ACLS CE -Part I of III -ECG STRIP INTERPRETATION w Case Scenarios Supplemental
1. ACLS CE
Part I of III
ECG STRIP INTERPRETATION
Prepared and presented by
Marc Imhotep Cray, M.D.
2. Graphic from: http://ekgenius.net/Fundamentals/nsr.html
Features include:
•Regular rhythm at 60-100 bpm
•Normal P wave morphology and
axis (upright in I and II, inverted in
aVR)
•Narrow QRS complexes (< 100 ms
wide)
•Each P wave is followed by a QRS
complex
•The PR interval is constant
2
Review of ECG Basics
Normal ECG Morphology
3. EKG Paper
3
From: EKG - Practical Clinical Skills (Website).EKG Paper
ECG tracings are recorded on grid
paper. The horizontal axis of the
EKG paper records time, with black
marks at the top indicating 3 second
intervals.
Each second is marked by 5 large
grid blocks. Thus each large block
equals 0.2 second. The vertical axis
records EKG amplitude (voltage).
Two large blocks equal 1 millivolt
(mV). Each small block equals 0.1
mV.
Within the large blocks are 5 small
blocks, each representing 0.04
seconds.
4. Normal Sinus Rhythm
12 lead ECG in sinus rhythm
Source: http://upload.wikimedia.org/wikipedia/commons/f/f0/12_lead_generated_sinus_rhythm.JPG
4
5. Analyzing a Rhythm
5
Component Characteristics
Rate The bpm is commonly the ventricular rate.
If atrial and ventricular rates differ, as in a
3rd-degree block, measure both rates.
Normal: 60–100 bpm
Slow (bradycardia): <60 bpm
Fast (tachycardia): >100 bpm
Regularity Measure R-R intervals and P-P intervals.
Regular: Intervals consistent
Regularly irregular: Repeating pattern
Irregular: No pattern
P Waves If present: Same in size, shape, position?
Does each QRS have a P wave?
Normal: Upright (positive) and uniform
PR Interval Constant: Intervals are the same.
Variable: Intervals differ.
Normal: 0.12–0.20 sec and constant
QRS Interval Normal: 0.06–0.10 sec
Wide: >0.10 sec
None: Absent
QT Interval Beginning of R wave to end of T wave Varies with
HR. Normal: Less than half the R-R interval
Dropped beats Occur in AV blocks. Occur in sinus arrest.
Thaler MS. The Only EKG Book you'll Ever Need - 5th ed., 117-20
6. Basic Rhythm Analysis
• Rate – too fast or too slow?
• Rhythm – regular or irregular?
• Is there a normal looking QRS? Is it wide or
narrow?
• Are P waves present?
• What is the relationship of the P waves to the
QRS complex?
6
7. ACLS Rhythm Analysis
Lethal vs non-lethal?
Shockable vs. non-shockable? Too fast vs too slow?
Symptomatic vs. asymptomatic?
or
Unstable vs. stable?
7
8. Hemodynamically Significant Rhythm
=Symptomatic or Unstable
8
•Clinically, most important parameter to assess in any
patient with a cardiac dysrhythmia is rhythm
hemodynamically "significant"
•This holds true regardless of whether rhythm in
question is slow or fast
•A rhythm is "hemodynamically" significant
IF it produces signs or symptoms of concern as a direct
result of the rate
N.B. Is the Patient Stable? How to Assess:
9. Signs and Symptoms of Concern
(“Think hypoperfusion of heart-brains-kidneys”)
9
•Signs of Concern --include hypotension (i.e.,
systolic BP ≤80-90 mm Hg); shock; heart
failure/pulmonary edema; and/or acute infarction
•Symptoms of Concern -- include chest pain;
shortness of breath; and/or impaired mental
status
10. Hemodynamic stability
VT or SVT
10
•The definition of hemodynamic stability is equally
applicable for supraventricular tachyarrhythmias (SVT) as
it is for ventricular tachycardia (VT)
•patient with tachycardia who is symptomatic
(i.e., hypotensive; short of breath; confused) is in
need of immediate synchronized cardioversion —
regardless of whether rhythm is VT or SVT
•In contrast – a trial of medical therapy is justified
IF the patient is stable!
See SVT and Tachycardia Notes.pdf
11. TO SUMMARIZE
“Symptomatic = unstable”
• Any abnormal rhythm that produces signs or
symptoms of hypoperfusion
– Chest Pain/ischemic EKG changes
– Shortness of Breath
– Decreased level of consciousness
– Syncope/pre-syncope
– Hypotension
– Shock - decreased Urine output, cool extremities,
etc.
– Pulmonary Congestion/CHF
11
Treat the patient not the monitor!!!!!!
12. “Sometimes you just have to be there"
12
For example
• despite a systolic BP of 75 mm Hg-we would not
necessarily cardiovert a patient with tachycardia who was
otherwise tolerating the rhythm well (i.e., without chest
pain, dyspnea, or confusion).
•Some patients may remain stable for hours (or even
days) despite being in sustained VT
Again Treat the patient not the monitor!!!!
14. Non-Lethal Rhythms
• Too fast (tachycardias)
– Sinus
– Supraventricular (including a-fib/flutter)
– Ventricular
• Too slow (bradycardias)
– Sinus
– Heart block (1°, 2°, 3° AV block)
14
N.B. May have the potential to become lethal rhythms if
not treated appropriately
15. The following outlined ECG strips will be reviewed
OUTLINE:
• SINUS RHYTHMS
SINUS RHYTHM
SINUS BRADYCARDIA
SINUS ARREST
SINUS TACHYCARDIA
• ATRIAL RHYTHMS
SUPRAVENTRICULAR TACHYCARDIA
ATRIAL FIBRILLATION
ATRIAL FLUTTER
• VENTRICULAR RHYTHM
VENTRICULAR TACHYCARDIA
VENTRICULAR FIBRILLATION
• ATRIOVENTRICULAR BLOCKS
FIRST DEGREE HEART BLOCK
SECOND DEGREE HEART BLOCK TYPE 1
SECOND DEGREE HEART BLOCK TYPE 2
THIRD DEGREE HEART BLOCK
For full rationale see respective disorders at
http://emedicine.medscape.com/cardiology
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29. Name the rhythm & its
management …
ACLS Case Scenarios
Supplement to Part I- ECG STRIP INTERPRETATION
30. 63 yo man with a witnessed collapse
while mowing the lawn
What is the rhythm?
What is the management?
30
NB: Go through step-by-step analysis of rhythm – rate, rhythm,
qrs, p waves, intervals etc.
33. ACLS Algorithm
• Primary Survey
• Shock – 360 J
• Secondary Survey
• Vasopressor - Epi or Vasopressin IV
• Shock 360J
• Antiarrhythmic – Amiodarone, Lidocaine or
Magnesium Sulfate IV
• Shock 360J
33
NB: Don’t forget CPR in between shocks – don’t stop CPR for anything
except to assess patient (no longer than 10 seconds) or shock resume CPR
*immediately* after the shock for 5 cycles *without* checking a rhythm until
after 5 cycles of 30/2 CPR
34. 79yo man s/p NSTEMI
What is the rhythm?
What is the management?
34
36. Ventricular Tachycardia (2)
• Monomorphic VT
• Polymorphic VT
36
Note: Polymorphic is often associated with electrolyte
abnormalities or toxicities
http://www.txai.org/edu/irregular/ventricular_tachyarrhythmias.htm
http://www.txai.org/edu/irregular/ventricular_tachyarrhythmias.htm
37. Ventricular Tachycardia (3)
• Assume any wide complex tachycardia is VT
until proven otherwise
– SVT with aberrant conduction may also have wide
QRS complexes
• Attempt to establish the diagnosis
– Ischemia risk and VT go together
37
38. Treatment of VT (1)
• If pulseless - follow VF algorithm
• If stable try anti-arrhythmics
– Amiodarone
– Lidocaine
– Procainamide?
• If patient has a pulse, but is unstable or not
responding to meds - shock
38
39. Treatment of VT (2)
• Anti-arrhythmics are also pro-arrhythmic
• One antiarrhythmic may help, more than one
may harm
• Anti-arrhythmics can impair an already
impaired heart
• Electrical cardioversion should be the second
intervention of choice
39
40. 60yo diabetic man with chest pain
What is the rhythm?
What is the management?
40
41. Normal Sinus Rhythm
• Regular rate and rhythm
• Normal P waves and QRS
• Evaluate for cause of chest pain and monitor for
change in rhythm
41
42. 40 yo woman found down, pulseless and
apneic
What is the rhythm?
What is the management?
42
43. Pulseless Electrical Activity (PEA)
• Any organized (or semi-organized) electrical
activity in a patient without a detectable pulse
• Non-perfusing
• Treat the patient NOT the monitor
• Find and treat the cause!!!!!
43
45. PEA
Atropine 1 mg IVP
if PEA is slow
Epinephrine 1 mg IVP
repeat every 3-5 minutes
Search for and Treat Causes
Secondary Survey
Primary Survey
45
46. Find and Treat the Cause
• Remember Non-shockable rhythms include:
–Asystole
–Pulseless electrical activity (PEA)
– The most effective treatment is to find and fix
the underlying problem
46
47. Causes of PEA?
• #1 cause of PEA in adults is hypovolemia
• #1 cause in children is hypoxia/respiratory
arrest
• Other causes? The H’s and T’s (Next Slide)
47
48. The H’s and T’s
• Hypovolemia
• Hypoxia
• Hydrogen ion (acidosis)
• Hyper-/hypokalemia
• Hypothermia
• Hypoglycemia (rare)
• Toxins
• Tamponade
• Tension pneumothorax
• Thrombosis (coronary
or pulmonary)
• Trauma
48
N.B. There are now actually 6 H’s – hypoglycemia added on AHA
guidelines BUT only a few anecdotal reports of actually causing PEA
and usually in association with underlying chronic cardiac dysfunction
(severe heart failure)
49. Treat the H’s and T’s
• Hypovolemia
– Volume – IVF, PRBC’s
• Hypoxia
– Oxygenate/Ventilate
• Hydrogen ion (acidosis)
– Sodium bicarbonate
– Hyperventilation
• Hyper-/hypokalemia
– Sodium bicarbonate
– Insulin/glucose
– Calcium
• Hypothermia
– Warm -- invasive
• Hypoglycemia
– Dextrose
• Toxins
– Check levels
– Charcoal
– Antidotes
• Tamponade
– pericardiocentesis
• Tension pneumothorax
– Needle decompression
– Tube thoracostomy
• Thrombosis (coronary or
pulmonary)
– Thrombolytics
– OR/cath lab
• Trauma
49
Busy slide – the point is to TREAT whatever the problem is
50. 19 yo man with palpitations
What is the rhythm?
What is the management?
50
51. Supraventricular Tachycardia
• Rapid (usually 150-250 bpm) and regular
• P waves cannot be positively identified
• QRS narrow
51
Note: P waves either merge with preceding T waves or are buried in QRS
complexes so the differentiation between atrial and junctional
tachycardia is impossible.
P waves buried in T wave or QRS or not present – can’t differentiate
supraventricular from junctional – most are supraventricular
52. Treatment of Stable SVT (1)
• Consider vagal maneuvers
– Carotid sinus massage
– Valsalva
– Eyeball massage
– Ice water to face
– Digital rectal exam
• Adenosine
– 6 mg, 12 mg, 12 mg
52
NB: Carotid massage - Turn head, Locate maximal impulse of carotid
artery. Massage up and down massage for 5-10 seconds.
One side at a time. Do not do in someone you suspect carotid disease
53. Treatment of Unstable SVT (2)
• Electrical Cardioversion
• Cardioversion is not defibrillation
• Use defibrillator in “sync” mode
– prevents delivering energy in the wrong part of
the cardiac cycle (R on T phenomenon)
53
NB: Heart is not fibrillating
54. Electrical Cardioversion (1)
• Energy level – somewhat controversial
• 100 J→200J→300J→360J
• Atrial flutter may convert with lower energy
– 50J
• For polymorphic VT – start with 200J
54
55. Electrical Cardioversion (2)
• Be prepared
– Patient on monitor, IV, Oxygen
– Suction ready and working
– Airway supplies ready
• Pre-medicate whenever possible
– Conscious sedation
– Electrical shocks are painful!
55
60. Atrial fibrillation/flutter
• Treatment based on patient’s clinical picture
– Unstable = Immediate electrical cardioversion
– Stable
• Control the rate
– Diltiazem
– Esmolol (not if EF < 40%)
– Digoxin
• Provide anticoagulation
• Treat the patient NOT the monitor!!!
60
61. 78yo man found down, pulseless and
apneic, unknown duration
What is the rhythm?
What is the management?
61
62. Asystole
• Is it really asystole?
• Check lead and cable connections.
• Is everything turned on?
• Verify asystole in another lead.
• Maybe it is really fine v-fib?
62
63. 68 yo woman with h/o hypertension
presents with dizziness
What is the rhythm?
What is the treatment?
63
65. Bradycardias (1)
• Many possible causes
– Enhanced parasympathetic tone
– Increased ICP.
– Hypothyroidism
– Hypothermia
– Hyperkalemia
– Hypoglycemia
– Drug therapy
65
66. Bradycardias (2)
• Treat only symptomatic bradycardias
– Ask if the bradycardia causing the symptoms
• Recognize the red flag bradycardias
– Second degree type II block
– Third degree block
66
67. Transcutaneous pacing (1)
• Class I for all symptomatic bradycardias
• Always appropriate
• Doesn’t always work
• Technique
– Attach pacer pads
– Set a rate to 80 bpm
– Turn up the juice (amps) until you get capture
• Painful – may need sedation / analgesia
67
68. Transvenous Pacing (2)
• Invasive
• Time-consuming to establish
• Skilled procedure
• Better long-term than transcutaneous
• May have better capture than transcutaneous
pacing
68
70. 29 yo asymptomatic female
What is the rhythm?
What is the management?
70
71. 1° AV block
• Regular rate and rhythm
• Normal P wave with long PR interval (>0.2msec/1 big
box)
• Normal QRS
71
Note: No emergent treatment needed (asymptomatic)
Follow bradycardia algorhitm if unstable
73. 2° AV Block - Type I
• aka Wenckebach
• Regular rate and rhythm
• Normal P waves and QRS complexes
• Increasing PR interval until QRS dropped
73
May be bradycardic. No ACLS interventions indicated
(asymptomatic)
74. 80 yo man with syncope
74
What is the rhythm?
What is the management?
75. 2° AV Block – Mobitz Type II
75
• Regular atrial rate with normal P wave
• Consistent PR interval
• Random QRS dropped
Note: Follow bradycardia algorithm – prepare for pacing even if
asymptomatic as this block can worsen
76. Another 80 yo man with syncope
What is the rhythm?
What is the management?
76
77. 3° AV Block
• Normal P waves
• Normal QRS
• No relationship between P and QRS
• aka complete heart block
77
NB: Follow bradycardia algorithm. Will need to be paced.
May also have wide complexes if escape rhythm is present
78. Know When To Stop
• With return of spontaneous circulation
• No ROSC during or after 20 minutes of
resuscitative efforts
– Possible exceptions include near-drowning, severe
hypothermia, known reversible cause, some
overdoses
• DNR orders presented
• Obvious signs of irreversible death
78
N.B. For out-of-hospital providers also include
transfer of care, danger to providers, etc
79. Take Home Points
• Assess and manage at every step before
moving on to the next step
• Rapid defibrillation is the ONLY effective
treatment for VF/VT
• Search for and treat the cause
• Treat the patient not the monitor
• Reassess frequently
• Minimize interruptions to chest compressions
79
80. 80
End of ACLS CE Part I
THANK YOU FOR YOUR ATTENTION
To follow:
Part II
ACLS in Acute Coronary Syndromes / Cardiac Arrest
•Importance of CPR / BLS in most current (2010) AHA ACLS
•Relationship of the chain of survival to successful resuscitation of the
cardiac arrest patient
•Discuss the interventions required to ensure good outcomes with
Return of Spontaneous Circulation (ROSC)
Part III
•Defibrillation and ACLS Drug Therapy
Reference resources and further study:
ACLS Study Guide - ECG STRIP INTERPRETATION.pdf
ACLS Rhythms for the ACLS Algorithms.pdf
http://acls-algorithms.com/ Website, including Megacode simulator