11. Introduction
• History
– no repair possible in the digit
– “no man’s land”
• Current evidence
12. Introduction
• History
– no repair possible in the digit
– “no man’s land”
• Current evidence
– 1970s: repair is possible without delayed grafting,
reconstruction
13. Introduction
• History
– no repair possible in the digit
– “no man’s land”
• Current evidence
– 1970s: repair is possible without delayed grafting,
reconstruction
– Now: adhesion-free, primarily healed tendon repair is
possible
17. Introduction
• General goals of repair
– primary tendon repair – avoid grafting
– sufficient strength for passive motion rehab which will
allow for
18. Introduction
• General goals of repair
– primary tendon repair – avoid grafting
– sufficient strength for passive motion rehab which will
allow for
• minimum adhesion formation
19. Introduction
• General goals of repair
– primary tendon repair – avoid grafting
– sufficient strength for passive motion rehab which will
allow for
• minimum adhesion formation
• restoration of gliding surface
20. Introduction
• General goals of repair
– primary tendon repair – avoid grafting
– sufficient strength for passive motion rehab which will
allow for
• minimum adhesion formation
• restoration of gliding surface
• facilitation of repair site healing
25. Anatomy
• FDS
– O: volar humerus, radius, ulna
– divided muscle belly in mid forearm (FA)
• super cial – long and ring ngers (LF, RF)
26. Anatomy
• FDS
– O: volar humerus, radius, ulna
– divided muscle belly in mid forearm (FA)
• super cial – long and ring ngers (LF, RF)
• deep – index and small ngers (IF, SF)
27. Anatomy
• FDS
– O: volar humerus, radius, ulna
– divided muscle belly in mid forearm (FA)
• super cial – long and ring ngers (LF, RF)
• deep – index and small ngers (IF, SF)
• FDP
28. Anatomy
• FDS
– O: volar humerus, radius, ulna
– divided muscle belly in mid forearm (FA)
• super cial – long and ring ngers (LF, RF)
• deep – index and small ngers (IF, SF)
• FDP
– O: anteromedial ulna, IO membrane
29. Anatomy
• FDS
– O: volar humerus, radius, ulna
– divided muscle belly in mid forearm (FA)
• super cial – long and ring ngers (LF, RF)
• deep – index and small ngers (IF, SF)
• FDP
– O: anteromedial ulna, IO membrane
– dorsal to FDS in volar FA
30. Anatomy
• FDS
– O: volar humerus, radius, ulna
– divided muscle belly in mid forearm (FA)
• super cial – long and ring ngers (LF, RF)
• deep – index and small ngers (IF, SF)
• FDP
– O: anteromedial ulna, IO membrane
– dorsal to FDS in volar FA
– four tendons usually arise from common belly
34. Anatomy
• In the hand
– ve zones
• V: musculo-tendinous junction to
proximal edge of carpal canal
35. Anatomy
• In the hand
– ve zones
• V: musculo-tendinous junction to
proximal edge of carpal canal
• IV: beneath (dorsal to) the
transverse carpal ligament
36. Anatomy
• In the hand
– ve zones
• V: musculo-tendinous junction to
proximal edge of carpal canal
• IV: beneath (dorsal to) the
transverse carpal ligament
– IF, SF FDS are dorsal to LF, RF FDS
here
39. Anatomy
• Zones (cont.)
– III: distal edge of transverse
carpal ligament to origin of
bro-osseous sheath at the
distal palmar crease
40. Anatomy
• Zones (cont.)
– III: distal edge of transverse
carpal ligament to origin of
bro-osseous sheath at the
distal palmar crease
– II: origin of bro-osseous
sheath to FDS insertion
41. Anatomy
• Zones (cont.)
– III: distal edge of transverse
carpal ligament to origin of
bro-osseous sheath at the
distal palmar crease
– II: origin of bro-osseous
sheath to FDS insertion
– I: distal to FDS insertion
44. Anatomy
• Super cialis
– FDS divides into two slips, wrapping around
FDP; reunite at Camper’s chiasma
45. Anatomy
• Super cialis
– FDS divides into two slips, wrapping around
FDP; reunite at Camper’s chiasma
– continues to insert along the proximal half of the
middle phalanx (P2)
49. Anatomy
• Profundus
– passes through chiasma to insert at proximal base
of distal phalanx (P3)
50. Anatomy
• Profundus
– passes through chiasma to insert at proximal base
of distal phalanx (P3)
– FDP = primary digital exor
51. Anatomy
• Profundus
– passes through chiasma to insert at proximal base
of distal phalanx (P3)
– FDP = primary digital exor
– FDS + intrinsics combine for forceful exion
56. Anatomy
• Sheath
– visceral and parietal synovial layers
enclose the tendons
57. Anatomy
• Sheath
– visceral and parietal synovial layers
enclose the tendons
– Pulleys
58. Anatomy
• Sheath
– visceral and parietal synovial layers
enclose the tendons
– Pulleys
• A2 and A4 arise from periosteum of
proximal P1 and midportion of P2 (prox-
prox and mid-middle)
88. Nutrition
• Vascularity
– damage to vascular and uid nutritional systems
may further hamper healing of repaired exor
tendons
89. Nutrition
• Vascularity
– damage to vascular and uid nutritional systems
may further hamper healing of repaired exor
tendons
– their integrity must be respected during all
reparative efforts
95. Biomechanics
• Excursion
– 9 cm of excursion for composite wrist, full digital
exion
– 2.5 cm required for full digital exion with wrist
at neutral
98. Biomechanics
• Moment arm
– increased distance of tendon from joint center of
rotation = higher moment arm = less motion per
muscle contraction force
102. Biomechanics
• Moment arm
– pulley system constraint governs these parameters
– loss of portions of pulley system may alter balance
between exor, intrinsic, and extensor tendons
103. Biomechanics
• Moment arm
– pulley system constraint governs these parameters
– loss of portions of pulley system may alter balance
between exor, intrinsic, and extensor tendons
– loss of A2 or A4 pulleys may diminish motion/power
or lead to contractures of IP joints
107. Tendon Healing
• Phases
– in ammatory – 48-72 hours
• repair strength reliant upon suture itself
108. Tendon Healing
• Phases
– in ammatory – 48-72 hours
• repair strength reliant upon suture itself
– collagen-producing phase – 5 days to 4 weeks
109. Tendon Healing
• Phases
– in ammatory – 48-72 hours
• repair strength reliant upon suture itself
– collagen-producing phase – 5 days to 4 weeks
• strength accelerates rapidly here
110. Tendon Healing
• Phases
– in ammatory – 48-72 hours
• repair strength reliant upon suture itself
– collagen-producing phase – 5 days to 4 weeks
• strength accelerates rapidly here
– remodeling phase – until 112 days
111. Tendon Healing
• Phases
– in ammatory – 48-72 hours
• repair strength reliant upon suture itself
– collagen-producing phase – 5 days to 4 weeks
• strength accelerates rapidly here
– remodeling phase – until 112 days
• Predominance of extrinsic over intrinsic
healing leads to more adhesions
130. Tendon Healing
• How is healing related to motion?
– passive mobilization enhances healing by
stimulating tendon wound maturation and scar
remodeling* , 110
*Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA
(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:
American Academy of Orthopaedic Surgeons, 1988, pp 5-40.
131. Tendon Healing
• How is healing related to motion?
– passive mobilization enhances healing by
stimulating tendon wound maturation and scar
remodeling* , 110
– use a strong, gap-resistant suture technique +
controlled-motion stress postoperatively
*Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA
(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:
American Academy of Orthopaedic Surgeons, 1988, pp 5-40.
132. Tendon Healing
• How is healing related to motion?
– passive mobilization enhances healing by
stimulating tendon wound maturation and scar
remodeling* , 110
– use a strong, gap-resistant suture technique +
controlled-motion stress postoperatively
– 3-5 mm of excursion needed at repair site to
prevent adhesions 144
*Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA
(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:
American Academy of Orthopaedic Surgeons, 1988, pp 5-40.
137. Tendon Repair
• Contraindications
– severe multiple tissue injuries to ngers, palm
– gross wound contamination
– signi cant skin loss over the exor system
138. Tendon Repair
• Contraindications
– severe multiple tissue injuries to ngers, palm
– gross wound contamination
– signi cant skin loss over the exor system
• Reduce fractures, repair neurovascular injuries
concomitantly
139. Tendon Repair
• Contraindications
– severe multiple tissue injuries to ngers, palm
– gross wound contamination
– signi cant skin loss over the exor system
• Reduce fractures, repair neurovascular injuries
concomitantly
• Repair tendons acutely/subacutely to avoid
deterioration of tendon ends and extrinsic muscle
shortening
141. Examination
• High index of suspicion for multiple structures
injured through a small laceration or crush injury
142. Examination
• High index of suspicion for multiple structures
injured through a small laceration or crush injury
• Complete neurovascular exam
143. Examination
• High index of suspicion for multiple structures
injured through a small laceration or crush injury
• Complete neurovascular exam
– division of two digital nerves indicates arterial division as
well
144. Examination
• High index of suspicion for multiple structures
injured through a small laceration or crush injury
• Complete neurovascular exam
– division of two digital nerves indicates arterial division as
well
– dysvascularity could compromise repair or skin ap
healing +/- cold intolerance
160. Surgery
• Planning
– inform the patient of the injury severity and potential
need for more surgery
161. Surgery
• Planning
– inform the patient of the injury severity and potential
need for more surgery
– incisions
162. Surgery
• Planning
– inform the patient of the injury severity and potential
need for more surgery
– incisions
• keep tendon retraction in mind
163. Surgery
• Planning
– inform the patient of the injury severity and potential
need for more surgery
– incisions
• keep tendon retraction in mind
• no bonus points for small incisions
164. Surgery
• Planning
– inform the patient of the injury severity and potential
need for more surgery
– incisions
• keep tendon retraction in mind
• no bonus points for small incisions
• maintain ap viability
165. Surgery
• Planning
– inform the patient of the injury severity and potential
need for more surgery
– incisions
• keep tendon retraction in mind
• no bonus points for small incisions
• maintain ap viability
• avoid skin/scar contracture
172. Surgery - principles
• Ideal repair
– easy placement of sutures in the tendon
– secure suture knots
173. Surgery - principles
• Ideal repair
– easy placement of sutures in the tendon
– secure suture knots
– smooth junction of tendon ends
174. Surgery - principles
• Ideal repair
– easy placement of sutures in the tendon
– secure suture knots
– smooth junction of tendon ends
– minimal gapping at the repair site
175. Surgery - principles
• Ideal repair
– easy placement of sutures in the tendon
– secure suture knots
– smooth junction of tendon ends
– minimal gapping at the repair site
– minimal interference with tendon vascularity
176. Surgery - principles
• Ideal repair
– easy placement of sutures in the tendon
– secure suture knots
– smooth junction of tendon ends
– minimal gapping at the repair site
– minimal interference with tendon vascularity
– sufficient strength for early motion stress to the tendon
179. Surgery
• Core suture considerations
– strength of a repair is proportional to the number of
suture strands crossing the repair site
180. Surgery
• Core suture considerations
– strength of a repair is proportional to the number of
suture strands crossing the repair site
– increasing crossing strands increases difficulty and risk of
tendon damage, vascularity compromise
181. Surgery
• Core suture considerations
– strength of a repair is proportional to the number of
suture strands crossing the repair site
– increasing crossing strands increases difficulty and risk of
tendon damage, vascularity compromise
– repairs usually rupture at the suture knots
182. Surgery
• Core suture considerations
– strength of a repair is proportional to the number of
suture strands crossing the repair site
– increasing crossing strands increases difficulty and risk of
tendon damage, vascularity compromise
– repairs usually rupture at the suture knots
– locking loop con gurations usually allow less gapping
than grasping types
183. Surgery
• Core suture considerations
– strength of a repair is proportional to the number of
suture strands crossing the repair site
– increasing crossing strands increases difficulty and risk of
tendon damage, vascularity compromise
– repairs usually rupture at the suture knots
– locking loop con gurations usually allow less gapping
than grasping types
– larger caliber sutures increase repair strength
189. Surgery
• Suture material
– absorbable materials seem attractive
• less foreign body reaction long-term
190. Surgery
• Suture material
– absorbable materials seem attractive
• less foreign body reaction long-term
• less stress-shielding of the host tissue
191. Surgery
• Suture material
– absorbable materials seem attractive
• less foreign body reaction long-term
• less stress-shielding of the host tissue
– optimal rates of material absorption, strength reduction
are unknown
192. Surgery
• Suture material
– absorbable materials seem attractive
• less foreign body reaction long-term
• less stress-shielding of the host tissue
– optimal rates of material absorption, strength reduction
are unknown
– 3-0, 4-0 braided polyester is most commonly used
198. Surgery
• Circumferential (peripheral) sutures
– may provide a 10-15% increase in exor tendon repair
strength
– reduces gapping between ends
– methods
• running lock loop (Lin)
199. Surgery
• Circumferential (peripheral) sutures
– may provide a 10-15% increase in exor tendon repair
strength
– reduces gapping between ends
– methods
• running lock loop (Lin)
• horizontal mattress method (Mashadi)
200. Surgery
• Circumferential (peripheral) sutures
– may provide a 10-15% increase in exor tendon repair
strength
– reduces gapping between ends
– methods
• running lock loop (Lin)
• horizontal mattress method (Mashadi)
• Halsted continuous horizontal mattress method (Wade)
201. Surgery
• Circumferential (peripheral) sutures
– may provide a 10-15% increase in exor tendon repair
strength
– reduces gapping between ends
– methods
• running lock loop (Lin)
• horizontal mattress method (Mashadi)
• Halsted continuous horizontal mattress method (Wade)
• cross-stich technique (Silfverskiold)
208. Surgery
• Strickland conclusion: Any 4-strand core suture +
running lock or horizontal mattress circumferential
suture should permit light composite grip during the
entire healing period
211. Surgery
• Sheath repair
– cumulative work of several investigators fails to give clear
direction
212. Surgery
• Sheath repair
– cumulative work of several investigators fails to give clear
direction
– advantages
213. Surgery
• Sheath repair
– cumulative work of several investigators fails to give clear
direction
– advantages
• provides a barrier to extrinsic adhesions
214. Surgery
• Sheath repair
– cumulative work of several investigators fails to give clear
direction
– advantages
• provides a barrier to extrinsic adhesions
• quicker return of synovial nutrition
215. Surgery
• Sheath repair
– cumulative work of several investigators fails to give clear
direction
– advantages
• provides a barrier to extrinsic adhesions
• quicker return of synovial nutrition
• serves as a mold for the remodeling tendon
216. Surgery
• Sheath repair
– cumulative work of several investigators fails to give clear
direction
– advantages
• provides a barrier to extrinsic adhesions
• quicker return of synovial nutrition
• serves as a mold for the remodeling tendon
• results in better sheath-tendon biomechanics
217. Surgery
• Sheath repair
– cumulative work of several investigators fails to give clear
direction
– advantages
• provides a barrier to extrinsic adhesions
• quicker return of synovial nutrition
• serves as a mold for the remodeling tendon
• results in better sheath-tendon biomechanics
– disadvantages
218. Surgery
• Sheath repair
– cumulative work of several investigators fails to give clear
direction
– advantages
• provides a barrier to extrinsic adhesions
• quicker return of synovial nutrition
• serves as a mold for the remodeling tendon
• results in better sheath-tendon biomechanics
– disadvantages
• difficult
219. Surgery
• Sheath repair
– cumulative work of several investigators fails to give clear
direction
– advantages
• provides a barrier to extrinsic adhesions
• quicker return of synovial nutrition
• serves as a mold for the remodeling tendon
• results in better sheath-tendon biomechanics
– disadvantages
• difficult
• may restrict tendon gliding
223. Surgery
• Partial lacerations
– lacerations of 50% or less do not need to be sutured
– Grewal: nonrepaired partial lacerations had higher
ultimate load and stiffness than repaired tendons
224. Surgery
• Partial lacerations
– lacerations of 50% or less do not need to be sutured
– Grewal: nonrepaired partial lacerations had higher
ultimate load and stiffness than repaired tendons
– entrapment, rupture, triggering of unrepaired partial
severed tendons has been reported (Schlenker)
227. Surgery
• Zone I injury – FDP avulsion
– occurs w/ forcible extension during strong DIP
exion
228. Surgery
• Zone I injury – FDP avulsion
– occurs w/ forcible extension during strong DIP
exion
– three types (Leddy and Packer) – relevant to
nding the fragment/tendon end and to repair
timing
229. Surgery
• Zone I injury – FDP avulsion
– occurs w/ forcible extension during strong DIP
exion
– three types (Leddy and Packer) – relevant to
nding the fragment/tendon end and to repair
timing
– tendon repair to cancellous bone provides the
best result
230. Surgery
• Zone I injury – FDP avulsion
– occurs w/ forcible extension during strong DIP
exion
– three types (Leddy and Packer) – relevant to
nding the fragment/tendon end and to repair
timing
– tendon repair to cancellous bone provides the
best result
• done with sutures exiting dorsally tied over a button
231. Surgery
• Zone I injury – FDP avulsion
– occurs w/ forcible extension during strong DIP
exion
– three types (Leddy and Packer) – relevant to
nding the fragment/tendon end and to repair
timing
– tendon repair to cancellous bone provides the
best result
• done with sutures exiting dorsally tied over a button
• OR with suture anchors
237. Surgery
• FDP laceration – Zone I
– laceration distal to FDS insertion = Zone I injury
– distal stump < 1 cm long = perform FDP advancement
and tendon to bone repair
238. Surgery
• FDP laceration – Zone I
– laceration distal to FDS insertion = Zone I injury
– distal stump < 1 cm long = perform FDP advancement
and tendon to bone repair
– > 1 cm distally = repair primarily
239. Surgery
• FDP laceration – Zone I
– laceration distal to FDS insertion = Zone I injury
– distal stump < 1 cm long = perform FDP advancement
and tendon to bone repair
– > 1 cm distally = repair primarily
• laceration may be near A4 difficult repair
240. Surgery
• FDP laceration – Zone I
– laceration distal to FDS insertion = Zone I injury
– distal stump < 1 cm long = perform FDP advancement
and tendon to bone repair
– > 1 cm distally = repair primarily
• laceration may be near A4 difficult repair
• advancement of FDP > 1 cm may result in the quadrigia effect
248. OITE
2004 #10
A partial laceration of the exor tendon should be repaired when the
percentage of tendon lacerated is more than
1. 10%
2. 20%
3. 40%
4. 60%
5. 80%
249. OITE
2004 #10
A partial laceration of the exor tendon should be repaired when the
percentage of tendon lacerated is more than
1. 10%
2. 20%
3. 40%
4. 60%
5. 80%
253. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
254. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
255. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
256. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
257. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
3. Musculotendinous rupture in the forearm
258. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
3. Musculotendinous rupture in the forearm
4. Stretch injury to the ulnar nerve at the wrist
259. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
3. Musculotendinous rupture in the forearm
4. Stretch injury to the ulnar nerve at the wrist
5. Ulnar artery thrombosis
260. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
3. Musculotendinous rupture in the forearm
4. Stretch injury to the ulnar nerve at the wrist
5. Ulnar artery thrombosis
265. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
266. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
267. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
268. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
269. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
3. Musculotendinous rupture in the forearm
270. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
3. Musculotendinous rupture in the forearm
4. Stretch injury to the ulnar nerve at the wrist
271. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
3. Musculotendinous rupture in the forearm
4. Stretch injury to the ulnar nerve at the wrist
5. Ulnar artery thrombosis
272. OITE
2005 #246
A 21 year-old left-handed college student was playing rugby 3 days ago and
felt the sudden onset of pain in his right ring nger when another
played pulled away from him. A clinical photo is shown in Figure 86.
What is the most likely diagnosis?
1. Volar plate avulsion of the proximal interphalangeal joint
2. Avulsion of the distal exor digitorum profundus tendon
3. Musculotendinous rupture in the forearm
4. Stretch injury to the ulnar nerve at the wrist
5. Ulnar artery thrombosis
274. OITE
2003 #10
Active mobilization following exor tendon repair is best accomplished
with the wrist in
1. exion and the metacarpophalangeal joints in exion.
2. exion and the metacarpophalangeal joints in extension.
3. neutral and the metacarpophalangeal joints in extension.
4. neutral and the metacarpophalangeal joints in exion.
5. extension and the metacarpophalangeal joints in exion.
276. OITE
2003 #10
Active mobilization following exor tendon repair is best accomplished
with the wrist in
1. exion and the metacarpophalangeal joints in exion.
2. exion and the metacarpophalangeal joints in extension.
3. neutral and the metacarpophalangeal joints in extension.
4. neutral and the metacarpophalangeal joints in exion.
5. extension and the metacarpophalangeal joints in
exion.
278. OITE
2003 #126
What is the major advantage of allowing early active motion of a repaired
zone II exor tendon injury?
• 1- Increased tendon excursion
• 2- Greater repair strength
• 3- Less postoperative pain
• 4- Better patient compliance
• 5- Faster tendon healing
280. OITE
2003 #126
What is the major advantage of allowing early active motion of a repaired
zone II exor tendon injury?
• 1- Increased tendon excursion
• 2- Greater repair strength
• 3- Less postoperative pain
• 4- Better patient compliance
• 5- Faster tendon healing
282. OITE
2005 #21
e ideal exor tendon rehab protocol that minimizes peritendinous
adhesions includes
1. Casting for 6 weeks
2. A synergistic wrist and digit motion rehabilitation protocol
3. Dorsal blocking splint with unrestricted active nger exion
4. Dynamic extension outrigger splinting
5. Early aggressive active motion and a strengthening program
284. OITE
2005 #21
e ideal exor tendon rehab protocol that minimizes peritendinous
adhesions includes
1. Casting for 6 weeks
2. A synergistic wrist and digit motion
rehabilitation protocol
3. Dorsal blocking splint with unrestricted active nger exion
4. Dynamic extension outrigger splinting
5. Early aggressive active motion and a strengthening program
286. OITE
2005 #103
A 3 year-old child undergoes repair of lacerated exor tendons of the
ring and little ngers in zone II. Postoperative rehabilitation should
consist of
1. Passive exion and extension exercises
2. Passive exion and active extension exercises
3. Active exion and extension exercises
4. Cast immobilization for 4 weeks
5. Cast immobilization for 8 weeks
288. OITE
2005 #103
A 3 year-old child undergoes repair of lacerated exor tendons of the
ring and little ngers in zone II. Postoperative rehabilitation should
consist of
1. Passive exion and extension exercises
2. Passive exion and active extension exercises
3. Active exion and extension exercises
4. Cast immobilization for 4 weeks
5. Cast immobilization for 8 weeks
290. OITE
2002 #70
A 4 year old girl undergoes repair of both exor tendons in zone II.
Initial postoperative physical therapy should consist of
1. passive mobilization only.
2. active extension and passive exion mobilization.
3. active place and hold mobilization.
4. immobilization.
5. unrestricted active mobilization.
292. OITE
2002 #70
A 4 year old girl undergoes repair of both exor tendons in zone II.
Initial postoperative physical therapy should consist of
1. passive mobilization only.
2. active extension and passive exion mobilization.
3. active place and hold mobilization.
4.immobilization.
5. unrestricted active mobilization.
293. Bibliography
Trumble, ed. Hand Surgery Update 3., 2003.
Green, ed. Green’s Operative Hand Surgery, 5th ed., 2003.
Canale, ed. Campbell’s Operative Orthopaedic Surgery, 10th ed., 2003.
294. Bibliography
Trumble, ed. Hand Surgery Update 3., 2003.
Green, ed. Green’s Operative Hand Surgery, 5th ed., 2003.
Canale, ed. Campbell’s Operative Orthopaedic Surgery, 10th ed., 2003.
296. Bibliography
Schmidt. Surgical Anatomy of the Hand. 2004
Notas do Editor
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achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
achievement\nof a primary tendon repair of sufficient\ntensile strength to allow application of\na postoperative passive-motion rehabilitation\nprotocol that inhibits formation\nof intrasynovial adhesions,\nstimulates restoration of the gliding\nsurface, and facilitates healing of the\nrepair site48.\n
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chiasm&#xA0;(chi&#xB7;asm) (ki&#xB4;az-&#x259;m) [L., Gr. chiasma] &#xA0;a decussation or X-shaped crossing; see chiasma.\nc. of digits of hand &#xA0;chiasma tendinum digitorum manus. \noptic c. &#xA0;chiasma opticum. \ntendinous c. of fingers &#xA0;chiasma tendinum digitorum manus. \nchiasma&#xA0;(chi&#xB7;as&#xB7;ma) (ki-az&#xB4;m&#x259;) &#xA0;pl. chias&#xB4;mata [L., from Gr. &#x201C;a cross, crosspiece&#x201D;, from the shape of the letter chi, (X)] &#xA0;1.&#xA0; [TA]&#xA0;a general term in anatomical nomenclature for a decussation or X-shaped crossing, such as of nerves. &#xA0;2.&#xA0;in genetics, the places where pairs of homologous chromatids remain in contact during late prophase to anaphase of the first meiotic division, indicating where an exchange of homologous segments has taken place between non-sister chromatids by crossing over. \noptic c. ,&#xA0;&#xA0;c. op&#xB4;ticum &#xA0;[TA] &#xA0;optic chiasm: the part of the hypothalamus formed by the decussation, or crossing, of the fibers of the optic nerve from the medial half of each retina; called also optic decussation.Click here to view imagec. ten&#xB4;dinum digito&#xB4;rum ma&#xB4;nus &#xA0;[TA] &#xA0;tendinous chiasm of fingers: the crossing of the tendons of the flexor digitorum profundus through the tendons of the flexor digitorum superficialis; called also chiasm of digits of hand.\n
chiasm&#xA0;(chi&#xB7;asm) (ki&#xB4;az-&#x259;m) [L., Gr. chiasma] &#xA0;a decussation or X-shaped crossing; see chiasma.\nc. of digits of hand &#xA0;chiasma tendinum digitorum manus. \noptic c. &#xA0;chiasma opticum. \ntendinous c. of fingers &#xA0;chiasma tendinum digitorum manus. \nchiasma&#xA0;(chi&#xB7;as&#xB7;ma) (ki-az&#xB4;m&#x259;) &#xA0;pl. chias&#xB4;mata [L., from Gr. &#x201C;a cross, crosspiece&#x201D;, from the shape of the letter chi, (X)] &#xA0;1.&#xA0; [TA]&#xA0;a general term in anatomical nomenclature for a decussation or X-shaped crossing, such as of nerves. &#xA0;2.&#xA0;in genetics, the places where pairs of homologous chromatids remain in contact during late prophase to anaphase of the first meiotic division, indicating where an exchange of homologous segments has taken place between non-sister chromatids by crossing over. \noptic c. ,&#xA0;&#xA0;c. op&#xB4;ticum &#xA0;[TA] &#xA0;optic chiasm: the part of the hypothalamus formed by the decussation, or crossing, of the fibers of the optic nerve from the medial half of each retina; called also optic decussation.Click here to view imagec. ten&#xB4;dinum digito&#xB4;rum ma&#xB4;nus &#xA0;[TA] &#xA0;tendinous chiasm of fingers: the crossing of the tendons of the flexor digitorum profundus through the tendons of the flexor digitorum superficialis; called also chiasm of digits of hand.\n
chiasm&#xA0;(chi&#xB7;asm) (ki&#xB4;az-&#x259;m) [L., Gr. chiasma] &#xA0;a decussation or X-shaped crossing; see chiasma.\nc. of digits of hand &#xA0;chiasma tendinum digitorum manus. \noptic c. &#xA0;chiasma opticum. \ntendinous c. of fingers &#xA0;chiasma tendinum digitorum manus. \nchiasma&#xA0;(chi&#xB7;as&#xB7;ma) (ki-az&#xB4;m&#x259;) &#xA0;pl. chias&#xB4;mata [L., from Gr. &#x201C;a cross, crosspiece&#x201D;, from the shape of the letter chi, (X)] &#xA0;1.&#xA0; [TA]&#xA0;a general term in anatomical nomenclature for a decussation or X-shaped crossing, such as of nerves. &#xA0;2.&#xA0;in genetics, the places where pairs of homologous chromatids remain in contact during late prophase to anaphase of the first meiotic division, indicating where an exchange of homologous segments has taken place between non-sister chromatids by crossing over. \noptic c. ,&#xA0;&#xA0;c. op&#xB4;ticum &#xA0;[TA] &#xA0;optic chiasm: the part of the hypothalamus formed by the decussation, or crossing, of the fibers of the optic nerve from the medial half of each retina; called also optic decussation.Click here to view imagec. ten&#xB4;dinum digito&#xB4;rum ma&#xB4;nus &#xA0;[TA] &#xA0;tendinous chiasm of fingers: the crossing of the tendons of the flexor digitorum profundus through the tendons of the flexor digitorum superficialis; called also chiasm of digits of hand.\n
chiasm&#xA0;(chi&#xB7;asm) (ki&#xB4;az-&#x259;m) [L., Gr. chiasma] &#xA0;a decussation or X-shaped crossing; see chiasma.\nc. of digits of hand &#xA0;chiasma tendinum digitorum manus. \noptic c. &#xA0;chiasma opticum. \ntendinous c. of fingers &#xA0;chiasma tendinum digitorum manus. \nchiasma&#xA0;(chi&#xB7;as&#xB7;ma) (ki-az&#xB4;m&#x259;) &#xA0;pl. chias&#xB4;mata [L., from Gr. &#x201C;a cross, crosspiece&#x201D;, from the shape of the letter chi, (X)] &#xA0;1.&#xA0; [TA]&#xA0;a general term in anatomical nomenclature for a decussation or X-shaped crossing, such as of nerves. &#xA0;2.&#xA0;in genetics, the places where pairs of homologous chromatids remain in contact during late prophase to anaphase of the first meiotic division, indicating where an exchange of homologous segments has taken place between non-sister chromatids by crossing over. \noptic c. ,&#xA0;&#xA0;c. op&#xB4;ticum &#xA0;[TA] &#xA0;optic chiasm: the part of the hypothalamus formed by the decussation, or crossing, of the fibers of the optic nerve from the medial half of each retina; called also optic decussation.Click here to view imagec. ten&#xB4;dinum digito&#xB4;rum ma&#xB4;nus &#xA0;[TA] &#xA0;tendinous chiasm of fingers: the crossing of the tendons of the flexor digitorum profundus through the tendons of the flexor digitorum superficialis; called also chiasm of digits of hand.\n
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The thin, condensable cruciate\nsections of the sheath&#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
The thin, condensable cruciate\nsections of the sheath&#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
The thin, condensable cruciate\nsections of the sheath&#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
The thin, condensable cruciate\nsections of the sheath&#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
The thin, condensable cruciate\nsections of the sheath&#x2014;C1\n(between A2 and A3 ), C2 (between A3\nand A4), and C3 (between A4 and\nA5)&#x2014;collapse to permit the annular\npulleys to approximate each other\nduring digital flexion. The flexor tendons\nare weakly attached to the sheath\nby filmy mesenteries composed of\nvincula\n
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from L. imbibere "absorb, drink in, inhale," from in- "in" + bibere "to drink," \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
from L. imbibere "absorb, drink in, inhale," from in- "in" + bibere "to drink," \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
from L. imbibere "absorb, drink in, inhale," from in- "in" + bibere "to drink," \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
from L. imbibere "absorb, drink in, inhale," from in- "in" + bibere "to drink," \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
from L. imbibere "absorb, drink in, inhale," from in- "in" + bibere "to drink," \n\nSynovial fluid diffusion provides an effective alternative\nnutritional and lubricating pathway for\nflexor tendons. The rapid delivery of nutrients is\napparently accomplished by a pumping mechanism\nknown as imbibition in which fluid is forced into the\ninterstices of the tendon through small conduits in\nthe tendon surface as the digit is flexed and extended.\n
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Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
Gelberman R, Goldberg V, An KN, et al: Tendon, in Woo SLY, Buckwalter JA\n(eds): Injury and Repair of the Musculoskeletal Soft Tissues. Park Ridge, Ill:\nAmerican Academy of Orthopaedic Surgeons, 1988, pp 5-40.\n
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factors:\n\nposition, length, and direction of original laceration,\n\nneed to gain access to other injured structures\n\npersonal experience and preferences\n
factors:\n\nposition, length, and direction of original laceration,\n\nneed to gain access to other injured structures\n\npersonal experience and preferences\n
factors:\n\nposition, length, and direction of original laceration,\n\nneed to gain access to other injured structures\n\npersonal experience and preferences\n
factors:\n\nposition, length, and direction of original laceration,\n\nneed to gain access to other injured structures\n\npersonal experience and preferences\n
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Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic "immaturity" of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic "immaturity" of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic "immaturity" of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic "immaturity" of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic "immaturity" of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
Repair of Zone I Lacerations or Avulsions \n\nRecent in-vivo experiments on the canine FDP-bone repair site have led to several new concepts of flexor tendon repair to bone.43,45,47,397 First, the tendon-bone repair site does not appear to accrue strength during the 3 to 6 week period postoperatively. 397 Second, even at 6 weeks postoperatively (the time at which the grasping suture and dorsal button are usually removed) there is substantial inflammatory tissue still present at the repair site, signifying a potential biologic "immaturity" of the repair site. 45 In addition, there is a substantial tendency for the repair site to elongate during the immediate postoperative period, which may not be apparent clinically. These findings suggest that tendon-bone healing does not follow the same time course as the healing of intrasynovial flexor tendon repairs.\n\n Laceration of the FDP tendon distal to the insertion of the FDS tendon or avulsion from its insertion at the base of the proximal aspect of the distal phalanx is by definition an injury in zone I of the flexor sheath. If the tendon is lacerated and the distal tendon stump is less than 1 cm long, FDP tendon advancement and primary repair to bone are indicated. If more than 1 cm of FDP stump is available for suture, then primary tenorrhaphy is indicated because shortening of the FDP tendon by greater than 1 cm may result in a quadrigia effect on attempted composite flexion of the digits. 458 In this clinical situation, the laceration may lie near or beneath the A4 pulley, making the repair technically difficult. \n\nTendon to Bone Repair\n\n Many techniques of core suture placement have been advocated for affixing the FDP tendon stump to bone. Theoretically, most of the techniques employed for tendon-tendon repair can be utilized for tendon-bone repair; however, several of these have been accepted more widely since removal of the dorsal button and transfixing suture at 6 weeks postoperatively has been advocated generally. The utilization of suture material of high tensile strength that is absorbed after months rather than weeks has led to increased advocacy of core suture techniques that need not be placed in the proximal stump with an intention toward their eventual removal.43,47,397 In addition, the increased utilization of intraosseous anchors in hand surgery has led some investigators to advocate their utilization in this clinical setting.61,62,151,365,403 If bone quality is good and experimental data are supported with clinical efficacy, their use could become more widespread.\n
Figure 7-7 Profundus avulsion classification of Leddy and Packer. \n\nType I: FDP is avulsed from its insertion and retracts into the palm. \n\nType II: the profundus tendon is avulsed from it insertion but the stump remains within the digital sheath, implying that the vinculum longum profundus is still intact. \n\nType III: a bony fragment is attached to the tendon stump, which remains within the flexor sheath. Further proximal retraction is prevented at the distal end of the A4 pulley. \n\nLeddy and Packer classified FDP avulsions into three types (Fig. 7-7).221,222 In type I avulsions, the FDP tendon retracts into the palm. By definition, the vincular blood supply of the tendon has been disrupted. The sheath may, after a few days, be noncompliant and not permit passage of the FDP tendon through it in an attempt to repair the stump to bone. Additionally, proximal muscle contracture may prevent tendon stump advancement. In type II avulsions, the tendon stump retracts to the level of the PIP joint. The sheath is not compromised, and proximal muscle-tendon contracture does not develop substantially. Attempts to advance the tendon stump through the sheath, especially the A4 pulley, may require gentle pulley dilation as well. Repair may be attempted for 6 weeks, or longer, after injury. A large bone fragment is attached to the stump of the FDP tendon in type III injuries. This fragment usually prevents tendon retraction proximal to the distal edge of the A4 pulley. Bony repair using Kirschner wire or screw fixation is often all that is required. A type III injury may be deceptive preoperatively after clinical and radiographic examination because, on occasion, the FDP stump itself is avulsed from the bony fragment (a so-called type IV injury, not initially classified by Leddy and Packer). 222 The stump may be located either within the tendon sheath or within the palm. Repair of the fracture is done first, after which the tendon is advanced and affixed to the distal phalanx. Immobilization of the DIP joint is required, and the range of motion that may be expected after repair is substantially less. Repair of Zone II Lacerations\n
Figure 7-7 Profundus avulsion classification of Leddy and Packer. \n\nType I: FDP is avulsed from its insertion and retracts into the palm. \n\nType II: the profundus tendon is avulsed from it insertion but the stump remains within the digital sheath, implying that the vinculum longum profundus is still intact. \n\nType III: a bony fragment is attached to the tendon stump, which remains within the flexor sheath. Further proximal retraction is prevented at the distal end of the A4 pulley. \n\nLeddy and Packer classified FDP avulsions into three types (Fig. 7-7).221,222 In type I avulsions, the FDP tendon retracts into the palm. By definition, the vincular blood supply of the tendon has been disrupted. The sheath may, after a few days, be noncompliant and not permit passage of the FDP tendon through it in an attempt to repair the stump to bone. Additionally, proximal muscle contracture may prevent tendon stump advancement. In type II avulsions, the tendon stump retracts to the level of the PIP joint. The sheath is not compromised, and proximal muscle-tendon contracture does not develop substantially. Attempts to advance the tendon stump through the sheath, especially the A4 pulley, may require gentle pulley dilation as well. Repair may be attempted for 6 weeks, or longer, after injury. A large bone fragment is attached to the stump of the FDP tendon in type III injuries. This fragment usually prevents tendon retraction proximal to the distal edge of the A4 pulley. Bony repair using Kirschner wire or screw fixation is often all that is required. A type III injury may be deceptive preoperatively after clinical and radiographic examination because, on occasion, the FDP stump itself is avulsed from the bony fragment (a so-called type IV injury, not initially classified by Leddy and Packer). 222 The stump may be located either within the tendon sheath or within the palm. Repair of the fracture is done first, after which the tendon is advanced and affixed to the distal phalanx. Immobilization of the DIP joint is required, and the range of motion that may be expected after repair is substantially less. Repair of Zone II Lacerations\n
Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n
Quadrigia\n\nQuadrigia syndrome manifests as a decrease in flexion of an adjacent normal finger after the proximal excursion of the FDP tendon of the involved digit has been limited. 392 It was first described by Bunnell 60 and later by Verdan471,473 after the Roman four-horse chariots driven by one charioteer who controlled four horses through individual reins. If injury or a surgical procedure prevents normal proximal excursion of a single FDP, a tethering effect will be experienced by the other FDP tendons that share a common musculotendinous origin. The adjacent FDP tendons then cannot fully flex their respective digits. The adjacent fingers will lose some distal flexion besides creating a flexion deformity in the operated finger.\n\nThe quadrigia syndrome may occur in a number of settings such as when one FDP is advanced too far distally in a reattachment procedure in zone I. 271 Other causes include a tendon graft that is too short, a distal finger amputation in which the flexor is sutured over the tip to the extensor tendon, or an amputation in which the FDP adheres to the proximal phalanx. The patient will complain not only of the flexion deformity in the injured finger but also of weakness of grasp in the adjacent fingers. The adjacent fingers would, on clinical examination, show weakness and deficiency in their FDP function. The treatment is to lengthen, tenolyse, or even sever the offending tendon to allow the other normal adjacent fingers to flex fully.\n