal palmar crease – II: origin of bro-osseous sheath to FDS insertion
  • 41. Anatomy • Zones (cont.) – III: distal edge of transverse carpal ligament to origin of bro-osseous sheath at the distal palmar crease – II: origin of bro-osseous sheath to FDS insertion – I: distal to FDS insertion
  • 44. Anatomy • Super cialis – FDS divides into two slips, wrapping around FDP; reunite at Camper’s chiasma
  • 45. Anatomy • Super cialis – FDS divides into two slips, wrapping around FDP; reunite at Camper’s chiasma – continues to insert along the proximal half of the middle phalanx (P2)
  • 49. Anatomy • Profundus – passes through chiasma to insert at proximal base of distal phalanx (P3)
  • 50. Anatomy • Profundus – passes through chiasma to insert at proximal base of distal phalanx (P3) – FDP = primary digital exor
  • 51. Anatomy • Profundus – passes through chiasma to insert at proximal base of distal phalanx (P3) – FDP = primary digital exor – FDS + intrinsics combine for forceful exion
  • 56. Anatomy • Sheath – visceral and parietal synovial layers enclose the tendons
  • 57. Anatomy • Sheath – visceral and parietal synovial layers enclose the tendons – Pulleys
  • 58. Anatomy • Sheath – visceral and parietal synovial layers enclose the tendons – Pulleys • A2 and A4 arise from periosteum of proximal P1 and midportion of P2 (prox- prox and mid-middle)
  • 61. Anatomy • Sheath – Pulleys
  • 62. Anatomy • Sheath – Pulleys • A1, A3, and A5 arise from volar plates of MCP, PIP, and DIP joints respectively
  • 65. Anatomy • Sheath – Pulleys
  • 66. Anatomy • Sheath – Pulleys • cruciate pulleys collapse to allow annular pulley apposition during exion
  • 68. Nutrition • Two sources of tendon nutrition
  • 69. Nutrition • Two sources of tendon nutrition – vascular
  • 70. Nutrition • Two sources of tendon nutrition – vascular – synovial
  • 71. Nutrition • Two sources of tendon nutrition – vascular – synovial • Vascularity sources
  • 72. Nutrition • Two sources of tendon nutrition – vascular – synovial • Vascularity sources – longitudinal vessels
  • 73. Nutrition • Two sources of tendon nutrition – vascular – synovial • Vascularity sources – longitudinal vessels – proximal synovial fold vessels
  • 74. Nutrition • Two sources of tendon nutrition – vascular – synovial • Vascularity sources – longitudinal vessels – proximal synovial fold vessels – segmental branches from paired digital vessels (vincular system)
  • 75. Nutrition • Two sources of tendon nutrition – vascular – synovial • Vascularity sources – longitudinal vessels – proximal synovial fold vessels – segmental branches from paired digital vessels (vincular system) – insertional vessels at FDP, FDS insertions
  • 80. Nutrition • Avascular segments – FDP and FDS: over proximal phalanx
  • 81. Nutrition • Avascular segments – FDP and FDS: over proximal phalanx – FDP: short zone over middle phalanx
  • 82. Nutrition • Avascular segments – FDP and FDS: over proximal phalanx – FDP: short zone over middle phalanx • Synovial uid diffusion
  • 83. Nutrition • Avascular segments – FDP and FDS: over proximal phalanx – FDP: short zone over middle phalanx • Synovial uid diffusion – imbibition
  • 88. Nutrition • Vascularity – damage to vascular and uid nutritional systems may further hamper healing of repaired exor tendons
  • 89. Nutrition • Vascularity – damage to vascular and uid nutritional systems may further hamper healing of repaired exor tendons – their integrity must be respected during all reparative efforts
  • 94. Biomechanics • Excursion – 9 cm of excursion for composite wrist, full digital exion
  • 95. Biomechanics • Excursion – 9 cm of excursion for composite wrist, full digital exion – 2.5 cm required for full digital exion with wrist at neutral
  • 98. Biomechanics • Moment arm – increased distance of tendon from joint center of rotation = higher moment arm = less motion per muscle contraction force
  • 101. Biomechanics • Moment arm – pulley system constraint governs these parameters
  • 102. Biomechanics • Moment arm – pulley system constraint governs these parameters – loss of portions of pulley system may alter balance between exor, intrinsic, and extensor tendons
  • 103. Biomechanics • Moment arm – pulley system constraint governs these parameters – loss of portions of pulley system may alter balance between exor, intrinsic, and extensor tendons – loss of A2 or A4 pulleys may diminish motion/power or lead to contractures of IP joints
  • 106. Tendon Healing • Phases – in ammatory – 48-72 hours
  • 107. Tendon Healing • Phases – in ammatory – 48-72 hours • repair strength reliant upon suture itself
  • 108. Tendon Healing • Phases – in ammatory – 48-72 hours • repair strength reliant upon suture itself – collagen-producing phase – 5 days to 4 weeks
  • 109. Tendon Healing • Phases – in ammatory – 48-72 hours • repair strength reliant upon suture itself – collagen-producing phase – 5 days to 4 weeks • strength accelerates rapidly here
  • 110. Tendon Healing • Phases – in ammatory – 48-72 hours • repair strength reliant upon suture itself – collagen-producing phase – 5 days to 4 weeks • strength accelerates rapidly here – remodeling phase – until 112 days
  • 111. Tendon Healing • Phases – in ammatory – 48-72 hours • repair strength reliant upon suture itself – collagen-producing phase – 5 days to 4 weeks • strength accelerates rapidly here – remodeling phase – until 112 days • Predominance of extrinsic over intrinsic healing leads to more adhesions
  • 112. Tendon Healing inflammatory
  • 113. Tendon Healing inflammatory
  • 114. Tendon Healing collagen-producing
  • 115. Tendon Healing collagen-producing
  • 116. Tendon Healing remodeling
  • 117. Tendon Healing remodeling
  • 119. Tendon Healing • Adhesion formation and control
  • 120. Tendon Healing • Adhesion formation and control – Contributing factors
  • 121. Tendon Healing • Adhesion formation and control – Contributing factors • trauma (from injury and surgery)
  • 122. Tendon Healing • Adhesion formation and control – Contributing factors • trauma (from injury and surgery) – tendon surface crushing
  • 123. Tendon Healing • Adhesion formation and control – Contributing factors • trauma (from injury and surgery) – tendon surface crushing – vincula disruption
  • 124. Tendon Healing • Adhesion formation and control – Contributing factors • trauma (from injury and surgery) – tendon surface crushing – vincula disruption • tendon ischemia
  • 125. Tendon Healing • Adhesion formation and control – Contributing factors • trauma (from injury and surgery) – tendon surface crushing – vincula disruption • tendon ischemia • tendon immobilization
  • 126. Tendon Healing • Adhesion formation and control – Contributing factors • trauma (from injury and surgery) – tendon surface crushing – vincula disruption • tendon ischemia • tendon immobilization • repair gapping
  • 127. Tendon Healing • Adhesion formation and control – Contributing factors • trauma (from injury and surgery) – tendon surface crushing – vincula disruption • tendon ischemia • tendon immobilization • repair gapping • excision of tendon sheath components
  • 129. Tendon Healing • How is healing related to motion?
  • 130. Tendon Healing • How is healing related to motion? – passive mobilization enhances healing by stimulating tendon wound maturation and scar remodeling* , 110 *Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA (eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill: American Academy of Orthopaedic Surgeons, 1988, pp 5-40.
  • 131. Tendon Healing • How is healing related to motion? – passive mobilization enhances healing by stimulating tendon wound maturation and scar remodeling* , 110 – use a strong, gap-resistant suture technique + controlled-motion stress postoperatively *Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA (eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill: American Academy of Orthopaedic Surgeons, 1988, pp 5-40.
  • 132. Tendon Healing • How is healing related to motion? – passive mobilization enhances healing by stimulating tendon wound maturation and scar remodeling* , 110 – use a strong, gap-resistant suture technique + controlled-motion stress postoperatively – 3-5 mm of excursion needed at repair site to prevent adhesions 144 *Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA (eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill: American Academy of Orthopaedic Surgeons, 1988, pp 5-40.
  • 135. Tendon Repair • Contraindications – severe multiple tissue injuries to ngers, palm
  • 136. Tendon Repair • Contraindications – severe multiple tissue injuries to ngers, palm – gross wound contamination
  • 137. Tendon Repair • Contraindications – severe multiple tissue injuries to ngers, palm – gross wound contamination – signi cant skin loss over the exor system
  • 138. Tendon Repair • Contraindications – severe multiple tissue injuries to ngers, palm – gross wound contamination – signi cant skin loss over the exor system • Reduce fractures, repair neurovascular injuries concomitantly
  • 139. Tendon Repair • Contraindications – severe multiple tissue injuries to ngers, palm – gross wound contamination – signi cant skin loss over the exor system • Reduce fractures, repair neurovascular injuries concomitantly • Repair tendons acutely/subacutely to avoid deterioration of tendon ends and extrinsic muscle shortening
  • 141. Examination • High index of suspicion for multiple structures injured through a small laceration or crush injury
  • 142. Examination • High index of suspicion for multiple structures injured through a small laceration or crush injury • Complete neurovascular exam
  • 143. Examination • High index of suspicion for multiple structures injured through a small laceration or crush injury • Complete neurovascular exam – division of two digital nerves indicates arterial division as well
  • 144. Examination • High index of suspicion for multiple structures injured through a small laceration or crush injury • Complete neurovascular exam – division of two digital nerves indicates arterial division as well – dysvascularity could compromise repair or skin ap healing +/- cold intolerance
  • 160. Surgery • Planning – inform the patient of the injury severity and potential need for more surgery
  • 161. Surgery • Planning – inform the patient of the injury severity and potential need for more surgery – incisions
  • 162. Surgery • Planning – inform the patient of the injury severity and potential need for more surgery – incisions • keep tendon retraction in mind
  • 163. Surgery • Planning – inform the patient of the injury severity and potential need for more surgery – incisions • keep tendon retraction in mind • no bonus points for small incisions
  • 164. Surgery • Planning – inform the patient of the injury severity and potential need for more surgery – incisions • keep tendon retraction in mind • no bonus points for small incisions • maintain ap viability
  • 165. Surgery • Planning – inform the patient of the injury severity and potential need for more surgery – incisions • keep tendon retraction in mind • no bonus points for small incisions • maintain ap viability • avoid skin/scar contracture
  • 167. Surgery - planning • Volar incisions
  • 168. Surgery - planning • Volar incisions • Mid-axial incision
  • 170. Surgery - principles • Ideal repair
  • 171. Surgery - principles • Ideal repair – easy placement of sutures in the tendon
  • 172. Surgery - principles • Ideal repair – easy placement of sutures in the tendon – secure suture knots
  • 173. Surgery - principles • Ideal repair – easy placement of sutures in the tendon – secure suture knots – smooth junction of tendon ends
  • 174. Surgery - principles • Ideal repair – easy placement of sutures in the tendon – secure suture knots – smooth junction of tendon ends – minimal gapping at the repair site
  • 175. Surgery - principles • Ideal repair – easy placement of sutures in the tendon – secure suture knots – smooth junction of tendon ends – minimal gapping at the repair site – minimal interference with tendon vascularity
  • 176. Surgery - principles • Ideal repair – easy placement of sutures in the tendon – secure suture knots – smooth junction of tendon ends – minimal gapping at the repair site – minimal interference with tendon vascularity – sufficient strength for early motion stress to the tendon
  • 178. Surgery • Core suture considerations
  • 179. Surgery • Core suture considerations – strength of a repair is proportional to the number of suture strands crossing the repair site
  • 180. Surgery • Core suture considerations – strength of a repair is proportional to the number of suture strands crossing the repair site – increasing crossing strands increases difficulty and risk of tendon damage, vascularity compromise
  • 181. Surgery • Core suture considerations – strength of a repair is proportional to the number of suture strands crossing the repair site – increasing crossing strands increases difficulty and risk of tendon damage, vascularity compromise – repairs usually rupture at the suture knots
  • 182. Surgery • Core suture considerations – strength of a repair is proportional to the number of suture strands crossing the repair site – increasing crossing strands increases difficulty and risk of tendon damage, vascularity compromise – repairs usually rupture at the suture knots – locking loop con gurations usually allow less gapping than grasping types
  • 183. Surgery • Core suture considerations – strength of a repair is proportional to the number of suture strands crossing the repair site – increasing crossing strands increases difficulty and risk of tendon damage, vascularity compromise – repairs usually rupture at the suture knots – locking loop con gurations usually allow less gapping than grasping types – larger caliber sutures increase repair strength
  • 188. Surgery • Suture material – absorbable materials seem attractive
  • 189. Surgery • Suture material – absorbable materials seem attractive • less foreign body reaction long-term
  • 190. Surgery • Suture material – absorbable materials seem attractive • less foreign body reaction long-term • less stress-shielding of the host tissue
  • 191. Surgery • Suture material – absorbable materials seem attractive • less foreign body reaction long-term • less stress-shielding of the host tissue – optimal rates of material absorption, strength reduction are unknown
  • 192. Surgery • Suture material – absorbable materials seem attractive • less foreign body reaction long-term • less stress-shielding of the host tissue – optimal rates of material absorption, strength reduction are unknown – 3-0, 4-0 braided polyester is most commonly used
  • 195. Surgery • Circumferential (peripheral) sutures – may provide a 10-15% increase in exor tendon repair strength
  • 196. Surgery • Circumferential (peripheral) sutures – may provide a 10-15% increase in exor tendon repair strength – reduces gapping between ends
  • 197. Surgery • Circumferential (peripheral) sutures – may provide a 10-15% increase in exor tendon repair strength – reduces gapping between ends – methods
  • 198. Surgery • Circumferential (peripheral) sutures – may provide a 10-15% increase in exor tendon repair strength – reduces gapping between ends – methods • running lock loop (Lin)
  • 199. Surgery • Circumferential (peripheral) sutures – may provide a 10-15% increase in exor tendon repair strength – reduces gapping between ends – methods • running lock loop (Lin) • horizontal mattress method (Mashadi)
  • 200. Surgery • Circumferential (peripheral) sutures – may provide a 10-15% increase in exor tendon repair strength – reduces gapping between ends – methods • running lock loop (Lin) • horizontal mattress method (Mashadi) • Halsted continuous horizontal mattress method (Wade)
  • 201. Surgery • Circumferential (peripheral) sutures – may provide a 10-15% increase in exor tendon repair strength – reduces gapping between ends – methods • running lock loop (Lin) • horizontal mattress method (Mashadi) • Halsted continuous horizontal mattress method (Wade) • cross-stich technique (Silfverskiold)
  • 208. Surgery • Strickland conclusion: Any 4-strand core suture + running lock or horizontal mattress circumferential suture should permit light composite grip during the entire healing period
  • 211. Surgery • Sheath repair – cumulative work of several investigators fails to give clear direction
  • 212. Surgery • Sheath repair – cumulative work of several investigators fails to give clear direction – advantages
  • 213. Surgery • Sheath repair – cumulative work of several investigators fails to give clear direction – advantages • provides a barrier to extrinsic adhesions
  • 214. Surgery • Sheath repair – cumulative work of several investigators fails to give clear direction – advantages • provides a barrier to extrinsic adhesions • quicker return of synovial nutrition
  • 215. Surgery • Sheath repair – cumulative work of several investigators fails to give clear direction – advantages • provides a barrier to extrinsic adhesions • quicker return of synovial nutrition • serves as a mold for the remodeling tendon
  • 216. Surgery • Sheath repair – cumulative work of several investigators fails to give clear direction – advantages • provides a barrier to extrinsic adhesions • quicker return of synovial nutrition • serves as a mold for the remodeling tendon • results in better sheath-tendon biomechanics
  • 217. Surgery • Sheath repair – cumulative work of several investigators fails to give clear direction – advantages • provides a barrier to extrinsic adhesions • quicker return of synovial nutrition • serves as a mold for the remodeling tendon • results in better sheath-tendon biomechanics – disadvantages
  • 218. Surgery • Sheath repair – cumulative work of several investigators fails to give clear direction – advantages • provides a barrier to extrinsic adhesions • quicker return of synovial nutrition • serves as a mold for the remodeling tendon • results in better sheath-tendon biomechanics – disadvantages • difficult
  • 219. Surgery • Sheath repair – cumulative work of several investigators fails to give clear direction – advantages • provides a barrier to extrinsic adhesions • quicker return of synovial nutrition • serves as a mold for the remodeling tendon • results in better sheath-tendon biomechanics – disadvantages • difficult • may restrict tendon gliding
  • 222. Surgery • Partial lacerations – lacerations of 50% or less do not need to be sutured
  • 223. Surgery • Partial lacerations – lacerations of 50% or less do not need to be sutured – Grewal: nonrepaired partial lacerations had higher ultimate load and stiffness than repaired tendons
  • 224. Surgery • Partial lacerations – lacerations of 50% or less do not need to be sutured – Grewal: nonrepaired partial lacerations had higher ultimate load and stiffness than repaired tendons – entrapment, rupture, triggering of unrepaired partial severed tendons has been reported (Schlenker)
  • 226. Surgery • Zone I injury – FDP avulsion
  • 227. Surgery • Zone I injury – FDP avulsion – occurs w/ forcible extension during strong DIP exion
  • 228. Surgery • Zone I injury – FDP avulsion – occurs w/ forcible extension during strong DIP exion – three types (Leddy and Packer) – relevant to nding the fragment/tendon end and to repair timing
  • 229. Surgery • Zone I injury – FDP avulsion – occurs w/ forcible extension during strong DIP exion – three types (Leddy and Packer) – relevant to nding the fragment/tendon end and to repair timing – tendon repair to cancellous bone provides the best result
  • 230. Surgery • Zone I injury – FDP avulsion – occurs w/ forcible extension during strong DIP exion – three types (Leddy and Packer) – relevant to nding the fragment/tendon end and to repair timing – tendon repair to cancellous bone provides the best result • done with sutures exiting dorsally tied over a button
  • 231. Surgery • Zone I injury – FDP avulsion – occurs w/ forcible extension during strong DIP exion – three types (Leddy and Packer) – relevant to nding the fragment/tendon end and to repair timing – tendon repair to cancellous bone provides the best result • done with sutures exiting dorsally tied over a button • OR with suture anchors
  • 232. Surgery – FDP avulsion
  • 233. Surgery – FDP avulsion • Leddy and Packer classi cation
  • 236. Surgery • FDP laceration – Zone I – laceration distal to FDS insertion = Zone I injury
  • 237. Surgery • FDP laceration – Zone I – laceration distal to FDS insertion = Zone I injury – distal stump < 1 cm long = perform FDP advancement and tendon to bone repair
  • 238. Surgery • FDP laceration – Zone I – laceration distal to FDS insertion = Zone I injury – distal stump < 1 cm long = perform FDP advancement and tendon to bone repair – > 1 cm distally = repair primarily
  • 239. Surgery • FDP laceration – Zone I – laceration distal to FDS insertion = Zone I injury – distal stump < 1 cm long = perform FDP advancement and tendon to bone repair – > 1 cm distally = repair primarily • laceration may be near A4  difficult repair
  • 240. Surgery • FDP laceration – Zone I – laceration distal to FDS insertion = Zone I injury – distal stump < 1 cm long = perform FDP advancement and tendon to bone repair – > 1 cm distally = repair primarily • laceration may be near A4  difficult repair • advancement of FDP > 1 cm may result in the quadrigia effect
  • 246. Talk Summary • Basic science – anatomy – nutrition – biomechanics – healing • Surgical considerations – lacerations – avulsions • Rehabilitation
  • 247. OITE
  • 248. OITE 2004 #10 A partial laceration of the exor tendon should be repaired when the percentage of tendon lacerated is more than 1. 10% 2. 20% 3. 40% 4. 60% 5. 80%
  • 249. OITE 2004 #10 A partial laceration of the exor tendon should be repaired when the percentage of tendon lacerated is more than 1. 10% 2. 20% 3. 40% 4. 60% 5. 80%
  • 250. OITE
  • 253. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis?
  • 254. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis?
  • 255. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint
  • 256. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon
  • 257. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon 3. Musculotendinous rupture in the forearm
  • 258. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon 3. Musculotendinous rupture in the forearm 4. Stretch injury to the ulnar nerve at the wrist
  • 259. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon 3. Musculotendinous rupture in the forearm 4. Stretch injury to the ulnar nerve at the wrist 5. Ulnar artery thrombosis
  • 260. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon 3. Musculotendinous rupture in the forearm 4. Stretch injury to the ulnar nerve at the wrist 5. Ulnar artery thrombosis
  • 261.
  • 262. OITE
  • 265. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis?
  • 266. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis?
  • 267. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint
  • 268. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon
  • 269. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon 3. Musculotendinous rupture in the forearm
  • 270. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon 3. Musculotendinous rupture in the forearm 4. Stretch injury to the ulnar nerve at the wrist
  • 271. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon 3. Musculotendinous rupture in the forearm 4. Stretch injury to the ulnar nerve at the wrist 5. Ulnar artery thrombosis
  • 272. OITE 2005 #246 A 21 year-old left-handed college student was playing rugby 3 days ago and felt the sudden onset of pain in his right ring nger when another played pulled away from him. A clinical photo is shown in Figure 86. What is the most likely diagnosis? 1. Volar plate avulsion of the proximal interphalangeal joint 2. Avulsion of the distal exor digitorum profundus tendon 3. Musculotendinous rupture in the forearm 4. Stretch injury to the ulnar nerve at the wrist 5. Ulnar artery thrombosis
  • 273. OITE
  • 274. OITE 2003 #10 Active mobilization following exor tendon repair is best accomplished with the wrist in 1. exion and the metacarpophalangeal joints in exion. 2. exion and the metacarpophalangeal joints in extension. 3. neutral and the metacarpophalangeal joints in extension. 4. neutral and the metacarpophalangeal joints in exion. 5. extension and the metacarpophalangeal joints in exion.
  • 275. OITE
  • 276. OITE 2003 #10 Active mobilization following exor tendon repair is best accomplished with the wrist in 1. exion and the metacarpophalangeal joints in exion. 2. exion and the metacarpophalangeal joints in extension. 3. neutral and the metacarpophalangeal joints in extension. 4. neutral and the metacarpophalangeal joints in exion. 5. extension and the metacarpophalangeal joints in exion.
  • 277. OITE
  • 278. OITE 2003 #126 What is the major advantage of allowing early active motion of a repaired zone II exor tendon injury? • 1- Increased tendon excursion • 2- Greater repair strength • 3- Less postoperative pain • 4- Better patient compliance • 5- Faster tendon healing
  • 279. OITE
  • 280. OITE 2003 #126 What is the major advantage of allowing early active motion of a repaired zone II exor tendon injury? • 1- Increased tendon excursion • 2- Greater repair strength • 3- Less postoperative pain • 4- Better patient compliance • 5- Faster tendon healing
  • 281. OITE
  • 282. OITE 2005 #21 e ideal exor tendon rehab protocol that minimizes peritendinous adhesions includes 1. Casting for 6 weeks 2. A synergistic wrist and digit motion rehabilitation protocol 3. Dorsal blocking splint with unrestricted active nger exion 4. Dynamic extension outrigger splinting 5. Early aggressive active motion and a strengthening program
  • 283. OITE
  • 284. OITE 2005 #21 e ideal exor tendon rehab protocol that minimizes peritendinous adhesions includes 1. Casting for 6 weeks 2. A synergistic wrist and digit motion rehabilitation protocol 3. Dorsal blocking splint with unrestricted active nger exion 4. Dynamic extension outrigger splinting 5. Early aggressive active motion and a strengthening program
  • 285. OITE
  • 286. OITE 2005 #103 A 3 year-old child undergoes repair of lacerated exor tendons of the ring and little ngers in zone II. Postoperative rehabilitation should consist of 1. Passive exion and extension exercises 2. Passive exion and active extension exercises 3. Active exion and extension exercises 4. Cast immobilization for 4 weeks 5. Cast immobilization for 8 weeks
  • 287. OITE
  • 288. OITE 2005 #103 A 3 year-old child undergoes repair of lacerated exor tendons of the ring and little ngers in zone II. Postoperative rehabilitation should consist of 1. Passive exion and extension exercises 2. Passive exion and active extension exercises 3. Active exion and extension exercises 4. Cast immobilization for 4 weeks 5. Cast immobilization for 8 weeks
  • 289. OITE
  • 290. OITE 2002 #70 A 4 year old girl undergoes repair of both exor tendons in zone II. Initial postoperative physical therapy should consist of 1. passive mobilization only. 2. active extension and passive exion mobilization. 3. active place and hold mobilization. 4. immobilization. 5. unrestricted active mobilization.
  • 291. OITE
  • 292. OITE 2002 #70 A 4 year old girl undergoes repair of both exor tendons in zone II. Initial postoperative physical therapy should consist of 1. passive mobilization only. 2. active extension and passive exion mobilization. 3. active place and hold mobilization. 4.immobilization. 5. unrestricted active mobilization.
  • 293. Bibliography Trumble, ed. Hand Surgery Update 3., 2003. Green, ed. Green’s Operative Hand Surgery, 5th ed., 2003. Canale, ed. Campbell’s Operative Orthopaedic Surgery, 10th ed., 2003.
  • 294. Bibliography Trumble, ed. Hand Surgery Update 3., 2003. Green, ed. Green’s Operative Hand Surgery, 5th ed., 2003. Canale, ed. Campbell’s Operative Orthopaedic Surgery, 10th ed., 2003.
  • 296. Bibliography Schmidt. Surgical Anatomy of the Hand. 2004

Notas do Editor

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  13. achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
  14. achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
  15. achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
  16. achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
  17. achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
  18. achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
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  39. chiasm&amp;#xA0;(chi&amp;#xB7;asm) (ki&amp;#xB4;az-&amp;#x259;m) [L., Gr. chiasma] &amp;#xA0;a decussation or X-shaped crossing; see chiasma.\nc. of digits of hand &amp;#xA0;chiasma tendinum digitorum manus. \noptic c. &amp;#xA0;chiasma opticum. \ntendinous c. of fingers &amp;#xA0;chiasma tendinum digitorum manus. \nchiasma&amp;#xA0;(chi&amp;#xB7;as&amp;#xB7;ma) (ki-az&amp;#xB4;m&amp;#x259;) &amp;#xA0;pl. chias&amp;#xB4;mata [L., from Gr. &amp;#x201C;a cross, crosspiece&amp;#x201D;, from the shape of the letter chi, (X)] &amp;#xA0;1.&amp;#xA0; [TA]&amp;#xA0;a general term in anatomical nomenclature for a decussation or X-shaped crossing, such as of nerves. &amp;#xA0;2.&amp;#xA0;in genetics, the places where pairs of homologous chromatids remain in contact during late prophase to anaphase of the first meiotic division, indicating where an exchange of homologous segments has taken place between non-sister chromatids by crossing over. \noptic c. ,&amp;#xA0;&amp;#xA0;c. op&amp;#xB4;ticum &amp;#xA0;[TA] &amp;#xA0;optic chiasm: the part of the hypothalamus formed by the decussation, or crossing, of the fibers of the optic nerve from the medial half of each retina; called also optic decussation.Click here to view imagec. ten&amp;#xB4;dinum digito&amp;#xB4;rum ma&amp;#xB4;nus &amp;#xA0;[TA] &amp;#xA0;tendinous chiasm of fingers: the crossing of the tendons of the flexor digitorum profundus through the tendons of the flexor digitorum superficialis; called also chiasm of digits of hand.\n
  40. chiasm&amp;#xA0;(chi&amp;#xB7;asm) (ki&amp;#xB4;az-&amp;#x259;m) [L., Gr. chiasma] &amp;#xA0;a decussation or X-shaped crossing; see chiasma.\nc. of digits of hand &amp;#xA0;chiasma tendinum digitorum manus. \noptic c. &amp;#xA0;chiasma opticum. \ntendinous c. of fingers &amp;#xA0;chiasma tendinum digitorum manus. \nchiasma&amp;#xA0;(chi&amp;#xB7;as&amp;#xB7;ma) (ki-az&amp;#xB4;m&amp;#x259;) &amp;#xA0;pl. chias&amp;#xB4;mata [L., from Gr. &amp;#x201C;a cross, crosspiece&amp;#x201D;, from the shape of the letter chi, (X)] &amp;#xA0;1.&amp;#xA0; [TA]&amp;#xA0;a general term in anatomical nomenclature for a decussation or X-shaped crossing, such as of nerves. &amp;#xA0;2.&amp;#xA0;in genetics, the places where pairs of homologous chromatids remain in contact during late prophase to anaphase of the first meiotic division, indicating where an exchange of homologous segments has taken place between non-sister chromatids by crossing over. \noptic c. ,&amp;#xA0;&amp;#xA0;c. op&amp;#xB4;ticum &amp;#xA0;[TA] &amp;#xA0;optic chiasm: the part of the hypothalamus formed by the decussation, or crossing, of the fibers of the optic nerve from the medial half of each retina; called also optic decussation.Click here to view imagec. ten&amp;#xB4;dinum digito&amp;#xB4;rum ma&amp;#xB4;nus &amp;#xA0;[TA] &amp;#xA0;tendinous chiasm of fingers: the crossing of the tendons of the flexor digitorum profundus through the tendons of the flexor digitorum superficialis; called also chiasm of digits of hand.\n
  41. chiasm&amp;#xA0;(chi&amp;#xB7;asm) (ki&amp;#xB4;az-&amp;#x259;m) [L., Gr. chiasma] &amp;#xA0;a decussation or X-shaped crossing; see chiasma.\nc. of digits of hand &amp;#xA0;chiasma tendinum digitorum manus. \noptic c. &amp;#xA0;chiasma opticum. \ntendinous c. of fingers &amp;#xA0;chiasma tendinum digitorum manus. \nchiasma&amp;#xA0;(chi&amp;#xB7;as&amp;#xB7;ma) (ki-az&amp;#xB4;m&amp;#x259;) &amp;#xA0;pl. chias&amp;#xB4;mata [L., from Gr. &amp;#x201C;a cross, crosspiece&amp;#x201D;, from the shape of the letter chi, (X)] &amp;#xA0;1.&amp;#xA0; [TA]&amp;#xA0;a general term in anatomical nomenclature for a decussation or X-shaped crossing, such as of nerves. &amp;#xA0;2.&amp;#xA0;in genetics, the places where pairs of homologous chromatids remain in contact during late prophase to anaphase of the first meiotic division, indicating where an exchange of homologous segments has taken place between non-sister chromatids by crossing over. \noptic c. ,&amp;#xA0;&amp;#xA0;c. op&amp;#xB4;ticum &amp;#xA0;[TA] &amp;#xA0;optic chiasm: the part of the hypothalamus formed by the decussation, or crossing, of the fibers of the optic nerve from the medial half of each retina; called also optic decussation.Click here to view imagec. ten&amp;#xB4;dinum digito&amp;#xB4;rum ma&amp;#xB4;nus &amp;#xA0;[TA] &amp;#xA0;tendinous chiasm of fingers: the crossing of the tendons of the flexor digitorum profundus through the tendons of the flexor digitorum superficialis; called also chiasm of digits of hand.\n
  42. chiasm&amp;#xA0;(chi&amp;#xB7;asm) (ki&amp;#xB4;az-&amp;#x259;m) [L., Gr. chiasma] &amp;#xA0;a decussation or X-shaped crossing; see chiasma.\nc. of digits of hand &amp;#xA0;chiasma tendinum digitorum manus. \noptic c. &amp;#xA0;chiasma opticum. \ntendinous c. of fingers &amp;#xA0;chiasma tendinum digitorum manus. \nchiasma&amp;#xA0;(chi&amp;#xB7;as&amp;#xB7;ma) (ki-az&amp;#xB4;m&amp;#x259;) &amp;#xA0;pl. chias&amp;#xB4;mata [L., from Gr. &amp;#x201C;a cross, crosspiece&amp;#x201D;, from the shape of the letter chi, (X)] &amp;#xA0;1.&amp;#xA0; [TA]&amp;#xA0;a general term in anatomical nomenclature for a decussation or X-shaped crossing, such as of nerves. &amp;#xA0;2.&amp;#xA0;in genetics, the places where pairs of homologous chromatids remain in contact during late prophase to anaphase of the first meiotic division, indicating where an exchange of homologous segments has taken place between non-sister chromatids by crossing over. \noptic c. ,&amp;#xA0;&amp;#xA0;c. op&amp;#xB4;ticum &amp;#xA0;[TA] &amp;#xA0;optic chiasm: the part of the hypothalamus formed by the decussation, or crossing, of the fibers of the optic nerve from the medial half of each retina; called also optic decussation.Click here to view imagec. ten&amp;#xB4;dinum digito&amp;#xB4;rum ma&amp;#xB4;nus &amp;#xA0;[TA] &amp;#xA0;tendinous chiasm of fingers: the crossing of the tendons of the flexor digitorum profundus through the tendons of the flexor digitorum superficialis; called also chiasm of digits of hand.\n
  43. \n
  44. \n
  45. \n
  46. \n
  47. \n
  48. \n
  49. \n
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  53. \n
  54. \n
  55. \n
  56. \n
  57. \n
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  59. \n
  60. The thin, condensable cruciate\nsections of the sheath&amp;#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&amp;#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
  61. The thin, condensable cruciate\nsections of the sheath&amp;#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&amp;#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
  62. The thin, condensable cruciate\nsections of the sheath&amp;#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&amp;#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
  63. The thin, condensable cruciate\nsections of the sheath&amp;#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&amp;#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
  64. The thin, condensable cruciate\nsections of the sheath&amp;#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&amp;#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
  65. \n
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  75. from L. imbibere &quot;absorb, drink in, inhale,&quot; from in- &quot;in&quot; + bibere &quot;to drink,&quot; \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
  76. from L. imbibere &quot;absorb, drink in, inhale,&quot; from in- &quot;in&quot; + bibere &quot;to drink,&quot; \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
  77. from L. imbibere &quot;absorb, drink in, inhale,&quot; from in- &quot;in&quot; + bibere &quot;to drink,&quot; \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
  78. from L. imbibere &quot;absorb, drink in, inhale,&quot; from in- &quot;in&quot; + bibere &quot;to drink,&quot; \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
  79. from L. imbibere &quot;absorb, drink in, inhale,&quot; from in- &quot;in&quot; + bibere &quot;to drink,&quot; \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
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  118. Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
  119. Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
  120. Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
  121. Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
  122. Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
  123. Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
  124. Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
  125. \n
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  156. factors:\n\nposition, length, and direction of original laceration,\n\nneed to gain access to other injured structures\n\npersonal experience and preferences\n
  157. factors:\n\nposition, length, and direction of original laceration,\n\nneed to gain access to other injured structures\n\npersonal experience and preferences\n
  158. factors:\n\nposition, length, and direction of original laceration,\n\nneed to gain access to other injured structures\n\npersonal experience and preferences\n
  159. factors:\n\nposition, length, and direction of original laceration,\n\nneed to gain access to other injured structures\n\npersonal experience and preferences\n
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  208. Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic &quot;immaturity&quot; of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
  209. Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic &quot;immaturity&quot; of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
  210. Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic &quot;immaturity&quot; of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
  211. Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic &quot;immaturity&quot; of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
  212. Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic &quot;immaturity&quot; of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
  213. Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic &quot;immaturity&quot; of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
  214. Figure 7-7 Profundus avulsion classification of Leddy and Packer. \n\nType I: FDP is avulsed from its insertion and retracts into the palm. \n\nType II: the profundus tendon is avulsed from it insertion but the stump remains within the digital sheath, implying that the vinculum longum profundus is still intact. \n\nType III: a bony fragment is attached to the tendon stump, which remains within the flexor sheath. Further proximal retraction is prevented at the distal end of the A4 pulley. \n\nLeddy and Packer classified FDP avulsions into three types (Fig. 7-7).221,222 In type I avulsions, the FDP tendon retracts into the palm. By definition, the vincular blood supply of the tendon has been disrupted. The sheath may, after a few days, be noncompliant and not permit passage of the FDP tendon through it in an attempt to repair the stump to bone. Additionally, proximal muscle contracture may prevent tendon stump advancement. In type II avulsions, the tendon stump retracts to the level of the PIP joint. The sheath is not compromised, and proximal muscle-tendon contracture does not develop substantially. Attempts to advance the tendon stump through the sheath, especially the A4 pulley, may require gentle pulley dilation as well. Repair may be attempted for 6 weeks, or longer, after injury. A large bone fragment is attached to the stump of the FDP tendon in type III injuries. This fragment usually prevents tendon retraction proximal to the distal edge of the A4 pulley. Bony repair using Kirschner wire or screw fixation is often all that is required. A type III injury may be deceptive preoperatively after clinical and radiographic examination because, on occasion, the FDP stump itself is avulsed from the bony fragment (a so-called type IV injury, not initially classified by Leddy and Packer). 222 The stump may be located either within the tendon sheath or within the palm. Repair of the fracture is done first, after which the tendon is advanced and affixed to the distal phalanx. Immobilization of the DIP joint is required, and the range of motion that may be expected after repair is substantially less. Repair of Zone II Lacerations\n
  215. Figure 7-7 Profundus avulsion classification of Leddy and Packer. \n\nType I: FDP is avulsed from its insertion and retracts into the palm. \n\nType II: the profundus tendon is avulsed from it insertion but the stump remains within the digital sheath, implying that the vinculum longum profundus is still intact. \n\nType III: a bony fragment is attached to the tendon stump, which remains within the flexor sheath. Further proximal retraction is prevented at the distal end of the A4 pulley. \n\nLeddy and Packer classified FDP avulsions into three types (Fig. 7-7).221,222 In type I avulsions, the FDP tendon retracts into the palm. By definition, the vincular blood supply of the tendon has been disrupted. The sheath may, after a few days, be noncompliant and not permit passage of the FDP tendon through it in an attempt to repair the stump to bone. Additionally, proximal muscle contracture may prevent tendon stump advancement. In type II avulsions, the tendon stump retracts to the level of the PIP joint. The sheath is not compromised, and proximal muscle-tendon contracture does not develop substantially. Attempts to advance the tendon stump through the sheath, especially the A4 pulley, may require gentle pulley dilation as well. Repair may be attempted for 6 weeks, or longer, after injury. A large bone fragment is attached to the stump of the FDP tendon in type III injuries. This fragment usually prevents tendon retraction proximal to the distal edge of the A4 pulley. Bony repair using Kirschner wire or screw fixation is often all that is required. A type III injury may be deceptive preoperatively after clinical and radiographic examination because, on occasion, the FDP stump itself is avulsed from the bony fragment (a so-called type IV injury, not initially classified by Leddy and Packer). 222 The stump may be located either within the tendon sheath or within the palm. Repair of the fracture is done first, after which the tendon is advanced and affixed to the distal phalanx. Immobilization of the DIP joint is required, and the range of motion that may be expected after repair is substantially less. Repair of Zone II Lacerations\n
  216. Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
  217. Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
  218. Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
  219. Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
  220. Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
  221. Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
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