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 Largest   tendon in the
  body
 Origin from
  gastrocnemius and
  soleus muscles
 Insertion on
  calcanealtuberosity
Lacks a true synovial
    sheath-


 Paratenon has visceral
  and parietal layers
 Allows for 1.5cm of
  tendon glide
Paratenon
   Anterior – richly vascularized
   The remainder – multiple thin
    membranes
Blood supply
1)   Musculotendinous junction
2)   Osseous insertion on calcaneus
3)   Multiple mesotenal vessels on
     anterior surface of paratenon (in
     adipose)
     –   Transverse vincula
           Fewest @ 2 to 6 cm proximal to
            osseous insertion
   Remarkable response to stress
     Exercise induces tendon diameter
      increase
     Inactivity or immobilization causes
      rapid atrophy
   Age-related decreases in cell
    density, collagen fibril diameter and
    density
       Older athletes have higher injury
        susceptibility
   Gastrocnemius-soleus-Achilles
    complex
       Spans 3 joints
         Flex knee
         Plantar flex tibiotalar joint

         Supinatesubtalar joint




   Up to 10 times body weight through
    tendon when running
1.   Close injury/rupture
2.   Open injury/rupture

     • Acute injury
     • Neglected injury
1.   Accidental cut injury
     (bath room injury, road
     traffic injury)
2.   Social/political
     Violence
1. Diagnosis and
assessment of extend
of injury.
2. Primary care
3. Operative treatment
      Pathophysiology
     Repetitive microtrauma
      in a relatively
      hypovascular area.
     Reparative process
      unable to keep up
     May be on the
      background of a
      degenerative tendon
 Antecedent tendinitis/tendinosis in 15%
 75% of sports-related ruptures happen
  in patients between 30-40 years of
  age.
 Most ruptures occur in watershed area
  4cm proximal to the calcaneal
  insertion.
History
 Feels like being kicked in the leg
 Case reports of fluoroquinolone use,
  steroid injections
 Mechanism
    Eccentric loading (running backwards in

     tennis)
    Sudden unexpected dorsiflexion of ankle

    (Direct blow or laceration)
Prone   patient with feet over edge of
 bed
Palpation of entire length of muscle-
 tendon unit during active and
 passive ROM
Compare tendon width to other side
Note tenderness, crepitation,
 warmth, swelling, nodularity,
 palpable defects
 Partial
  Localized tenderness +/-

   nodularity
 Complete
  Defect

  Cannot heel raise

  Positive Thompson test
 Diagnostic Pitfalls
 23% missed by Primary Physician
  (Inglis&Sculco)
     Tendon defect can be masked by hematoma
     Plantar-flexion power of extrinsic foot flexors
      retained
     Thompson test can produce a false-negative
      if accessory ankle flexors also squeezed
This lateral x-ray of the
calcaneus shows an
avulsion fracture at the
insertion of the Achilles
tendon, with marked
separation of fragments.
.
 Inexpensive, fast, reproducable,
  dynamic examination possible
 Operator dependent
 Best to measure thickness and gap
 Good screening test for complete
  rupture
   Expensive, not dynamic
   Better at detecting partial
    ruptures and staging
    degenerative changes,
    (monitor healing)
 Restore
  musculotendinous length
  and tension.
 Optimize gastro-soleous
  strength and function
 Avoid ankle stiffness
CAM Walker or cast with
                        2 wks      plantarflexion q 2 wks

                                                 4 weeks

Start physio for ROM            Allow progressive weight-
exercises                       bearing in removable cast

                         When WBAT and          2- 4 weeks
                         foot is plantigrade

Start a strengthening           Remove cast and walk with shoe
program                         lift. Start with 2cm x 1 month,
                                then 1cm x1 month then D/C
 Preserve anterior paratenon blood
  supply
 Beware of sural nerve
 Debride and approximate tendon ends
 Use 2-4 stranded locked suture
  technique
 May augment with absorbable suture
 Close paratenon separately
 Acute  case : usually end
  to end repair is enough
 Neglected case:
  Advancement plasy (V-Y)
  or reconstruction by
  other tendons
 Assess strength of repair, tension and
  ROM intra-op.
 Apply long leg cast with ankle in the least
  amount of planterflexion(gravityequinus) &
  knee 60 degree flexion with window at
  operated site.
 Stitch removal after 2 wks.

 Short leg cast after 3 wks with partial
  equinus correction
 2 weekly plaster change with
  gradual equinus correction (4-6
  episode ).
 Walking with heel raised shoe &
  regular physiotherapy.
 Reverse ankle stop brace up to 6
  months.
Acute rupture of tendon Achilles. A prospective randomised
           study ofcomparison between surgical and non-surgical treatment.
             Moller M, et al. J Bone Joint Surg Br. 2001 Aug;83(5):863-8



                          112 patients


Casted x 8 wks                                   Surgery +
                                                 Early functional rehab in
                                                 brace


21 % re-rupture                                       1.7% re-rupture
                                                      5%     infection
                       No difference in
                       functional outcome             2% Sural nerve inj.
Achilles tendon for presentation
Achilles tendon for presentation
Achilles tendon for presentation
Achilles tendon for presentation

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Achilles tendon for presentation

  • 1.
  • 2.  Largest tendon in the body  Origin from gastrocnemius and soleus muscles  Insertion on calcanealtuberosity
  • 3. Lacks a true synovial sheath-  Paratenon has visceral and parietal layers  Allows for 1.5cm of tendon glide
  • 4. Paratenon  Anterior – richly vascularized  The remainder – multiple thin membranes
  • 5. Blood supply 1) Musculotendinous junction 2) Osseous insertion on calcaneus 3) Multiple mesotenal vessels on anterior surface of paratenon (in adipose) – Transverse vincula  Fewest @ 2 to 6 cm proximal to osseous insertion
  • 6. Remarkable response to stress  Exercise induces tendon diameter increase  Inactivity or immobilization causes rapid atrophy  Age-related decreases in cell density, collagen fibril diameter and density  Older athletes have higher injury susceptibility
  • 7. Gastrocnemius-soleus-Achilles complex  Spans 3 joints  Flex knee  Plantar flex tibiotalar joint  Supinatesubtalar joint  Up to 10 times body weight through tendon when running
  • 8. 1. Close injury/rupture 2. Open injury/rupture • Acute injury • Neglected injury
  • 9. 1. Accidental cut injury (bath room injury, road traffic injury) 2. Social/political Violence
  • 10. 1. Diagnosis and assessment of extend of injury. 2. Primary care 3. Operative treatment
  • 11. Pathophysiology  Repetitive microtrauma in a relatively hypovascular area.  Reparative process unable to keep up  May be on the background of a degenerative tendon
  • 12.  Antecedent tendinitis/tendinosis in 15%  75% of sports-related ruptures happen in patients between 30-40 years of age.  Most ruptures occur in watershed area 4cm proximal to the calcaneal insertion.
  • 13.
  • 14. History  Feels like being kicked in the leg  Case reports of fluoroquinolone use, steroid injections  Mechanism  Eccentric loading (running backwards in tennis)  Sudden unexpected dorsiflexion of ankle  (Direct blow or laceration)
  • 15.
  • 16. Prone patient with feet over edge of bed Palpation of entire length of muscle- tendon unit during active and passive ROM Compare tendon width to other side Note tenderness, crepitation, warmth, swelling, nodularity, palpable defects
  • 17.  Partial Localized tenderness +/- nodularity  Complete Defect Cannot heel raise Positive Thompson test
  • 18.
  • 19.
  • 20.
  • 21.  Diagnostic Pitfalls  23% missed by Primary Physician (Inglis&Sculco)  Tendon defect can be masked by hematoma  Plantar-flexion power of extrinsic foot flexors retained  Thompson test can produce a false-negative if accessory ankle flexors also squeezed
  • 22. This lateral x-ray of the calcaneus shows an avulsion fracture at the insertion of the Achilles tendon, with marked separation of fragments. .
  • 23.  Inexpensive, fast, reproducable, dynamic examination possible  Operator dependent  Best to measure thickness and gap  Good screening test for complete rupture
  • 24. Expensive, not dynamic  Better at detecting partial ruptures and staging degenerative changes, (monitor healing)
  • 25.  Restore musculotendinous length and tension.  Optimize gastro-soleous strength and function  Avoid ankle stiffness
  • 26. CAM Walker or cast with 2 wks plantarflexion q 2 wks 4 weeks Start physio for ROM Allow progressive weight- exercises bearing in removable cast When WBAT and 2- 4 weeks foot is plantigrade Start a strengthening Remove cast and walk with shoe program lift. Start with 2cm x 1 month, then 1cm x1 month then D/C
  • 27.  Preserve anterior paratenon blood supply  Beware of sural nerve  Debride and approximate tendon ends  Use 2-4 stranded locked suture technique  May augment with absorbable suture  Close paratenon separately
  • 28.
  • 29.  Acute case : usually end to end repair is enough  Neglected case: Advancement plasy (V-Y) or reconstruction by other tendons
  • 30.
  • 31.
  • 32.
  • 33.  Assess strength of repair, tension and ROM intra-op.  Apply long leg cast with ankle in the least amount of planterflexion(gravityequinus) & knee 60 degree flexion with window at operated site.  Stitch removal after 2 wks.  Short leg cast after 3 wks with partial equinus correction
  • 34.  2 weekly plaster change with gradual equinus correction (4-6 episode ).  Walking with heel raised shoe & regular physiotherapy.  Reverse ankle stop brace up to 6 months.
  • 35. Acute rupture of tendon Achilles. A prospective randomised study ofcomparison between surgical and non-surgical treatment. Moller M, et al. J Bone Joint Surg Br. 2001 Aug;83(5):863-8 112 patients Casted x 8 wks Surgery + Early functional rehab in brace 21 % re-rupture 1.7% re-rupture 5% infection No difference in functional outcome 2% Sural nerve inj.