1. Inflammatory Heart Disease

4. Pericarditis

5. Pathophysiology Inflammation of the pericardium  Ventricular filling and emptying  Intrapericardial pressure  Arterial pressure Compression of the heart  CO  Venous pressure Pericardial effusion Fluid accumulation (serous, purulent, blood) in the pericardial sac

6. Acute Pericarditis – result to exudate formation (if severe, can lead to cardiac tamponade) Chronic Pericarditis – result to fibrosing (hardening) of the pericardial sac - the thick fibrous pericardium tightens around the heart and  efficiency as a pump (Constrictive Pericarditis)

22. Pathophysiology Organism travels in the blood stream forms vegetations (clumps of bacteria, fibrin, cellular debris, platelets) growth of vegetation on heart valves attaches to the endocardial lining of a normal heart or an area of defect (heart valves) Emboli that can lodge to various organs (kidney, coronary artery, spleen, lungs, brain) deforms, thicken, stiffen, perforate the valve leaflets infected clots may break free and travel through the bloodstream Dysfunctional heart valves obstruct blood flow and produce organ damage

24. Janeway lesions

35. Interventions (cont.) 4. Diuretics, digitalis if w/ signs of CHF 5. Prevent recurrence – teach pt. on good nutrition, proper hygiene practices, adequate rest, immediate treatment of sore throat - taking prophylactic doses of Penicillin to prevent recurrence of attacks of RF – 5 years after 1 st attack - take prophylaxis of antibiotics before & after surgery or dental procedures - Severe RHD – Penicillin IM (Penadur) 1-2 x a month or oral penicillin for lifetime