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Dental caries

By : Dr. Ammar khaled
        BDS, IUA
Dental caries:
•
  def :
• infectious microbiological diseases of the tooth
  structure result in the localized dissolution and
  destruction of calcified tissue
  caries lesion only occur under a mass of bacteria
  capable of producing a sufficiency acid
  environment to demanerialize tooth structure
  Lactic acid : organic weak acid
Plaque
• Plaque: Gelatinous mass of bacteria adhering to
  the surface is treated
  the plaque bacteria metabolize refined
  carbohydrates for energy and produce organic
  acid as byproduct These acids may cause carious
  lesion by dissolution of the tooth surface
  Carious lesion progress as series of exoneration
  and remission as the PH acid at the tooth surface
  various with the change in the plaque
  metabolism
The groups of oral bacteria :
1. No bacteria no caries
2. Caries developed with bacteria inoculated.
3. Antibiotics inhibited caries
4. Bacteria found in all caries specimens
Conclusion
dental caries may be caused by members of
  the commensal oral microflora
But
• not any microbe to be considered responsible
  for a given condition
• produce great amounts of acids (acidogenic),
• tolerant of acidic environments (aciduric)
               MS and lactobacilli
the hypotheses concerning the
       etiology of dental caries :
• There are two hypotheses concerning the
  pathogenicity of plaque.
1- nonspecific plaque hypothesis: which
  assumes that all plaque is pathogenic,
2- specific plaque hypothesis : plaque as
  pathogenic only when signs of associated
  disease are present.
Etiology of dental caries :

  a. pathogenic bacterial plaque :
• soft, translucent, and tenaciously adherent
  material accumulating on the surface of teeth
  is commonly called plaque. It is more
  accurately described as bacterial plaque
  because it is composed almost completely of
  bacteria and their by-products.
•
    - early stage plaque

    -late stage plaque
early stage plaque
• After professional removal of all organic
  material and bacteria from a tooth surface, a
  new coating of organic material begins to
  accumulate immediately. Within 2 hours a
  cell-free, structureless organic film, the
  pellicle, can completely cover the previously
  denuded area. The pellicle is formed primarily
  from the selective precipitation of various
  components of saliva.
The functions of the pellicle

are believed to be:
 (1) protect the enamel,
(2) reduce friction between the teeth,
(3) possibly provide a matrix for remineralization.
• early secondary succession: S. sanguis along with
   Actinomyces viscosus, Actinomyces naeslundii, and
   Peptostreptococcus are the main pioneering
   species and are capable of attaching to the pellicle
   within 1 hour after tooth cleaning."
late stage plaque
• The late stages of ecologic succession in
  plaque are responsible for causing either
  caries or periodontal disease.

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Dental caries

  • 1. Dental caries By : Dr. Ammar khaled BDS, IUA
  • 2. Dental caries: • def : • infectious microbiological diseases of the tooth structure result in the localized dissolution and destruction of calcified tissue caries lesion only occur under a mass of bacteria capable of producing a sufficiency acid environment to demanerialize tooth structure Lactic acid : organic weak acid
  • 3. Plaque • Plaque: Gelatinous mass of bacteria adhering to the surface is treated the plaque bacteria metabolize refined carbohydrates for energy and produce organic acid as byproduct These acids may cause carious lesion by dissolution of the tooth surface Carious lesion progress as series of exoneration and remission as the PH acid at the tooth surface various with the change in the plaque metabolism
  • 4. The groups of oral bacteria : 1. No bacteria no caries 2. Caries developed with bacteria inoculated. 3. Antibiotics inhibited caries 4. Bacteria found in all caries specimens Conclusion dental caries may be caused by members of the commensal oral microflora
  • 5. But • not any microbe to be considered responsible for a given condition • produce great amounts of acids (acidogenic), • tolerant of acidic environments (aciduric) MS and lactobacilli
  • 6. the hypotheses concerning the etiology of dental caries : • There are two hypotheses concerning the pathogenicity of plaque. 1- nonspecific plaque hypothesis: which assumes that all plaque is pathogenic, 2- specific plaque hypothesis : plaque as pathogenic only when signs of associated disease are present.
  • 7. Etiology of dental caries : a. pathogenic bacterial plaque : • soft, translucent, and tenaciously adherent material accumulating on the surface of teeth is commonly called plaque. It is more accurately described as bacterial plaque because it is composed almost completely of bacteria and their by-products.
  • 8. - early stage plaque -late stage plaque
  • 9. early stage plaque • After professional removal of all organic material and bacteria from a tooth surface, a new coating of organic material begins to accumulate immediately. Within 2 hours a cell-free, structureless organic film, the pellicle, can completely cover the previously denuded area. The pellicle is formed primarily from the selective precipitation of various components of saliva.
  • 10. The functions of the pellicle are believed to be: (1) protect the enamel, (2) reduce friction between the teeth, (3) possibly provide a matrix for remineralization. • early secondary succession: S. sanguis along with Actinomyces viscosus, Actinomyces naeslundii, and Peptostreptococcus are the main pioneering species and are capable of attaching to the pellicle within 1 hour after tooth cleaning."
  • 11. late stage plaque • The late stages of ecologic succession in plaque are responsible for causing either caries or periodontal disease.