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Diagnosis of Pulpal pathology
Abdullah Karamat
The process of making a diagnosis has five
stages:
1- Patient tells the clinician why the patient is
seeking advice
2- The clinician questions the patient about
the symptoms and history that led to patient’s
visit
3- The clinician performs objective clinical
tests.
4- The clinician correlates the objective
findings with the subjective details and
creates a tentative differential diagnosis.
5- The clinician formulates a definitive
diagnosis
History
• Presenting complaint :
Patients should be
questioned sympathetically and asked to
describe their complaint in their own words;
this should then be documented.
• History of presenting complaint :
The history of
presenting complaint is divided into five basic
directions of questioning :
1- Localization: “Can you point to the
offending tooth?”
2- Commencement: “When did the symptoms
first occur?”
3- Intensity: “How intense is the pain?”
4- Provocation and Relief of Pain: “What
produces or reduces the symptoms?”
5- Duration: “Do the symptoms subside
shortly, or do they linger after they are
provoked?”
• Dental history :
- Past dental clinic attendance
- Possible contributing factors towards
patient’s present condition.
- Proper documentation
• Medical history :
Examination
• Vital signs :
- Blood pressure normal for people under 60
years age = 120/80 mmHg and for people
above 60 years age = 130/90 mmHg
- Pulse rate normal = 60-100/min
- Respiratory rate normal = 16-18 breaths/min
- Temperature = 98.6 F
• Extra oral examination :
Examine for :
1- localized swellings
2- facial asymmetry
3- change in color
4- lymph node examination
5- trauma
6- sinus tract
7- cancer screen ( soft tissue examination
, lumps and white spots )
8- temporomandibular joint
Intra oral examination :
Examine for :
1- intra oral swellings
2- soft tissue examination
3- intra oral sinus
4- hard tissues
To locate the source of an infection, the sinus tract can
be traced by threading the stoma with a gutta-percha point
Palpation :
• The buccal/labial and
palatal/lingual mucosa are palpated .
Light finger pressure is applied in a rolling
motion on the soft tissues
using, normally, an index finger . Signs of
tenderness usually indicate inflammation
of the underlying tissue.
Light finger pressure is applied in a rolling
motion to palpate the soft tissues
Percussion
Teeth are percussed in an axial and buccal
direction using a forefinger or the end of a
mirror handle. Tenderness to gentle
percussion indicates inflammation of the
periodontal ligament surrounding the tooth;
or this may be of pulpal or periodontal in
origin .
Tooth percussion performed using a forefinger
Periodontal probing :
Probing depths should be assessed by
‘walking’ the periodontal probe around the
entire circumference of the tooth . The
probing profile for root fractures and
iatrogenic perforations is, characteristically, an
isolated localised loss of attachment
The periodontal probe must be walked around
the tooth to ensure that any isolated narrow periodontal
defects are not missed.
Mobility :
Like percussion testing, an increase in tooth
mobility is not an indication of pulp vitality. It
is merely an indication of a compromised
periodontal attachment apparatus.
Mobility testing of a tooth, using the back ends of two
mirror handles.
INVESTIGATIONS:
1- Pulp sensitivity tests :
Currently available sensitivity tests assess the
neural response, and not the vascular supply
of the pulp . The assumptionwith these tests is
that the neural status reflects the blood
supply status of the tooth.
a- Cold test :
- ice sticks 0 C
- ethyl chloride -50 C
- Frozen Carbon dioxide
-78.5 C
•
False positve :
If cold contacts gingiva or is
transferred to adjacent teeth with vital pulps.
False-negative :
Is often obtained when cold is
applied to teeth with calcified canals,
b – Heat tests :
- Hot water
- Hot burnisher
- Hot green stick compound
- Heated GP
c- Electrical pulp testing :
The probe tip will be coated with a medium such as
toothpaste and placed in contact with the tooth surface.
The patient will activate the unit by placing a finger on
the metal shaft of the probe .
These testers other electrical testers but are
more user friendly. High readings indicate
necrosis. Low readings indicate vitality. Testing
normal control teeth establishes the
approximate boundary between the two
conditions. The exact number of the reading is
of no significance and does not detect subtle
degrees of vitality, nor can any electrical pulp
tester indicate inflammation
d – Laser Doppler flowmetry :
A diode is used to project an infrared light
beam through the crown and pulp chamber of
a tooth. The infrared light beam is scattered as
it passes through the pulp tissue. The Doppler
principle states that the light beam will be
frequency-shifted by moving red blood cells
but will remain unshifted as it passes through
static tissue
e- Pulse oximeter :
It is designed to measure the oxygen
concentration in the blood and the pulse
rate.
f- Test cavity preparation
Historically, test cavity preparation has been
suggested as a technique to assess the pulp
status when all other tests are inconclusive. Local
anaesthetic is not administered and a small bur is
used with copious irrigation to prepare a small
cavity down the centre of the tooth into dentine.
If the patient feels sensitivity, this may indicate
that the tooth is vital; alternatively, it may
indicate that the tooth is unhealthy as Aδ fibres
may still be viable in necroticpulp tissue.
g - Bite/cusp loading tests
Tenderness to bite is indicative of
inflammation of the periodontium and a common
presenting symptom. The more specific cusp
loading bite test using some form of wedging
device is indicated for patients with a suspected
cusp, tooth or root fracture presenting with
poorly localized pain on biting.The patient is
instructed to bite firmly on a cotton roll or a
commercially available ‘Tooth Slooth’
h- Staining and Transillumination :
To determine the presence of a longitudinal
fracture of the tooth, the application of a stain
to the area is often of great assistance.
Applying a bright fiberoptic light probe to the
surface of the tooth is also helpful
i- Radiographs :
- Radioisuography
- Xeroradiography
- Digital subtraction radiography
- CT
- MRI
- CBCT
Liquid Crystal Testing :
Cholesteric liquid crystals have
been used by investigators to show the
difference in tooth temperature between
teeth with vital (hotter) pulps and necrotic
(cooler) pulps.
Hughes Probeye camera :
Is capable of detecting
temperature changes as small as 0.1°C, has
also been used to measure pulp vitality
experimentally
Selecting the Appropriate Pulp Test :
The selection depends on the situation. When
cold (or hot) food or drink initiates a painful
response, a cold (or hot) test is conducted in
place of other vitality tests. Replication of the
same symptoms in a tooth often indicates the
offender. Overall, electrical stimulation is similar
to cold (refrigerant) in identifying pulp necrosis;
heat is the least reliable stimulus.
Differential diagnosis :
• Pulpal conditions
a- Normal pulp
A tooth with a normal pulp will be symptom-
free. The results of clinical examination will be
unremarkable, and the tooth will respond
normally to sensitivity testing
b- Reversible pulpitis
There is mild or transient pulpal inflammation.
This may result in the tooth causing sharp pain
lasting for up to 5–10 seconds, which does not
linger, after the applied stimulus has been
removed. Common causes of reversible
pulpitis include caries and coronal leakage
c- Irreversible pulpitis
The pulp has suffered a more severe insult and is
irreversibly inflamed; therefore, the tooth cannot
be treated conservatively. Symptoms of
irreversible pulpitis may range from a throbbing
pain, initiated by hot or cold stimuli and lasting
minutes to hours, to spontaneous intermittent
bouts of aching pain lasting for hours. Symptoms
may be exacerbated when the patient lies down
or bends over ( Barodontalgia )
Hyperplastic Pulpitis
Hyperplastic pulpitis (pulp polyp)
is a form of irreversible pulpitis that originates
from overgrowth of a chronically infl amed young
pulp onto the occlusal surface. It is usually found
in carious crowns of young patients . Ample
vascularity of the young pulp, adequate exposure
for drainage, and tissue proliferation are
associated with the formation of hyperplastic
pulpitis.Usually asymptomatic
d- Pulp necrosis :
This term describes the partial or complete
necrosis of the pulp caused by a loss of, or
inadequate blood supply. If the necrotic tissue
has not become infected, then the periapical
tissues will appear normal radiologically. Until
the periodontium is involved, the tooth is
usually symptom free.
Periapical conditions
a- Normal periapical tissues
The tooth is symptom-free and there is no
tenderness to palpation or percussion.
Radiological examination will reveal a normal
periodontal ligament space and no evidence
of periapical pathosis.
b- Acute apical periodontitis
The tooth in question will be exquisitely
tender to touch, biting or percussion.
Radiological examination may reveal a slight
widening of the periodontal ligament space. A
negative response to sensitivity testing
indicates an endodontic cause.
c- Acute periapical abscess
Patients suffering from acute periapical
abscess will usually present complaining of an
intense throbbing pain. The tooth in question
will be very tender to touch, percussion and
palpation. There may be discernible mobility
as the tooth is elevated from its bony socket.
The tooth will not respond to sensitivity tests.
An intra or extraoral swelling may be present
d- Chronic apical periodontitis
Patients may be symptom-free, alternatively, they
may report that the tooth feels different or it may
be slightly tender to chewing. Clinically, the tooth
may be tender to percussion or palpation and
does not respond to sensitivity testing.
Radiologically, there may be a widening of the
periodontal ligament space or more usually, a
periapical radiolucency may be present
e- Chronic periapical abscess
The tooth is usually symptom-free, not sensitive
to biting pressure but may ‘feel different’ to the
patient upon percussion. It is not responsive to
pulp sensitivity tests and radiologically, there will
be a periapical radiolucency. Chronic periapical
abscess may be distinguished from chronic apical
periodontitis because the former will usually be
associated with a draining sinus tract.
References :
1- Harty
2- Torabinejad
3- Ingle
4- Weine
5- Jayshree and Hedge
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )
Diagnosis of pulpal  pathology ( Abdullah karamat )

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Diagnosis of pulpal pathology ( Abdullah karamat )

  • 1. Diagnosis of Pulpal pathology Abdullah Karamat
  • 2. The process of making a diagnosis has five stages: 1- Patient tells the clinician why the patient is seeking advice 2- The clinician questions the patient about the symptoms and history that led to patient’s visit
  • 3. 3- The clinician performs objective clinical tests. 4- The clinician correlates the objective findings with the subjective details and creates a tentative differential diagnosis. 5- The clinician formulates a definitive diagnosis
  • 4. History • Presenting complaint : Patients should be questioned sympathetically and asked to describe their complaint in their own words; this should then be documented.
  • 5. • History of presenting complaint : The history of presenting complaint is divided into five basic directions of questioning : 1- Localization: “Can you point to the offending tooth?” 2- Commencement: “When did the symptoms first occur?”
  • 6. 3- Intensity: “How intense is the pain?” 4- Provocation and Relief of Pain: “What produces or reduces the symptoms?” 5- Duration: “Do the symptoms subside shortly, or do they linger after they are provoked?”
  • 7. • Dental history : - Past dental clinic attendance - Possible contributing factors towards patient’s present condition. - Proper documentation
  • 9. Examination • Vital signs : - Blood pressure normal for people under 60 years age = 120/80 mmHg and for people above 60 years age = 130/90 mmHg - Pulse rate normal = 60-100/min - Respiratory rate normal = 16-18 breaths/min - Temperature = 98.6 F
  • 10. • Extra oral examination : Examine for : 1- localized swellings 2- facial asymmetry 3- change in color 4- lymph node examination
  • 11. 5- trauma 6- sinus tract 7- cancer screen ( soft tissue examination , lumps and white spots ) 8- temporomandibular joint
  • 12.
  • 13.
  • 14. Intra oral examination : Examine for : 1- intra oral swellings 2- soft tissue examination 3- intra oral sinus 4- hard tissues
  • 15.
  • 16.
  • 17. To locate the source of an infection, the sinus tract can be traced by threading the stoma with a gutta-percha point
  • 18.
  • 19.
  • 20. Palpation : • The buccal/labial and palatal/lingual mucosa are palpated . Light finger pressure is applied in a rolling motion on the soft tissues using, normally, an index finger . Signs of tenderness usually indicate inflammation of the underlying tissue.
  • 21. Light finger pressure is applied in a rolling motion to palpate the soft tissues
  • 22. Percussion Teeth are percussed in an axial and buccal direction using a forefinger or the end of a mirror handle. Tenderness to gentle percussion indicates inflammation of the periodontal ligament surrounding the tooth; or this may be of pulpal or periodontal in origin .
  • 23. Tooth percussion performed using a forefinger
  • 24. Periodontal probing : Probing depths should be assessed by ‘walking’ the periodontal probe around the entire circumference of the tooth . The probing profile for root fractures and iatrogenic perforations is, characteristically, an isolated localised loss of attachment
  • 25. The periodontal probe must be walked around the tooth to ensure that any isolated narrow periodontal defects are not missed.
  • 26. Mobility : Like percussion testing, an increase in tooth mobility is not an indication of pulp vitality. It is merely an indication of a compromised periodontal attachment apparatus.
  • 27. Mobility testing of a tooth, using the back ends of two mirror handles.
  • 28. INVESTIGATIONS: 1- Pulp sensitivity tests : Currently available sensitivity tests assess the neural response, and not the vascular supply of the pulp . The assumptionwith these tests is that the neural status reflects the blood supply status of the tooth.
  • 29. a- Cold test : - ice sticks 0 C - ethyl chloride -50 C - Frozen Carbon dioxide -78.5 C •
  • 30. False positve : If cold contacts gingiva or is transferred to adjacent teeth with vital pulps. False-negative : Is often obtained when cold is applied to teeth with calcified canals,
  • 31. b – Heat tests : - Hot water - Hot burnisher - Hot green stick compound - Heated GP
  • 32. c- Electrical pulp testing : The probe tip will be coated with a medium such as toothpaste and placed in contact with the tooth surface. The patient will activate the unit by placing a finger on the metal shaft of the probe .
  • 33. These testers other electrical testers but are more user friendly. High readings indicate necrosis. Low readings indicate vitality. Testing normal control teeth establishes the approximate boundary between the two conditions. The exact number of the reading is of no significance and does not detect subtle degrees of vitality, nor can any electrical pulp tester indicate inflammation
  • 34. d – Laser Doppler flowmetry : A diode is used to project an infrared light beam through the crown and pulp chamber of a tooth. The infrared light beam is scattered as it passes through the pulp tissue. The Doppler principle states that the light beam will be frequency-shifted by moving red blood cells but will remain unshifted as it passes through static tissue
  • 35. e- Pulse oximeter : It is designed to measure the oxygen concentration in the blood and the pulse rate.
  • 36. f- Test cavity preparation Historically, test cavity preparation has been suggested as a technique to assess the pulp status when all other tests are inconclusive. Local anaesthetic is not administered and a small bur is used with copious irrigation to prepare a small cavity down the centre of the tooth into dentine. If the patient feels sensitivity, this may indicate that the tooth is vital; alternatively, it may indicate that the tooth is unhealthy as Aδ fibres may still be viable in necroticpulp tissue.
  • 37. g - Bite/cusp loading tests Tenderness to bite is indicative of inflammation of the periodontium and a common presenting symptom. The more specific cusp loading bite test using some form of wedging device is indicated for patients with a suspected cusp, tooth or root fracture presenting with poorly localized pain on biting.The patient is instructed to bite firmly on a cotton roll or a commercially available ‘Tooth Slooth’
  • 38.
  • 39. h- Staining and Transillumination : To determine the presence of a longitudinal fracture of the tooth, the application of a stain to the area is often of great assistance. Applying a bright fiberoptic light probe to the surface of the tooth is also helpful
  • 40.
  • 41. i- Radiographs : - Radioisuography - Xeroradiography - Digital subtraction radiography - CT - MRI - CBCT
  • 42. Liquid Crystal Testing : Cholesteric liquid crystals have been used by investigators to show the difference in tooth temperature between teeth with vital (hotter) pulps and necrotic (cooler) pulps.
  • 43. Hughes Probeye camera : Is capable of detecting temperature changes as small as 0.1°C, has also been used to measure pulp vitality experimentally
  • 44. Selecting the Appropriate Pulp Test : The selection depends on the situation. When cold (or hot) food or drink initiates a painful response, a cold (or hot) test is conducted in place of other vitality tests. Replication of the same symptoms in a tooth often indicates the offender. Overall, electrical stimulation is similar to cold (refrigerant) in identifying pulp necrosis; heat is the least reliable stimulus.
  • 45. Differential diagnosis : • Pulpal conditions a- Normal pulp A tooth with a normal pulp will be symptom- free. The results of clinical examination will be unremarkable, and the tooth will respond normally to sensitivity testing
  • 46. b- Reversible pulpitis There is mild or transient pulpal inflammation. This may result in the tooth causing sharp pain lasting for up to 5–10 seconds, which does not linger, after the applied stimulus has been removed. Common causes of reversible pulpitis include caries and coronal leakage
  • 47. c- Irreversible pulpitis The pulp has suffered a more severe insult and is irreversibly inflamed; therefore, the tooth cannot be treated conservatively. Symptoms of irreversible pulpitis may range from a throbbing pain, initiated by hot or cold stimuli and lasting minutes to hours, to spontaneous intermittent bouts of aching pain lasting for hours. Symptoms may be exacerbated when the patient lies down or bends over ( Barodontalgia )
  • 48. Hyperplastic Pulpitis Hyperplastic pulpitis (pulp polyp) is a form of irreversible pulpitis that originates from overgrowth of a chronically infl amed young pulp onto the occlusal surface. It is usually found in carious crowns of young patients . Ample vascularity of the young pulp, adequate exposure for drainage, and tissue proliferation are associated with the formation of hyperplastic pulpitis.Usually asymptomatic
  • 49. d- Pulp necrosis : This term describes the partial or complete necrosis of the pulp caused by a loss of, or inadequate blood supply. If the necrotic tissue has not become infected, then the periapical tissues will appear normal radiologically. Until the periodontium is involved, the tooth is usually symptom free.
  • 50. Periapical conditions a- Normal periapical tissues The tooth is symptom-free and there is no tenderness to palpation or percussion. Radiological examination will reveal a normal periodontal ligament space and no evidence of periapical pathosis.
  • 51. b- Acute apical periodontitis The tooth in question will be exquisitely tender to touch, biting or percussion. Radiological examination may reveal a slight widening of the periodontal ligament space. A negative response to sensitivity testing indicates an endodontic cause.
  • 52. c- Acute periapical abscess Patients suffering from acute periapical abscess will usually present complaining of an intense throbbing pain. The tooth in question will be very tender to touch, percussion and palpation. There may be discernible mobility as the tooth is elevated from its bony socket. The tooth will not respond to sensitivity tests. An intra or extraoral swelling may be present
  • 53. d- Chronic apical periodontitis Patients may be symptom-free, alternatively, they may report that the tooth feels different or it may be slightly tender to chewing. Clinically, the tooth may be tender to percussion or palpation and does not respond to sensitivity testing. Radiologically, there may be a widening of the periodontal ligament space or more usually, a periapical radiolucency may be present
  • 54. e- Chronic periapical abscess The tooth is usually symptom-free, not sensitive to biting pressure but may ‘feel different’ to the patient upon percussion. It is not responsive to pulp sensitivity tests and radiologically, there will be a periapical radiolucency. Chronic periapical abscess may be distinguished from chronic apical periodontitis because the former will usually be associated with a draining sinus tract.
  • 55. References : 1- Harty 2- Torabinejad 3- Ingle 4- Weine 5- Jayshree and Hedge